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Parasitology Exam II

QuestionAnswer
General characteristics of trematodes dorso-ventrally flattened, leaf-like, oral and ventral suckers, indirect life cycle, usually genitally independent
What are the final hosts for Alaria species? dog, cat, fox, mink
What are the intermediate hosts for Alaria species? snails and tadpoles
What are the paratenic hosts of Alaria species? frogs, snakes, mice, rats, birds, reptiles, raccoons, opossums, wild boars, humans
Identification of Alaria species egg: oval, honey color, distinct operculum, up to 130um Adult: pink or brown, 10mm
Alaria - sites of infection adults in small intestine and immature stages in lungs
Alaria - pathogenesis and lesion severe duodenitis; lung migration causes clinical illness
Alaria - clinical signs not in final host, but occur in paratenic host
Alaria - diagnosis fecal sedimentation
Alaria - treatment and prevention cestocides
Nanophyetus salmincola salmon fluke poisoning
What are the final hosts of Nanophyetus salmincola? dogs, cats, minks, raccoons, other fish-eating mammals
What are the intermediate hosts of Nanophyetus salmincola? snails and fish
Nanophyetus salmincola identification adults: ovoid and creamy white, up to 2mm egg: oval, yellowish brown, thick shell, 80um
Nanophyetus salmincola site of infection adults in small intestine
Nanophyetus salmincola pathogenesis and lesions extremely pathogenic. Flukes are vectors of Neorickettsia helminthoeca, which causes salmon poisoning in dogs, severe hemorrhagic enteritis
Nanophyetus salmincola clinical signs enteritis, sudden fever, vomiting, diarrhea, high mortality
Nanophyetus salmincola diagnosis fecal sedimentation
Nanophyetus salmincola treatment and prevention anthelmintics, tetracycline for rickettsia
Paragonimus kellicotti lung fluke
Paragonimus kellicotti final hosts dog, cat, carnivores
Paragonimus kellicotti intermediate hosts snails, crayfish Some species infect humans
Paragonimus kellicotti identification adult - ovoid, reddish brown, up to 16mm egg - oval, yellowish brown, distinct operculum, 110um
Paragonimus kellicotti site of infection lung parenchyma
Paragonimus kellicotti pathogenesis and lesions developing flukes cause formation of a cyst
Paragonimus kellicotti clinical signs intermittent cough can occur; heavy infections lead to severe cough, pneumonia, and death
Paragonimus kellicotti diagnosis radiographs reveal cysts; fecal sedimentation
Paragonimus kellicotti treatment and prevention praziquantel
Platynosomum fastosum lizard poisoning
Platynosomum fastosum final host cat
Platynosomum fastosum intermediate hosts snail, crustacean (crayfish)
Platynosomum fastosum paratenic host obligate paratenic host - lizard, skink, toads, geckos
Platynosomum fastosum identification Egg- oval, brownish, medium thick shell, operculate, up to 50um Adult - 8mm in length
Platynosomum fastosum sites of infection bile ducts
Platynosomum fastosum pathogenesis hyperplastic bile ducts
Platynosomum fastosum clinical signs lizard poisoning - palpable liver, icterus, death
Platynosomum fastosum diagnosis fecal sedimentation
Platynosomum fastosum treatment and prevention praziquantel, surgery
General characteristics of insects 3 pairs of legs, a head, distinch thorax and abdomen, pair of antennae
Characteristics of family Oestridae Adults - large hairy flies, nonfunctional mouthparts, short-lived Larvae - obligatory parasites, host specific
Cuterebra spp. rodent or rabbit bot fly, New World skin bot flies
Cuterebra spp. hosts rodents, rabbits, squirrels, chipmunks, mice, dogs, cats. Zoonosis.
Cuterebra spp. identification third stage larva and season
Cuterebra spp. site of infection subcutaneous CT, can locate to nasal and oral regions and migrate to brain
Cuterebra spp. clinical signs lump in neck, fur constantly wet, hole drips fluid
Cuterebra spp. diagnosis larva can be removed whole from breathing hole, type I hypersensitivity reaction, would heals slowly
Cuterebra spp. treatment and prevention no approved prevention
Culicidae mosquitoes
Culicidae identification pair of wings, long thin legs, long proboscis, long antennae
Culicidae pathogenesis and lesions transmit many diseases and pathogens
Characteristics of suborder Ischnocera (Mallophaga) biting, chewing lice, wide mandible mouthparts, feed on skin, hair, feathers, other organic material, 3mm, yellow, rapid movement, often bird lice
Trichodectes canis dog biting louse, canine chewing louse
Trichodectes canis hosts dogs
Trichodectes canis identification yellowish color, biting/chewing mouthparts, active louse, adults 2mm
Trichodectes canis life cycle female lays eggs, develop to nymphs and adults. Host required to complete life cycle (same for Linognathus setosus)
Trichodectes canis sites of infection hair, skin
Trichodectes canis pathogenesis and lesions vector of D. caninum; heavy infestations in neglected and underfed animals. Affects young
Trichodectes canis clinical signs pruritus, dogs scratch, cause loss of hair, restless, scratch continuously, anemia can occur
Trichodectes canis diagnosis identification of louse
Trichodectes canis treatment and prevention avoid direct contact with brushes, combs, and blankets; recent products include MCL (same for Linognathus setosus)
Characteristics of suborder Anoplura bloodsucking, more pathogenic, pointed noses, large, 3-5mm, dark blue depending on amount of blood ingested, slow moving, clasping hair shaft, lice of mammals
Linognathus setosus dog sucking louse
Linognathus setosus hosts dogs
Linognathus setosus identification 3-5mm, head narrower than thorax, sucking mouthparts
Linognathus setosus site of infection skin
Linognathus setosus pathogenesis and lesions heavy infestation can cause severe anemia
Linognathus setosus clinical signs loss of body condition, damage to skin
Linognathus setosus diagnosis louse identification
Ctenocephalides felis cat flea (most common)
Ctenocephalides felis hosts dogs and cats
Ctenocephalides felis identification egg 0.5mm
Ctenocephalides felis sites of infection skin, back, ventral abdomen, inner thighs
Ctenocephalides felis pathogenesis and lesions/clinical signs flea allergy dermatitis, vectors of a wide range of parasitic and infectious agents
Ctenocephalides felis diagnosis fleas, flea feces, blood on host and in bedding
Ctenocephalides felis treatment and prevention requires breaking life cycle in several places. insecticides, environmental control
Characteristics of Reduviidae (assassin bugs) have mouthparts designed to pierce and suck blood, allergic reactions at site of bite, painful bites, develop wings, transmit trypanosoma cruzi
Characteristics of Cimicids (bed bugs) have mouthparts designed to pierce and suck blood, allergic reactions at site of bite, painless bites until later after feeding, no wings, does not transmit disease
Reduviidae and Cimicidae life cycle hide by day and attack by night; cimicids only nocturnal; can molt 5 times at weekly intervals, can endure starvation for months
Reduviidae and Cimicidae sites of infection skin
Reduviidae and Cimicidae pathogenesis and lesions can transmit disease (Reduviidae)
Reduviidae and Cimicidae clinical signs blood loss and allergic reactions
Reduviidae and Cimicidae diagnosis identification of bug
Reduviidae and Cimicidae treatment and prevention environmental and insecticide
Created by: Stacia1226
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