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RA drugs

Med Surg III

QuestionAnswer
DMARDS methotrexate Rheumatrex, Trexall
DMARDS penicillamine Cuprimine, Depen
DMARDS sulfasalazine Azulfidine, Salazopyrin
DMARDS leflunomide Arava
Antimalarial hydroxychloroquine Plaquenil
Biologic Targeted Therapy etanercept Enbrel
Biologic Targeted Therapy infliximab Remicade
Biologic Targeted Therapy adalimumab Humira
Biologic Targeted Therapy anakinra Kineret
Biologic Targeted Therapy abatacept Orencia
Biologic Targeted Therapy rituximab Rituxan
methotrexate rapid anti-inflammatory effect
penicillamine Cuprimine, Depen Inhibits T cell function, impairs antigen presentation.
Sulfasalazine, Azulfidine Salazopyrin Blocks prostaglandin formation
leflunomide Arava Inhibits proliferation of lymphocytes, blocks immune cell overproduction
Hydroxychloroquine Plaquenil suppress formation of antigens, inhibits lysosomal enzymes
Etanercept Embrel Engineered copy of the TNF cell receptor, Binds TNF before TNF can by to the cell surface receptor
Infliximab Remicade monoclonal antibody that binds to TNF
adalimumab Humira monoclonal antibody that binds to TNF reduces infilitration of inflammotory cells
anakinra Kineret blocks the action of interleukin decreased inflammatory response
Abatacept Orencia Modulates T cells activation, suppresses immune response
Rituximab Rituxan monoclonal antibody that targets b cells
Gold compounds alters immune response, inhibits T and B cell activity, suppressing synoviitis of active ra.
Corticosteriods inhibits synthesis/release of mediators of inflammtion
methotrexate rheumatrex trexall hepatoxicity, bone marrow suppression, nephrotoxicity sustained cough
penicillamine Cupramine Depen GI irritation, reduces alters taste, proteinuria, hamaturia, rash fe def anemia
Sulfasalazine Azulfidine Salazopyrin Gi effects, bleeding, bruising, jaundice, rash, HA, uticaria, purititis. Discoloration urine et skin.
Created by: 1161798020