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Hypertension 33

lewis HTN 33

QuestionAnswer
What is blood pressure determined by Systemic vascular resistance and cardiac output
What does abnormally high blood pressure lead to excessive contraction of arteries, kidney disease, stroke, and heart attack
What is cardiac output controlled by Renin-angiotensin-aldosterone system and Naturitic peptides
What is systemic vascular resistance controlled by Vasocontrictors-Angiotensin, norepinephrine Vasodilators-prostaglandins
What is pulse pressure difference between SBP and DBP
When is it increased atherosclerosis
When is it decreased CHF and Hypovolemia
What is Map and what does it measure perfusion of organs must be above 60
What accounts for 90-95% of cases of HTN Primary or essential
When is HTN classified as Pre-HTN 120-139 or 80-89
When is HTN classified as Stage 1 140-159 or 90-99
When is HTN classified as Stage 2 greater than 160 or greater than 100
What are risk factor for Primary HTN Age,African American, high sodium intake
What does altered angiotensin mechanism cause increased aldosterone and fluid retention
What does stress and SNS mechanism cause increased vasoconstriction, HR, and Renin release
What does insulin resistance and hyperinsulinemia cause stimulates SNS and vasodilator response
What is blood pressure determined by Systemic vascular resistance and cardiac output
What does abnormally high blood pressure lead to excessive contraction of arteries, kidney disease, stroke, and heart attack
What is cardiac output controlled by Renin-angiotensin-aldosterone system and Naturitic peptides
What are urinalysis indications of renal disease protein and hematuria
What is systemic vascular resistance controlled by Vasocontrictors-Angiotensin, norepinephrine Vasodilators-prostaglandins
What serum or blood studies indicate HTN serum electrolytes, glucose, creatnine, lipids,
What is pulse pressure difference between SBP and DBP
What does ECG show in HTN ischemic heart disease
When is it increased atherosclerosis
What does echo show LV hypertrophy
When is it decreased CHF and Hypovolemia
What is Map and what does it measure perfusion of organs must be above 60
What accounts for 90-95% of cases of HTN Primary or essential
When is HTN classified as Pre-HTN 120-139 or 80-89
When is HTN classified as Stage 1 140-159 or 90-99
When is HTN classified as Stage 2 greater than 160 or greater than 100
What are risk factor for Primary HTN Age,African American, high sodium intake
What does altered angiotensin mechanism cause increased aldosterone and fluid retention
What does stress and SNS mechanism cause increased vasoconstriction, HR, and Renin release
What does insulin resistance and hyperinsulinemia cause stimulates SNS and vasodilator response
What are symptoms of HTN Fatigue, reduced activity tolerance, palpitations, angina, dyspnea
What are complications of HTN CAD, LVH, HF, kidney damage, PAD, and retinal damage
What are urinalysis indications of renal disease protein and hematuria
What serum or blood studies indicate HTN serum electrolytes, glucose, creatnine, lipids,
What does ECG show in HTN ischemic heart disease
What does echo show LV hypertrophy
What does eye exam show retinal hemmorhage, AV nicking, and papilldoma
What eight loss is significant 10-20 lbs show decrease in BP of 5-20 mmHg
What exercise level is recommended 30 min aerobic 3 times per week
What is DASH diet plan enphasizes fruits and vegetables, fat free milk products, whole grains, fish and poultry, beans,seeds and nuts
What is the most important role of the nurse in HTN diagnosis TEACHING
Where do central adrenergic antagonist act vasomotor Block SNS response
Where do peripheral adrenergic antagosist act Sympathetic ganglion to vasomotor center Block SNS response
Where do Beta blockers act Inhibit Renin release
Where do ACE inhibitors act block production of angiotensin I
Where do angiotensin II blockers act block angiotensin II from constriction blood vessels
Where do diuretics act decrease sodium absorption
Where do calcium channel blockers, and direct arterial vasodilators act block vasoconstriction
What med is used to counteract the baroreceptor bet blocker
What med counteracts the concentrating effect of the kidneys Ace inhibitor, ARB, or aldosterone inhibitor
What symptoms are often the reason for noncompliance with cardiac meds Orthostatic hypotension, sexual dysfunction,dry mouth, and frequent urination
What is the result of diruetics decrease blood volume, preload, cardiac output, and BP
What are 4 types of diuretics and where do they act Loop-loop of henle Thiazide-proximal distal renal tubule Potassium sparing-distal renal tubule and collecting duct Osmotic-creates osmotic force in the lumen of the nephron (proximal convoluted tubule
Which diuretic is used for massive and quick diuresis and/or renal impairment Loop Diuretics
Which diuretics are only PO Thiazides
Which diuretics can increase glucose Loop and Thiazides
Which diuretics cause skin reactions thiazide and loop
Which diuretics can't be given with Sulfa allergy Loops
Which diuretics are dosed daily Thiazide and potassium sparing
Which diuretics are dosed twice daily loop
What are dietary sources of potassium bananas,orange or yellow fruits and vegetables, beans, potato
What teaching is needed with diuretics hyperglycemia, NSAIDs reduce effectiveness and gout flares
What labs are monitored with diuretics k+ and Mag
What are common diuretics Lasix, HCTZ, spiolactone,diuril,mannitol
What are central acting adrenergic inhibitors Catapres,aldomet,wytensin
What are peripheral acting adrenergic inhibitors Reserpine,beDta blockers
How do beta blockers work block catecholamine production which decreases HR and decreases renin
Why do you have to wean off BB in diabetics it blunts hypoglcemia symptoms
What are common beta blockers Tenormin, lopressor, toprol and olol's
What does hydralazine do dilate arterioles, decrease afterload, and increase CO
What does Nitro do dilates veins, decreases preload, CO,
What are adverse effects of vasodilators postural hypotension, reflex tachycardia, and increased blood voulume
What is drug of choice in hypertensive crisis Nipride-IV, immediate onset
What NI must be done when administering Nipride BP every minute
What are adverse effects of Nipride extreme hypotension, cyanide posioning, and thiocyanate toxicity
What is Angiotensin II Neurohormone that promotes sodium and water retention, stimulates SNS, and promotes atherosclerosis
What do ACE inhibitors do block the formation of angiotensin II
What are NI for Ace inhibitors or ARB Monitor electrolytes, creatnine, fluid status, and blood pressure
What is a common side effect of ACE inhibitors or ARB dry hacking cough
What teaching for ACE inhibitors or ARB avoid alcohol and salt subtitutes
What are Angiotensin Receptor Blocker (ARB) Block the receptor site of angiotensin II
What are ARB's Benicar, coozar, Diovan
What are drug interactions for ACE inhibitors or ARB potassium supplements, potassium sparing diuretics, and Lithium
What is the action of CCB dilation of arterioles, decreased HR and afterload
What are S/E of CCB Bradycardia, headache
What is a food interaction with CCB grapefruit juice
What are some of the reasons for hypertension resistant HTN Corticosteroids, licorice, erythropoetin, oral contraceptives, excessive alcohol intake
HTN in older persons wide gap between korotkoff and subsequent beats white coat hypertension
What consistutes a Hypertensive crisis or malignant HTN Severe increase in BP Evidence of acute target organ damage
How is malignant HTN treated Direct Vasodilators-titrated to MAP monitor cardiac and renal function Neuro checks Bring BP down gradually investigate cause Teaching
Created by: garrisonl