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FINAL EXAM FOR A & P

ALL 16 CHAPTERS

QuestionAnswer
MOTOR NEURON MOTOR CELL
MOTOR UNIT NEURON + MUSCLE FIBER
MOTOR END PLATE END OF AXON WHICH ACETYLCHOLINESTINE EMPTIS INTO
CURARE BLOCKS ACETYLCHOLINE FROM RECEPTOR SITES NOT CONTRACTION DUE TO NO MEMBRANE CHANGE DEATH BY ASPHYXIATION
SOME ORGANOPHOSPHATES DOES WHAT ? FOUND IN PESTICIDES + BIO WARFARE CHEM.INHIBIT ACETYLOCHOLINESTERASE BIO. WARFACE CHEM.
BOTULIN FOOD POISONING BACTERIA BLOCK RELEASE OF ACETYLCHOLINE
MYASTHENIA GRAVIS MUSCLE WEAKNESS-DUE TO DECREASE IN RECEP.SITES FOR
MOTOR UNIT MOTOR NEURON + ALL MUSCLE FIBER THAT IT CONTROLLS
FEWER FIBERS MEAN? BETTER CONTROL I.E EYE
WHAT IS MUSLCE FATIQUE? DUE TO NO BLOOD ACETYLCHOLINE EXHAUSTED LACTIC ACID BUILD UP--PH DOWN (PREVENT CONTRACTION)
DEFINE MUSCLE CRAMP IS DUE TO LACK OF ATP NEEDED TO RMOVE CALCIUM (ACTIVE TRANSPORT TO PULL CALCIUM OUT)
MUSCLE RIGORMORTIS IS DUE TO LACK OF ATP MAY LAST FOR 72 HOURS
SUMMATION AN INCREASE IN RESPONSE TO STIMULI IN RAPID SUCCESSION
TETANUS MAXIMAL SUMMATION ( LOCK JAW)
TREPPE STAIRCASE OF SUMMATION
FATIQUE DROP IN TENSION AFTER PROLONGED STIMULATION,BUT IF ATP SOLUTION ADDED WILL RELIEVE MUSLCE FATIQUE
THRESHOLD STIMULUS MINIMAL STRENGHT OF STIMULUS TO ELICIT A RESPONSE
REFRACTORY PERIOD TIME NEEDED FOR ELECTROLYTE BALANCE AGAAIN BEFOR NEXT RESPONSE
RHEOBASE INTENSITY (HIT HARDER) OF STIMULUS SUFFICIENT TO ELLICITE A RESPONSE
CHRONAXIE TWO TIMES RHEOBASE (HARDER)
MUSLCE TONE CONTINOUS CONTRACTION
MUSLCE HYPERTROPHY ENLARGEMENT DUE TO EXERCISE
SATELLITE CELL UNDIFFERENTIATED CELL IN MUSCLE WHICH REPLACES INJURED MUSCLE CELLS: MUSCLES CANNOT UNDERGO MITOSIS, SO SATELLITE CELLS REPAIR MUSCLES BY GOING THROUGH MITOSIS
RATCHET THEORY THEORY OF MUSCLE CONTRACION WHICH INDICATES RATCHETING OF MYOSIN FILAMENT
ISOMETRIC MUSCLE CONTRACTION CONTRACTION W/O LENGTH CHANGE (NO OVERLAPPING) NO INTER DIGITATION OF MYOFILAMENTS
ISOTONIC MUSCLE CONTRACTION CONTRACTION WITH LENGTH CHANGE MYOFILAMENTS OVERLAP
THERE ARE TWO WAYS TO STORE ENERGY INSIDE MUSCLES WHAT ARE THEY ? FIRS OF ALL MITOCHONDIRA (CELL RESPIRATION) PRODUCES ATP...
WHAT DOES THE --- IN A RESTING CELL STORE SARCOPLASM (MUSCLE CELL); WITH MITOCHONDRIA(RESPIRATION)
WHAT IS STORED FROM LIVER W/O OXYGEN ? CREATINE ALONG WITH A PHOSPHATE + ATP WHICH GIVES HIGH ENERGY PHOSPHATE BOND 4-6 TIMES MORE ABUNDANT THAN APT IN MUSLCE CELL
HOW MUCH ATP FROM THE SARCOPLASM IN STRESS HALF WAY
HOW MUCH ATP FROM CREATINE PHOSPH.+ ADP=CREATINE+ATP ? FEW SECONDS OF ENERGY (ONLY ENOUGH) FOR SECONDS AND REQUIRES RESPSIRATION FOR BOTH (SARCOPLASM, CREATINE PHOSPHATE)
TYPES OF SKELETAL MUSLCES SOME FIBERS HAVE HIGHER ATPASE ACTIVITY(FASTER) SOME CONTAIN MYOGLOBIN (SIMILAR TO HEMOGLOBIN) INDIVIDUAL MUSCLES CONTAIN A VARIETY OF DIFFERENT TYPES OF FIVERS-THUS MUSCLE MAY PERFORM DIFFERENT FUNCTIONS
WHAT IS SMOOTH MUSCLES VISCERAL MUSCLE
THREE TYPES OF FILAMENTS FROM SMOOTH MUSCLE ACTIN--THIN MYOSIN--THICK INTERMEDIATE ( CYTOSKELETON)
SMOOTH MUSCLE (DEMOGRAPHICS) NO STRIATIONS NO ZONES- H,I,M ETC. ACTIN FILAMENT ANCHORED TO CELL MEMBRANE OR "DENSE BODIES" IN CYTOPLASM(INSIDE) WHICH ATTACH FILLIMENTS.
SMOOTH MUSCLE DEMOGRAPHIS 2 NO TROPONIN CALCIUM REGULATES CROSS BRIDGE ACTIVITY BY INFLUENCING ATPASE ACTIVITY BY CALMOUDULIN(PROTEIN WHICH BINDS CALCIUM) ATP IS SPLIT(SLOWER) VISCERAL CONTRACTION TIME THAN SKELETAL MUSCLE
WHAT ARE THE CONTRIBUTORS TO SMOOTH MUSCLE CONTRACTION? SPONTANEOUS ELECTRICAL ACTIVITY OF CELL MEMBERAIN NEUROTRANSMITTER(AUTONOMIC SYSTEM) LOCAL ENVIRONMENTAL INFLUENCE,PH,OXYGEN, ETC. RAPID STRETCHING OF MUSCLE
DEMOGRAPHICS OF CARDIAC MUSCLES STRAITED WITH WELL DEVELOPED SARCOPLASMIC RETICULUM HOVEVER, LIKE SMOOTH MUSLCE IN THAT SOME FIBERS PRODUCE SPONTANEOUS ACTIVITY
WHAT LAYER DOES THE NERVOUS SYSTEM ARISE FROM ECTODERM
NERVOUS SYSTEM FUNCTIONS TRANSMIT BRAIN FAST TRANSMISSION
ENDOCRINE SYSTEM FUNCTIONS TRANSMITS PITUITARY SLOW
NERVOUS SYSTEM PERFORMS WHAT? EXCITABILITY CONDUCTIVITY
DEFINE EXCITABILITY CAPABLE OF REACTING TO STIMULUS
DEFINE CONDUCTIVITY CAPABLE OF TRANSMITTING IMPULSES
WHAT TWO CELLS ARE ASSOCIATED WTH THE NERVOUS SYSTEM CENTRAL PERIPHERAL
THE CENTRAL NERVOUS SYSTEM CONSISTS OF BRAIN AN SPINAL CORD
THE PERIPHERAL NERVOUS SYSTME CONISIST OF NERVES FROM THE CNS TO MUSCLES, GLANDS ETC.
WHAT ARE THE COVERINGS OF THE (PNS) SCHWANN CELLS
WHAT IS THE FUNCTION OF SCHWANN CELLS FORMS A JELLY ROLL COVERING AROUND NERVE FIBERS
WHAT IS THE INNER COVERING OF THE SCHWANN CELL MYENLIN= LIPOPROTEIN
WHAT IS THE OUTER COERING OF THE SCHWANN CELL NEUROLIMMA W/O MYELIN CONTAIN NUCLEUS
IN THE BRAIN AND SPINAL CORD WHAT IS WHITE MATTER (COVERING AROUND MYELIN) GROUPS OF MYELINATED FIVERS (PRODUCED BY NEUROGLIAL CELLS
GREY MATTER UNMYELINNATED NERVE FIVERS AND CELL BODIES
NODES OF RANVIER GAP BETWEEN SCHWANN CELLS
WHAT ARE THE ELECTRICAL ARCING OR JUMPING FROM NODE TO NODE SALTATORY CONDUNCTION
WHAT IS THE DEMOGRAPHICS OF THE NEURON? STRUCTURAL FUNCTIONAL UNIT NERVOUS SYSTEM (INVOLVED WITH THE REFLEX ARC
WHAT IS THE CELL BODY? (INSIDE THE VERTEBRA)MOST CELL BODIES ARE WITHIN THE CNS
CELL BODY PARTS ARE ? 1. GANGLIA= GROUP OF CELL BODIES IN A CLUSTER COVERED WITH CONNECTIVE TISSUE 2. NUCLEI=GOUP OF CELL BODIES IN THE BRAIN +SPINAL CORD 3.CENTER=GRP OF NUCLEI ASSOCIATED WITH A PARTICULAR CENTER
WHAT ARE THE TWO PROCESSES THAT EXTENDS FROM THE CELL BODY?` DENDRITE AXON
DESCRIBE DENDRITE MAIN RECEPTIVE SURFACE OF THE NEURON (GOES INTO THE NEURON BODY) SHORT HIGHLY BRANCHED
DESCRIBE AXON SINGLE FIBER FROM NERVE BODY MAY HAVE MANY BRANCHES AT TERMINALS (AWAY FROM NERVE BODY NEURON)
AXON PLASMIC FLOW, FLOWS FROM CYTOPLASM DOWN THE AXON-THOUGHT TO SUPPLY NEEDED MATERIALS(CARRY NUTRITION DOWN TO THE NERVE FIBER OF THE AXON
AXON HILLOCK IS ALSO A PART OF THE NEURON, CONICAL ELEVATION WHERE AXON LEAVES THE CELL BODY RIGHT AT THE HUMP
AXON COLLATERAL IS ALSO PART OF THE NEURON BODY, SIDE BRANCHES FROM AXON WHICH DIVIDES INTO TERMINALS KNOWN AS TELODENDRIA
TELODENDRIA IS ALSO PART OF THE NEURON BODY, FINE FILAMENTS AT TERMIANL END OF AXON
AXON X.S CONSIST OF NEUROLEMMA-OUTER COVERING MYELIN- AXON SCHWANN CELL NUCLEUS
MYELINATION PROCESS OF SCHWANN CELLS MYELINATION FIBERS NOT COMPLETE UNTIL ADOLESCENCE NERVOUS SYSTEM NOT AS EFFICIENT
PARTS OF CELL BODY LRG NUCLEUS NUMEROUS NISSL BODIES NEURO FIBRILS INCLUSION
LARGE NUCLEOLUS STORES MRNA
NUMEROUS NISSL BODIES MEMBRANOUS SACS SIMILAR TO ROUGH E.R.
NEURO FIBRILS FINE FILAMENTS WHICH EXTEND INTO FIBERS
INCLUSIONS BY PRODUCT OF LYSOSOME ACCUMULATE IN NEURON+PUSH ASIDE OTHER ORGANELLES MAY BE AGING PROCESS
WHAT ARE NEUROGLIAL CELLS (GLIAL CELLS) CAPABLE OF REPRODUCTION FOUND ONLY IN BRAIN+SPINAL CORD MORE NUMEROUS THAN NEURONS IN CNS TUMORS OF CNS FILL EMPTY SPACES
FUNCTION OF GLIAL CELLS FILL SPACES SUPPORT NEURON HOLD(CONNECT)ORGANS TOGETHER PROTECTION AND CLEANING (PHAGOCYTIC)
ASTROCYTES ARE TWINE AROUND NEURONS TO FORM SUPPORT ATTACH NEURONS TO THEIR BLOOD VESSELS RMOVE DEBRIS AND SEAL OFF DMAGED AREAS
OLIGODNEDROCYTES ARE NEROGLIAL CELLS; FROM MYELIN IN CNS FORM SEMI RIGID CONNECTIVE TISSUE ROWS BETWEEN NEURONS
MICROGLIA CELLS ARE NEROGLIAL CELLS HELP IN REMOVING DEBRI BECOME MOR NUMEROUS IN BRAIN INFLAMMATION SOMTIMES MIGRATE INTO INFLAMED AREA
EPENDYMAL CELLS ARE NEUROGLIAL CELLS CUBOIDAL/COLUMNAR SOMETIMES WITH CILIA FORM (ONE CELL LAYER) MEMBRANES
MEMBRANES OF THE NEUROGLIAL CELLS LINES CAVITIES: VENTRICLES(SPACES OF THE BRAIN) CENTRAL CANAL ( SPINAL CORD CANAL) CHOROID PLEXUS: SPECIAL CAPILLARIES WHICH SECRETE INTO VENTRICLES , FORMS THE BRAIN FLUID
SOMATIC SENSES OF TOUCH CORPUSCLES OF TOUCH (MEISNER'S CORPUSCLES) HAIR ROOT PLEXI HAIR ROOT PLEXI
SHALLOW TOUCH MERKEL DISCS (TOUCH STRATUM BASALE
DEEPER TOUCH RUFFINI CORPUSCLES (IN DERMIS)
PRESSURE LAMELLATED (PACINIAN) CORPUSCLE ONION
LOW FREQUENCY VIBRATION MEISNER'S CORPUSCLE
HIGH FREQUENCY VIBRATION LAMELLATED CORPUSCLES (PACINIAN CORPUSCLES)
PROPRIOCEPTORS MUSCLES ASSOCIATED WITH BLIND BALANCE REFLEXES
REFACTORY PERIOD IN NURVE IMPULES DELAY BEFORE ANOTHER IMPULSE
ABSOLUTE IMPULSE NO IMPUSE(REESTABLISHING RESTING POTENTIAL)
RELATIVE REFRACTORY W/HIGH INTENSITY "HIT HARDER" STUMULUS IMPULSE TRIGGER (POLARIZATION NOT COMPLETE)
CONDUCTION DIFF TYPES OF NERVE IMPULSES
SALTATORY NODES OF RAVIER
DIAMETER LARGER DIAMETER=FASTER IMPULSE
WHAT'S THE BENEFIT FOR CALCIUM ION NEEDED FOR CLOSURE OF SODIUM CHANNELS
CALCIUM DEFICIENCY MAY RESULT FROM PRGNANCY OR DIARHEA
WHAT'S A PRESYNAPTIC CELL & POST SYNAPTIC CIRCUITS I-IV
DIVERGING CIRCUIT ONE IMPULSE--SEVERAL RESPONSES
CONVERGING CIRCUIT MORE STIMULUS FROM ONE SOURCE OR FROM DIFFERENT SOURCES
REVERBERATING CIRCUIT CONTINOUS IMPULSES
PARALLES CIRCUIT STREAM OF IMPULSES
WHAT'S THE SPINAL CORD BEGINS AT THE FORAMEN MAGNUM ENDS AT 2ND LUMBAR VERT. 31 SEGMENTS- EACH WITH SPINAL NERVES (PAIR)
WHAT ARE THE 2 ENLARGEMENTS OF THE SPINAL CORD CERVICLE--NERVES TO ARMS LUMBAR--NERVES TO LEGS
WHAT ARE THE FUNCTIONS OF THE SPINAL CORD CONDUCT IMPULSES BY ASCENDING--DESCENDING TRACTS CENTER FOR SPINAL REFLEXES
WHAT DOES SPINAL TRACTS CONTAIN ORIGINATIN + TERMINATING NEURONS-NAMED ACCORDINGLY SPINO THALMIC TRACT.....
WHAT IS THE WHITE PORTION OF THE SPINAL CORD ANTERIOR, POSTERIOR,LATERAL FUNCICULUS
GRAY PORTION, ANTERIOR, LATERAL, POSTERIOR HORN
MONOSYNAPTIC INTERCONNECTIONG NEURON PAIR
POLYSYNAPTIC 2 OR MORE INTERCONNECTING NEURONS
SPINAL SHOCK DERPESSED ACTIVITY WITH LESSENING OF SENSATIONS FOR DAYS TO WEEKS
PARALYSIS COMPLETE LOSS OF MUSCULAR CONTROL
FLACCID P. TOTAL LOSS OF TONE ATROPHY OF MUSCLES CAUSED BY DAMAGE OF CORTICO SPINAL TRACT
SPATIC P. MUSLCE TONE NOT ATROPHY UNCOORD. RELFEX DURING MOVEMENT MUSLCE SPASMS
Created by: Orzealyea
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