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cardiovascular SCC

meds, diagnostic test, disorder, nursing implications

Goal serum cholesterol level <200mg/dl
What is the main lipid associated with arteriosclerotic vascular disease? cholesterol
No alcohol for 24 hours before which test? serum lipid
Which foods should be avoided in a low cholesterol diet? animal sources such as animal fats, eggs, organ meats, and dairy products. Increase intake of fruits and veggies.
Increased levels of cholesterol are associated with what? hypertension, MI, atherosclerosis, stress.
digoxin Lanoxin
chlorothiazide Diuril
Hydrothiazide Microzide
furosemide Lasix
bumetanide Bumex
spironolactone Aldactone
amiloride Midamore
enalapril Vasotec
lisinopril Prinivil, Zestril
captopril capoten
nitroglycerin nitrostat
isordil nitrobid
propranolol Inderal
atenolol Tenormin
nifedipine Procardia
verapamil Calan, Isoptin
streptokinase Streptase
alteplase TPA
warfarin Coumadin
aspirin ASA, empirin
ticlopidine Ticlid
clopidogrel Plavix
Inotropic Cardiac Glycosides digoxin (Linoxin)
Thiazides diuretics chlorothiazide (Diuril)
Thiazides diuretics hydrothiazide (Microzide)
Loop diuretics furosemide (Lasix)
Loop diuretics bumetadine (Bumex)
Potassium sparing diuretics spironolactone (Aldactone)
Potassium sparing diuretics amiloride (Midamore)
ACE inhibitor enalapril (Vasotec)
ACE inhibitor lisinopril (Prinivil, Zestril)
ACE inhibitor catopril (Capoten)
Beta blockers propranolol (Inderal)
Beta blockers atenolol (Tenormin)
Calcium Channel Blockers nifedipine (Procardia)
Calcium Channel Blockers verapamil (Calan, Isoptin)
Thrombolytics steptokinase (Streptase)
Thrombolytics alteplase (TPA)
anticoagulants heparin
anticoagulants warfarin (Coumadin)
antiplatelets aspirin (ASA, Empirin)
antiplatelets ticlopidine (Ticlid)
antiplatelets clopidogrel (Plavix)
Isotropic Cardiac Glycosides MOA slows and strengthen heart, increases myocardial contractility, exhange of ca and na ions into myocardial cells
diuretics MOA decreases circulating blood volume inhibits reabsorption of sodium and other metabolites except for in potassium sparing
ACE inhibitors MOA block the effect of renin-angiotensin-aldosterone system,reduces vasoconstriction, reduces sodium and water rentention, decreases cardiac workload and edema
Nitrates MOA relaxes smooth muscle and blood vessels resulting in vasodialation= decreased venous pressure, decrease venous return decrease blood volume, decrease o2 need and workload of the heart
beta blockers MOA blocks SNS stimulation, decreases HR, decreases BP, decreases myocardial contraction= decreased myocardial 02 demand
calcium channel blockers MOA improves blood supply to myocardium by dialating coronary arteries, decreases work load of heart by dialating peripheral arteries, prevents movement of extracellular ca into cells, decreases myocardial contralitity and slows av node conduction.
thrombolytics MOA act by stimulating conversion of plasminogen to plasmin. Plasmin is a proteolytic enzyme that breaks down fibrin famework of thrombus
anticoagulants MOA inactivates clotting factors and thrombin so that thrombus formation is prevented. Does not dissolve existing thrombus but prevents it from getting larger
Warfarin (Coumadin) antidote Vit K
Heparin antidote protamine sulfate
antiplatelets MOA inhibits platelet aggregation and thrombus formation
Created by: 1161798020