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F/E MCC Block 1

Chpt 52 Kozier and Kee

QuestionAnswer
Role of Sodium (Na+) (extracellular) osmolarity, affects water distribution between ECF and ICF. Regulates body fluids.
Na sources bacon, beef cubes, catsup, corned beef, ham, tomatoe juice, soda crackers, pickles
Hyponatremia lab values Na < 135 mEq/L
Symptoms of Hyponatremia muscle weakness, headaches, lethargy, confusion, siezures,abdominal cramps, tachycardia
Hyponatremia etiology vomiting, diarrhea, surgery, diuretics( water loss), kidney disease, excessive perspiration
Hyponatremia management fluid restictions, Na rich foods, monitor I/O, monitor labs
Hypernatremia lab values Na >145 mEq/L
Symptoms of Hypernatremia Flushed dry skin, agitation, elevated body temperature, increased BP, nausea, vomiting, hypertension, muscle twitching
Hypernatremia etiology excess Na intake leads to elevated serum sodium which leads to dehydration and increase in myocardial depolarization
Hypernatremia management strict I/O, encourage fluids, Na restriction, monitor labs
Normal lab values of Sodium (Na+) 135 - 145 mEq/L
Role of Calcium (Ca2+) Promote normal nerve and muscle activity. Promotes normal neve and muscl activity. Increases contraction of heart muscle. Maintain cell permeability, protes blood clotting,aides formation of bone and teeth
Sources of Ca dairy products, spinach, OJ, seeds, nuts
Hypocalemia etiology Vit. D deficient, pancreatisits, Alkalosis (high pH), chronic renal failure, chronic alcoholism
Hypocalcemia symptoms Chvostek's sign(facial nerves) , Trousseau's(carpal spasm from BP cuff)sign, numbness, tingling, hyperative reflex, tetany, muscle cramps, ECG abnormalities
Hypocalcemia management Calcium rich foods, supplements,
Hypercalcemia Etiology Osteometastasis, Paget's disease (brittle bones), osteoporosis, prolonged immobolization, acidosis (low pH)
Hypercalcemia symptoms anorexia, weekness, hypoactive reflex, flank pain, decreased LOC, personality change, cardiac arrest, THINK CARDIAC
Normal lab values of Calcium (Ca2+) serum ionized 4.5 (hypo) - 5.6 mg/dL (hyper) total serum 9.0 (hypo) - 10.5 mg/dL (hyper)
Role of Potassium (K+) Transmission and conduction of nerve impules and for contractioin of smooth cardiac and skeletal muscles, Changes carbs to energy and amino acids to protiein.
Sources of potassium fruits, fruit juice, veggies, bananas and dried fruits
Hypokalemia etiology When cells are damaged K+ leaks from the cell into the intravascular fluid and is excreted by the kidneys. With cellular lossof K+, K+ shifts from blood plasma into the cellto restore the cellular K+ balance. Vomit and diarrhea 80-90% excreted in Urine
Hypokalemia symptoms Nausea, vomiting, dysrhythmia, ab distention, soft flabby muscles.
Hypokalemia lab values K < 3.5 mEq/L
Hypokalemia management monitor heart rate/rhythm, oral/IV potassium, rich diet, education on K+ loss in diuretics
Hyperkalemia etiology Renal failure, hypoaldosteronism, potassium conserving diuretics, renal insufficiency
Hyperkalemia lab values K > 5.0 mEq/L
Hyperkalemia symptoms nausea, ab cramps, oliguria, tachycardia, weakness, nubness/tingling in extremities
Hyperkalemia management Monitor cardiac status and ECG, diuretics and glucose/insulin as ordered, Hold potassium supplements and K+ conserving diuretic
Function of Phospate (PO4-) primary ICf anion, essential to function of muscle, red blood cells & nervous system. Deposited wth Ca for bone and tooth structure, inversly related to Ca. Energy transfer in cells, celolular osmotic pressure, component of nucleic acids (DNA/RNA)
Function of Magnesium (MG2+) ICF intracellular metabolism, especially in the production and use of ATP. Necessary for protien and DNA synthesis within cells. transmits neuro muscular activity. myocardial contraction
Function of Chloride (Cl-) ECF. functions with Na+ to regulate sodium osmolity and blood volume. major gastric juice component as hydrochloric acid (HCI), regulates acid base balance
Function of Bicarbonate (HCO3-) ICF & ECF. regulates acid base balance as a component of the arbonic acid-bicarbonate buffering system
Phosphate lab values (PO4)_ 1.8 - 2.6 mEq/L or 2.5 - 4.5 mEq/L
Magnesium lab value (MG2+) 1.5 - 2.5 mEq/L or 1.6 - 2.5 mEq/L
Chloride lab value (Cl-) 95 - 108 mEq/L
oliguria low urine output <30cc/hr
ICF intracellular fluids fluids with in cell membreanes, 40 % body weight, provides cells with internal aqeous medium used for chemical functions
ECF extracellular fluid fluid found outside of cell membrane, 15 - 20% of body weight, transport system
Components of ECF plasma (intravascular fluid) 20% of ECF and intersistal fluid (in tissues) 75% of ECF
List four ways body fluids move filtration, diffusion, osmosis & active transport
Filtration movement of fluid through a membrane as a result of hydrostatic pressure
Diffusion process by which solid, particulate matter moves from an area of higher concentration to an area of lower concentration
Osmosis water moves through semipermeable membrane from a solution of lower concentration to a high concentration
active transport metabolic energy (Na/K pump) is expended to move cells from less consentrated solution to more concentrated one
thirst center of brain regulated by hypothalamus. stimulated by increased serum osmolarity and decreased blood volume to kidneys
four routes of fluid output urine, feces, insensible loss (lungs, perspiration)
sensible loss perceived by individual
insensible loss thorugh skin and lungs. not noticeable to individual
ADH Antidiuretic hormone released from hypothalamus stimulation, increases the reabsorption of water into blood decreasing serum osmolality.
Aldosterone mineralcorticoid produced by adrenal cortex, causes kidneys to reabsorb Na and excrete K (increased Na = Increased water retention)
Fluid Volume Deficit FVD loss of water and electrolytes fom ECF AKA: hypovolemia
FVD causes abormal loss through skin, GI or kidney, decreased fluid intake, movement of fluid to third space
Fluid Volume Excess FVE retention of water and electrolytes in equal portions to normal ECF. AKA: hypervolemia
Symptoms of FVD dry skin and mucous membrane, poor skin tugor, coated tongue, low BP, collapsed vein, weak pulses, oliguria
Symptoms of FVE increased Hgb and Hct, Increased BUN, increased specific gravity, increased serum osmolarity
BUN Blood Urea Nitrogen - renal/liver function 10 - 20 mg/dL or 3.6 - 7.1 mmol/L (SI units)
RBC Red Blood Count - hemoglobin and hematocrite Male: 4.7 - 6.1 million Female: 4.2 - 5.4 million
WBC Leukocytes - evaluates for infection 1. Tota number of WBC: 5000 - 10000mm3 2.Differential (% of each type of WBC)
Creatinine Impaired renal function Male: 0.5 - 1.1 mg/dl Femle: 0.6 - 1.2 mg/dl
HgB Amount of HgB in blood as part of CBC rapid measurement of RBC Male: 14 - 18 g/dl Female: 12 - 16 g/dl
Hct Hematocrit % of RBC found in 100 ml Male: 0.42 - 0.52 volume fraction (%) Female: 0.37 - 0.47 volume fraction (%)
K Potassium electrolyte/cardiac essential Blood: 3.5 - 5.0 mEq/L
Na Sodium electrolyte Blood: 135 - 145 mEq/L
Ca Calcium parathyroid function and Ca metabolism Adult Total: 9.0 - 10.5 Adult Ionized: 4.5 - 5.6
ABG Arterial Blood Gas pH: 7.35 - 7.45 PaCO2: 35 - 45 mmHg HCO3: 22 - 26 mEq/L PaO2: 80 - 100 mmHg
list four types of acid/base imbalances Respitory Acidosis, Respitory Alkalosis, Metabolic Acidosis, Metbolic Alkalosis
Respitory Acidosis ABG pH<7.35, PaCo2>45, HCO3 about normal 22 - 26
Respitory Alkalosis ABG pH>7.45, PaCo2<35
Metabolic Acidosis ABG pH<7.35, HCO3<22
Metabolic Alkalosis ABG pH>7.45, HCO3>26
S/S Respiratory Acidotic state Increased HR & RR, headache, diziness, confusion, decreased LOC, convulsions, warm flushed skin
S/S Metabolic Acidotic state Kussmal respirations (deep/rapid), lethargy, confusion, headache, weakness, nausea, vomiting
S/S Resiratory Alkalotic state SOB, tight chest, light head, parathesis (burning/prickling), difficulty concentrating, tremulous, blurred vision
S/S Metabolic Alkalotic state decreased RR/depth, diziness, parathesis (burning/prickling), Tetany
What happens to the serum osmoality in a dehydrated patient? Increase. Decrease in fluid, leads to an increase proportion of solutes to fluid volume in the blood.
What happens to the serum hematocrit level in a patient with a FVE? Decreases. Increased fluid leads to a decrease in the proportion of a solutes to fluid volume in the blood.
Cations (+) Potassium K+ ; Sodium Na+ ; Calcium Ca2+ ; Magnesium Mg2+
Anions (-) Chloride Cl- ; Bicarbonate HCO3- ; Phosphate PO4- ; Sulfate SO4-
Adult Body Fluid Volume ICF 40%, ECF 20% - Intracellular 15% & Intravascular 5%, Total body fluid 60%
platelets thrombocytes: 150K - 400Kmm3
R.O.M.E. Acid Base Balance interpret RESPIRATORY = OPPOSITE (Ph/PaCO2 values opposed means respiratory problem) METABOLIC = EQUALS (pH/PaCO2 values both high or low means a metabolic problem)
pH acid or base A=Acid <7.35 B=Base >7.45 Alkaline
Metabolic Acidosis Excessive blood acidity caused by and overabundance of acid or loss of bicarbonate from the blood
Respiratory Acidosis a buildup of carbon dioxide in the blood that results from poor lung function
Metabolic Alkalosis overabundance of bicarbonate in the blood or a loss of acid in the blood
Resiratory Alkalosis carbon dioxide in the blood that results from rapid or deep breathing
Created by: lucky duck
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