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375 Exam 1

COPD, Pulmonary Hypertension

TermDefinition
high CO2 hypercapnic, give a bipap
copd exacerbation avoid opioids (lead to less ventilation), no benzodiazepines
copd diet teaching diet, oral hygiene before meals, small frequent meals, high calories and protein, avoid exercise 1 hour before and after meals, avoid gassy foods and carbonation, avoid high fiber (causing bloating and more pressure0
fluid copd teaching increase fluid intake to thin mucus, avoid drinking fluids while eating
infection copd teaching report increase in sputum, fever and worsened dyspnea
meds copd teaching albuterol used for shortness of breath
pursed lip breathing inhale for 2 seconds, exhale 4 seconds with pursed lips; helps decrease shortness of breath, prevents air way collapse and airway trapping
huff coughing cough out thick mucus; sit upright in chair, feet shoulder width, lean forward, deep slow inhalation through mouth and diaphragm muscle, hold breath 2 -3 seconds and forcefully exhale, repeat once or twice to clear
chronic bronchitis daily productive cough for three months or more in at least two consecutive years, damaging to the airways
bronchitis manifestations overweight and cyanotic, elevated hemoglobin, peripheral edema, rhonchi and wheezing
emphysema permanent enlargement and destruction of airspaces distal to the terminal bronchiole
emphysema manifestations older and thin, severe dyspnea, quiet chest
copd manifestations underweight, dyspnea on exertion, SOB, progressive cough and sputum, air hunger, tripod position and barrel chest
diagnosis copd pulmonary function's test, chest xray, test cat, echo
forced vital capacity (FVC) maximum volume of air that can be forcibly expired (3 seconds)
forced expiratory volume in 1 second (FEV1) volume of air that can be expired in the first second of a forced expiration, 70% or above
increased residual volume copd air left in the lungs after forced exhalation
spirometry confirms presence of airflow obstruction
copd alterations in ABG decreased O2, increased CO2, decreased pH, increased HCO3
6 minute walk test measures distance an individual is able to walk over a total of 6 minutes, self paced, exercise is a predictor of risk for mortality, can determine functional status and response to treatment
risk modification copd smoking cessation, hand washing, avoid irritants, vaccines, Tdap vaccination
bronchodilators preferred to improve ventilation, decrease dyspnea and increase FEV
SABA short acting bronchodilator agonist - rescue inhaler; albuterol, makes you anxious
LABA long acting bronchodilator agonist, maintenance inhaler; salmeterol
anticholinergic, muscarinic block acetylcholine, decrease mucus production (end in ium)
SAMA short acting muscarinic antagonist; ipratropium
LAMA long acting muscarinic antagonist; tiotripium
corticosteroids decrease inflammation, inhalers are used in a stepwise approach; SABA, LABA, SAMA, LAMA, corticosteroid then combination inhaler
phosphodiesterase inhibitor for severe copd and chronic bronchitis; roflumilast
long term o2 therapy improves survival, exercise capacity, cognitive performance, sleep
nasal cannula patient's can eat, talk and cough; most common and comfortable
simple face mask low flow system, delivers 5 - 8L/min = 40 - 60% fiO2 depending on fit and oxygen flow; never run at less than 5LPM or CO2 may buildup in mask; must remove to eat and oral care
non rebreather valves going to the reservoir bag prevent CO2 being re inhaled, highest FiO2 without becoming invasive (intubation)
venturi mask delivers precise amount of oxygen by use of adapters or dial selector making it most accurate non invasive method; liter flow as indicated on the dial or adapter, no humidification necessary
goal of pursed breathing prolong exhalation and increase airway pressure during expiration; improves oxygen transport; do not puff cheeks
airway clearance technique loosen mucus and secretions to be cleared by coughing, huff coughing
chest physiotherapy postural drainage (2-4 x/day), percussion, vibration
copd malnutrition increased metabolic rate, lack of appetite, altered taste, weight loss
goals for copd reduce symptoms, reduce risk, prevent progression
lung volume reduction surgery (LVRS) reduce size of hyper inflated lungs, decrease airway obstruction and increase room for normal alveoli
bronchoscopy lung volume reduction surgery valves used for endoscopic lung volume reduction; removable one way flow devices placed by flexible bronchoscopy into selected airways supplying emphysematous lung
pulmonary hypertension elevated pulmonary arterial pressure from a increase in resistance to blood flow through the pulmonary circulation
normal pulmonary pressure 12 - 16 mmHg
risk factors for pulmonary hypertension 30 - 60 years of age, higher among women, history of blood clots, diseases that change the chest wall, hepatitis b or c, liver disease and thyroid disease
idiopathic PAH cause unknown, injury to endothelium leads to vascular scarring, smooth muscle proliferation and endothelial dysfunction
manifestations of idiopathic dyspnea on exertion, fatigue, exertional chest pain, dizziness; with progression abnormal s3 heart sound or ventricular gallop
secondary pulmonary hypertension chronic increase in artery pressures from another disease
assessment findings secondary PAH RV hypertrophy, s4 or atrial gallop, peripheral edema, increased peripheral venous pressure, hepatomegaly because the portal vein is unable to circulate
chest x ray PAH visualize pulmonary system, hypertrophy of heart; reveals no vasculature, this is to rule out other disease process and see enlargement of the heart
BNP hormone, an increase reflects heart failure
transthoracic echocardiogram (TTE) ultrasound looks at blood flow and measures pressures in the heart; mean pulmonary artery pressure; will report ejection fraction - normal 55 - 70%
pulmonary functions test look for other underlying conditions, COPD, pulmonary fibrosis
right heart catheterization accurate measurement of right atrial pulmonary artery pressure, cardiac output and pulmonary vascular resistance
cath procedure catheter is placed in the femoral, internal jugular or subclavian veins and advances into the vena cava into the right atrium; balloon is inflated on the tip of the catheter and wedges in the pulmonary artery
calcium channel blockers for PAH cause blood vessels to relax and widen, lowers artery pressure - dilitazem or amlodipine
CCB teach count HR and check BP, change position slowly, bad headaches because blood vessels in the brain vasodilate
endothelin receptor antagonists PAH block the constriction of pulmonary arteries, relaxing and decreasing the pressure; need to monitor LFT; bosentan
phosphodiesterase inhibitors PAH promote smooth muscle relaxation in lung vasculature; side effects of hypotension and bradycardia; sildenafil
inhaled vasodilators PAH dilates systemic and pulmonary arterial vasculature; nebulizer 6 - 9 days; need to check BP; iloprost
parenteral vasodilators PAH reduces pulmonary vascular resistance, continuous IV or subcutaneous, short half life; education is essential; epoprostenol
diuretics PAH used to manage peripheral edema
anticoagulants PAH prevent thrombus formation; patients are at risk for blood clots in the small arteries in the lungs
pulmonary thromboendarterectomy removes clots from the pulmonary arteries
Created by: ahommel
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