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Pharm10
| Question | Answer |
|---|---|
| -zosin | alpha adrenergic blocker |
| -lol | beta adrenergic blocker |
| -zepam, -zolam | BZO |
| -pril | ACEI |
| -sartan | angiotensin II receptor blocker |
| -dipine | Ca channel blocker |
| -statin | LDL-lowering HMG-CoA reductase inhibitor |
| -sone, -olone | corticosteroid |
| -parin | anticoagulant |
| -prazole | PPI |
| -terol, -phylline | bronchodilator |
| -semide | loop diuretic |
| How does alpha-2 stimulation differ from other sympathetic receptor sites? | Can inhibit release of epinephrine or NE from presynaptic neuron |
| Adrenergics, sympathomimetics, adrenergic agonists mimic the effects of? | NE |
| Sympatholytics, adrenergic blockers, and adrenergic antagonists block the effects of | NE |
| Cholinergics, parasympathomimetics, cholinergic agonists, cholinomimmetics mimic the effects of | Ach |
| Anticholinergics, parasympatholytics, and cholinergic antagonists block the effects of | Ach |
| Sx of sulfite sensitivity | Dizziness, loss of consciousness, hypotension, syncope, clammy flushed skin, pruritus, cyanosis, bronchospasm, wheezing, SOB, laryngeal edema, respiratory arrest Epinephrine, NE, phenylephrine, methoxamine, dopamine, dobutamine, metaraminol, fenoldopam |
| Pt edu for pts receiving clonidine (Catapres) | Never stop abruptly- can cause rebound HTN |
| How does dopamine (Intropin) affect renal hemodynamics? | High doses (bind alpha-1 receptors) cause vasoconstriction in kidneys, can cause renal failure. Low doses (binds dopaminergic receptors) causes increased GFR, renal blood flow, and sodium excretion (overall, increased renal output) |
| Why is dopamine (Intropin) and fenoldopam (Corlopam) only admined in acute care envts? | Need to closely monitor BP, CO, urine output, pulmonary wedge pressure b/c can cause severe dysrhythmias and vasoconstriction (dopamine) or acute cerebral infarction or hemorrhage (fenoldopam) |
| AE of prazosin (Minipress) | 1st dose syncope |
| Why are beta blockers used cautiously in pts with resp disorders? | Can cause development of obstructive pulmonary disorders |
| Cholinergic crisis | overstimulation at nm junction due to too much Ach, give atropine |
| Myasthenic crisis | lack of Ach due to undermedication, give more prostigmin can cause inability to speak, breathe, or swallow in pts with myasthenia gravis |
| Common AEs of atropine | blurred vision, urinary retention, constipation, dry mouth |
| Location of beta 2 receptors | lungs |
| location of beta 1 receptors | heart, kidneys |
| location of alpha 1 receptors | prostate, peripheral vasculature |
| location of alpha 2 receptors | terminal end of presynaptic neuron |
| propranolol (Inderal) effects on blood glucose | None, but inhibits perception of hypoglycemic sx |
| Body SA | sqrt [weight (kg) x height (cm)]/3600 |
| -triptans | serotonin agonists |
| LFTs | bilirubin, AST, ALT, alk phosphatase, PTT, aPTT |
| renal function tests | BUN, serum creatinine, creatinine clearance |
| hematopoietic function tests | CBC- Hgb, Hct, WBC, platelets |
| 6 rights of drug admin | Right: patient, time, drug, dose, route, documentation |
| IM sites | deltoid, rectus femoris, vastus lateralis, dorsogluteal, ventrogluteal |
| half-life | time required to remove 50% of the blood [drug] |
| steady state | 4-5 half lives, admin rate=excretion rate |
Created by:
lmr24
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