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243 CH 27

243 EXAM 3

TermDefinition
pericardium double layer serous membrane isolates heart from other thoracic structures
pericarditis inflammation of the pericardium
pericarditis manifestations chest pain, pericardial friction rub, ECG changes, dyspnea, fever, malaise
pericardial effusion accumulation of fluid in the pericardial cavity, leads to cardiac tamponade
cardiac tamponade medical emergency, slow or rapid compression of heart due to accumulation of fluid, pus or blood in pericardial sac
cardiac tamponade manifestations pulsus paradoxus, hypotension, muffled heart sounds, jugular venous distention
pulsus paradoxus drop in SBP by at least 10 mmHg during inspiration
jugular venous distension fluid causing pressure
rheumatic fever acute inflammatory, follows strep throat, involves immune attack on self tissues
acute stage rheumatic fever mesenchymal connective tissue of the heart, blood vessels, joints and subcutaneous tissue
acute rheumatic fever manifestations carditis, rash, joint pain, chorea, fever, subcutaneous nodules
infective endocarditis invasion and colonization of endocardial structures
predisposing factors of infective endocarditis valvular disease, prosthetic heart valves, pacemaker, congenital heart defects, portal of entry to circulatory system, bacteria in bloodstream
acute infective endocarditis fever, chills, malaise, petechiae, splinter hemorrhage
subacute infective endocarditis low grade fever, fatigue, weight loss, flu symptom's, positive blood cultures
kawasaki disease acute febrile disease of young children, acquired heart disease, vasculitis is small vessels, progresses to involve large arteries
acute kawasaki fever, conjunctivitis, rash, oral mucosa, redness and swelling hands and feet, enlarged cervical nodes
subacute kawasaki defervescence and desquamation
convalescent kawasaki complete resolution of symptoms until all signs of inflammation disappear (after 8 weeks)
post kawasaki complication 15 - 25% develop, myocardial infarction, sudden death, chronic coronary insufficiency
valvular disorders stenosis and regurgitation - congenital, rheumatic, degenerative calcification, infective
stenosis impeding forward flow, valve restricted creates pressure gradient, increases volume and work of chamber emptying through
aortic stenosis obstruction of flow from left ventricle to aorta during systole
left ventricular hypertrophy increased workload, pressure greater, reduced CO
aortic stenosis manifestations angina, syncope, heart failure, systolic crescendo - decrescendo murmur
heart failure occurs when valve orifice in 1/3 normal size
aortic stenosis causes bacterial endocarditis, trauma, aortic dissection, rheumatic heart disease, congenital bicuspid aortic valve, syphilis
mitral stenosis large left atrial to left ventricular pressure gradient during ventricular diastole, left atrial hypertension and pulmonary congestion, right sided heart failure
mitral stenosis manifestations dyspnea, palpitations, fatigue, diastolic murmur, CVA, emboli from stagnant blood in atrium
regurgitation incomplete closure of valve leaflets, backward flow of blood
aortic regurgitation retrograde flow from ascending aorta to left ventricle, volume overload, pulmonary hypertension and right ventricular failure develop
aortic regurgitation manifestations diastolic murmur, cardiovascular collapse, weakness, dyspnea, orthopnea, paroxysmal nocturnal dyspnea, hypotension
mitral regurgitation due to myocardial infarction, chronic rheumatic heart disease, isolated rupture of chordae tendinae, mitral valve prolapse, ischemic papillary muscle dysfunction, infective endocarditis
mitral regurgitation manifestations some degree of left ventricular failure, weakness, fatigue, dyspnea, orthopnea, paroxysmal nocturnal dyspnea, pan-systolic murmur, abnormal flow during systole
mitral prolapse structure abnormality of mitral valves and papillary muscles or chordae that allow leaflets to backflow into left atrium during systole causing enlargement
mitral prolapse causes redundant mitral valve leaflets, elongated chordae tendinae, enlarged mitral annulus, abnormal left ventricular contraction
mitral prolapse manifestations decreased CO, dizziness, arrhythmias, tachycardia, palpitations, chest pain
cyanotic heart defects blue due to deoxygenated blood going to left side of heart then to blood stream
tetralogy of fallot birth defect ventricular septal defect, overriding aorta, pulmonary stenosis, right ventricular hypertrophy
tet spells sudden decrease in blood flow to the lungs, hands feet and mouth
tetralogy of fallot manifestations polycythemia from chronic hypoxemia, increase risk for thrombus
transposition of the great vessels life threatening position of the two major vessels that carry blood away from the heart (aorta and pulmonary artery) is switched
acyanotic oxygenated blood enters right side of heart, second pass through the lungs, not cyanotic
atrial septal defect septal opening between atriums, more common in males, causes left to right shunt
ventricular septal defect opening between ventricles, left to right shunt, associated with other cardiac defects
eisenmenger's syndrome (reverse) begins left to right shunt, increased pulmonary blood flow, pulmonary hypertension, right ventricular hypertrophy, increase right ventricle pressure, reverse shunt right to left - cyanosis
ductus arteriosus communication/ passage between aorta and pulmonary artery, blood from right heart bypass lungs into systemic circulation, more pressure in aorta, left to right shunt
fetal circulation oxygenation occurs through placenta, pulmonary circulation is bypassed
formamen ovale incomplete fusion of septal wall, allows right to left shunt
coronary arteries surround heart
left main coronary artery supplies to anterior and left lateral portions of the left ventricle
right main coronary artery supplies majority of right ventricle and posterior LV that supplies the node
coronary blood flow openings for the coronary arteries to originate in the root of the aorta near the aortic valve
aortic blood flow main factor responsible for perfusion of coronary arteries
Created by: ahommel
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