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pathophysiology
| Question | Answer |
|---|---|
| ESR | ESR is the rate at which erythrocyte sediment in one hour in a test tube. length\time(1 hour) 15mm\1 hour ESR value man< 50yrs- <15mm\hr female <50 yrs< 20mm\hr it increases as you grow older. factor that affect high ESR> plasma cell, fibrinogen |
| why are there negative charges on the rbc membrane, and what is the source of ABO system. | the main source of negative charges are the carbohydrate(glycolyx) of the cell membrane. and is the main source of ABO system |
| what is the biggest immunoglobin | IgM, which get activated in cold called cold agglutins when warm it reduced Raynauds phenomenon. |
| what condition increases ESR | LUPUS inflammation-fibrinogen IgG TB-infection-fibrinogen IgG multiple myeloma , rouleux, IgG waldenstrom macroglobulinemia IgM Anemia pregnancy macrocytosis |
| what condition decreases ESR | hyperviscosity syndrome sickle cell microcytosis spherocytosis polycthemia-high erythrocyte, and low EPO |
| Hematocrit rate Uterine bleeding will haave | is the volume of rbc to the volume of the whole blood. reduced hematocrit rate |
| the is reticulocyte and the normal rate of reticulocyte | reticulocyte are immature red blood cells (in the process of EPO) in the bone marrow 0.5 to 2.5% reticulocyte depends on your hemoglobin(protein) , it is higher if hemoglobin is low from bleeding or lysis of rbc |
| resection of pyloric part of the stomach with periodic dark shadows beneath his eyes, hb-70g/l, erythrocyte 3.0, CI 0.7 what is your diagnosis | microcytic anemia |
| Activation of a number of hemostatic factors occurs through joining with calcium ions. What structure component allows adjoining of calcium ions | Gamma carrboxglutamic acid |
| Diabetes .ellitus cause ? That has a conical induration, reddish blue with a yellow center on the skin of the neck what is your diagnosis . | Furuncles |
| A change in the heart force and vessel can be detected and be represented into a graphical recording of carotid pulse waves what method will be applied | Sphygmography |
| An athlete has hemoglobin of 180g\l first of all, it will stimulate what | erythropoiesis(EPO) |
| A vomit has colour of coffee ground with a melena due to gastric ulcer CBC is very low what complication is this. | Hemorrhage<> hemosiderin |
| vomiting result into decrease fluid>>increase in erythrocyte, decrease blood plasma volume>hypovolemia and finally reduced circulation of blood what changes happens in the blood volume | increase in erythrocyte is polycthemia, and reduced circulation of blood is hypovolemia. polycthemia(increase in the number of Rbc in the body) hypovolemia |
| 5TH DAY after acute blood loss a hypochromia anemia( means less color of RBC ) what is the main mechanism of hypochromia development. | less color of RBC can be seen in immature RBC been produced from the bone marrow especially when there is loss of blood to compensate for that loss rbc is release. |
| a man has car accident that causes lost of blood and the bleeding continue to the 2nd day how will it affect the peripheral blood. | if bleeding occurs up to the 2nd day, it then means that there will be a decrease in RBC leading to 1.erythropenia due to this persistant blood loss, immature RBC is release that causes hypochromia and also a change in the size (anisocytosis) of the cell. |
| A high mountain climber will have hypoxia that stimulate what | EPO |
| A trauma with blood loss causes fatique, weakness, pale skin, pulse is 90beats per minute, how can this leads to anemia when | Hemoglobin concentration is low, because it a carrier of oxygen, it does it matter if blood volume and color index is reduced but if hemoglobin is reduced it will is the first to lead to anemia |
| what is 1.Aplastic anemia 2.Posthemorrhagic anemia 3.Hemolytic anemia 4.Anemia of chronic dx | 1. insufficient of EPO 2. Blood loss that will result decrease of Erythrocyte and Hemoglobin 3.premature destructure of mature RBC sickel, thala, toxins 4. Abnormal increased demand for new erythrocyte e.g chronic infection, inflammation, malignancy |
| iron deficiency anemia, check for color index and ferritin, total iron binding capacity or transferrin. | 1. blood loss 2. insufficient of dietary intake 3. poor absorption of iron |
| A child birth causes complication which result into massive blood loss and hypochromia anemia was diagnosed what pathological form of erythrocyte will develop. | less color of RBC can be seen in immature RBC(Anisocytosis) they are smaller than mature RBC, been produced from the bone marrow especially when there is loss of blood to compensate for that loss rbc is release |
| A moderate menorrhagia with eryth 3.6, HB-60g/l, reticulocyte(0.5 to 2.5%) 0.1%, in the smear has hypochromia, anisocytosis, microcythemia, poikilocytosis, what type of anemia is this. | Iron deficiency. |
| living in the highlands will cause increase in EPO, RBC, hemoglobin in the peripheral blood, what cause polycythemia. | is caused as a result of exogenous hypoxia |
| in the case of anemia, there are degenerative and regenarative forms of erythrocyte in the peripheral blood, name the regenative forms of erythrocyte | 1. Multipotenthematopoietic stem cells 2. Common myeloid progenitor 3. proerythroblast 4. Basophilic erythroblast 5. polychromatic erythroblast 6.Normoblast 7.polychromatic erythrocyte(Reticulocyte) is the ANSWER. |
| in the case of anemia, there are degenerative and regenarative forms of erythrocyte in the peripheral blood, name the Degenative forms of erythrocyte | Poikilocyte(acantocyte, target cell, ovalocyte, stomatocyte and sickle cell) Anisocytosis(schistocyte, microcyte, normocyte, macrocyte, megalocyte) |
| Due to injury, a patient loss 25% of circulating blood volume, name the urgent mechanism of compensation of blood loss. | will be increasein the movement of fluid from the extracellular matrix of tissuebetween blood and lymphatic to the INTRACELLULAR\INTERSTITIAL FLUID INFLOW IN THE VESSELS |
| Due to thermal burns up to 40% of the body surface due to careless fire handling, hematocrit reveal a violation of plasma and element ratio, what form of violation of the total blood volume is observed at the same time. | polycythemia hypovolemia burning result into decrease fluid>>increase in erythrocyte(polycythemia), decrease blood plasma volume(hypovolemia)>hypovolemia and finally reduced circulation of blood increase in erythrocyte. |
| what is absolute erythrocytosis in which erythrocyteis 9X10 and hematocrit is 60% what pathological condition will this leads to | Absolute Erythrocytosis is when Red cell mass(erythrocyte) is raised and hematocrit is elevated above prescribe limits, it could be due to intrinsic problem in the bone marrow seen in vaquez disease. KROK SAYS VEKEZA |
| Vitamn B 12 defiency anemia, which of the criteria is an indication of increase erythropoiesis | Vitamn B 12 is need to make RBCs, which carries oxygen through our body, not having enough Vit B12 will cause hypoxia that will leads to increase in erythropoiesis and increase in the number of reticulocyte (polychromatic erythrocyte) |
| A patient has hypochromic anemia with a whipped hair fall, there is an increased in fragility of nails and taste disturbance, what is the mechanism of these symptoms | ferriferous enzymes deficiency. |
| 26 years of man has 18% of erythrocyte of the spherical, ballshaped, flat and thorn like shape and a concavo-concave disks how is this phenomenon called. | Physiological poikilocytosis |
| 26 years of man has 20% of erythrocyte of the spherical, ballshaped, flat and thorn like shape and a concavo-concave disks how is this phenomenon called. | pathological poikilocytosis |
| A patient was diagnosis with iron deficiency, with hyperpigmentation of the skin, pigment of the liver cirrhosis, damage of the pancrease, and heart was seen, iron content in the serum was increased what violation of iron metabolism caused this disease. | first of all iron defic is due to hemorrhage(hemolytic anemia) that leads to hemosiderin that causes those hyperpigmentation you see on the skin, ANSWER THIS LEADS TO DISUSE OF IRON AND DEPOSITION IN NTISSUE. |
| A patient has Anephric patients has anemia symptoms. what is the cause. | Reduced synthesis of eryhropoietins will leads to anemia |
| A patient has autoimmune hemolitic cytotoxic anemia what substances are antigens in type2 allergic reaction | modified receptors of cell membrane |
| An increase in permeability of RBC in a patient with microspherocytic anemia(minkowsky shauffard disease) received sodium ions and water, erythrocyte takes the form of spherocyte that be easily be broken down , what is the leading mechanism damge. | minkowsky shauffard disease is a spherocytic (membranopathy) anemic disease the reason why the sphere like shaped of RBC can be easily be broken down is becuase of electrolytic osmotic due to increase surface-permeability of blood. |
| A 52 years female has decrease in the amount of RBCs and an increase in free hemoglobin in the blood plasma(hemoglobinemia), CI 0.85 what type of anemia is this. | decrease in the amount of RBCs is as a resuly acquired hemolytic anemia, that rupture rbc causing a seperation of hemoglobin that is un-bound with oxygen called free or naked HgB. this name Hgb Ccan be protected with haptoglobin(a protein gene HP) . |
| degenerative changes seen in th posterior and lateral column of the spinal cord(funicular myelosis) causes methylmalonic acid accumulation occurs in a patients with vitB 12 def anemia. this result in synthesis disruption of the following substance | myelin |
| A blood drop has been put into a test tube with 0.3% solution of Nacl what will happen to the erythrocyt | Osmotic hemolysis due to nacl causes increase osmotic pressure and loss of hemoglobin. |
| a man was bitten my a snake hemolysis will occur where | blood stream |
| A pin and needles like pain on the extremities, with long atropic gastritis , tongue looks crimson, Hb 60g\l, RBC 1.5\l, color index 1.2, isolated macrocyte, significant anisocytosis or erythrocyte what anemia has occur | B12-folic acid def anemia |
| A neonate is 3 weeks old, father is rh tve, mother is rh -ve, a clear jaundice of the skinand visible mucous , erythrocyte 3.7 X10, Hb 105g\l, elevated(free) bilirubin of blood. what is this anemia by pathogenesis | Acquired hemolytic |
| A 36 yrs CC of general weakness and bleeding gums, nasal bleeeding, symptoms occurs after having respiratory viral infection using rx of sulfonamide, hyporegenaratory, (normochromic anemia), leukopenia, thrombocytopeni, decrease of myelocyte, what anemia | hypoplastic or Aplastic Anemia |
| blood analysis of a pt hb 58%, erythrocyte 1.3, (CL 1.3), leukocyte 2.8, platelets-110 X 10, reticulocyte, ESR 35mm\h, determine (hypersegmented neutrophils) and also jolly corpuscles and kebot rings, what is the mechanism of anemia | vitamin B 12 folic acid def. |
| Hmolytic anemia caused by a genetic defect in the enzymes glucose 6 phosphate dehydrogenase in the erythrocyte, formation of what susbstance of pentose phosphate pathway is most violated in this case | NADPH |
| A mutated gene responsible for the sysnthesis of hemoglobin leads to a sickle cell anemia what pathological hemoglobin has developed. | HbS |
| A significant reduction of hemoglobin and erythrocyte in blood is due to consumption of FABA BEAN, what form of anemia is in this patients | fermentopathy this is a pathological disruption of the functioning of the structure of the human body enzymes. |
| A 40 years was diagnosis with sickle cell anemia what is the mechanism causing reduction of RBC in a patients | intracellular hemolysis>(NOT intravascular hemolysis this occur inside the vessel) erytrocyte can be destroyed by macrophages(extravascular hemolysis or intracellular hemolysis) of the mononuclear phagocytic system of the spleen, liver and bone marrow. |
| A patient has fermentopathy, what is the leading mechanism of hemolysis at defic of glucose 6 phosp dehydrogenase | Decrease in antioxidant protection |
| A tunisia pt has alpha thalasemia with hemolysis and jaundice, with disproportional increase in the ratio of surface membrane area to volume what is your diagnosis | Target cells or codocytes they are RBC that have the appearances of a shooting target with a bull eye. |
| A pt has hemolytic jaundice with a (microspherocyte) 1-6 insight, what is the possible reason of hemolysis of erythrocyte causing the emergence of this form of jaundice. | hereditary defect of their membranes. |
| A 35 yrs developed immune microspherocytic hemolytic anemia what rate of serum will mostly be increase | indirect bilirubin the reason is becuase there is increase in reticulocyte which causes increase immature RBC which then leads to indirect bilirubin. |
| A man working in gas discharge lamp, lead processing shop, blood test shows, HBG and RBC level is low, serum iron concentration exceeds the norm by several times, specify the type of anemia | iron refractory anemiai this is due to reduced ferroportin expression which then lead to def of iron in the blood stream, which does not improve even with oral fe2 |
| Gastric resection Billroth ll method with CL 0.69 | iron deficiency |
| Expressed iron def anemia appearance in the blood will be like | Anilocytosis |
| Violation of the synthesis of prophyrins | serum iron increases 37mmol, anisocytosis,poikilocytosis, HgB 74g, CS|CI 0.74 |
| DESIRE TO EAT CHALK and some kids receiving COWS MILK | IRON DEF |
| A resection of the pyloric stomach with an appearance of dark circles under the eyes, shortness of breath, Hb 70g, ER 3X10 what changes in the pheripheral blood smear will be seen | Hypochromic erythrocyte. |
| Castle instrinsic factor | is needed for Vitamin B12 absorption |
| Atrophic and inflammatory with a diagnosis of Addison biermer anemia what type of anemia is this | megaloblastic anemia |
| Serum iron of 80mkmol\L what pathology is it typical | Iron refractory anemia |
| Leukogram analysis has leukocyte of 16,000\L, juvenile form is 2%, stab nuclear is 8%, segmentonuclear is of 59%, lymphocyte is 25%, monocyte is 4% what your diagnosis | Left shift Reg, first of all Leukocyte is within 4500 to 11,000 this is a case of Neutrophilia, though there are present of immature segmented neutrophil but is just 59% compare to the high number of Juvenile form 2% (Normal is 0.6%), StabN 8%(N 0.6), |
| A pt has appendicitis, with basophil, eosinophil, myelocyte, juvenile are all 0, stab neutrophil is 20, segmentonuclear neutrophil 53%, lymphocyte 21, monocyte 5 what your diagnosis | Left shift Degeneration this is when immature neutrophil outnumber the present of mature segmented neutrophil, as you can see all mature neutropil are 0 esp stab Neutrophil but Immature segmentonuclear is 53. |
| What is Right shift Neutrophil | this is an increased number of mature or hyper segmented neutrophil and it occur in megaloblastic and aplastic anemia, aleukia, radiation dx, addison biermers anemia, giant polysegmented neutrophil. |
| A appendectomy pt present with neurophilic leukocytosis with regenarative shift what is the mechanism of leukocytosis development. | this is a case of Neutrophilia in which leukocytosis stimulate or amplify leukopoiesis. |
| A pt has rise tempt, 38 to 40 degree, liver, spleen are all enlarged, Hb is 100g\l, erythrocyte is 2.9, leukocyte is 4.4, thrombocyte is 4.8, segmented neutrophil is 17%, lymphocyteis 15%, blast cells 68%, all cytochemical reaction are negative. | Acute undifferentiated leukemia (AUL) is rare and by definition has neither lymphoid nor myeloid lineage specific markers, you can see eryth is low, thromb is low, abundant blast cell and all cytochemical reactions are negative. |
| Apparent leukopenia, granulocytopenia( WBCs that has small granules that contains proteins esp Neutropile that helps to fight infectn) but in this case is low after taking antibiotic to prevent enteric infections, what mechanism of blood change occur. | Leukopenia and granulocytopenia occurs after myelotoxic therapy is taken, which causes a potentially dangerous adverse effect. Myelotoxicity can be dose limiting and life threatening. In adults leukopenia is a total WBC count <3700 cells/mm3. |
| A traumatic shock cause AP 140\90mmgh, pulse is 120bm, he is fussy, talkative, pale what phase of shock is this. | Erectile shock patients feels pains and his talkative |
| After a traumatic shock a patient blood was 3.2 with leukopenia that causes the patient to start having drowsiness, loss appetite and feels slugguish and loss consciousness what phase of shock | Topid shock will cause redistribution of leukocyte in the blood that may result into leukocyte. |
| A paraprotein in the zone of gamma globulins that confirm myeloma diagnosis | bence jones protein |
| A nuclear plant workers has irradiation dose of 500 roentgen that result in headache, nausea, dizziness, what change will you observe in the leukocyte quantity after 10.5 hours after this suffering. | Neutrophilic Leukocytosis |
| 1.in a case leukemia what happen 2.if lumph nodes are enlarged 3.if smudge cells or gumcrht shows chronic case 4cytochemical reactions are negative 5. oragomegaly like liver 5kg, spleen 3kg 6. if promyelocyte, myelocyte or metamyelocyte are increas | 1. blast cells will be increase 2.lymphoblastic leukemia 3.lymphoblastic leukamia 4.undifferentiated leukamia 5. Chronic myelogenous leukemia 6.myelosis |
| if monocyte is increase | monocytosis |
| physiological poikilocytosis | till 20% |
| What help Myosin head to bind to actin in order to stimulate a muscle contraction | L-CALCIUM |
| BCR-ABL gene | is a place where immature myeloid cells makes RBCS, platelets excepts lymphocyte. |
| Obliteration of bone marrow with fibrosis, a teradrop rbcs, bone marrow cries because it is fibrosed and is a dry tap | Myelofibrosis |
| A pt has Chronic myeloleukamia, decrease of eryhrocyte, HB, oxyphilic and polychromatophilic normocytes, and microcytes, what is the leading pathogenetic mechanism of anemia development. | substitution of hemoblast |
| Eaten of strawberries develop rash and itching what was found in the child leukogram | Eosinophilia |
| Activation of a number of hemostatic factors occurs through their joing with calcium ions, what structural component allows for adjoining of calcium ions | Gamma carboxyglutamic acid |
| A man of 27 years has acute myelogenous leukamia , despite ongoing therapy thas an amplify anemia, what is the anemia by pathogenesis A. Hemolytic B. Hypoplastic | As the number of immature cells increases, the amount of healthy rbcs and platelets decrease. this causes the fall that leads to hypoplastic(when immune system is attacking the stem cells) and leukamia symptoms. it con cause by radiation. |
| What is acute myelogenous leukamia | caused by a DNA mutation in the stem cells BM that produce red blood cells, platelets and infection-fighting white blood cells.The mutation causes the stem cells to produce many more white blood cells than are needed. |
| Pathogenicity of acute myelogenous leukamia | The wbcs produced are still immature, so they do not have the infection-fighting properties of fully developed white blood cells. As the number of immature cells increases, the amount of healthy rbcs and platelets decrease. |
| Surgical pt was diagnosis with appendicitis , local peritonitis, analysis shows neutrophil is 77%, segmented neutrophil is 55% what type of nucleus shift occur | Matured Neutrophil is having 77% which is higher than Segmented neutrophil this is Regeneration shift to the left. |
| A man of has liquidator plant accident that causes pale skin, visible mucous membrane, respiratory rate is 22, pulse is 95, blood eythrocytes is 2.4/L, Hemoglobin is 70g, Leukocytes is 3.3, platelets is 130/L what medical condition is this. | Pancytopenia is a condition that occurs when a person has low counts for all three types of blood cells: red blood cells, white blood cells, and platelets. Pancytopenia is usually due to a problem with the bone marrow that produces the blood cells. |
| A pt has MI blood analysis shows neutrophil leukocytosis with a left shift what factors can determine such phenomenon | Neutrophils contribute to tissue injury by amplifying the inflammatory response and direct release of toxic effectors. which leads to decomposition of heart tissue product. |
| Violation of leukopoiesis occurs as a result of deficiencies of cyanocobalamin and folic acid what changes may this case be | Leukopenia |
| Chronic myelogenous leukamia | CML cauase early immature leucocyte production this changes form an abnormal gene called BCR-ABL which turns the cell into CML, there will be alot immature and myeloid stem cells like stab, segmented, metamyelocyte, promyelocyte, eosinophil etc ALL HIGH. |
| Acute myelogenous leukamia | Segemnted(immature) neutrophil will be HIGHER than Stab(mature) neutrophi. |
| An operator of liquidator plant has an accident after been irradiated with 500 xrays, he CC of headache, nausea, dizziness what change in the number of leukocytes in a pt in 10 hours after the irradiation | There will be an increase redistribution of leucocyt(LEUKOCYTOSIS) |
| A pt that has Chronic Leukemia, has sharply increaase obody tempt, shortness of breath, several weakness of muscles, cough, pneumonia occur, what mechanism of complication of chronic Leukemia has occur | immunodeficiency. |
| punctura hemorrage is seen in a patient after tourniquet what dysfunction of what blood cells it is connected | Platelets |
| extrinsic pathway | 3,7, 10a |
| instrinsic pathway | 12,11,9,10a |
| intestinal dysbacteriosis which resulted in hemorragic syndrome, what is the most likely cause of hemorrhage of the child | Vitamin K insufficiency |
| A patient has bile acid obstruction suffer from inhibition of blood coagulation bleeding as a result of low level of vitamin assimilation, what vitamin is in def. | Vitamin K |
| A Patient with burn dx was complicated by DIC syndrome. what stage of DIC is seen if pt blood coagulate in less than 3 minute | Hypercoagulation |
| A pt has cyts on pancreas, after then development of hemorrhagic syndrome with apparent disorder of blood coagulate development of this complication can be explain by | Activation of Fibrolytic system convert plasminogen to plasmin in the circulation, then to fibrin . |
| A 70 yrs has Atherosclerosis complicated with lower limb thrombosis that causes gangrene on the left toes what is the most likely cause of the thrombosis origin | Thrombocyte adhension explain |
| Chronic persistent hepatitis with a prolonged hemorrage syndrome what is the reason | decrease in thrombin production |
| implanted heart takes indrect anticoaqulant, his state is complicated with hemorrage, what subst is decrease | prothrombin |
| A Disaster fighter of a nuclear power planr has hemorragic syndrome due to acute radiation dx what is the most important factor of syndrome of pathogenicity | Thrombocytopenia |
| A 3 yrs old boy has pronounced hemorragic syndrome and does not have anti-hemphilic globulin A (factor Vlll 8) in the blood plasma. hemostasis has been impaired at the following stage | Ans>Internal mechanism of Prothrombinase activation after then to conversion of prothrombin to thrombin, then fibrinogen to fibrin. |
| Hemoarthrosis of the knee joint with frequent bleeding dx | Hemophilia |
| Hereditary coagulopathy with factor 8 def, specify the phase of blood clotting during coaqulation will be disrupted | Thromboplastin formation |
| Hemophila B what deficiency of whcih coagulation factors cause it | factor 9 IX |
| Chronic hepatitis causes bleeding while removing his teeth, didnt stop for 2hrs, a decrease in the content of several blood clotting factors was observe, what type of hemostasis is violated A. Coaqulative platelet B. Coaqulation C. Platelet | B.Coagulation |
| Multiple bruises with increase bleeding time 25 minute , the number of platelet is 25,000( Normal 150=440,000) what dx is characteristed by such symptoms | Hereditary defect of platelet formation |
| A pt taking Analgin has persistent Migraine with skin hemorrage, platelets count is 30X10, bleeding time is increasd, what causes these disorders | Autoimmune thrombocytopenia |
| A pt has hereditary form of coaqulopathy due defect of blood coaqulation Vlll, PLEASE INDICATE in what PHASE OF BLOOD COAGULATION PRIMARY HEMOSTATIC DISORDERS OCCUR IN THIS CASE | Formation of thromboplastin |
| A violation of the 3rd phase of blood coaqulation in a pancrease surgical pt specify the mecanism of hemostatic disorder in this case | 3rd phase will be the stage of fibrin formation Ans increased fibrinolysis |
| A coaqulation time was 6minute, bleeding time by dengua was 15 minute, violation of which link of hemostasis is possible to assume in this patient A. Vascular platelet B.Coagulative phas 1 C. fibrinolysis | A.Vascular platelet |
| Artificial heart had tendency to bleed, platelet count in the blood isbreduced, thrombocytopenia in this pt can be explained by | Mechanical destruction of platelets |
| A pt during examination has thrombocytopenia, which occur after increased damge of platelets to what group of thromocytopenia does it belong A. Myelotoxic b. Immune | immune |
| Thrombocytopenia, a decrease in fibrinogen with petechial hemorrage dX | DIC |
| A pt has hemophilia, a change in some parameters of blood which of the following symptoms correspond to this dx | increase\prolonged in clotting time. |
| Nitrate poisoning causes what complication on Hemoglobin | Nitrate oxidize iron component of RBC hemoglobin rendering them unable to carry o2 this lead to Methemoglobin |
| A pt came from Tunis has Alpha Thalasemia with hemolysis of erythrocyte and icterus, what will be seen in the cell | Target like erythrocyte |
| Sulphanilamide drugs , revealed hypogenrative normochromia anemia , leukopenia, thrombocytopenia, myelokariocyte was seen in bone marrow | Hypoplastic or Aplastic |
| Hereditary hemolytic microspherocytic anemia(Minovski shauffard anemia) what is the cause of hemolysis of erythrocyte | is an hereditary dx with a mutation that affect and destroys the RBC membrane in the spleen and removal (hemolytic anemia) (Membranopathia). |
| A blood has leukocytosis, lymphocytosis, Botkin-Gumpreht shade also called smudge cell diagnosis | Chronic Lymphoid Leukamia |
| Thrombocytopenia is cause by autoimmune process, it will leads to | increased destruction of thrombocyte |
| A pt has Sicklemia what is the mechanism of decrease RBC number in the pt | Intracellular hemolysis |
| Decrease amount of RBC and increase amount of free hemoglobin in the blood plasma | Acquired hemolytic |
| Glomerulonephritis suffer from which anemia | EPO destruction |
| Regenerative RBC | Reticulocyte |
Created by:
Greatman Israel
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