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Fail of Neurotrans O

What would happen if a toxin prevented release of inhibitory neurotransmitters in the spinal cord that normally reduce background activity of motor neurons? inhibition of MN means increased excitation = increased AP, M = increased contraction - hypertonia, S= normal, R = hyporreflexia
What would happen if a drug or toxin prevented axons from changing their permeability to sodium (by inactivating sodium channels)? no polarization, no AP, S = analgesia or anesthesia, M = paralysis paresis, R = hyporreflexia
What would happen if a drug, toxin or antibody inactivated the postsynaptic (ACh) receptor at the neuromuscular junction? inactivation at receptor means no contraction, M = paresis or paralysis, S = normal, R = decreased or absent/ areflexia
What would happen if a toxin prevented release of ACh at the neuromuscular junction? lack of excitatory NT, reduced contractions, M - paralysis or paresis, S= normal, R = areflexia
What would happen if an individual had an acute fall in the amount of calcium in the extracellular fluid? no Ca means no release of NT or binding at postsynaptic membrane, M = paresis or paralysis, S = analgesia or anesthesia, R = areflexia
What would happen if a drug increased the function of the GABA receptor (allowed it to admit more chloride ion)? increased inhibition and hypo polarized, M = paresis or paralysis, S = analgesia or anesthesia, R = areflexia
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Created by: eew888



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