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Fail of Neurotrans O
Question | Answer |
---|---|
What would happen if a toxin prevented release of inhibitory neurotransmitters in the spinal cord that normally reduce background activity of motor neurons? | inhibition of MN means increased excitation = increased AP, M = increased contraction - hypertonia, S= normal, R = hyporreflexia |
What would happen if a drug or toxin prevented axons from changing their permeability to sodium (by inactivating sodium channels)? | no polarization, no AP, S = analgesia or anesthesia, M = paralysis paresis, R = hyporreflexia |
What would happen if a drug, toxin or antibody inactivated the postsynaptic (ACh) receptor at the neuromuscular junction? | inactivation at receptor means no contraction, M = paresis or paralysis, S = normal, R = decreased or absent/ areflexia |
What would happen if a toxin prevented release of ACh at the neuromuscular junction? | lack of excitatory NT, reduced contractions, M - paralysis or paresis, S= normal, R = areflexia |
What would happen if an individual had an acute fall in the amount of calcium in the extracellular fluid? | no Ca means no release of NT or binding at postsynaptic membrane, M = paresis or paralysis, S = analgesia or anesthesia, R = areflexia |
What would happen if a drug increased the function of the GABA receptor (allowed it to admit more chloride ion)? | increased inhibition and hypo polarized, M = paresis or paralysis, S = analgesia or anesthesia, R = areflexia |
7 | 7 |
8 | 8 |
9 | 9 |
10 | 10 |