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Session 2 CM derm7
CM- Derm -7- scaling lesions 2- dermatitis
| Question | Answer |
|---|---|
| Name the 9 types of eczema/xerosis | atopic dermatitis allergic contact dermatitis irritant contact dermatitis stasis dermatitis nummular dermatitis lichen simplex chronics (neurodermatitis) Asteatotic eczema Dyshidrotic eczema seborrheic dermatitis |
| this is a common immune mediated inflammatory skin disorder with a 10-20% prevalence in western population | Atopic Dermatitis |
| What are the s/sx of atopic dermatitis | pruritis, facial and extensor involvement, flexural lichenification in adults, chronic or relapsing dermatitis |
| What are the associated features of atopic dermatitis | xerosis, skin infections, non-specific dermatitis on hands and feet, pityriasis alba, white dermatographism, ELEVATED IgE, early age of onset, Dennie-Morgan infraorbital folds |
| What is the atopic triad | Asthma, Hay fever, atopic dermatitis |
| what are the theories on why there is more atopic dermatitis | too much hygiene, vaccination, lack of bacterial viral infections, changes in diet, obesity decreased physical activity and responses to enivironmental allergens, pollution |
| What are some complications of atopic dermatitis | ocular- eyelid dermatitis, conjuctivitis, keratoconus, cataracts Hand Dermatitis infections- bacterial 90% staph, viral (herpes, molluscum, HPV), superficial fungal infection |
| what is the treatment for atopic dermatitis | identify and control flare factors topcial glucocorticoids, and non steroidal TIMs emollients- oatmeal baths, moisturizers Systemic treatments- oral antihistamine oral antibiotics systemic steroids immunosuppression |
| what are the topical immune modulators | tacrolimus and pimecrolimus |
| How do topical immune modulators tacrolimus and pimecrolimus work | derived from fungal polypeptide it inhibits t-lymphocyte activation potent immunosuppresive |
| What is the benefit to using tacrolimus and pimecrolimus over topical steroids | don't cause atrophy of the kin or other steroid side effects. Slow acting anti inflammatory |
| scaling skin disorder caused by T-lymphocyte mediated type IV allergic reaction | Allergic Contact Dermatitis |
| How common is allergic contact dermatitis | commononest specific skin disorder encountered and second commenest dermatosis encountered in dermatologists office |
| what are the s/sx of allergic contact dermatitis with exposure to poison ivy or oak | 2-3 days post exposure you get erythematous, vesicular pruritic rash caused by urshiol in sap of leaves you can get reinfection from sap on clothing and tools, you can get cross reaction with poison summac cashews mango rinds and japanese lacquer |
| how can you determine what would cause allergic contact dermatitis in a patient | patch testing |
| what is the treatment protocol for allergic contact dermatitis | eliminate contact with allergen topical steroids protopic topical calcineurin inhibitor which targets t cell activation PO steroids |
| What is the most occupational caused skin disease | irritant contact dermatitis |
| HOw is irritant contact dermatitis different than allergic contact dermatitis | irritant is a non-immunological inflammatory reaction of the skin which is different than allergic CD. |
| what are common causes of irritant contact dermatitis | soaps and detergents, acids, alkalis, metal salts, solvents, fabrics and plants |
| what is the treatment for irritant contact dermatitis | define/remove exposure to irritant, moisturizers, barrier creams, topical glucocorticoids, oral antihistamines |
| dermatitis marked by increased hydrostatic pressure in lower extremities, caused by valve incompetence, CHG, obesity, liver failure, DVT and more | Stasis Dermatitis |
| Fibrinongen, RBC breakdown products, Toxic metabolites, trapped wbc, trapped growth hormones | all causes of stasis dermatitis |
| What are the S/Sx of stasis dermatitis | red dry and scaly patches and plaques on lower extremities, which can develop into venous ulcers, infections, and pain prone to develop allergic and irritant contact dermatitis |
| what is the treatment for stasis dermatitis | emmolients, petrolatum, mineral oil, aquaphor topical steroids support stockings elevation of extremities prevention of ulcers!!!!!! |
| The etiology of this disease is unknown peaks in women 15-25 and men 55-65 characterized by circular or oval coin-shaped plaques some with vesicles and pustules on extensor surfaces of extremities especially pretibial areas | nummular dermatitis |
| what is the treatment for nummular dermatitis | hydration, topical steroids, protopic, and antibiotics |
| Another disease of unknown etiology that starts with pruritus and stimulation of the itch scratch cycle that results in constant rubbing and scratching | lichen simplex chronicus |
| HOw does lichen simples chronicus progress | starts with itching, then small red macules to papules and then to lichenified plaques found only on accessible areas of the body |
| what is the treatment for lichen simplex chronicus | potent topical steroids antihistamines doxepin protopic emollients |
| eczema craquelatum or winter itch usually on lower extremities clinically dry cracked skin treatment of emollients | asteatotic eczema |
| irritation of skin on the palms and soles of the feet characterized by clear deep blisters w/o erythema that itch and burn and worsens after contact with soap, water, or irritating substances | dyshidrotic eczema |
| What is the female to male ration of dyshidrotic eczema | 2:1 peak between 20-40 yrs of age |
| what is the treatment for dyshidrotic eczema | steroids emollients during the drying/scaling phase drying products during the vesicular phase |
| Greasy yellow scales on erythematous background generally on scalp, eyebrows, gabella, eyelid margins, cheeks, nasolabial folds, beard and sternal areas | seborrheic dermatitis |
| dermatitis that is common in parkinson's disease | seborrheic dermatitis |
| treatment for seborrheic dermatitis | ketoconazole, selsun and low potency steroid shampoos |
| a chronic skin disease characterized by itchy, inflames skin | atopic dermatitis |
| a red, weepy reaction when the skin comes into contact with a substance the immune system react to | allergic contact dermatitis |
| a localized reaction including redness itching and burning where the skin contacts irritants such as an acid, cleaning agent or other chemical | irritant contact dermatitis |
| skin irritation on the lower legs usually due to circulatory problems | stasis dermatosis |
| coin shaped patches or palques commonly on the arms, back, buttocks and lower legs may be crusted scaling and extrememly pruritic | nummular derm |
| generalized dry skin or winter itch | asteatotic |
| irritation on the palms and soles with clear blisters that itch and burn | dyshidrotic |
| skin inflammation of unknown cause presenting with yellow oily scaly patches on the scalp and face primarily | seborrheic |
Created by:
smaxsmith
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