Busy. Please wait.

show password
Forgot Password?

Don't have an account?  Sign up 

Username is available taken
show password


Make sure to remember your password. If you forget it there is no way for StudyStack to send you a reset link. You would need to create a new account.
We do not share your email address with others. It is only used to allow you to reset your password. For details read our Privacy Policy and Terms of Service.

Already a StudyStack user? Log In

Reset Password
Enter the associated with your account, and we'll email you a link to reset your password.
Don't know
remaining cards
To flip the current card, click it or press the Spacebar key.  To move the current card to one of the three colored boxes, click on the box.  You may also press the UP ARROW key to move the card to the "Know" box, the DOWN ARROW key to move the card to the "Don't know" box, or the RIGHT ARROW key to move the card to the Remaining box.  You may also click on the card displayed in any of the three boxes to bring that card back to the center.

Pass complete!

"Know" box contains:
Time elapsed:
restart all cards
Embed Code - If you would like this activity on your web page, copy the script below and paste it into your web page.

  Normal Size     Small Size show me how

F & E

Goljan's F & E and Hemodynamic disorders

State 2 functions of K+ 1) Regulate neuromuscular excitability and muscular contraction 2) Regulate insulin secretion
What are the ECG changes in a) Hypo-Kalemia? b) Hyper-Kalemia? a) Hypo-K+ = U wave b) Hyper-K+ = Peaked T waves
Is sweating a hypo/iso/hypertonic loss of Na+? Hypotonic loss of Na+
Is adult diarrhea a hypo/iso/hypertonic loss of Na+? Isotonic loss of Na+
Are pitting edema states (RHF, cirrhosis, neprotic) a hypo/iso/hypertonic loss/gain of Na+? Hypotonic gain of Na+
Is salicylate intoxication a metabolic acidosis or respiratory alkalosis? It is both. It is a primary metabolic acidosis, and a primary respiratory alkalosis.
What are the differences between transudate and exudate? Transudate is protein-poor and cell-poor, produce pitting edema. Exudate is protein-rich and cell-rich. No pitting edema.
State the 3 pathogenesis of thrombi. (Virchow's triad) 1) Endothelial cell injury 2) Stasis of blood flow 3) Hypercoagulability
List the causes of Fat Embolism. 1) MC due to fracture of long bones 2) trauma to fat-laden tissues, or fatty liver
Dx : During labor, abrupt onset of dyspnea, cyanosis, hypotension, and bleeding. Amniotic fluid embolism
List 2 acute complications of scuba diving. 1) Pneumothorax 2) Pulmonary embolism 3) bends
List the changes during hypovolemic shock to : a) CO b) LVEDP c) PVR a) decrease CO b) decrease LVEDP c) increased PVR
List the changes during cardiogenic shock to : a) CO b) LVEDP c) PVR a) decreased CO b) increased LVEDP c) increased PVR
List the changes during septic shock to : a) CO b) LVEDP c) PVR a) increased CO b) decreased LVEDP c) decreased PVR
What is the most common organism to cause septic shock? Escherichia coli
Dx : Warm skin, bounding pulse, ARDS, DIC Septic shock
Created by: lemontea88