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Perioperative Nsg
Nsg
| Question | Answer |
|---|---|
| Diagnostic | Anxiety/Knowledge Deficit |
| Curative surgery | upbeat optimistic |
| Pallative Surgery | Slow Recovery |
| Restorative | Body Images |
| Emergent | Immediately. (appendectomy) |
| Urgent | Within 24-30hrs |
| Required | Few Weeks to Months |
| Pre-Op Assessment | Pre-op Allergy Assessment Medications/Chemicals/Foods/Latex/Anes-thesia Confounding Risk factors Smoking Obesity Age Skeletal deformities Alcohol/Drug Nutritional and Fluid Status |
| Steroids | Don’t D/C suddenly/Suppress immune system |
| Diuretics | Cause respiratory depression |
| Antipsychotics | Hypotension |
| Insulin | May effect meds |
| Anticoagulants | May increase bleeding |
| AntiSeizure | Decrease threshold |
| Aspirin/NSAIDS | Increased bleeding potential |
| Cardiac | Indicated for good control |
| Treatment of Respiratory Alkalosis | (hyperventilation) Anxiety-Paper bag. High Altitude-02, Pregnancy- Monitor breathing, Fever-temp. |
| Depolarization | electrical activation of a cell caused by influx of Na into cell while K+ leaves cell. |
| Anxiety and Panic Disorders PAIN | Pain around heart. Whole Chest. |
| Managment of Cardiogenic Shock | Correct underlying problem. Administer diuretics. Circulatory assist devices. |
| Patient Teaching CHF | Meds, Diet, Signs of excess fluid, hypotension, daily wt., exercise, stress, infection |
| Hemiplegia | Weakness/paralysis of one side of the body |
| Treatment of respiratory acidosis | (Hypoventilation) D/O: Administer antidote(narcan) Chest Trauma: Splint Chest/assist ventilation Pulmonary edema: Remove secretions. Sit up. o2 Airway Obstruction: Remove obstruction. COPD: increase breathing efforts Neuromuscular Disease: Vent. |
| Apraxia | Inability to perform previously learned motor tasks on voluntary basis. |
| Medical Management of Heart Failure | eliminate or reduce contributory factors Reduce workload of heart Prevent exacerbation |
| Activity Intolerance Related to CHF | Bedrest for acute pain. Encourage regular activity. 30-45min reg. Exercise training Pacing activities Wait 2hrs after eating before doing activity |
| CVP | Central Venous Pressure(Ride Side) Normal CVP 2-10cm water. Amount of blood returning to heart and ability of heart to pump blood into arterial system. |
| High CVP Indicates | (CVP of 14)Right sided heart failure. Hypervolemic |
| Low CVP Indicates | (1) Hypovolemic. Hemorrhaging. |
| Pulmonary disorder PAIN | pain below heart. sides radiating over |
| Receptive Aphasia | inability to understand what someone else is saying. Damage to temporal lobe(wernike's area) |
| Objective Liver Exam | Dilated abdominal veins. Spider anginomas Hepatomegaly Protruding belly button Janudice Fector Hepaticus Edema MSC |
| Liver Function Test | More than 70% of parenchyma may be damaged before positive test. |
| ALT | Liver Function test |
| AST | High metabolic tissue test(non-specific livertest) |
| Jaundice | High bilirubin(over 2.5)Hepatocellular-damaged cells, hepatitis, meds. Obstructive |
| Renal Insufficency | Kidney Function~14-40% Metabolic waste begins to accumulate. |
| Acute Renal Failure Risk factors | Age Preexisting renal disease Diabetes Dehydration nephrotoxic |
| Preload | Degree of stretch of cardiac muscle fibers @ end of diastole. Greater the volume of blood entering during diastole, greater the ejected |
| Compensation | PH normal Abnormal PaCo2. Use 7.40pH |
| Metabolic Acidosis | pH low. PCO2 low. DKA. Diarrhea. Renal Failure. Shock. Salicylate Overdose. |
| Esophageal Varices | Enlarged tortuous vein or artery. Related to impaired drainage of the esophageal veins into portal veins. Added pressure causes vessel wall to weaken and rupture. |
| Asses Esophageal Varices | ETOH, liver disease, pain, occult blood, hematemesis, hemopysis, jaundice, cirroses |
| Surgery Varices | Sclerotherapy, ligation, balloon, shunting |
| Constipation Assesment Surgery Interventions | History: Stool patterns, Onset, Duration, Severity. Ever had it before? Diet habits. disease, Abdominal sounds/distention. Surgery: Resection, fiber supp. laxatives |
| Constipation NSG interventions | Diet, High Fiber, High Fluids, Activity, Ambulation. |
| Ulcer Complications | Dumping: Rapid gastric emptying. Abdominal Pain, NVD. Explosive diarrhea, malnutrition, small freq. meals. High protein, no carbs. |
| Appendicitis | inflammation of the appendix. Lumen of the appendix is obstructed by e-coli, resulting in infection. |
| Assess Appendicitis | Fever, Pain, High WBC, Anorexia, N/V, lower RQ pain. Rebound tenderness. |
| Intervention for Appendicitis | Emergent Surgery, Monitior S/S of infection, pulmonary status, Drains, Dressings. |
| Diverticula | sac like protrusions of the intestinal wall |
| Gastritis | inflammation of the stomach mucosa when stomach has been exposed to irritants. ex. H.Pylori |
| Assessing of Gastritis | Epigastric pain, burning, nausea, Pain w food, occult blood. |
| Medicine and NSG interventions for gastritis | Upper GI, Antacids, Proton Pump, Monitor pain, admin meds, Monitor response, teach meds. and lifestyle. |
| Emetic Med | Ipecac. Irritates Gastric mucosa. Induces Vomiting. Side Effects: Aspiration, seizure. |
| Subjective Liver Exam | Peripheral Edema, Fatigue, dry skin, abd. pain, change in mental status, anorexia, early morning N/V |
| Hydroureter | Dilation of ureter |
| Ileus NSG Assess. | Abdominal pain, Nausea, Constipation, bloating, distention, low Na and K+, high BUN, Vomiting, Bowel Resection |
| Ileus Intervention | encourage ambulation, encourage turn and deep breathe q2h, Monitor I&O, IV manage, NG tube |
| Cushing's Syndrome | too much cortisol wide range of effects, moon face, obese trunk |
| Addison's Disease | hyposecretion of adrenal cortex hormones, corticosteroid (hydrocortisone) and mineralocorticoids (aldosterone). The condition is fatal if untreated. |
| Assessment Addison's Disease | Lethargy, fatigue, and muscle weakness Gastrointestinal (GI) disturbances Weight loss Menstrual changes in women; impotence in men Hypoglycemia Hyperkalemia Postural hypotension Dehydration Emotional disturbances/MS Δ’s (60-80 %) |
| Chronic fatigue Muscle weakness Anorexia and weight loss Symptoms of Addison's Cause | Decreased cortisol, improper metabolism. |
| Symptoms of Addison's:Hyponatremia (demonstrated by salt craving) | Caused by: Nausea Vomiting. |
| Symptoms of Addison's: Orthostatic hypotension Hyponatremia (decreased sodium in blood) Hyperkalemia (elevated potassium in blood) | Alsdosterone deficiency and hypovolemia limit BP control and effective excretion of proper waste products. |
| Addison's:Hyperpigmentation of skin | Increased corticotropin levels due to decreased feedback. |
| Addison's Disease Implementation | Monitor V/S particularly BP, weight, and I&O Monitor blood glucose and K levels Administer glucocorticoid or mineralocorticoid medications a/o Observe for Addisonian Crisis secondary to stress, infection, trauma, or surgery. |
| PT. Teaching Addison's | Avoid individuals with infections Avoid stress Need for lifelong glucocorticoid therapy Wear a Medic-Alert bracelet |
| Addisonian Crisis | A life-threatening disorder caused by acute adrenal insufficiency Precipitated by stress, infection, trauma, of surgery Can cause hyponatremia, hyperkalemia, hypoglycemia, and shock |
| Addisonian Crisis Assessment | Severe headache Severe abdominal, leg, and lower back pain Generalized weakness Irritability and confusion Severe hypotension Shock |
| Hyperthyroidism Crisis | Can also occur from: severe infection, stress, diabetic acidosis, surgery, abrupt withdrawal of antithyroid medications, or other vigorous palpitation of the thyroid. |
| Asses Hyperthyroidism Crisis | Hyperplasia: >101.3F Tachycardia: >130bpm Exaggerated symptoms of hyperthyroidism Altered MS: confusion to coma |
| Thyroid Crisis Implementation | Implications: Maintain airway Ice packs/cooling blanket Tylenol O2 Monitor O2 Sat Iodine to decrease preformed thyroid hormone secretion. |
| Type I diabetes | Destruction of pancreatic beta cells Genetic susceptibility is a key factor Autoimmune may also be involved. Resulting in:Decreased insulin production Fat breakdown resulting in ketone bodies as byproducts Fasting hyperglycemia |
| Type II Diabetes | Insulin resistance ( glucose metabolism at the cellular level) Impaired insulin secretion Results in: Increased need for insulin production ( ability to secrete exists) No marked fat breakdown (no ketones) May go undetected for years 75% of cases a |
| Clinical Manifestation of Diabetes | Polyuria(increased urine), Polydipsia(Increased Thirst), Polyphagia(increased appetite)Fatigue, weakness, tingling in limbs, slow healing wounds |