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transient lingual papillitis enlargement of fungiform papilla on dorsal tongue
localized form of transient lingual papillitis one enlarged fungiform papilla on anterior tongue
diffuse form of transient lingual papillitis many enlarged, painful fungiform papilla; sometimes with fever and lymphadenopathy
'papulokeratotic variant' of transient lingual papillits diffuse and asymptomatic enlargement of fungiform papillae; most likely frictional hyperkeratosis
recurrent apthous stomatitis/ulceration (RAS/RAU) oral ulcers mediated by CD8 T-cells
1. antigenic stimulus 2. immunodysregulation 3. decreased mucosal barrier 3 pathologic mechanisms of apthous stomatitis
simple apthosis most common type of RAS; self-limiting without other disease processes
complex apthosis patients that have several apthous ulcers with significant disease and recurrences
minor apthous stomatitis mildest form of RAU; a couple of outbreaks each year, usually in adolescents
major apthous stomatitis large ulcers with long healing time and a later onset
herpetiform apthous stomatitis numerous small ulcers like herpes but not on gingiva or systemic symptoms
Behcet syndrome systemic vasculitis with irregular oral ulcers on soft palate, genital ulcers, and ocular problems
sarcoidosis multisystem granulomatous disorder, probably autoimmune; lungs and lymphoid tissues are most affected -oral involvement of mass or papules is seen first in 2/3 of patients
Heerfordt syndrome acute form of sarcoidosis; parotid gland enlargement, facial nerve paralysis, uveitis and fever
orofacial granulomatosis variety of clinical presentations that show non-specific granulomatous inflammation; called Melkersson-Rosenthal syndrome when with facial paralysis and fissured tongue
Wegener granulomatosis necrotizing granulomatosis lesions of respiratory tract; effects in kidneys and vasculitis
classic/generalized Wegener granulomatosis fatal kidney involvement after initial respiratory infection
limited Wegener granulomatosis disease stays localized to respiratory tract without renal lesions
'superficial' Wegener granulomatosis only have skin and oral mucosal lesions
strawberry gingivitis early manifestation of Wegener granulomatosis
PR3-ANCA antibodies detected in 90% of patients with Wegener's and can be used to monitor disease activity
stomatitis medicamentosa mucosal reactions to systemic drugs
anaphylactic stomatitis IgE mediated erythema and ulceration 24 hours after exposure to drug
fixed drug eruptions reactions that reappear at same site after taking drug
lichenoid drug reactions mucosal changes from systemic drug that mimic lichen planus
pemphigus-like drug reactions mucosal desquamation and bullae formation like pemphigus
pemphigoid-like drug reactions mucosa comes off in sheets and mimics pemphigoid
lupus erythematous-like drug reactions fever, flu-like symptoms and lupus-like changes
nonspecific vesiculoerosive or apthous-like lesions nonspecific mucosal changes due to drug reaction
allergic contact stomatitis long list of agents that cause hypersensitivity reaction; {from artificial cinnamon flavoring}
perioral dermatitis inflammatory reaction around mouth from antigen or corticosteroid cream
lichenoid contact reaction to dental restorative materials reaction adjacent to old, corroding amalgam fillings
angioedema diffuse edematous swelling of soft tissue; minor trauma can precipitate reaction
IgE-mediated type I hypersensitivity reactions most common cause of angioedema; mast cells degranulate and histamine is released
angiotensin-converting enzyme (ACE) inhibitors drug used to treat hypertension that also causes angioedema due to increased levels of {bradykinin}
angiotensin II receptor blockers drugs developed to avoid inhibition of bradykinin breakdown and angioedema
activation of complement pathway mechanism of causing angioedema; can be hereditary or acquired
type I complement pathway inherited from decreased {C1 esterase inhibitor} that enhances complement pathway
Type II complement pathway normal C1-INH levels but enzyme does not work
acquired type of C1-INH deficiency can be seen in lymphoproliferative diseases that inhibit C1-INH
Created by: aharnold