Busy. Please wait.

show password
Forgot Password?

Don't have an account?  Sign up 

Username is available taken
show password


Make sure to remember your password. If you forget it there is no way for StudyStack to send you a reset link. You would need to create a new account.
We do not share your email address with others. It is only used to allow you to reset your password. For details read our Privacy Policy and Terms of Service.

Already a StudyStack user? Log In

Reset Password
Enter the associated with your account, and we'll email you a link to reset your password.
Didn't know it?
click below
Knew it?
click below
Don't know
Remaining cards (0)
Embed Code - If you would like this activity on your web page, copy the script below and paste it into your web page.

  Normal Size     Small Size show me how


Comprehensive Pharm 9

which receptors are associaed with Gq HAVe 1 M&M H1 alpha 1 V1 M1, M3
which receptors are associated with Gi MAD 2's M2 alpha 2 D2
which receptors are associated with Gs B1, B2, D1, H2, V2
major fxns of M2 decreases HR
major fxns of M3 increase exocrine gland secretion
major fxns of D1 relaxes vascular smooth muscle
major fxns of D2 modulates transmimtter release in brain
major fxns of H1 ubcreases basak abd bronchial mucus production, contraction of bronchioles, pruritis, pain
major fxns of H2 increase gastric acid secretion
major fxn of V1 constricts vascular smooth muscle
major fxn of V2 increas water permeability and reabsorption in CT of kidney
MOA hemicholinium blocks the transport of choline into cholinergic neurons, blocking the production of ACh
MOA vesamicol blocks the transporter that brings Acetyl CoA + Choline CHAT into vesicles
MOA botulinum blocks the release of ACh vesicles
MOA metyrosine blocks conversion of tyrosine into DA
MOA reserpine blocks DA transporter into vesicles that form NE
MOA guanethidine inhiits release of NorE from noraderenergic neurons
MOA amphetamine increase release of NorE from vesicles
MOA pralodoxime reactivates AChE after it's been inhibited by pesticides
treatment for salicylate OD alkalinize urine dialysis
treatment of antimuscarinics OD physostigmine salicyate
treatment of b-blocker od glucagon
tx of digitalis od stop dig normalize K \lidocaine anti-dig Fab fragments Mg
tx iron toxicity deferoxamine (chelating agent)
tx fo lead poisoning EDTA dimercaprol succimer penicillamine
tx of arsenic toxicity dimercaprol succimer
tx hg toxicity dimercaprol succimer
tx au toxicity dimercaproli succimer penicillamine
tx cu toxicity penicillamine
tx cn toxicyt nitrite hydroxocobalamin thiosulfate
tx methemoglobin toxicity methylene blue
tx CO toxcity 100% o2, hyperbaric pressure
tx of methanol od ethanol dialysis fomepizole
tx of ethylene glycol od etoh dialysis foempizole
tx of opiod toxicity nalaxone naltrexone
tx of benzo od flumazenil
tx of ca od NaHCO3
tx of heparin toxicity protamine sulfate
tx of warfarin toxicity vitamin k ffp
x tpa toxicity aminocaproic acid
tx streptokinase toxicity aminocaproic acid
sx of iron od fever sweating abdominal pains diarrhea cyanosis weakness
examples of insulin drugs (and give duration of action) lispro (short) insulin (short) NPH (intermediate) lente and ultralente (long acting)
clinical uses of insulin analogs DM I life-threatening hyperkalemia (insulin increases K entry into cells) stress induced hyperglycemia
examples of 1st generation sulfonylureas tolbutaminde chlorpropamide
examples of 2nd generation sulfonylureas glyburide glimepiride glipizide
MOA sulfonylureas when glucose enters the cell, the ATP level rises high ATP:ADP closes K channel this causes Ca influx --> insulin release these drugs enoucrage this process by closing k channels (basically stimulates the release of endogenous insulin)
uses of sulfonylureas DM II reqires some islet cell fxn, so useless in DM I
toxicity associated with sulfonylureas (1st gen) diulfuram effects
toxicity associated with 2nd generation sulfonylureas hypoglycemia
examples of biguanides metformin
MOA metformin unknown, but might decrease gluconeogenesis, increase glycolysis and decrease serum glucose levels
clinical use of metformin can be used in pts without islet cell fxn
adverse effects of metformin lactic acidosis
MOA glitazones incresaes target cell response to insulin
clinical use for glitazones DM II
toxicity associated with glitazones weight gain edema hepatotoxicity
examples of alpha-glucosidase inhibitors acarbose miglitol
MOA alpha-glucosidase inhibitors inhibits intestinal brush border alpha-glucosidases delays sugar hydrolysis and glucose absorption decreased post-prandial hyperglycemia
clinical use of alpha glucosidase inhibitors DM II
Created by: Asclepius



Use these flashcards to help memorize information. Look at the large card and try to recall what is on the other side. Then click the card to flip it. If you knew the answer, click the green Know box. Otherwise, click the red Don't know box.

When you've placed seven or more cards in the Don't know box, click "retry" to try those cards again.

If you've accidentally put the card in the wrong box, just click on the card to take it out of the box.

You can also use your keyboard to move the cards as follows:

If you are logged in to your account, this website will remember which cards you know and don't know so that they are in the same box the next time you log in.

When you need a break, try one of the other activities listed below the flashcards like Matching, Snowman, or Hungry Bug. Although it may feel like you're playing a game, your brain is still making more connections with the information to help you out.

To see how well you know the information, try the Quiz or Test activity.

Pass complete!

"Know" box contains:
Time elapsed:
restart all cards