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AP MOD. II-Urinary

urethra passage of urine from bladder to exterior of body
micturition urination or voiding
urge incontinence associated with smooth muscle overactivity in the bladder wall
urinary retention urine produced but not voided
urinary suppression no urine produced but bladder is normal
urinary incontinence (enuresis)-urine is voided involuntarily
stress incontinence associated with weakened pelvic floor muscles
overflow incontinence associated with urinary retention and overdistended bladder
nocturnal enuresis nighttime bed wetting
neurogenic bladder periodic but unpredictable voiding
ADH promotes water reabsorption, reduces urinary volume
aldosterone promotes sodium and water reabsorption, reduces urinary volume
ANH promotes loss of sodium and water into kidney tubules, increases urine volume
hydronephrosis enlargement of renal pelvis and calyces
renal calculi (kidney stones)-mineral chunks in renal pelvis of calyces, may block ureters
renal colic intense kidney pain
urinary tract infections (UTIs)-caused by gram-negative bacteria
urethritis inflammation of the urethra
cystitis inflammation or infection of the urinary bladder
pyelonephritis inflammation of the renal pelvis and connective tissues of the kidney
proteinuria protein in the urine
hypoalbuminemia low plasma protein caused by loss of proteins to urine
edema tissue swelling, resulting from hypoalbuminemia
acute glomerulonephritis caused by delayed immune response to a strep infection
chronic glomerulonephritis a slow inflammatory conditioncaused by immune mechanisms
acute renal failure abrupt reduction in kidney function, usually reversible
chronic renal failure slow, progressive loss of nephrons
polycystic kidney disease (PKD)-heridary condition of numerous fluid-filled cysts that destroy kidney tissue
renal insufficiency loss of kidney function resulting in uremia
uremic syndrome complete kidney failure
Created by: ddoyon