Busy. Please wait.
or

show password
Forgot Password?

Don't have an account?  Sign up 
or

Username is available taken
show password

why


Make sure to remember your password. If you forget it there is no way for StudyStack to send you a reset link. You would need to create a new account.
We do not share your email address with others. It is only used to allow you to reset your password. For details read our Privacy Policy and Terms of Service.


Already a StudyStack user? Log In

Reset Password
Enter the associated with your account, and we'll email you a link to reset your password.

Remove ads
Don't know
Know
remaining cards
Save
0:01
To flip the current card, click it or press the Spacebar key.  To move the current card to one of the three colored boxes, click on the box.  You may also press the UP ARROW key to move the card to the "Know" box, the DOWN ARROW key to move the card to the "Don't know" box, or the RIGHT ARROW key to move the card to the Remaining box.  You may also click on the card displayed in any of the three boxes to bring that card back to the center.

Pass complete!

"Know" box contains:
Time elapsed:
Retries:
restart all cards




share
Embed Code - If you would like this activity on your web page, copy the script below and paste it into your web page.

  Normal Size     Small Size show me how

LD Final

QuestionAnswer
Fetal movement couting kick counts- should have 4 kicks in 1 hour
Non Stress Testing (NST) evaluation of FHR pattern in absence of regular uterine contractions to determine fetal oxygenation, neurologic, and cardiac functioning
benefits of NST noninvasive, short, safe
indications for NST fetus not moving as frequently as usual, post date, placenta may not be functioning adequately, high risk pregnancy
Criteria for reactive NST 2 or more 15x15 accels in 20 minute period (10x10 ok for extreme premature)
Non reactive NST no accels, one accel that meets 15x15 OR 2 or more accels that do not meet 15x15 criteria in any 20 minute period of time during a total monitoring period of 40 minutes
fetal acoustic stimulation test stimulation of fetus with loud sound and identification of FHR response
Contraction Stress Test determine how the fetus responds to decreased oxygen delivery during contractions; should be performed in a facility with emergency csection capability
benefits of CST indirectly determines placental function and fetal oxygen reserves
limitations of CST longer and more expensive than NST/FAST, invasive (requires IV with pitocin)
interpretation of CST when there are 3 contractions that last 40-60 seconds during a 10 minute period the CST can be interpreted
negative CST no late decels in 10 minute period
positive CST late decels with 2 or more contractions in a 10 minute period
biophysical profile (BPP) evaluation to determine acute and chronic developmental markers to indirectly measure fetal CNS function and oxygenation
scoring of BPP 2 points for each variable: NST, fetal breathing, fetal movement, fetal tone, amniotic fluid volume
amniotic fluid volume - criteria for oligohydramnios AFI <5
amniotic fluid volume - criteria for polyhydramnios AFI>25
Biophysical monitoring techniques daily fetal movement count, ultrasonagraphy (heart activity, gestational age, fetal growth, fetal anatomy, genetic anomalies, placental position/function), MRI
Biochemical monitoring techniques alpha fetoprotein, amniocentesis, percutaneous umbilical blood sampling, chorionic villus sampling
alpha fetoprotein (AFP) measured in maternal serum levels to screen for neural tube defects
amniocentesis obtains amniotic fluid, which contains fetal cells
amniocentesis indications genetic concerns (mom >35 yom previous child with chromosomal abnormality, family hx xsome anomalies), fetal maturity (lungs), fetal hemolytic disease, meconium
chorionic villus sampling removal of small tissue specimen from fetal portion of placenta (reflects genetic makeup of fetus), used for genetic studies between 10-12 weeks of gestation
placental abruption detachment of part or all of placenta from implantation site after week 20 and before birth of fetus
causes of placental abruption maternal hypertension, cocaine use, trauma, cigarette use, history of abruption, preterm PROM, thrombophilias
clinical manifestations of placental abruption abdominal pain, uterus tender, increased uterine tone/tetany, uterus feels firm/board like
management of placental abruption csection, vag delivery if no fetal distress of if fetus is dead, blood transfusions
placenta previa placenta implants in the lower uterine segment near or over the internal os of cervix (causes bleeding when cervix dilates or effaces)
complete/total previa placenta totally covers the internal os
marginal previa edge of the placenta is seen on transvaginal ultrasound to be 2.5 cm or closer to the internal os
risk facotrs for placental previa age, prior placenta previa, drug use (smoking)
clinical manifestations of placental previa PAINLESS, bright red vaginal bleeding
expectant management of placental previa if fetus is <36 weeks, has normal tracing, bleeding is mild and stops and mother is not in labor- observation and bedrest, ultrasounds every 2-3 weeks, NST/BPP once or twice weekly
active management of placental previa fetus >36 weeks or if bleeding is excessive/persistent 0 immediate csection
preterm labor cervical changes and uterine contractions occurring at 20-37 weeks of pregnancy
preterm birth occurs before completion of the 36th week of pregnancy
late preterm 34-36 weeks
early preterm <32 weeks
low birth weight describes only the weight at the time of birth (<2500 grams)
intrauterine growth restriction inadeqaute fetal growth caused by various complications of pregnancy that interfere with uteroplacental perfusion (GDM, HTN, poor nutrition)
small for gestational age less than 10th percentile for babies of that gestational age
risk factors for preterm labor/birth low SES, low prepregnancy weight, smoking, alcohol, drugs, little/no PNC, uterine condiitons, previous preterm delivery, multiple gestation, hx of 2 TAB, PPROM, amnionitis, oligo, previa, abruption, incompetent cervix, PIH, fetal anomalies, IUGR, etc
risk factors for preterm PROM previous preterm birth, amnionitis, mulitple gestation, abrutpion, smoking, maternal low body weight, family predisposition
fetal fibronectins biomarker for preterm labor/birth- test between 2436 weeks, poor positive predictive value but used to determine who will NOT go into preterm labor
subjective signs of preterm labor menstural like cramps, dull low back pain, suprapubic pressure/pain, pelvic pressure or heaviness, change in character of vaginal discharge, diarrhea
objective signs of preterm labor uterine contractions- 4 in 20 min, 8 in 60 min, cervical dilation >1 cm, cervical effacement >80%, preterm PROM
interventions for preterm labor tocolytics to stop contractions (terbutaline, magnesium sulfate), corticosteroids to help lung maturation, bedrest, restriction of sexual activity, activity restriction (betamethasone)
terbutaline stops uterine contractions by relaxing uterine smooth muscle by stimulating beta 2 receptors in uterine smooth muscle (beta 2 agonist)
adverse effects of terbutaline beta 1 stimulation (tachycardia), beta 2 stimulation (hyperglycemia)
contraindications to terbutaline known or suspected heart disease, severe preeclampsia, eclampsia, pre-GDM,GDM, hyperthyroidism, migraine headaches
magnesium sulfate most commonly used tocolytics, promotes relaxation of smooth muscle by competing with calcium in cells
betamethasone corticosteroid that assists in lung maturation/surfactant formation
Induction of labor stimulation of uterine contractions before spontaneous onset
indications for labor induction pregnancy dangerous for mom/baby, health risk, pelvis adequate, no contraindications to artifical ROM, post dates and hypertension
bishop score used to evaluate ability to induce, scored on dilation, effacement, station, cervical consistency, and cervix position. Score of >8 indicates that induction will be successful
cervical ripening agents- pharm. prostaglandins (misoprostil, cerividil)
mechanical ways of cervical ripening balloon catheter (foley), laminaria tent (seaweed), hydroscopic dilators, sex, nipple stimulation, walking
pitocin synthetic oxytocin used to iduce or augment labor that is progressing slowly because of inadequate uterine contractions
augmentation stimulation of uterine contractions after labor has started spontaneously but is progressing unsatisfactorily
how the powers affect dystocia primary powers- hypertonic uterus/ineffective contractions, hypotonic uterus/insufficient for dilation/effacement, secondary powers compromise with lots of drugs/epidurals/exhaustion
how the passage affect dystocia pelvic dystocia (contractures of pelvis tht reduce capacity), soft tissue dystocia (obstruction of birth passage by anatomic abnormality other than pelvis)
how the passenger causes dystocia cephalopelvic disproportion, abnormal presentation or position, anomalies, excessive size, number of fetuses
how the position (maternal) can cause dystocia recumbent and lithotomy may compromise progress
how the psyche can cause dystocia psychologic responses of mother to labor r/t past experiences, prep, culture, and support system can hurt labor process- stress hormones released in response to stress can cause dystocia
shoulder dystocia head is born, but anterior shoulder cannot pass under pubic arch
turtle sign head goes in and out continuously
risk of shoulder dystocia fetal asphyxia, brachial plexus damage, fracture, maternal blood loss, lacerations, extension of episiotomy, endometritis
risk factors for shoulder dystocia maternal obesity, abnormal pelvic shape, postterm regnancy, large baby, prolonged 1st/2nd stage of labor, short maternal stature, GDM, vacuum.forceps birth, induction, increased parity, etc
Interventions for shoulder dystocia GET HELP!, Mcrobert's maneuver, suprapubic pressure, gaskin maneuver (hands and knees), ruben's maneuver (suprapubic, intravaginally), wood screw maneuver
prolapsed umbilical cord occurs when the cord lies below the presenting part of the fetus
risk factors for cord prolapse iatrogenic (artificial ROM), long cord, malpresentation, transverse lie, unengaged presenting part
risk of prolapsed cord fetal hypoxia resulting from prolonged cord compression (occlusion of blood flow for more than 5 minutes)- can result in CNS damage or death of the fetus
interventions for cord prolapse call for help!, do not attempt to put cord back inside uterus, keep hand in place, lift fetal head off the cord, monitor fetal heart tones, immediate csection, hand must stay in place until baby is delivered and cord is retrieved through uterus
complications of infants of diabetic mothers congenital anomalies, macrosomia, birth trauma, perinatal asphyxia, stillbirth, preterm birth, respiratory distress syndrome, hypoglycemia, hypoclacemia, hypomagnesemia, cardiomyopathy, hyperbilirubinemia, polycythemia
what causes congenital anomalies in infants of mothers with DM? fluctuations in BG levels and episodes of ketoacidosis early in pregnancy are believed to cause congenital anomalies
macrosomia- patho mothers pancreas cant release enough insulin to meet demands, maternal hyperglycemia results in increased amounts of glucose crossing placenta and stimulates fetal pancreas to release insulin --> results in excessive fetal growth
physical characteristics of macrosomia round, cherubic face, chubby body, flushed complexion, large internal organs and increased body fat
perinatal asphyxia/oxygen deprivation in fetus of DM mother normally maternal blood is more alkaline than fetal blood that contains CO2 waiting to be removed. When mother is hyperglycemic and gets ketoacidosis, blood becomes more acidotic than fetal blood and little exchnge of CO2 and O2 occurs, causing hypoxia
reason for respiratory distress syndrome in infants of DM mothers high fetal serum levels of insulin and or glucose can delay surfactant production
L/S ratio in DM mothers 3:1
hypoglycemia (infant) infant blood glucose level less than 40. Caused by hyperinsulinemia. Regulation of insulin can take a few days. Signs= jittery, apnea, tachypnea, cyanosis
hyperbilirubinemia increased total serum bilirubin (commonly due to polycythemia, esp. in DM babies)
pathologic jaundice occurs within 24 hours of birth, lasts for longer than 10 days in term infant and 21 days in preterm
risk factors for pathological jaundice mother with DM, hemolytic disease of newborn, delayed cord clamping, altered hepatic clearance
kernicterus encephalopathy caused by neonatal hyperbilirubinemia. Basal ganglia and other areas of brain and spial cord are infiltrated with bilirubin, can result in death
Chronic Hypertension hypertension that is present before pregnancy or develops before 20 weeks of gestation; also, htn that lasts more than 6 weeks postpartum
gestational hypertension (PIH) onset of hypertension without proteinuria after week 20 of pregnancy
risk factors associated with PIH first pregnancy or pregnancy of new genetic makeup, history of vascular disease, multiple gestation, women age <20 or >40, primigravida, morbid obesity, multiple gestation
pre-eclampsia pregnancy specific syndrome in which hypertension and proteinuria develop after 20 weeks of gestation in a previously normotensive woman
risk factors for preeclampsia primigravidity, multifetal pregnancy, morbid obesity, <20 yo, >40 yo
preeclampsia patho vascular remodeling to inc capacity of vessels of uterus does not occur, dec placental perfusion adn hypoxia occur, placental ischemia causes endothelial cell dysfunction, generalized vasospasm,poor tissue perfusion in all organs, inc BP, protein loss
diagnosis of preeclampsia 0.3 g protein or greater in 24 hr period, +2 or greater on urine dipstick, increased blood pressure
mild preeclampsia elevated blood pressure (>140 S, >90 D), .3 g protein, +2 urine dip
severe preeclampsia BP >160 S >110 D, proteinuria 5 g, 3+ urine dip, oliguria, CNS changes (vision, HA, scotomata, mental status), pulmonary edema, epigastric/RUQ pain
HELLP Syndrome variant of severe preeclampsia that involves hepatic dysfunction. Hemolysis, Elevated Liver enzymes, Low Platelet count
eclampsia onset of seizure activity or coma in a woman with preeclampsia with no history of preexisting pathology that could result in seizures
postpartum hemorrhage loss of 500 ml or more after vag birth, loss of 1000 ml after csection, 10% change in hematocrit from labor admin values, early is w/in 24 hr, late is 24 hr-28 days
causes of PP hemorrhage tone (uterine atony), trauma (lacerations, episiotomy, hematomas, inversion, rupture), tissue (retained placenta), thrombin (coagulopathies)
hypovolemic shock assessments rapid shallow respirations, rapid/weak/irregular pulse, decreasing blood pressure, cool/pale/clammy skin, decreasing urinary output, altered LOC, mental status change, dec CVP, poor turgor, dry MM
interventions for hypovolemic shock restore circulating blood volume (IV crystalloids, pRBCs), treat underlying cause of hemorrhage, oxygen per nonrebreather mask 10-12 L
drugs to stop PP hemorrhage methergine, hemabate
uterine atony marked hypotonia of uterus, results in brisk venous bleeding, impaired coagulation
risk factors for uterine atony high parity, hydramnios, macrosomic fetus, multifetal gestation, uterine atony in previous pregnancy, overstretched uterus (poorly contracts after birth)
subinvolution failure of uterus to return to pre-pregnancy state
signs and symptoms of subinvolution boggy uterus, reddish/brown oozing from vagina/irregular heavy vaginal bleeding in 6 weeks PP, low persistent backache, abdominal pain or tenderness, fatigue, foul smelling discharge d/t infection, persistent low grade temperature
risk factors of subinvolution retained placental fragments, pelvic infection
complete inversion of uterus large, red, rounded mass protrudes 20-30 cm outside introitus
causes of uterine inversion traction on cord before placenta separates, fundal implantation of placenta, manual extraction of placenta, short umbilical cord, uterine atony, leimyomas, abnormally adherent placental tissue
signs and symptoms of uterine inversion large PP hemorrhage 800-1800 ml, shock, pain, absence of palpable fundus
idiopathic thrombocytopenia purpura autoimmune disorder in which antiplatelet antibodies decrease lifespan of platelets, can cause severe hemorrhage after csection, vag delivery, cervical lecerations
von willebrand disease hereditary bleeding disorder, factor VIII deficiency and platelet dysfunction
disseminated intravascular coagulation pathological clotting throughout body, coagulation proteins and platelets used up, normal coagulation disrupted, bleeding occurs in skin, GI tratc, resp. tract, etc
risk factors for DIC placenta abruption, placenta previa, severe preeclampsia, HELLP syndrome, hemorrhage, amniotic fluid embolism, septic abortion, massive hemorrhage
DIC lab markers PT prolonged, PTT normal/shortened, fibrinogen decreased, platelets decreased, fibrin split products increased, coagulation factors decreased
manifestations of DIC severe bleeding, shock, dyspnea, hemoptysis, acute renal failure
thrombophlebitis inflammation of superficial deep veins
risk factors for venous thrombosis varicosities, immobility, obesity, advanced maternal age, smoking
s/s thrombosis pain, tenderness in lower extremity, warmth, redness, swelling, tenderness over vein, positive homan's sign
treatment of superficial venous thrombosis NSAIDs, rest, elevation of extremity, stockings, moist heat
treatment of DVT anticoagulants (heparin), bed rest, elevation, analgesia
pulmonary embolism complication of DVT, clot breaks off to pulmonary artery, causes cough, dyspnea, shallow/rapid breathing, adventitious lung sounds
tx for PE IV heparin, oxygen, blood products, etc
puerperal infection infection of genital tract or a would that occurs 28 days after miscarriage, induced abortion, childbirth, or csection (100.4 x2 consecutive days during first 10 days PP)
endometritis infection of decidua (pregnancy endometrium)
risk factors for endometritis prolonged labor, prolonged ROM, multiple vag exams, internal monitors, maternal DM, manual removal of placenta
endometritis s/s fever, uterine tenderness, foul lochia, leukocytosis, bacteremia
UTI risk factors urinary catheterization, frequent pelvic exams, epidural anesthesia, hx of UTI
s/s UTI dysuria, frequency, urgency, urinary retention, hematuria, pyuria, CVA tenderness
mastitis prevention support breasts, comfort, promote brestfeeding, prevent cracked nipples
Created by: alexadianna