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PEDS final

study guide

QuestionAnswer
hypopituitarism management hormone replacement therapy, perform pituitary function test, caused by tumor, infection or idiopathic
precocious puberty pathophysiology and standard management excess production of LH, FSH, estradiol or testosterone, administer GnRHa, Depo Lupron, Synarel
common causes of Cushing’s Syndrome excess or prolonged steroid use or tumor
cleft lip palette feeding management no straws, pacifiers or oral suction. encourage breast-feeding or use specialized nipples if bottle-feeding. high risk for aspiration
clinical signs and pathophysiology of pyloric stenosis projectile vomiting, weight loss, dehydration. hardening of pyloric muscle/canal
management of GER modification of feeding (thickening foods, upright feeding position), use of H2 antagonists
pathophysiology of gastroschisis bowel herniation, NOT covered by a membrane
pathophysiology of Hirschsprung’s congenital, absence of ganglion nerves in parts of colon
pathophysiology of intussusception telescoping of intestine
clinical signs of intussusception abrupt abdominal pain, jelly-like stools, hallmark sign -palpation of sausage-like mass in upper-mid abdomen
diaphragmatic hernia most severe, abdominal contents protrude through thoracic cavity
GI hernia omphalocele covered herniated abdominal organs
GI hernia gastroschisis more severe than omphalocele, no membrane covering
diagnostics for appendicitis elevated WBC, ultrasound, McBurney’s point tenderness, no rebound test
clinical management of NEC (necrotizing enterocolitis) fluid and nutritional maintenance, TPN, encourage breast milk
IBD, management inflammation of GI tract, caused by infectious organisms or dietary habits, treated with corticosteroids (to control inflammation), nutrition, possible surgery, anti-tumor necrosis factor may be used
gastroenteritis inflammation of stomach and intestines, vomiting, diarrhea, etc.
Created by: BrandiLynn
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