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platelets&hemostasis

MTSU Stegall-Faulk Anatomy 2

TermDefinition
platelets cell fragments of megakarycytes.
platelets large amounts in the spleen. initiate and control clotting process form a patch with myosin and actin fibers.
glycoproteins stick on the surface
thrombopoietin hormone stimulates formation of platelets
hemostasis cessation of bleeding
3 mechanisms that prevent blood loss from damaged vessels vascular spasm, platelet plug formation, and coagulation
vascular spasms wall of blood vessel, smooth muscle within the wall contracts, reducing the diameter of the vessel and reducing the blood flow.
platelet adhesion stick to the exposed collagen and other endothelial cells.
platelet activation activated and release several substances by exocytosis
ADP attract more platelets, cause platelets to swell
thromboxanes stimulate more vascular spasm and sttract more platelets
platelet activation ADP, thromboxanes, serotonin, clotting factors, calcium ions, and control of platelet activation and aggregation.
clotting phase the ultimate goal is the conversation of circulating soluble fibrinogen into insoluble fibrin.
extrinsic pathway initiated by tissue and or vessel injury. release from vessel walls>tissue factor CF3> activate CF10> common pathway
intrinsic pathway initiated by substances within the blood. released from platlets> inactive CF7 exposed collegen and activates CF7> activate CF10> common pathway
common pathway activates CF10
Prothrombinase catalyzes the conversion of prothrombin into thrombin
thrombin and calcium catalyze the conversion of fibrinogen into fibrin(insoluble)
clot retraction 30-60 minutes. actin and myosin contract
fibrinolysis scab reducing in size by breaking down insoluble fiber
plasminogen proenzyme for breaking down activation by thrombin
IV streptokinase breakdown clot
t-PA issue plasminagen activator to break down
thrombocytopenia platelet count below 100,000
thrombosis abnormal clotting in a closed blood vessel
Created by: deleted user
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