Busy. Please wait.

show password
Forgot Password?

Don't have an account?  Sign up 

Username is available taken
show password


Make sure to remember your password. If you forget it there is no way for StudyStack to send you a reset link. You would need to create a new account.
We do not share your email address with others. It is only used to allow you to reset your password. For details read our Privacy Policy and Terms of Service.

Already a StudyStack user? Log In

Reset Password
Enter the associated with your account, and we'll email you a link to reset your password.

Remove Ads
Don't know
remaining cards
To flip the current card, click it or press the Spacebar key.  To move the current card to one of the three colored boxes, click on the box.  You may also press the UP ARROW key to move the card to the "Know" box, the DOWN ARROW key to move the card to the "Don't know" box, or the RIGHT ARROW key to move the card to the Remaining box.  You may also click on the card displayed in any of the three boxes to bring that card back to the center.

Pass complete!

"Know" box contains:
Time elapsed:
restart all cards

Embed Code - If you would like this activity on your web page, copy the script below and paste it into your web page.

  Normal Size     Small Size show me how

Stack #121109

a MCPHS- Provider I- Ch 41- Assessment & Management of Pts w/DM

Group of diseases characterized by hyperglycemia Diabetes Mellitus
Hyperglycemia can be d/t defects in Insulin secretion, Insulin action, Both
Insulin functions r/t glucose Transport, Metabolism, Stimulates storage, Inhibits breakdown, Signals liver to stop release
Storage sites of glucose Liver & muscles
Other functions of insulin Enhances storage of dietary fat in adipose tissue, Transport of amino acids into cells, Inhibits breakdown of protein and fat
Type 1 DM d/t Autoimmune destruction of Beta cells in pancreas
2 factors causing Type 2 DM Decreased sensitivity to insulin (resistance), Impaired beta cell function
DM that can be controlled by diet Type 2 DM
Fasting blood glucose > 126 mg/dL
Random glucose > 200 mg/dL
Three P's r/t Clinical Manifestation of DM Polyuria, Polydipsia, Polyphagia
Polydipsia vs. Polyphagia Dip:excessive thirst, Phagia:excessive eating
Dietary goals r/t DM Optimal nutrition, Meet energy needs, Reasonable weight, Prevent wide glucose fluctuations, Decrease serum lipids
Diabetic ketoacidosis(DKA) caused by Type I diabetes
Main causes r/t DKA Noncompliance w/insulin regimen, Illness/infection, Undiagnosed/untreated diabetes
Hyperglycemic Hyperosmolar Nonketonic Syndrome(HHNS) caused by Type II diaetes
Complication r/t Diabetes can occur in anyone w/ Type I and Type II diabetes, Not only Pts using insulin
Glucagon function Stimulates liver to release glucose
Pathophysiology effects r/t Type I diabetes Decreased insulin production, Rampant glucose production by liver, Fasting hyperglycemia, Glucose not stored in liver
Ketone bodies are byproducts of Fat breakdown
S/Sx r/t DKA Abd pain, N/V, Hyperventilation, Acetone breath(fruity odor)
Insulin problems r/t Type II diabetes Insulin resistance, Impaired insulin secretion
Primary treatment r/t Type II diabetes Weight loss
Main goal r/t Diabetes treatment Normalize insulin activity and blood glucose levels, Reduce development of complications
Glycated hemoglobin(A1C) reflects Average blood glucose levels over the last 2-3 months
A1C binds to RBC's for the cells entire life
Rapid-acting vs. Short-acting vs. Intermediate-acting vs. Very long-acting r/t Insulin names R:Lispro, S:Regular, I:NPH, V:Lantus
Rapid-acting vs. Short-acting vs. Intermediate-acting vs. Very long-acting r/t Onset period R:15 min, S:30 min, I:2 hrs, V:1 hr
Insulin regimens goal Mimic normal pattern of insulin secretion d/t food intake and activity patterns
Pt requirements r/t Complex insulin regimens Commitment, Intensive education, Close follow-up w/health care team
Complications r/t Insulin therapy Local/systemic allergic reactions, Insulin lipodystrophy, Insulin resistance, Morning hyperglycemia
3 types of Morning hyperglycemia Dawn phenomenon, Insulin waning, Somogyi effect
Alternative methods r/t Insulin delivery Insulin pen, Jet injector, Insulin pump, Implantable insulin delivery, Pancreatic cell transplants
First skills taught r/t Newly diagnosed diabetics Needle insertion & insulin injection
Absorption rates(Fastest to slowest) r/t Injections sites Abd(fastest), arm, thigh, hip(slowest)
Hypoglycemia r/t Blood glucose level Less than 60 mg/dL
Hypoglycemia causes Too much insulin or physical activity, Too little food
Mild vs. Moderate vs. Severe hypoglycemia r/t Manefestations Mild:adrenergic symptoms(sweating, tremor, tachycardia, hunger), Moderate:adrenergic symptoms plus some CNS impairities, S:extremely impaired CNS function(Pt needs assistance)
Immediate treatment r/t Hypoglycemia 15g fast-acting concentrated CHO
Emergency treatment r/t Hypoglycemia 1 mg glucagon
Time r/t Snacking Peak insulin activity
3 main clinical feature r/t DKA Hyperglycemia, Dehydration/electrolyte loss, Acidosis
DKA treatment factors that reduce serum K levels Rehydration, Insulin administration
Precipitating events r/t HHNS Acute illness, Medications exacerbating hyperglycemia, Recent history of polyuria w/adequate fluid intake
Ketosis & acidosis r/t HHNS Generally do not occur
Manifestations r/t HHNS Hypotension, Severe dehydration, Tachycardia, Variable neurologic signs
Initial treatments r/t DKA & HHNS Fluid replacement, Correct electrolyte imbalances, Insulin administration
Cause r/t Diabetic retinopathy Changes in blood vessels of retina
Autonomic neuropathy r/t Adrenergic symptoms of hypoglycemia Symptoms are diminshed/absent
Subtle sign r/t Hypoglycemia Numbness around mouth, Difficulty concentrating
Manifestations r/t Sudomotor neuropathy Decreased/absence of sweating in extremities, Increased upper body sweating
Created by: rpclothier