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Neural Exam 2

Ch. 5, 6, and 7

QuestionAnswer
Somatosensation Bodily sensations of pain, temperature, touch and proprioception. Specialized receptors convert sensory stimuli to neural signals and transmit to parietal lobe to analyze information.
Mechanoreception touch, pressure, vibration, and kinesthesia
Thermoreception Cold, hot, temperature
Nociception Pain
Three-neuron organization Dorsal root ganglion, brainstem/spinal cord, and thalamic nuclei to primary sensory cortex (parietal lobe).
Conscious sensation occurs at the cortical level
primary sensory cortex Analyzes the quality of sensory information; Broadmann areas 1, 2, 3.
Somesthetic association cortex Superior and part of the inferior parietal lobe and lies at the crossroads of the temporoparietal (occipital regions).
Lesion in the somesthetic association cortex Cognitive impairments and Cross-modality integration
Trigeminal Nerve (5) Principal sensory nerve of face; innervates different regions of the head, face and intraoral structures.
Analgesia absence of pain while consciousness.
Anesthesia insensitivity to pain with/without consciousness
Graphesthesia feel writing on the skin
Kinesthesia Awareness of position, weight, and movement.
Proprioception sense of where your limbs are in space
Stereognosis perceiving an object via touch
referred pain pain perceived at a site NOT the pain stimulus site.
Phantom pain perception of pain on a limb that is no longer there.
Visual Cortex; Broadmann area 17 midsagittal surface of the occipital lobe
Damage to visual cortex blind spots in the visual field or total blindness
Damage to secondary visual areas (visual memory is stored here) visual agnosia
visual agnosia problem with giving meaning to what is seen by relating what is seen to experiences and knowledge
Vision area key for reading of words, and in the recognition of objects. Seen in MRI and PET scans
Island of Reil or INSULA lies below the fissure of Sylvius (5th lobe). may be involved in programming for speech (apraxia).
Pupillary Light Reflex light = constricting the pupils. Both pupils constrict in response to light entering one eye
Direct Response pupillary reaction in the eye exposed to light
Consensual Response Reflexive change in other eye
Occuulomotor N (CN 3) Damage alters pupillary light reflex
Temporal Lobe responsible for auditory processing (Heschl's gyrus BA: 41), sense of smell, holds Wernicke's area
Outer ear pinna and eternal auditory canal
Middle ear tempanic membrane (ear drum), Occicles, Eustachian tube.
Inner ear Bony and membranous labyrinth, cochlea, and CN 8
Frequency pitch
Intensity loudness
Wernicke's area BA: 44, Understanding and producing MEANINGFUL speech. Short-term memory, speech recognition spoken written and signed lnguage.
Wernicke (44) and 39 damage 44 = Wernickes Aphasia: Issues in comprehension and neither understandable meaning nor syntax. Lack of self awareness, rarely respond to treatment. 39 = Anomia
Alexia loss of the ability to read
Agraphia loss of the ability to write or spell
Acalculia loss of the ability to perform basic mathematical calculations
Paraphasia substituting one word for another
Secondary Auditory Association BA: 21,42; involved in comprehension of speech and language memory
Vestibular System Reflexive sensorimotor system controls: equilibrium, upright posture, coordinate head, body movements, and eye fixation.
Semicircular /canals Balance organ; Superior, posterior, and lateral; at right angles to each other, contain fluid and can sense movement
Vestibule (saccule & utricle)
Vestibular nuclei
Sensory receptors (hair cells) In semicircular ducts respond to changes head and body position
Dynamic regulates body and head positions during rotational and angular movement.
Static regulates straight-line movements and regulates both head and body position during rest.
Motion Sickness Most common vestibular system; vertigo, dizziness, nausea (especially with up and down movements).
Vertigo Sensation of spinning, associated with Meniere Disease.
Labyrinthitis irritation of the intercommunicating semicircular ducts after a viral infection or inflammation. Vertigo, nystagmus, dysequilibrum, nausea.
Benign positional vertigo Common in the elderly, caused by head movement.
Spinal Cord (Motor System 1) Most important structure btwn body and brain. Extends from the base of the skull to the lower back Increase in cord size at the cervical and lumbar levels cause greater NN supply is needed there.
Spinal Nerves 31; C:8 T:12 L:5 S:5 C:1
Neurotransmitters released by the activity of brainstem (Acetylcholine, Norepinephrine, Serotonin, and Epinephrine)
Acetylcholine the primary neurotransmitter of the autonomic nervous system, regulates voluntary or reflexive motor movements, Myasthenia Gravis
Lower Motor Neuron Syndrome (LMN) (peripheral NN damage) Complete muscle weakness and total flaccidity.
Cerebellum (Motor System 2) makes ongoing modifications to the motor plan, error control device, Coordinates input and output systems during RAPID movement.
Cerebellum NOT concerned with: conscious appreciation of sensations and cognitive processing
Cerebellum participates in: Motor learning, Motor memory, Movement execution
Cerebellar Malfunction Hits target but trajectory is jerky and requires corrections
Cerebellar Lesion evident on the body on the ipsilateral to the site of lesion Sure sign it's cerebellar lesion: Vision cannot compensate
Romberg Test Evaluates proprioception. Arms extended in front, feet together and eyes closed. If one arm drifts down and/or patient tilts. Cause may be a cerebellar (input or outpt), vestibular or proprioceptive abnormality.
Ataxia Lack of order and coordination in muscle activities. Segment movements.
Dysdiadochokinesia Can't do /pa ta ka/
Dysarthria Slow, slurred and disjointed language
Dysmetria Error in judgment of a movement’s range/distance to the target.
Intention Tremor (Motion Tremor) Tremors during a skilled movement sequence; more severe as the target closer
Hypotonia Normal muscle tension is decreased and the muscle becomes floppy -ipsilateral
Rebounding Person looses the ability to predict, stop or dampen movement
Cerebellar Pathologies Cerebrovascular Accident and Toxicity (alcoholism and PCD)
Progressive Cerebellar Degeneration PCD fredreich ataxia: autosomal recessive genetic degenerative condition.
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