NVRN Word Scramble
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Question | Answer |
Stroke is _______ leading cause of death | 5th |
What percent is Ischemic events? Hemmorrhagic events? | I - 80% H - 20% |
What race has highest hemm strokes | Asian & Finns, AA > white |
Define Stroke | rapid syndrome of sudden onset, non-epileptic neurologic deficit |
what is ischemic infarct | dead tissue at core |
What is common to happen after infarction? | Hemmorrhagic transformation HT` |
What is penumbra | potentially viable brain tissue surrounding core infarction |
What is Framingham Study | 1950s, Mass, about risk factors for stroke and CAD HTN single most imp risk factor, Incr total chol, decr HDL, whole grain reduce risk, Elev C protein in women risk for carotid atherosclerosis, New L BBB, |
What is GCNKSS Gr. Cinn/North KY study | First ever stroke in AA > whites young/middle age, HTN/DM risks, prior stroke, 1st degree relative with hx of ICH |
Northern Manhattan Stroke Study (NOMASS) | Carotid atherosclerosis White>AA/non hispanics, risks: HTN, DM, smoke, hyperchol, abd obesity, afib white/hisp > AA |
Strong Heart Study | Native Am, >DM, smoke, obesity HTN = Arizona > Oklahoma Native Am, binge drinking |
Southwest US Native Am. Stroke Study | Long time to get to hospital, common HTN, DM, alcohol consumption, prev. stroke/TIA, obesity |
ReGARDS | born in stroke belt incr risk for stroke |
What % of strokes are first time? What % need institutional care? | 70% 20% |
Non-modifiable risk factors for stroke | Age: doubles 10y after 55y, >AA Race: AA greatest risk Gender: men > women Family hx: paternal side > risk |
Modifiable risk factors | CAD, HTN, smoke, DM, afib, diet, sickle cell, obesity, (post menopausal) hormone replacement, incr chol. < mod. risks = alcohol/oral contrac/migraine/sleep disorders/hypercoag/inf dis/acute inf |
ABCD2 Score | prediction for severity stroke with TIA: 1 - >60y 1 - BP >140/90 2 - wkn, 1 - speech w/o wkn 2 - >60min, 1 - 10-59min 1 - DM <4 = low, 4-5 = mod, >5 = high risk |
What r 5 cat of TOAST - Trial of org in acute stroke tx | way to categorize what caused ischemic stroke. 5 categories: 1.Lg aa atherosclerosis - occl/stenosis lg major aa 2.Cardioembolism - afib/MI/inf endocarditis/myxoma 3. Lacunar - htn,dm,smoke,hyperchol |
What r 5 cat of TOAST | 4. other etiology: hypercoag/venous thromb/vasculitis/art diss/cocaine/fat emboli 5. undertermined cause (cryptogenic): incomplete workup/workup with no source |
What included in work up for stroke | brain imaging ( ct,mri) vasc imaging cta/mra/tcd/cranial ultra cardiovasc H/P: hrt snds/holman/12 ecg/caps /pulses/jug dist/bp Duplex ultra: carotid/vertebral Echo/TEE Labs: Protein C,S/antithrombin III incr |
IPH/ICH most common cause | HTN #1, #2 Cocaine (ICH) trauma, amyloid angiopathy/aneurysm/vasculitis/alc abuse/cocaine/neoplasms/infections |
Where are IPH's usually | subcortical region, near lateral ventricles (BG/thal) |
2nd most common hemm stroke | aneurysmal SAH, >women, ICA |
Studies to incr public detection | FAST - 80% s/s face/arm/speech/time FLASH - 90% s/s face/leg/arm/speech/HA BE FAST - improve posterior stroke balance/eyes |
Who uses EMS more? | AA, hemm stroke, incr in stroke severity, unemployed |
Stroke Severity Scales | Los Angeles Motor Score - LAMS: lg vessel occl: face 0-1/grip 0,1,2/arm strength 0,1,2 Shortened NIH, sNIHSS 8 items |
What should EMS not do with stroke | lower bp, give glucose IV, give excessive IV volume |
What is Golden Hour for tx stroke? | initial MD eval - 10 min stroke team notified - 15 min CT - 25 min CT/labs interpreted - 45 min tPA - 60m |
Golden 1/2 Hour | MD/stroke - arrival CT - 10m CT/labs read - 25m tPA - 30m |
How many triage levels? What level need to have for stroke/TIA? | 5 levels Level 2 for stroke/TIA |
What is med risk for cardio-embolism | PFO, L atrial turbulence (smoke), post op, mitral prolapse |
What is TIA | sudden focal neurologic disablity, resolves in 24h, 300,000 annually |
CHADS2 | 1 - CHF 1 - HTN 1 - >75y 1 - DM 2 - hx stroke/TIA |
ED assessment/work up | ABCs, disability, non-contrast CT/MRI, labs, ecg, h/p: LKN, handedness, allergies, prev stroke/TIA, risk for CAD, drug/alcohol use, anticoag meds, hx brain hem/trauma, sx hx |
Stroke mimics: can you use tPA | Yes. Conv. Dis, htn enceph, hypogly, complicated migraine, sz, AMS |
What scale is used for Hemm? | GCS 3- unresponsive, 15- normal Eyes: 1 no open, 2 pain, 3 speech, 4, spont Verbal: 1 no verbal, 2 incomp sounds, 3 inapp words, 4 confused, 5 oriented Motor: 1 none, 2 Decer, 3 decor, 4 withdraw, 5 localize pain, 6 obeys commands |
Which neuroimaging is 100% sensitive for blood? | Non-contrast CT |
CTA is good to determine | Lg vessel occl, not lacunar |
Which part of MRI is best for acute stroke confirmation | DWI - diffusion weighted imaging, w/in min. Ischemia = White MRP - detect presence/abscence viable tissue |
which type of stroke is TCD used with | SAH - blood flow in lg aa |
Which lab test is needed prior to tPA? | Blood glucose Coumadin - need INR |
When is a lumbar puncture usually ordered, what kind of stroke? | SAH |
Gen mgmt of all acute stroke pts | temp, NPO, card enzymes, arrhythmias, bp parameters, hyperglycemia, HOB |
What are BP parameters for hemm & ischemic | H: <160/90 I: untreated = permissive 220/110 treated= <185/110, then <180/105 after bolus tPA |
What is correct glycemic control | 80 - 140 mg/dL |
What is correct HOB for hemm & ischemia | H: 30 deg I: 0 deg, pt turned to side 20% incr blood flow Zodiac trial - only did lg vessels |
How many bones in skull and name | 8 bones: frontal/temporal(2)/parietal(2)/occipital/sphenoid/ethmoid |
What is total cranial volume made up of and how much | 1400-1500ml: CSF, brain tiss, blood |
What are sutures of skull | Sagittal - b/n 2 parietal Coronal - parietal to frontal Lambdoidal - parietal to occipital Squamosal - joins parietal, temp, occ |
How many total vertebrae? How many cervical, thoracic, sacral, coccygeal? | total 33: 24 vertebral & 2 fused ( atlas C1, axis C2) Cerv 7, thor 12, sacral 5, coccy 4 |
Name meningeal coverings of brain and spinal cord | Dura mater - strong, b/n bone & dura mater Arachnoid mater - thin, loose surround brain, contains lg vessels, below subarachnoid space Pia mater - adheres to brain, choroid plexus |
Where is CSF manufactured | choroid plexus in Pia mater |
Name and locate three ventricles in brain | Lateral (2): horns Third ventricle: b/n laterals & connected by foramen of Monro Fourth ventricle: connected by aqueduct of Syvius |
What is communicating vs non-communicating hydrocephalus | comm: obstruction/lack of absorption non comm: obstruction in ventricle system |
How much CSF is produced hour? day? total system volume? | Hour: 20ml/h day: 500ml/day, total vol: 150ml |
What is cellular structure/fx of neuron | Dendrites: impulse reception Cell body: metabolic fx of cell Axon: carries impulse away |
What makes up the Circle of Willis | Anterior and Posterior circulation Connection of 3 comm aa (AcomA, PcomA (2) |
Where does the ant. and post. circ originate | common carotid CCA, R CCA: innominate aa/L CCA: aorta Post: subclavian aa, R innominate, L aorta |
What is considered the Ant. circulation and leads off ICA | Ophthalmic aa OA PcomA: post comm. aa MCA: supply to deep subcortical area ACA: linked together by AcomA |
What is posterior circulation include | Vertebral aa give off post inf cerebellar aa PICA Vertebral fuse to Basilar aa Basilar gives off to PCA (thalm), SCA, pontine, AICA |
What % of circle of willis is ideal? Most common missing segments? | Only 50% 1. ACA, 2. One of the PcomA, 3. 1st seg of PCA |
What is the cerebral cortex | 80% of brain wt. 2 hemispheres separated by corpus collosum 4 lobes: frontal/temp/parietal/occipital |
What is major fx of Brodmanns Area 4 | Voluntary motor fx: Motor strip ACA territory ( esp leg) and some MCA (lower facial wkn) |
Where is Area 44: Broca's area | Left MCA, frontal lobe Expressive aphasia: spoken/written lang. word finding, |
Where is 9-11, cognitive fx area | ACA behind forehead seen in SAH affects orientation, memory, insight, judgement, arithmetic/abstract thinking |
Where is Area 1-3, primary sensory | Parietal lobe sup ACA, inf MCA |
Where is area 5 & 7: somesthetic assoc | MCA extinction, stereognosia (tell what object it is), graphesthesia (write on skin and decipher) |
What is are 39 & 40, Wernicke's area | Left MCA receptive lang. |
Where is area 41 & 28 | Temporal lobe, MCA |
What does Occipital lobe show | visual integration & pathways |
What does Area 17 & 18 primary visual cortex affect | PCA territory |
Where would you find cortical blindness stroke | top of the BA or PCA occl. |
Where is homonymous hemianopia found | always post. to chiasm Lg parietal - MCA Medial occipital - PCA |
Double vision would indicate which part of brain | brain stem infarct |
Internal capsule is which distrubution | MCA |
Thalami is | PCA distribution |
What does cerebellum control | fine motor coordination, equilibrium/balance, ataxia, ipsilateral side affected stroke, vertigo |
Brainstem stroke affects | LOC, double vision, cardiac, resp, vomiting, sneezing, hiccups |
What does cerebrum control | association, motor, sensory, contralateral |
Name cranial nerves | 1 - smell, 2 - vision, 3,4,6 - EOM extraocular mvmt, 5-corneal reflex(S)/maxillary(S)/Mandibular(S)(M) teeth clench, 7-facial(S)(M), 8-acoustic/balance, 9,10-glosso/vagal, swallow, 11-spinal acc, 12-hypogloss, tongue mvmt |
Where is midbrain and what are infarct findings | From thalamus to pons, CN3,4 pupil dilate, EOM dysfx, decr LOC, (M)(S) disrupt |
Where is Pons and infarct findings | from pons to medulla oblongata, CN5,6,7,8 Pneumotaxic ctr: inhibit depth of resp/incr rate apneustic ctr: promotes inspiration gaze/diplopia, EOM dysfx, decr LOC, (M)(S), resp arrest |
Where is Medulla oblongata and infarct findings | from pons to spinal cord, CN8,9,10,11,12 cardiac/vasomotor ctrs decr LOC, card/vasomotor dysfx, hearing loss, dysphagia, quadriplegia |
What is important about basiar art thrombosis | Most misdiagnosed, hiccups, weird s/s, diplopia, vertigo, cortical blindness |
Diff bn cortical and brainstem infarct | Above brainstem: loss (M)(S) same side with face brainstem: CN deficit same side as infarct, but opp extremity (M)(S), sudden loss of consciousness in non-hemm |
When should you do NIH? Scoring is from 0 - _______ Best NIH monitoring p tPA | admission, discharge, 1 x shift, and any changes Score: 0 - 42 baseline pretx, post tPA, q4h q15-30 focused neuros, deterioration/improvemnet |
What does ICH score predict | death in 30 day, 0- best prognosis, 6- dead. contains: GCS, ICH vol, Intraventricular component, Supra vs Infratentorial, Age |
what is Modified Rankin Score for | disablility score, 0-6, intervention when score 0-1 |
What are absorptions/color for CT | air - black/none, CSF - black/low, White matter - darker gray/low med, Gray matter - lighter gray/high med, Blood - white/high, Bone - bright white/high |
How to read CT | start at base of brain and moves superiorly to top of skull, looking through bottom of feet to top of head, mirror image - L side reflects right brain, vice versa |
Adv/Disadv for CT | Adv: fast, good for lesions dis: poor resolution, 6-8h delay b4 stroke shows |
What are early/late signs for stroke on CT | early: clot in vessel, hyperdense aa late: hypodensity, darkened, subacute inf |
What is scoring for Modified Ranking Score | 0- no s/s 1- able to carry out usual activities/duties 2- slight disable, can look after self w/o assistance 3- mod disable, some help, can walk w/o assistance |
What is scoring for MRs | 4- mod severe disable, not walk w/o assist, not do bodily needs w/o assist 5- Severe disable, bedridden, incontinent 6- dead |
NIH scoring LOC | 0- alert 1- not alert, arousable 2- needs repeated stimulation, obtunded, req strong painful stimuli to make mvmnts 3- Reflex motor response, totally unresponsive, flaccid |
NIH Questions: Month/Age | 0-answers mo/age 1- ans one correctly, intubated, sev dysarthria 2-both incorrect/no answer, aphasic/stuporous not responding |
NIH Commands to open/close hand/eyes | 0- obeys both 1- obeys one 2- Not obey/respond |
NIH EOMs: horizontal eye mvmt, doll's eyes used if needed | 0- Normal 1- Partial gaze palsy, can come to ctr, but goes back 2- Forced deviation/total gaze paresis |
NIH Visual Fields: upper/lower, confrontation | 0-no loss 1- partial hemianopia 2- complete hemianopia 3- bil hemianopia, blindness/cortical |
NIH Facial Palsy: smile, raise eyebrows, grimace with noxious stimuli | 0- normal symmetric mvmt 1- minor paralysis, flattened nasolabial fold 2- Partial paralysis: total paralysis lower face only 3- complete paralysis lower/upper face |
NIH Motor Arm: Count of 10 Left & right | 0- No drift 1- drift 2- some effort against gravity 3- No effort against gravity 4- No mvmt |
NIH Motor Leg: Count of 5, Left & right | 0-No drift 1- drift 2- some effort 3- no effort 4- no mvmnt |
NIH Limb ataxia: Finger to nose, heel to shin | 0- absent 1- present in one limb 2- present in two limbs |
NIH Sensory: sensation, grimace, withdrawal | 0- Normal, no loss 1- mild to mod, dull, less sharp 2- severe to total loss, not aware of being touched |
NIH Language: aphasia | 0- no aphasia 1- mild to mod 2- severe 3- mute, global: no speech/auditory comprehension |
NIH Dysarthria: slurred speech | 0- normal 1- mild to mod: slur some words 2- unintelligible |
NIH Extinction/Inattention | 0- No abnomality 1- visual, tactile, auditory, spatial, personal inattention, or to bil sensory 2- profound in >1 sensory, not recognize hand, etc |
What is watershed infarct | b/n regions |
which area is worst/best outcome for ICH: cortical, BG, thalamus, Pontine, Cerebellar | Worst: Pontine Best: Basal Ganglia, most common IPH, |
What should you r/o with Intraventricular hemm | Presence of ACA or AcomA aneurysm, usually is expansion of hemm in BG, IPH |
Where is SAH | Bleeding in subarachnoid space, usually from aneurysm rupture or trauma, "star" appearance or "hanging chicken" |
What is 1st s/s SAH | thunder clap HA |
What is tx and appearance of amyloid angiapathy hemm | more round and no tx can be done, more on surface |
what are CTP scans for | Looking to save penumbra and ischemic penumbra, measures tiss perfusion, usuall for "wake up" strokes |
In MRI, which is better for disease definition, T1 or T2 | T2, CSF- white, Fat- dark |
What is GRE, gradiant Recall Echo in MRI | Rules out blood, microbleeds may be detected |
What does the FLAIR show in MRI | older stroke, dating/timing of stroke onset, if neg and DWI pos, give tPA, shows 4-6h after stroke |
Name MRI sequences | 1- GRE id blood 2- DWI ischemia 3- FLAIR timing of onset, old isch changes 4- MRA vascular imaging` |
What is gold standard imaging for vascular | Catheter Angiography, IR, uses 1/2 of dye of CTA, invasive, can have IA rescue, safe to due p tPA |
What is HOB for sheath mgmt | <15 deg |
What is doppler shift | ultrasound to describe mvmt of blood away/towards probe |
What is duplex imaging | image lg vessels in neck, see plaque & blood, stable(hyperechoic)/unstable(hypoechoic) plaque, |
What is transcranial dopper TCD | flow of blood thru lg vessels in circle of willis, detect vasospasm (risk @ 5-7d), monitor tPA, brain death, sickle cell |
What is risk of vasospasm? Tx of vasospasm | ischemia Tx: Triple H therapy: hemodilution/hypervolemia/htn Meds: Nimodipine(neuroprotective effect, not lessen spasm, Mg, statin Intra Art tx: angioplasty, verapamil inf |
What was result of NINDs tPA stroke study | Got tPA FDA approved and standard of care |
What is tPA dosing | 0.90mg/kg, total dose 90mg Waste 10cc, give 10% as bolus, then remaining 90% inf over 1 hour. |
What should BP be b4 tPA and during/after | b4: 185/110 after: 180/105 |
what are inclusion/exclusion criteria for tPA | incl: clear time of onset w/n 180 min of tx, deficits on NIH, neg CT for hemm Excl: prev stroke/trauma w/n 3 mos onset, major sz w/n 14 days, hx ICH, SBP>185/110, improve TIA |
What are more excl of tPA | s/s SAH, hx GI/urin hemm past 21d, art px prev 7d, Sz Todd's paralysis(mimic), incr PTT, <100,000platelet, BG <50; >400 |
At 3 mos, what % of pt w/ no deficits after tPA | 12%, no incr death, age not a factor, severe NIH not a factor, sICH rate 6.4%, 30% more likely to have mRS 0-1 @ 3 mos |
What time frame should IV tPA be adm to acute stroke | w/n 4.5h |
What are the drug choices to manage BP wtih tPA | labatelol 10-20mg q 5-15m up to total 150mg IVP Nicardipine drip: rapid onset/offset, less titrations Cleviprex: incr med 1-2mg = decr 2-4mg of BP, rapid |
what are contraindications of labatelol | asthma, CHF, heart block |
what is Poiseuille's Law | incr in extremity size = decr BP, cuff too lg decr extremity = incr BP, cuff too small |
What is BP monitoring after tPA | BP q 15 x 2h, 30m x 6h, 1h x 16h, q4h |
Post tPA mgmnt | No invasive procedure, No antiplatelet/anticoag for 24h |
What is % for recanalization wtih tPA | 13-38%, better if tx is started earlier |
Lg vs sm vessel occl tx wtih tPA results | Lg vessel: less likely to respond, need IR Sm. vessel: very well tx |
What rare complication with tPA is more common with AA and pt taking ACE/ARBs? | oropharyngeal edema, may need intubation/drugs |
What defines a symptomatic ICH during tx of tPA? | >4 pts from original NIH + IPH on non-contrast CT |
Pt can't get tPA, what r IA tx, (intra-arterial) options? | thrombectomy, drip tPA directly on clot, angioplasty, stenting |
List Guideline Classification for IA tx | Class 1: (strong) Benefit >>> risk Class IIa: benefit>>risk (tx reasonable) Class IIb: benefit > risk (tx may be considered) Class III: risk > benefit (harm) no tx |
What is the TICI score? | Done for Thrombolysis cerebral infusion after tx Grade 0: no perfusion G1: penetration w min. perf G2: partial perf, G2a/2b: better G3: complete perf Goal is 2b/3 w/n 6h of stroke onset |
What high risks are included with ICH | HTN, >55y, anticoag therapy, amyloid angiopathy, smoke, alcohol use, asians (highest), AA 65-74y, white older age |
What is 6 mos mortality rate post-ICH | 30-50%, < independence in 6 mos, |
Describe scoring for ICH score | Score 0 = 0 death in 30 days 1 = 14% death 2= 28% death 3=70% death 4= 95% death 5&6 = 100% death |
Indicators for ICH scoring | GCS 3/4 - 2pts, 5-12 - 1 pt, 13-15 - 0pt ICH volume: >30ml 1 pt, <30ml - 0 pt IVH: Yes - 1pt, No - 0pt Infratentorial: Yes - 1pt, No - 0pt Age: >80 - 1pt, <80 - 0pt |
What is significance of CT "spot sign" | probably active bleeding expansion, 72% in 24h, most common loaction for ICH is BG d/t HTN. 2nd: amyloid angiopathy in cortical/parieto/occipital (surface) |
What are clinical s/s of ICH | impaired LOC, vomiting, severe HA, severe HTN |
Ways to manage ICH | airway mgmt, INR >1.4 needs reverse anticoag, BP reduction 160/90, CPP 60-80mm Hg (MAP-ICP=CPP) |
What is first tier therapy to manage ICP | ventriculostomy w CSF drainage, then manitol, hypertonic solutions, HOB 30deg, hyperventilation, sedation |
What is second tier therapy to manage ICP | Hemicraniectomy, hypothermia, barbituate coma |
Are prophylactic sz medication necessary in ICH | No, only if see sz. |
When is surgery benefit in ICH? | Sx not usually help Sx better in cerbellar hemm >3cm to prevent herniation |
Who is most common for SAH | women, 40-60y, Japanese, black>white, htn, smoke, alcohol, cocaine |
Most common aa affected by SAH | Base of Circle of Willis: PcomA from ICA, AcomA - ACA, MCA |
What are most common forms of aneurysms? Which aneurysm type most likely to rupture? What size is an aneurysm needing sx | Berry/sacular - 80-90% >7mm, worst HA |
What diagnostics determine SAH? What is gold standard? | Non-contrast CT If CT neg, then lumbar px CTA Gold standard: catheter angiography |
What scales are used to grade SAH severity | Hunt & Hess Grading Scale World Federation of Neurological Surgeons Scale |
What tx is best to prevent re-bleed | coiling/clipping |
What monitoring is needed for SAH rupture | monitor cardiac enzymes/telemetry, monitor for SIRS(HR>90, RR>20, WBC<4/>12, T >38C), hydrocephalus, hyponatremia (AcomA), 30%, cerebral salt wasting - decr free water, isotonic IV fluids |
Pt 40y has sz, mass effect on CT and hemm w/ HA....what likely is cause | AVM |
What is formula for cardiac output CO | CO = SV + HR |
what is SV: stroke volume | amt of blood ejected by heart with ea contraction |
What is Frank-Starling Law | Preload stretches heart muscle, incr degrees of stretch result in improved contractility until muscle is over stretched, dilated, & flaccid |
What is afterload | resistance in vasculature that heart muscle must overcome to eject volume` |
What causes resistance to flow in heart | atheroscloerosis, art pressure, metabolic factors(incr CO2 = dilate/decr CO2=constrict), intracranial pressure (ICP), extracranial pressure (stenosis in carotids/vertebrals), blood viscosity (incr hematocrit) |
What is primary method to autoregulation | Vasomotor reactivity |
Where is the phlebostatic axis and what is it for? What is leveled at the tragus? | 0 line for BP monitoring tragus: ICV |
Normal hemodynamic values: cardiac output CO/MAP/SvO2/ICP/CPP | CO - 4-8L/min MAP - 70-105 mm Hg SvO2 - 60-75% ICP - 0-15mm Hg CPP - 70-100mm Hg |
What is tidal volume | 5-8mf/Kg, volume of gas moved into/out of lungs in single normal insp or exp. Start with 8010ml/Kg and titrate down |
What is Vital capacity and who benefits from monitoring this | volume of gas exhaled after the deepest possible inhalation. Guillan Barre pts |
What factors reduce diffusion | pulm edema/thick membrane, fibrosis/surface area for gas exchange... |
What improves ventilation/perfusion | HOB 45 deg, good lung down, prone |
What is diff b/n PaO2 & SaO2 | PaO2: oxygen transported dissolved in blood serum SaO2: oxygen transported in combination w hemoglobin |
With FiO2, what is goal | PaO2 >60% produces SaO2 >90%, use FiO2<50%. Add PEEP to incr SaO2 b4 incr FiO2 |
pH/paCO2/PaO2/HCO norms | pH: 7.35-7.45 paCO2: 35-45 paO2: 90-100 HCO3: 22-26 |
in resp acidosis/alkolosis how do you adjust RR with paCO2 of 55, pH 7.34 | acidosis: incr RR alkolosis: decr RR |
What are factors with weaning success | LOC, hemodynamic stability, physiologic stability, sats, spontaneous ventilation, pulm mechanics |
What are two non-invasive ventilations | Bipap: have to be able to take mask off and not be restrained. PS @ insp/ PEEP@ end of exp Cpap: provided PEEP w/o pressure support |
What is most preventalbe cause of death in hospitals | VTE: DVT/PE/post-thrombotic syndrome |
What is Virchow triad | factors of DVT: change in vessel wall (injury), pattern of blood flow(venous stasis), constituency of blood(hypercoag states) |
What is best method for VTE/PE prevention? What is best dx of VTE? what is best dx of PE? | 1st: Medical mgmt: anticoags: lovenox 2nd: mechanical SCD Dx: LE dopplar PE dx: CT |
What is Braden Scale for predicting pressure ulcer | 6 -skin breakdown risk 32 - no risk for skin breakdown |
Factors influencing intensity/duration in Braden: Factors in skin/support tissues in Braden: | int/dur: Mobility/Activity/Sens perception skin/tiss: moisture/friction/nutrition/age/art press |
A pt with an infarct with left neglect, visual field deficits &/or tactile neglect, & incontinence likely is right or left brain | Right brain asoc with falls |
Normal BP/prehtn/high stage 1/high stage 2 | normal BP 120-80 prehtn: 120-139/80-89 high Stage 1: 140-159/90-99 Hight Stage 2; >160/100 |
What is first line anti-htn drug givn with 2nd drug | thiazide diuretic w ACE in AA Pro: cheap, effective Con: dehydration, incr blood viscosity |
what is 2nd drug of choice to combine with thiazide diuretics | Ca. Ch. blockers |
When are BB best used for htn | CAD and afib Pro: cheap con: stopping can cause death, impotence/depression |
What is best BP drug in stroke prevention/mgmnt | ACE/ARB pro: stroke reduction/cardiac remodeling/renal protection Con: not good to decr BP in pt that lack renin-based htn, avoid in preg, cough |
When is okay to use Minoxidil | When no other agents lower BP, aggressive, monitor pericardal eff, combine with BB to prevent rebound tachy |
What dose is best for statins in stroke? When does statin reduce first ever stroke? | Dose: 80mg statin First ever stroke prevention: elevated C-reactive protein |
when ASA best for stroke pts? Which is better ASA or Aggrenox? | when not on ASA before Aggrenox bettern than ASA, risk is HA |
Which is best ASA or Plavix for stroke | Plavix to reduce event Plavix + ASA to lower risk of embolic events, but not long term except wtih stents |
When is anticoagulation best used in relation of stroke prevention/mgmnt | cardioembolic stroke/crescendo TIA/hypercoag states/art dissection assoc w embolization/cerebral venous thrombosis |
Warfarin is best tx for young/old when? | warfarin is superior to placebo in reducing stroke risk. Younger: no comorbidities than use ASA Older: w comorb, use warfarin |
How to determine ciggarette pack year hx | # of yrs smoked X # of cigg smoked / 20 |
medications to support smoking cessation | gum: up to 24 pcs/d, pack b/n teeth and gum patch: >10cig/21mg, in 2wks/14mg, in 2wks/7mg |
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