Inflammation 102 Test
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| A. Red (trauma or surgical wound: protect and clean)
Yellow (prescence of slough and exudate: cleaning and removal of non viable tissue)
Black (necrotic tissue: debridement)B. vasoconstriction (limit bleeding), followed by vasodilation (bring WBC to area, allows fluid to leak out of vascular) fibrin formsC. static, rash (up to 6 weeks), aplastic anemia, fatal blood abnormality, Gram + and -D. 2 days to 3 weeks. Granulation (fibroblasts lay new collagen, fills defects and makes caps). Contraction (wound edges pull together). Epitheliazation (crosses moist surfaces 3 cm from good cell in all directions)E. valtrex, treat virus. Liver toxicF. celebrex, block receptors. NOT antipyretic, no GI effects but possible cardiac effectsG. yeast, black tongue, caused by antibiotic useH. neutrophils, monocytes, I. lasts for a couple of days, renal toxic. Reye's syndrome in children J. 3 weeks to 2 years. New collagen forms (for strength) and scare tissue growsK. collagen and elastic fibers, 0.3-3.0mm glands and hair folliclesL. initial inflammatory responseM. static, GI and hepatic toxicity, OTO toxic and renal, take with water, no grapefruit juice, Gram +N. hospital acquiredO. cidal, GI issues, no toxicity, cross reacts with PCN, Gram + and -. Most common antibioticsP. shift weight every 20 minQ. arrive 3-5 days after injury, get rid of waste produced by phagocytes, macrophages release fibrenogin and starts to heal and knit togetherR. fat, varies from person to person, regulates body temperatureS. static, GI, dysrhythmias, hepatotoxicity, tendon ruptures, anaphylactic, BS elevation, Gram + and pseudomonusT. chemical response, all irritate skin and cause pain and swelling, part of healing |
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