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NUR 141 EXAM 3

Quiz yourself by thinking what should be in each of the black spaces below before clicking on it to display the answer.
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Question
Answer
for intestinal disorders you need to interrupt one of the following...   digestion, secretion, absorption, motility (segmentation (mixing), peristalsis (propulsion)), elimination  
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intestinal disorders occur d/t   inflam, inf, tumors, obstr, structural problems  
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manifestations of dysfunction   pain, hemorrhage, N/V, distention, constipation, diarrhea, abnormal fecal contents, incontinence  
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lactose intolerance   enzyme deficiency, missing lactace, afros and asians, undigested CHO  
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celia disease   automimmune damage to villi when gluten is consumed: wheat, oats, barley, rye  
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IBD   crohns and colitis  
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cystic fibrosis   genetic, mucus accumulation=pancreatic dysfunction, protein and fat digestion problems  
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steatorrhea   fatty stools  
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oxalates cause   kidney stones  
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inflam of hte gastrointestinal tract   gastroenteritis  
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gastroenteritis is caused by   inf organisms (virus, bacteria, parasites)  
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source of inf of gastro   contaminated food/water, contact w inf person, dirty utensils, fecal-oral,  
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patho of gastro   inflam alters secretion and absorption due to direct damage or production of toxins  
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s/s and dx of gastroenteritis   N/V, abd pain/flatulence, systemic fever, body aches, diarrhea, dehydration, electrolyte loss, stool  
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labs for dehydration   inc Hgb, inc Hct, inc BUN, inc Creat  
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labs for stool   ova/parasite, culture, WBCs, or toxins. test takes days d/t normal flora in stool  
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major health care associated inf   C. diff (clostridium difficele)  
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C. Diff occurs in..   immunocompromised pt after antibiotic tx that destroys normal flora, spores last 70 days on surfaces  
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Tx for C. Diff   constact isolation, metronidazole (fLAGYL), vancomycin, stool transplant (not in immunocompromised)  
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diet for gastroenteritis   NPO until V stops, tx N/V, I/O, isolation, antibiotics, antimicrobials, no anti-diarrheals  
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IBS   chronic functional GI disorder, more in women  
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s/s IBS   abd pain, alternating diarrhea or constipation, flatulence, foul breath, N, anorexia, anxiety/depression, mucus stools  
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risks for IBS   stress, ETOH, smoking, lactose intol, specific food intol, psych factors  
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tx for IBS   high fiber (20gm), probiotics, low fat, no carb beverages or ETOH  
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meds for IBS   SSEI, loperamide (Imodium) for diarrhea, lubipostone (Amitzia) for constipation, alosetron (Lotronex) restricted access program  
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Inflammatory bowel disease   ulcerative colitis/chrones; chronic inflam of the GI tract, relapses/remissions, overractionto normal bacteria, 20yo and 60yo, genetic, more in whites  
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s/s of inflam bowel disease   diarrhea, bloody stools, wt loss, abd pain, fever, fatigue, skidney stones, liver disease, thromboembolism  
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complications of inflam bowel desease   hemorrhage, stricture/obstruction, perforation, fistula, colonic dilatation, nurtritional probs, sclerosing cholangisits, gallstones, liver failure  
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DX of inflam bowel diesease   labs, stool-+for blood, pus, mucus, signmoidoscopy, colonoscopy, EGD, barium enema, CT w contrast  
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IBD collaborative care goals   rest bowel, induce remission, , inflam control, correct malnutrition, reduce stress, stop smoking  
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meds for IBD   5-aminosalicylate (saulfasalazine (Azulfidine)), antimicrobials, corticosteriods, immunosuppressants, biologic and targeted therapies, antidiarrheals (Imodium), hematologics and vit (iron, potassium, zinc)  
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nutritional therapy for IBD   fluid/electrolyte, high calorie, low fiber, residue, fat, small meals, no trigger foods, TPN  
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surgical intervention for chrones   common, high recurrence rate, short bowel syndrome, emergency situations  
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surgical intervention for ulcerative colitis   less common, total proctocolectomy is curative  
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dec freq, difficulty passing stools, dec volume, retained feces in the rectum   constipation  
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causes of contstipation   insufficient dietary fiber and fluids, dec physical activity, ignoring defecation urge, laxative abuse  
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causes of constipation related to age   dec secretion of digestive enzymes, dec muscle tone abd wall  
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s/s constipation   distention, bloating, flatulence, inc rectal pressure, hemorrhoid  
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related to venous engorgement with straining   hemorrhoids  
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related to Valsalva   cardiac problems  
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severe constipation, no stools/gas expelled   obstipation  
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accumulation of hardened stool that cant be expelled   fecal impaction-leaking of stool  
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tx for atonic constipation   high fiber, use natural foods, increase water intake , activity  
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diverticulosis   asymptomatice, seen w scope, high fiber diet, wt loss, no straining, tight clothes and lifting. no nuts/seeds  
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diverticulitis   pain, inc WBC, fever, CT scan, colon rest, NPO, IV fluids, progression of food first mush.  
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lesions of mucosal surface that progect into lumen of the bowel   colon polyp  
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non cancerous, limited growth   hyperplastic  
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linked to colorectal adenocarcenoma "mushroom"   adenomatous  
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third most common form of cancer in US, familia   colorectal cancer, second leading cause of death  
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risk factors for colorectal cancer   s/s dont appear until desease is advanced, obesity, smoling ETOH, large intake of processed meats  
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decrease risk for colorectal cancer   exercise, diet large mts of fruits, veggies and grains  
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s/s of colorectal cancer   pain, change in bowel habits, anemia, distention/obstruction, wt loss, N/V, black stools, blood in stool, hemorrhage  
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tx for colorectal cancer   resect tumor, restore bowel, ostomy, chemo, targeted biologics, radiation  
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targeted biologics for colorectal cancer   bevicizumb (Avastin)-prevents agiogenesis in the tumor, cetuximab (Erbitux) blocks epidermal growth factor receptor  
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skin assessment hx   onset/duration of problem, relationship (season, travel, etc), symptoms, hx oa associated illness  
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factors for assessment   good lighting, room comfortable temp, cosmetics removed, inspect mailbeds, oral mucous membranes on dark skinned pts.  
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decreased blood supply as in COPD, vascular disorders   cyanosis  
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vasicilation oas in fever cellutitis   flushing  
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skin lesion that drains from 1 hole, painful   furuncle  
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skin lesion that drains from any locations, usually staph, fever   carbuncle  
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purilant skin infection   pyoderma (pyo=puss)  
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furuncles/carbuncles need...   antibiotics and incision to drain  
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secondary skin inf   changes in skin due to scratching, irritation, inf, meds, invironment  
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blistering that is a response to a med   steven johnson  
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inflammation of subcutaneous tissue   cellulitis  
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s/s of cellulitis   hot, tender, red, edema, diffused borders  
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tx of cellulitis   moist heat, immobilization, elevation, antibiotic. can goto gangrene. no amb until under control  
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viral skin disease   very contagious, transmit easy, wore w immunosuppressed, avoid antibiotics (masks symptoms)  
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shingles   follows nerve pathways, pain before lesions, painful, fever, itch.  
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meds for shingles   Zovirax, Famvir, analgesics, steroids to relieve pain and itch  
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benign grown due to a virus   wart-verucca. bleeding can spread virus  
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skin fold appear moist and beefy red or brown, itch may have pustules on edge   fungal skin infections  
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tx for fungal skin inf   keep skin cool dry, leather, topicals  
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tenea capitis   craddle cap  
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candida   oral thrush  
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vitiligo   loss of skin pigment  
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chronic dermatitis, papular lesions with scalling borders, redness, mostly on scalp, elbow, knees   psoriasis  
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tx for psoriasis   UV light, tegison, methotrexate, corticosteroids  
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Tegison causes   birth defects, visual disturbances, hepititis  
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Methotrexate causes   liver toxicity, take no asprin  
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corticosteroids   apply after scales are removed, wet skin, cover w plastic  
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development of new pigment, sudden mole, loss/gain of hair, bleeding   seborrheic keratoses-mostly benign  
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tumor arising from melanocytes, metastasizes to any organ   melignant melanoma  
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gross distortion of reality, disturbances of language, communication, withdrawal, thought fragmentation   schizophrenia  
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s/s of schizophrenia   apathy, confusion, delusions, hallucinations, speech probs, bizarre behavior  
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persistent averant (incorrect) belief or perception that the pt cant b convinced otherwise despite of evidence to the contrary   delusional  
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sensory perception that doesnt result from an external stimulus in an awake state. auditory, gustatory, tactile, olfactory, visual   hallucinations  
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schizoid personality (schizophrenia phase 1)   loners, limited range of emotional expression, withdrawn  
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prodromal phase (2)   eccentric behavior, withdrawn, impaired role function, disturbances in communication, neglect of personal hygiene  
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schizophrenia (3)   delusions, hallucinations, disorganized speech, catatonic behavior  
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residual phase (4)   periods of remissions and exacerbations, depends on if they are taking meds or not  
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tx for schizophrenia   individual/group/behavioral therapy,  
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meds for schizophrenia   major tranquilizers, antipsychotic drugs. blocks dopamine in the brain  
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phenothiazines   thorazine, mellaril  
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thiozanthenes   navane  
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nonphenothiazines   haldol, zyprexa, resperdal  
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alteration in mood   depression  
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s/s depression   sadness, crying, withdrawn, cant concentrate, insomnia, anorexia, constipation, HA, fatigue  
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NS interventions   ask if they r thinking of killing self, make contract, meds, trust, make chage, set goals  
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anti depressio nmeds   effect neurotransmitter levels. MAOs, tricyclic antidepressants, slective serotonin, uptake inhibitors (SSRI),  
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MAOs   dont use much anymore, diet restrictions, many side effects  
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tricyclic antidepressants   Rememron, Desyrel  
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SSRIs   best ones. Clexa, Lexapro, Prozac, Paxil, Zoloft  
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Others   Wllbutrin, Cymbalta, Effexor  
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mood swings from profound depression to extreme euphoria (mania)   bipolar disorder  
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stage 1   hypomania-good mood, helpful  
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stage 2   everything is wonderful, euphoric, do inappropriate things, dec hygiene and sleep.  
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stage 3   exaggerated mood swings, stooperous, confused, most unsafe  
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Ns interventons for bipolar   reduce stimuli, safety, calmness, set limits, meds  
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meds for bipolar   major tranquilizers, phenothiazines and lithium for the acute manic phases. can easily od on lithium  
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diffuse apprehension that is vague in nature and is associated with feeling of uncertainty and helplessness   anxiety disorder  
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affects ability to respond, more restless   moderate anxiety  
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unable to concentrate, cant complete task, HA, heart palpitations, insomnia, dread, horror   severe anxiety  
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unable to focus, hallucinations, delusions, withdrawn, confusion, feeling of terror, poor decisions   panic anxiety  
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anxiety disorder meds action   act at the limbic, thalamic and hypothalamic levels to produce calming  
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benzodiazepines   Xanax, Valium, Ativan  
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non-benzodiazepines   BuSpar, Atarax, Vistaril  
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others   tranquilizers  
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NS interventions for Anxiety disorders   any additional probs, what stressors, family support, accept, educate, coordinate care  
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assessment of suicide   elicit info, have a plan, hx of previous attempts, refuse to sign contract, live alone, ETOH, drugs, psychosis  
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Kubler-Ross stages of grieving   denial/isolation, anger, bargaining, depression, acceptance  
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worden and harper basic tasks of grief   accept the reality of the loss, experience the pain of grief, adjust to an environment in which the individual is missing, refocus emotional energy  
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risk factors for complicated grief   sudden death, marked dependence, lack of support system, loss of a child, perception of preventable death  
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NS interventions for grieving   support, assess how pt reacts to loss, understand the normal grief process, assess s/s of maladaptive response  
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maladaptive response to grieving   preserving, intense/distorted occurrences, mental/physical disorders, suicide  
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s/s of approaching death   extremities cool/discolored (molded), inc sleeping, hard to arouse, lose bowel/bladder control, poor swallow, restless, resp patterns change, can hear  
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if an autopsy is to be done...   leave all lines in.  
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criteria for hospice   terminal illness, death within one year, DNR status  
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how to deal w death at work   get help, know your limitations, leave work at work  
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ascending symmetric paralysis that usually affects cranial and pereipheral nervous system   Guillain-Barre Syndrome  
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Guillain-Barre Syndrome   loss of mylein, edema, inflam of affected nerves that stops or slows nerve impulses  
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Guillain-Barre Syndrome occurs after a...   upper respiratory or GI inf-resp failure most serious  
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Dx for GBS   CSF-elevated protein, EMG-nerve and muscle function in affected extremities  
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NS management of GBS   monitor ascending paralysis, resp functin, arterial blood gases, gag and swallowing reflexes, BP, HR and rythm  
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amyotrophic lateral sclerosis   Lou Gehrigs desease-rare, loss of motor neurons resulting in degeneration of motor neurons in the brainstem and spinal cord.  
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s/s of ALS   weakness upper extremities, dysarthria(difficulty articulating words), dysphagia, muscle twitching, pain, spasicity, drooling,const, esophageal reflux  
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Dx of ALS   nothing rule out others  
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px of ALS   death in 2-6yrs from resp inf  
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disease disseminated demyelination of the nerve fibers of the brain and spinal cord.   MS-multiple sclerosis-disease of the young to middle aged adults.  
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Risk factors for MS   women (2-3x more than men), 5x more in US, Canada and Europe than tropical, family relative w MS  
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MS is marked by   exacerbations/remissions. attack of mylin sheath then regrow of. slerotic plaque on nerves  
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dx of MS   hx, physical exam, clinical mans, lesions in brain from MRI  
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teaching for MS management   avoid triggers, balanced exercise, rest and nutrition, no hot/cold climate, exposure to inf, fatigue, follow drug regimen .  
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Chronic, progressive neurodegnerative disease   Parkinson’s Disease-Slowness in initiation/execution of movement, bradykinesia)Increased muscle tone.(rigidity)Tremor at rest. Gait disturbance.  
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Clinical Manifestations of parkinsons   gradual/insidious, r/l sided, 70 yo, more males  
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Beginning stages of parkinsons   Mild tremor (first sign) Slight limp,Decreased arm swing.  
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Later Stages of parkinsons   Shuffling Gait Propulsive gait with arms flexed,Loss of postural reflexes,Slight change in speech  
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Tremors of parkinsons   First sign,Pt. may only notice, Effect in handwritting,Aggravated by emotional stress,Tremors can involve:Diaphram,Tongue,Lips,Jaw  
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Rigidity of parkinsons   Second sign,Sustained muscle contractions,Muscle soreness,Feeling tired Achy Pain head, upper body, spine, and legs Slowness of movement  
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Bradykinesia of parkinsons   Loss of automatic movements(involuntary),Stooped posture,Masked face,Drooling,Shuffling gait  
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Diagnostic Studies of parkinsons   Not set test.Must have 2 of 3 symptoms present:Tremor,Rigidity, Bradykinesia; Ultimate confirmation is positive response to antiparkinsonian drugs  
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med for Parkinsons-levodopa/carbidopa (Sinemet)   Blocks the effects of the overactive cholinergic neurons. Treats bradydinesia, tremors, and rigidity  
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Anticholinergics   Artane, Cogentin, Akineton. Treats tremors  
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Antihistamine   Benadryl Treats tremors and rigidity  
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MOI   Eldepryl, Crebex, Azilect Treats bradydinesia, tremors, and rigidity  
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disease that destroys the brain   alzheimers-60 yo, genetic, b-amyloid plaques, neurofibrillary tangles,  
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s/s alzheimers   subtle memory loss, final stage unresponsive, incontinent  
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TIA   A transient ischemic attack episode of neurologic dysfunction caused by focal brain, spinal cord, or retinal ischemia, but w/o acute infarction of the brain.  
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A stroke, caused by an event that causes more permanent damage to a larger area of the brain resulting in more widespread damage and more permanent disability   CVA Cerebral Vascular Accident-caused by plaque  
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A stroke that a patient is able to fully recover from within a few weeks.   RIND Reversible Ischemic Neurological Deficit  
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Etiology of a stroke   A stroke occurs when there is ischemia (inadequate blood flow) to a part of the brain or hemorrhage into the brain that results in death of the brain cells  
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Risk factors for stroke   doubles each decade over 55yrs, more common in men, but more women die from it, African/ American, family history/heredity  
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modifiable Risk factors for stroke   Hypertension, Heart disease, Diabetes Mellitus, High cholesterol Smoking,Alcohol,Obesity,  
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Results from bleeding into the brain tissue itself or into the subarachnoid splace or ventricles.   Hemorrhagic stroke  
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Results from inadequate blood flow to the brain from partial or complete occlusion of an artery.   Ischemic Stroke  
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right hemisphere stroke   left-sided weakness, rash in judgement, inc falls, "left neglect", impulsivity, cant recognize body parts, probs w depth perception, cant see left in each eye(homonymous hemianopsia)  
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left hemisphere stroke   right-sided weakness, receptive/global aphasia, dec decision making, cant see right in each eye(homonymous hemianopsia), behavioral changes, apraxia  
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aphasia traditionally associated with neurological damage to Wernicke’s area in the brain. language area   Receptive aphasia  
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inability to properly move the muscles of the tongue and mouth to produce speech.   Expressive aphasia  
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impairment of both comprehension and expression of language. often mute or reduced to a few stereotyped words or sounds   Global aphasia  
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Inability to perform learned movements despite having desire and physical ability to perform them.   Apraxia  
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a motor speech disorder resulting from neurological injury of the motor component of the motor-speech system   Dysarthria  
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a paroxysmal, uncontrolled electrical discharge of neurons in the brain that interrupts normal function.   Seizure  
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a condition in which a person has spontaneously recurring seizures caused by a chronic underlying condition   Epilepsy  
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involve both sides of the brain, Loose consciousness for a few seconds to several minutes.   GENERALIZED  
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occurs in children, staring spell   absence seizures  
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loss of consciousness and falling to the ground   tonic clonic  
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Dx of seizure   EEG  
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peculiar sensation that precedes a seizure   aura  
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interventions for seizure   ensure airway, protect from injury, time seizure, say w pt until gone, pt on side, remove tight clothing  
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drugs for seizure   anti-convulsived- Ativan, Valium, Tegritol, Felbatol  
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drugs given IV for staticus epilepticus (rapid acting)   Ativan and Valium  
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Don’t take with grapefruit, report visual abnormalities, abrupt withdrawal after long term use may precipitate seizures.   Tegritol  
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Use is limited. It can cause aplastic anemia and liver toxicity. Mostly used to treat patients who are resistant to other drugs.   Felbatol  
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(long acting) anti-convulsive durgs   phenytoin, phenobarbital, ethosuximide, lamotrigine, topiramate  
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Dilantin   good oral hygene, tack 2-3 hours of antacids, use additional contraceptive  
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LEADING CAUSE OF BLINDNESS WORLDWIDE!   CATARACTS:An opacity within then lens  
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NON-SURGICAL tx for cateracts   changing prescriptions, using magnifiers, increasing the amount of light, adjusting his/her lifestyle, reassurance  
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surgical tx for cateracts   NPO 6-8hrs, antibiotic drops, dilation of eye, removal of lens nucleus and coretex, corticosteroid drops  
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post surgical instuctions   no stooping, lifting, coughing, call dr for any drainage, analgesic for pain  
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FOUR DISEASES MOST COMMONLY CAUSING BLINDNESS IN OLDER PERSON:   Cataracts, Viral infections, Chlamydial infections, Glaucoma  
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Clinical Manifestions of cateracts   decrease in vision (gradual), abnormal color perception, and glare.  
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related to consequences of increased intraocular pressure (IOP), optic nerve atrophy and peripheral visual field loss   GLAUCOMA-Second leading cause of permanent blindness and leading cause of blindness in African Americans.  
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the outflow of aqueous humor is decreased in the trabecular mesh work. Most common type.   Primary open-angle glaucoma (POAG)  
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due to reduction in the outflow of aqueous humor that results from angle closure.   Primary angle-closure glaucoma (PACG)  
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clinical mans of Primary angle-closure glaucoma (PACG)   N&V, sudden, excruciating pain in/around eye, halos around lights, blurred vision, ocular redness  
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clinical mans of Primary open-angle glaucoma (POAG)   Tunnel vision (PAOG)  
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Separation of the sensory retina and the underlying pigment epithelium, with fluid accumulation between the two layers   RETINAL DETACHMENT  
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CLINICAL MANIFESTATIONS OF RETINAL DETACHMENT   Photopsia (light flashes), floaters, “cobwebs”,“hairnets”, Ring in the field of vision  
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The area of visual field loss is the center of the visual field (the macula) because of damage to the retina.   MACULAR DEGENERATION  
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Conductive Hearing Loss   cerumen (earwax build up, ear infection or sometimes tumors) to external/middle ear.  
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Damage to the inner ear functions can cause hearing and equillibrium problems   Sensori-Neural Hearing Loss-Can be abnormal at birth or caused by damage.  
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Hearing loss due to aging.   Sound transmission is diminished Loss of high pitched sound hearing Tinnitus Increased wax/hair production blocks sound transmission Loss of elasticity of cartilage Reduced blood supply to ear Decline in ability to filter out unwanted sound  
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Care of hearing aid   When not worn Remove or disconnect battery, Clean Ear molds weekly, ear tip w toothpick  
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determines hearing range of patient in terms of decibels (sound intensity), and Hertz (pitch) for diagnosis of conductive and sensorineural hearing loss.   Audiometry  
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can cause inconsistent results to audiometry   tinnitus  
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assessing vertigo ask...   hearing changes, dizziness or spinning, ringing in the ears, food allergies, staining during BM, changes in sleep, communication and during exercise  
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The cause is unknown but it results in an excessive accumulation of endolymph causing pressure in the membranous labyrinth (inner ear)   Meniere’s Disease  
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s/s of meniere's disease   severe attacks with nausea, vomiting, sweating and pallor, Tinnitus, fluctuating sensori-neural hearing loss, aural fullness.  
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meds for meniere's disease(during attacks)   Benadryl, atropine and ativan can be used to decrease the abnormal sensation and lessen N/V Antiemetics- Compazine Antivertigo- Antivert administered IV, orally or rectally  
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meds for meniere's disease(between attacks)   Antihistamines, calcium channel blockers and a low sodium diet Valium, fentanyl with innovar may be used to reduce the vertigo  
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Surgical interventions for meniere's   Decompression of endolyphatic sac and shunting, vestibular nerve resection, Labyrinthectomy  
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Patient care got meniere'sde   safe room, dark room (NO TV, lights on low),Assist patient to comfortable position,Have emesis basin at bedside,Keep bedrails up and bed in locked/low position, Instruct patient to use call light for assistance when getting up,Monitor I/O  
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Hydrops diet for meniere's   Restriction of sodium, caffeine, nicotine, alcohol and foods with MSG fluid buildups in the inner ear, lessen attacks  
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irritation of sensory branch of 5th cranial nerve   TRIGEMINAL NEURALGIA-in women >40, triggered by touch or temp  
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s/s of trigeminal neuralgia   intermittent, sudden-onset severe facial pain; unilateral; + blinking, tearing, jerking; unpredictable; clusters possible  
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tx for TN   antiseizure meds-decrease in neuron firing to block movement.  
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Cause: unknown, affects the motor aspects of the 7th CN (facial nerve), ? Inflammation RT Herpes virus   BELL’S PALSY  
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s/s of bells palsy   unilateral, drooping of mouth, eye rolls up when closing eyelid; can have difficulty with eating; tearing, lasts <6mtns  
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tx for bells palsy   MEDS: steroids, analgesics, eye protection PT/OT  
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abnormal, excessive, involuntary movements   HUNTINGTON’S DISEASE -chorea  
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autosomal dominant genetic disorder (men = women) that results in changes in neurotransmitters and degeneration of basal ganglia (onset most common 30-50 years)   HUNTINGTON’S DISEASE -chorea  
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s/s of huntingtons disease   progressive, death within 15-20 yrs Abnormal muscle movement: voluntary & involuntary impacted Emotional disturbances & personality change Intellectual decline  
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prion disorder (infectious protein) CNS disease; effects dependent on where proteins are active. Can be genetic or infectious. 4 – 21 yr incubation.   CREUTZFELDT-JAKOB DISEASE  
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s/s of CREUTZFELDT-JAKOB DISEASE   RAPID PROGRESSION (wks to months) Behavior and personality change CNS: memory, vision loss, dysphagia, abnormal movement  
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dx of CREUTZFELDT-JAKOB DISEASE   autopsy  
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bilateral, “pressing-tightening”, most common, mild-moderate; no triggering event. Duration 30 min – 7 days   tension HA  
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- rare, repeated HA over weeks or months; “sharp/stabbing”; periods of remission; swelling around eyes; congestion or tearing   cluster  
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recurring, often unilateral, “throbbing”; family tendency Improves with sleep. Can have N & V, photophobia and be preceded by prodrome.   migraine  
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meds and tx of HA   reduce inflammation of cerebral blood vessels & vasoconstrict, : diary to determine triggers, treat symptoms ASAP, rest, reduce stress, R/O other brain pathology  
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brain tumor arises from tissues within brain   primary  
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brain tumor metastatic, most common   secondary  
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s/s of brain tumor   HA, N & V (inc. ICP), Behavior change,Muscle weakness, Seizures, cognitive problems(Memory,Personality & mood) Sensory losses,Hydrocephalus  
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tx of brain tumors   Surgery, Radiation, Chemo, Symptom management: shunts  
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alterations in thought, acute, shorterm   confusion  
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temporary acuteconfusion,inattention Disorganized thinking Altered LOC   delirium  
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Chronic confusion Multiple memory problems Gradual decline   dementia  
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Records electrical activity of the brain using scalp electrodes. Noninvasive.   EEG- Electroencephalography-Clean hair, Check meds, NO CAFFEINE 8 hrs before test,  
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Records electrical activity associated with innervation of skeletal muscle. Needle electrodes into muscle. Normal skeletal muscle has no electrical activity at rest   EMG - Electromyography- looks at speed of depolarization  
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Computer-assisted xray of multiple cross sections. Noninvasive.   CT-Computed tomography-Some studies will utilize IV contrast media.  
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Uses magnetic field & radio waves to create detailed image   MRI – Magnetic resonance imaging, Greater contrast & better images of soft tissue. Can do 3D.Noninvasive but claustrophobic, noisy, and metal risks, Can use contrast media to enhance images, Pt must BE VERY STILL.  
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Measures the metabolic activity of tissue at the cellular level. (CT & MRI evaluate structural changes).   PET – Positron emission tomography-Requires IV injection of radioactive tracer.Provides 3D images. Can measure blood flow, O2 use, & glucose metabolism in tissues. Earlier detection of problems, Pt must BE VERY STILL.  
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Evaluates vascular status by inserting catheter (groin) & threading catheter to carotid and contrast inserted.   Angiogram (cerebral arteriogram-Serial images to evaluate flow of the dye, INVASIVE and risk of allergy due to contrast media.Aftercare of puncture site to prevent/detect bleeding  
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EMERGENCY TX OF ISCHEMIC STROKE   Recombinant tissue plasminogen activator(tPA): fibrinolysis – reestablishes circulation  
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ESSENTIAL MEDS FOR CVA   ANTI-PLATELET, ANTICOAGULANTS, Antihypertensives, Statins,Diabetic meds  
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interaction with the environment that promotes homeostasis   ADAPTATION:  
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loss of equilibrium, physical and mental illness, loss of identity and self-esteem   MALADAPTATION  
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Compensation   making up for a deficiency by emphasizing a feature considered an asset.  
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Conversion   unconsciously repressing an anxiety producing emotion conflict and causing difficulty sleeping, loss of appetite etc....  
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Denial   avoiding emotional conflicts  
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Displacement   transferring anger over an interpersonal conflict to a malfunction VCR  
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Identification   acting like someone else  
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Dissociation   subjective sense of numbing and reduced awareness of ones surroundings  
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Regression   coping with stressor by going back to childhood  
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PSYCHOPHYSIOLOGICAL and should be tx like a real illness. Drs feel like these are in your head   HA, backache, arthritis, asthma, wheezing, diarrhea, anorexia, HB, Colds, infection, RA, GU, agina, hypertension, hypoglycemia, lupus  
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s/s of ETOH withdrawl   TREMORS, ANOREXIA, ANXIETY, RESTLESSNESS, INSOMNIA, ABDOMINAL PAIN, NAUSEA, DIAPHORESIS,  
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s/s of delirium tremmens from ETOH   DISORIENTATION, NIGHTMARES, VISUAL/AUDITORY HALLUCINATIONS, HYPERTENSION, DELUSIONS, CARDIAC PROBLEMS  
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TREATMENT FOR WITH- DRAWAL SYMPTOMS   SUPPORT, REASSURANCE, MONITOR, PROTECT FROM INJURY, one to one supervision  
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tx with meds for withdrawl   VALIUM -SERAX LIBRIUM -ANTIVAN  
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CHRONIC ALCOHOLISM can Cause Degeneration brain tissue Deficiency of Thiamine   Wernicke’s Encephalopathy-s/s=Ataxia, confusion, nystagmus. tx=IV Thiamine, Glucose replacement  
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Untreated Wernicke’s encephalopathy   Korsakoff’s Psychosis=s/s shorterm memory loss. tx=IV Thiamine, Glucose replacement  
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NICOTINE EFFECTS   TACHYCARDIA, VASOCONSTRICTS, dry ORAL MM, COUGH, LUNG DAMAGE, EMPHYSEMA, CANCER  
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Nicotine Replacement:   Nicotine gum, lozenges, patches, nasal spray, inhaler  
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Non-nicotine agents:   Zyban, Chantix, Aventyl, Catapres  
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abnormal malignant proliferation of plasma cells   MULTIPLE MYELOMA, middle to old, age 60 years, more common males and blacks  
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s/s of multiple myeloma   bone pain, anorexia, numbness, weakness, CHF, visual problems, headache, confusion,  
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DIAGNOSIS of MULTIPLE MYELOMA   x-ray, hematology=Bence-Jones protein--affects renal tubules increased calcium levels  
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TREATMENT MULTIPLE MYELOMA   chemotherapy, malphalan, prednisone anticalcium agents, localized radiation  
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Nursing Interventions MULTIPLE MYELOMA   high risk for injury, increase fluids, pain relief, prevent infection, rest and avoid fatigue, spinal brace  
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stage 1 lymphoma   single lymph node involvement  
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Stage two   two—two or more—one side of diaphragm  
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Stage three   lymph node involvement above and below the diaphragm  
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Stage four   involvement outside the diaphragm  
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Acute non-lymphcytic leukemia   resp. infections, anemia, bleeding mm, lymphoblasts inc.,hepatomegaly, splenomegaly, bone pain, CNS symptoms  
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Chronic myelogenous leukemia   fatigue, weakness, anorexia, wt. Loss, blastic phase, anemia, thrombocytopenia, fever,  
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etiology of leukemia   cause unknown, genetic predisposition, viral origins, radiation and chemical exposure, medication/drug related  
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Methods of transmission of HIV   intimate contact with body secretions, blood exposure, maternal-child transfer via placental exchange or breast milk  
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HIV Infection Control   Good hand washing, Do Not recap used needles, Use of gloves when handling body secretions, Disposal of contaminated materials  
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HIV acute inf stage   Flu-like syndrome, Acute retroviral syndrome, cold or flu, self-limiting  
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HIV early chronic inf stage   Asymptomatic disease, vague symptoms persist Median interval between infection and diagnosis 11 years  
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HIV Intermediate chronic inf stage   early symptoms become worse, thrush, shingles  
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Late chronic infection or AIDS   CDC criteria Opportunistic infections and cancers  
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normal function of T-calls   immune system function. norm>500. <200=aids. HIV uses T-cells to replicate  
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The amount of virus per milliliter of blood   VIRAL LOAD. The higher the viral load, the higher the risk of disease progression and/or death. low risk<10000. high risk>50000.  
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The CD4 count tells you the distance to the cliff. The viral load gives you the speed of the train.   Antiretrovirals (ART’s) are the “brakes” for this train.  
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HIV Encephalopathy   Brain invaded with virus, 60% of AIDS patients, neurological symptoms (impaired memory, slowness of speech, loss of function, loss of mental process)  
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HIV meds   Nucleoside Reverse Transcriptase Inhibitors, Protease inhibitors, Nonnumcleoside Reverse Transcriptase Inhibitors, Fusion Inhibitor  
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Nucleoside Reverse Transcriptase Inhibitors:   AZT -- malaise, HA, fever, nausea, vomiting, pancreatitis, neuropathy, oral lesions, rash, diarrhea, etc. Viread  
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Protease inhibitors   Crixivan, Viracept, Norvir  
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Nonnumcleoside Reverse Transcriptase Inhibitors   Rescriptor, Viramune  
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Fusion Inhibitor   Fuzeon  
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CHALLENGES OF HIV MANAGEMENT   Pill Burden, Food restrictions, Side Effects, Chronicity of the disease, Development of drug resistance, lipodystrophy Syndrome  
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HIV Dietary Recommendations   Lean meat, high protein, high calorie, Six small feedings, High in vitamin A, Vitamin C, Beta carotene, supplements to diet  
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HIV CDC Guidelines   Thoroughly cook meats, Avoid spoiled food, Reheat food well, Use bottled or distilled water, Do not leave food out of refridgerator more than 20 min. No unpasturized milk or cheese  
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Types of strokes   embolitic, thrombolitic, hemorragic  
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embolitic stroke   flows there, tissue dies, anticoags, heart holding blood (clots), activity onset, tpA.  
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thrombolitic stroke   grows there, antiplatelet meds r used, preceded by TIA, slow onset of symptoms, happens in AM, tpA  
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hemmoragic stroke   leak, have HA, from trauma, inc ICP, sudden onset, surgery to repair, get BP under control  
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#1 modifiable risk factor for stroke   HTN management, smoking  
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3 main reasons a person seizes   hypoxia, fatigue, alcohol, stress  
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period of fatique with reduced responsiveness post siezure   postictal  
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how to report a seizure   clear scene, time it, lay on ground, add o2, conscious or not??  
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absence seizure   just stare off  
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grand mall, tonic clonic   on the floor twitching  
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epilepsy   recurring seizures  
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classic signs of parkinsons   rigidity, brady kinesia, tremors, stoop, shuffles, pill rolling, lip smacking  
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myscemia gravis   auto immune, causes breathing difficulties, slowly dec, muscle probs, exhaust easily, removal of thymus gland, can paralyze resp muscles  
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MS   many plaques on nerves, remission/exacerbations, muscle, motor, urinary, sexual, emotional, cognitive impairments  
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ALS (amyotrophic lateral sclerosis)   20-40 yo, no confusion, cant swallow, breath or eat.  
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