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NUR 141 EXAM 2

Quiz yourself by thinking what should be in each of the black spaces below before clicking on it to display the answer.
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Question
Answer
LPNs care for persons who are...   well, ill but stable, injured, convalescent, handicapped  
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LPNs work under...   own license and supervision, can not delegate tasks to unlicensed personnel  
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most facilities LPN does not...   start IVs, give IVP or IBPB meds, work with central lines, take verbal orders  
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LPN duties   caths, dressings, tube feedings, wound irrigation, med adm (not IV), pre-post mortom care  
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LPNs can direct, assign and supervise aides in extended care facilities    
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PT does   motor  
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OT does   upper extremities and ADLs  
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ST does   swallowing, speaking  
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"study learning principles"    
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domains of learning   cognitive, affective, psychomotor  
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cognitive   intellectual-understanding of risks and s/s of low blood sugar  
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affective   emotions, attitudes, values= values blood sugar control and health promotion  
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psychomotor   acquiring skills-can use glucometer to check blood sugar level  
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pain is the physical sensation, suffering is the emotional response to pain    
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pain lasting > 3 months   chronic, vs can b normal  
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sudden pain   acute, inc in BP and vs  
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nocioceptive pain   caused by damage to tissue, heals quicker  
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nocioceptive somatic pain   superficial (sharp, burning, prickly), deep (aching, throbbing)  
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nocioceptive visceral   internal organs (referred pain) cramping, pressure  
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nocioceptive pain responds to   opioid and non opioid meds  
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neuropathic pain   caused by damage to nerves or CNS (numbing, burning, stabbing)  
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causes for neuropathic pain   trauma, inflam, metabolic disease, ETOH, inf. Not well controlled by opioids alone.  
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incident pain   is ambulation, change in posiiton  
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pattern of pain   onset, duration, breakthrough pain  
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non opioids   for mild/mod pain, nonsalicylate, salicylate, NSAIDS  
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nonsalicylate   acetminophen-analgesic, antipyretic, hepatic toxicity  
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salicylate   ASA-analgesic, antipyretic, antiplatelet, antinflamm  
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NSAIDS   losts of side effects  
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characteristics of nonopioids   analgesic ceiling, no tolerance/dependence to, OTC, opioid sparing  
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opioids   bind to CNS receptors for mod/severe pain, no ceiling  
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opioids to avoid   propoxyphene (seizures), meperedine (neurotoxicity)  
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side effects of opioids   contstipation, N/V, sedation, resp depression, itching, urinary retention, confusion  
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adjuvants for opioids   corticosteroids, antidepressants, antizeizure, GABA receptor agonists, alpha adrnergic agonists, local anesthetics, cannabinoids  
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corticosteroids   dec edema/inflam. SE=dose/duration dependent  
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antidepressants   inhibit reuptake of serotonin &/or norepinepherine higher levels in synapse. inhibits pain impulses. good for neuropathic pain  
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antiseizure   affect peripheral nerves &CNS, good for neuropathic pain, gabapentin (neurotin), pregabalin (Lyrica)  
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GABA receptor agonists   muscle spasms  
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marrinol   pot in pill form  
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a chart of conversion for other meds that do same thing   equianalgesic dosing  
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PCA pumps are a high risk for resp depression    
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Tolerance   chronic exposure dec effect of drug. rotate  
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physical dependence   expected, experience withdrawl s/s when blood level drops. Taper off.  
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addiction   powerful drive to take substance for reasons other than the therapeutic purpose. Seeking SE instead of therapeutic effects  
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cancer pain   acute/chronic, r/t desease/tx,  
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palliative care   focus on pain control in incurable conditions  
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hospice   end of life care, focus on comfort/quality of life vs quantity  
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ethical concern with hospice   hasten death  
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nutrition in >50 yo   women=1920, men=2300, need more vit D, B12, protien, fat, CHO, fluids  
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dehydration in tne elderly   purposely avoid fluids, loose sense of thirst, forget to drink, can get fluids on their own  
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lack of nutrition in elderly   poor fitting teeth, taste/swallowing impaired, vision, constipation  
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white patches in mouth   leukoplakia  
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red velvety patches   erythroplackia  
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precursor to esphageal cancer   pharynxesphogitis from reflux  
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cancer that is mastatic at time of dx   esophageal cancer, to brain, lungs; removal of esophagus, stints, survial<20%,  
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after removal of esophagus   NG tube (do not touch), I/O, VS, pain, mobility, DB, cough  
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sign of fever, severe pain and NG contents change after esophagus removal   a leak  
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pts with inc temp after surgery   ateloactisis=DB and cough  
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gall stone blocking duct can cause   pancreantitis  
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lab the is relased with injury   LDL  
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lab the inc w other organ dmage   AST  
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lab that is liver specific   ALT  
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all fluids of body have billirubin except   CSF  
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hep A & E   trans w fecal oral  
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hep B   trans w body fluid, blood  
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hep C   trans blood  
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hep D   only have w B  
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drug of choice for liver disease   aldactone  
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what are some contributing causes for cirrhosis of liver   ETOH, hep C 7 B, chronic inflam, billary cirosis, obstructions  
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what labs are for cirrhosis of liver   ALT, LDH, bilirubin, albumin, amonia, PT  
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assessment data for cirrhosis liver   pain, fatique, brusing, acetises, LOC, what have been exposed to  
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what orders with sirrhosisi of liver   daily st, liver biopsy, parancitisis,, vit K, folic acid, thymiane, DTs, diuretics  
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wat liver does   stores carbs, protein, 1st pass, responsible for clotting factors, detoxes at 1st pass  
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must have PT prior to liver biopsy   want good clotting: after biopsy ly on R side  
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break down of protein   = amonia  
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jaundice   bilirubin is x3, clay stools, tea urine, skin itches, anorexic, fatique  
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ETOH need   thyamine and folic acid  
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common hepatotoxins   isoniazid, acetaminophen, thiazides, tetracycline, methotrexate, ETOH, poisons, heavy metals  
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pt adm w RUQ pain N/V, sudden onset, ate shell fis. wat tests dr order   hepatits profile (hep A)  
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how is HBV trans   blood, body fluid  
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how is HCV trans   blood (transfission, neeedles)_  
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how protect yourself from transmission   hep B vac  
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hep B more than C can cause   liver cancer  
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causes of pancreatitis   obstruction, EToh, gall stones, billiary tract disease  
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assessment data for pancreatitis   pain, N/V, fever, HTN, vs, left upper gastric pain  
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lab teszts for pancreatitis   amalyse, lipase, BG  
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gall stone blocking duct can cause   pancreantitis  
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lab the is relased with injury   LDL  
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lab the inc w other organ dmage   AST  
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lab that is liver specific   ALT  
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all fluids of body have billirubin except   CSF  
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hep A & E   trans w fecal oral  
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hep B   trans w body fluid, blood  
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hep C   trans blood  
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hep D   only have w B  
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drug of choice for liver disease   aldactone  
🗑
what are some contributing causes for cirrhosis of liver   ETOH, hep C 7 B, chronic inflam, billary cirosis, obstructions  
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what labs are for cirrhosis of liver   ALT, LDH, bilirubin, albumin, amonia, PT  
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assessment data for cirrhosis liver   pain, fatique, brusing, acetises, LOC, what have been exposed to  
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what orders with sirrhosisi of liver   daily st, liver biopsy, parancitisis,, vit K, folic acid, thymiane, DTs, diuretics  
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wat liver does   stores carbs, protein, 1st pass, responsible for clotting factors, detoxes at 1st pass  
🗑
must have PT prior to liver biopsy   want good clotting: after biopsy ly on R side  
🗑
break down of protein   = amonia  
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jaundice   bilirubin is x3, clay stools, tea urine, skin itches, anorexic, fatique  
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ETOH need   thyamine and folic acid  
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common hepatotoxins   isoniazid, acetaminophen, thiazides, tetracycline, methotrexate, ETOH, poisons, heavy metals  
🗑
pt adm w RUQ pain N/V, sudden onset, ate shell fis. wat tests dr order   hepatits profile (hep A)  
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how is HBV trans   blood, body fluid  
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how is HCV trans   blood (transfission, neeedles)_  
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how protect yourself from transmission   hep B vac  
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hep B more than C can cause   liver cancer  
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causes of pancreatitis   obstruction, EToh, gall stones, billiary tract disease  
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assessment data for pancreatitis   pain, N/V, fever, HTN, vs, left upper gastric pain  
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lab teszts for pancreatitis   amalyse, lipase, BG  
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what DO for pancreatitis   morphine, dilaudid, NPO, NG tube, previcid, Zofran  
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pancreas   endo/exocrine secretes insulin, glucagon, digestive enzymes  
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pancreatitis tx   NPO (cant excrete lipase,amalace until gone), NGT, morphine, fluid, low fat, bland, pancreatic enzymes, cant drink again or will b back in hosp  
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tests for a DM pt   DM diet, insulin, fluids, A1c  
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pts on beta blockers are tachy    
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repeated hypoglycemia causes memory loss    
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diabetic ketoacidosis only occurs in   type 1  
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somogyi effect   caused by too much insulin, tto much insulin at night  
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dawn phenomenon   hypergycemia d/t coungterregulatory hormones, hormone imbalance  
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DM and renal failure go together    
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DM causes dec in motility   give Regalan 1/2 hr before meals to inc motility  
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A1C   4-6 non diabetic  
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if hypoglycemic give..   no candy bars (too much fat, delays absorption)  
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if hypoglycemic give...   15-20gm x15min, 6 oj, 4 cola, 8 milk, 3 glucose pills, 5 lifesavers, 1 dry toast, 1/2 c ice cream  
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what drug you cant give with a dye study   metforman  
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medulla adrenal gland (sympathetic NS)   epinephrine, norepinephrine  
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Cortex adrenal gland   mineralcorticoids, glucocorticoids, androgens,  
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too many mineralcorticoids, glucocorticoids, androgens =   cushings-pituitary (inc cortisol)  
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too little mineralcorticoids, glucocorticoids, androgens =   addisons-adrenal (dec cortisol)  
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cushings syndrome is caused from   too much steroids (prenidone) brusing, muscle waste, fatty, poor wound healing, moon face, moods up/down, anmenorea, impotent, gynomastia  
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side effects of steroid therapy   osteoparosis, fat to belly, cataracts, moon face, muscle atrophy, DM. diet dec Na, inc Ca  
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goup of disorders caused by impared DNA synthesis   Megaloblastic Anemia  
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Megaloblastic anemia causes   defective RBC maturation, large RBCs, the RBC have fragile cell membranes which are easily destroyed  
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Instrinsic factor is secreated by   gastric mucosa  
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Need IF to absorb   cobalamin (B12)  
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Absence of intrinsic factor   pernicious anemia-onset after 40yo, large RBCs, dec B12 levels, SERUM FOLATE IS NORMAL,  
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parietal cell function test with radioactive cobalamin adm   shillings test  
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Upper GI and biopsy of gastric mucosa test for pernicious anemia can cause   inc risk of gastric cancer  
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assessment finding for pernicious anemia   sore tongue (red, beefy/shiny), N/V, abd pain, weakness,  
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drug therapy for pernicious anemia   dietary B12 doesnt work...use parenteral, intranasal, sublingual.  
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increased production of RBCs and impaired circulation dt inc blood viscosity   Polycythemia  
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chronic chromosomal mutation with inc RBCs, WBCs and platelets   Polycythemia Vera  
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Hoypozia driven, inc O2 demand   secondary polycythemia  
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ns assessment for polycythemia   Plethora (ruddy complexion), intermittent claudication (pain in butt, back of legs at rest)  
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managemnt of primary polycythemia vera   phlebotomy, monitor i/o, RBC, WBC, platelets, active/passive leg exercises, amb when ordered.  
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amb would not be ordered due to   clots  
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managment of secondary polycythemia   control chronic pulmonary disease, no smoking, high altitudes  
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prehypertension   SBP 120-139, DBP 80-89  
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HTN inc risks for   MI, hear failure, stroke, renal disease, silent killer, unknown cause  
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target organ diseases for HTN   heart, brain, pere\ipheral vascular disease, kidney, eyes  
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HTN heart disease   CAD, L ventricular hypertrophy, heart failure  
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HTN cerebrovascular disease   stroke, 4 times more likely to have one  
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HTN peripheral vascular disease   speeds up atherosclerosis in peripheral blood vessels  
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atherosclerosis can lead to   PVD, aortic aneurysm, aortic dissection intermittent claudication  
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death of nephrons from narrowing of the arteries and arterioles   nephrosclerosis, leading cause of ESRD  
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labs for nephrosclerosis   albumin, proteinuria, hematuria, elevated BUN and creatinine  
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s/s HTN   fatigue, reduced activity tolerance, dizziness, palpitations, angina dyspnea  
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ns mangement of HTN   inc fiber, dec fat, inc water, dec salt, dec wt  
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fall in bp when a person stands   orthostatic hypotension  
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3 factors of orthostatic hypotension   cardiac func, vascular status, intravascular blood and fluid volumes  
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orthostatic vs   bp from lying to sitting to standing q 2 min. SBP inc by 20, DBP inc by 10 and HR inc by 20  
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2 main actions of antihypertensives   dec CO or reduce systemic vascular resistance (the squeeze of vessels  
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pts on thiazide should eat foods rch in   potassium  
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pt taking thiazides should avoid what OTC   NSAIDs-dec diuretic effects  
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monitor what for loop and K+ sparing diuretics   orthostatic hpyotension, hypokalemia, i/o, wt  
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pt taking aldoctone (aldosterone receptor blockers) should not eat   fruit w potassium  
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avoid what with catapres (adrenergic inhibitors)   any sedative  
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pt on lopressor (beta blocker) requires monitoring of   BP and P  
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pt on Apresoline (direct vasodilators)and urine is brown and 1+ edema on ankle what going on   rapidly progressing glomerilonephritis  
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side effect of Vasotec (ACE inhibitor)   dry hacking cough, in the lungs causes cough. angioedema  
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swelling of face and tongue   angioedema, allergic reaction from ACE-inhibitors  
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pt on Cozzar (angiotension II receptor blocker) still has high BP   med takes days to weeks to work  
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elevated BP w a specific cause that can b identifies and corrected   secondary HTN  
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causes of secondary HTN   MAO inhibitors w tyramine foods (wine, cheese, cured meats), estrogen replacement therapy, the pill, NSAIDS  
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clinical finding of secondary HTN   hypokalemia, abd buit, variable BPs w tacy, sweating, tremors, fam hx of renal disease  
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CAD   fatty deposits on aterial wall that cause inflam "hrdening of the arteries"  
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dec risk for CAD   diet, lifestyle, exercise, no smoking, no ETOH  
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causes of CAD   cholesterol>240, SBP>160, >1 pack a day smoker  
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CAD med that causes Rhabdomyolosis   statins  
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rhabdomyolosis   breakdown of skeletal muscle, products get caught in kidneys and cause renal failure  
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common side effects of niacin   itching, hot flashes  
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fibric acid derivative increase the effects of   coumadin  
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bile acid sequestrants interfere with absorption of   digozin, thiazide diuretics, warfarin, penicillins  
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cholesterol absorption inhibitors should not be use with what condition   liver impairment  
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natural lipid lowering agents   niacin, garlic, omega-3 fatty, psyllium, soy, phytosterols (nuts, seeds), red yeast rice  
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leading cause of death   CAD  
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chest pain   angina pectoris-caused by dec supply of oxygen to heart muscle  
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noncardiac factors that dec O2 to myocardial cells   anemia, asthma, COPD  
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noncardiac factors that inc O2 demand of myocardial cells   anxiety  
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cardiac factos that dec O2 to myocardial cells   coronary attery spasm, thrombosis, dysrhymias, heart failure  
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cardiac factors that inc O2 demand of myocardial cells   aortic stenosis, cardiomyopathy, tachy  
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type of Angina   Chronic Stable Angina-secondary to CAD, tx w rest and nitrate  
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type of angina   Prinzmetals angina-occurs at rest due to coronary artery spasm. tx w Ca Channel blockers  
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Chronic stable angina   pain at rest in neck, jaw, shoulders, arms, between shoulders lasts 5-15min.  
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manifestations of CAD tend to occur during   early morning after wakening  
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questions for chest pain   Precipitating evens, Quality of pain, Radiation of pain, Severity of pain, Timing  
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tx for chronic stable angina   Antiplatelet/anticoagulant, ACE, antiotension receptor blocker; B-adreergic blocker (BP); Cigarette smoling, calcium channel blockers, cholesterol; Diet, DM; Exercise, education; Flu vacc  
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unstable angina   at rest, worsening pattern,  
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nitro   pain should be relieved in 3 min and last for 30-60min. can give another 5 mins apart x3.  
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nitro can cause   dec BP and HA due to vasodilation  
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teaching of nitro   accessible at all times, keep in dark bottle, replace q 6 mths, under tongue, feel tingle, no ED drugs, change positions slowly, HA  
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sustained ischemia causing myocardial death   Myocardial infarction (MI) by thrombus, pain not relieved  
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cardiac disorder that impairs the ability of the ventricle to fill with or eject blood   Heart failure  
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why HF instead of CHF   not all pts have volume overload at the time of intial or subsequent evaluations  
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s/s HF   ventricular dysfunction, reduced exercise tolerance, diminished quality of life, shortened life expectancy  
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most common reason for hospitalization adm in adults >65yo   HF  
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two most common causes of L sided failure   systolic failure, distolic failure  
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systolic failure   most common, L ventricle fails to contract/pump dec ejection fraction ends up w pulmonary congestion. listen to lungs  
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diastolic failure   ventricls stiffen, dec filling, selling feet, some lung congestion  
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sign of L sided heart failure   no urinary output  
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right sided failure   R ventricle fails pump enough blood to meet body needs, ventricle cant accept all the blood returning to the heart.  
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s/s of R sided failure   backs up into veins/capillaries, edema, dry lungs, generalized edema  
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Heart failure assessment   Fatigue, Activities limited, Chest congestion/cough, Edema, SOB  
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Heart failure assessment   dec blood flow to extremities less hair, chest pain, dusky cool skin, edema, tachycardia, SOB  
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s/s R sided HF   Jugular vien distention, pedal edema, wt gain, fatigue, dependent bilat edema, URQ pain  
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s/s L sided HF   crackles, changes in mental status, confusion, restlessness, weak, dyspnea, shallow resp, SOB in recumbant, cry hacking cough, nocturia  
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1+ edema   mild pitting, slight indentation, no perceptible swelling of the leg  
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2+ edema   moderate pitting, indentation subsides rapidly  
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3+ edema   deep pitting, indentation remains for a short time, leg looks swollen  
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4+ edema   very deep pitting, indentation last a long time, leg is very swollen  
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digoxin (Lanoxin)   dec and strengthens heart beat, check apical HR (>60) prior to adm  
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digoxin toxicity   yellow vision, halos. anecdote=digibindIV  
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tx for heart failure   diuretics (K+ waisting loops) watch electorlytes (K+, Na), morphine for pain causes vasodilation of lungs to inc capacity  
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major cause of PAD   athrerosclerosis  
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drug for intermittent claudication   trental, pletal  
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tx for PAD   Trental, Pletal, walking, inspect feet, exercise till discomfort than rest start again  
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surg tx for PAD   percutaneous transluminal ballon angioplasty, atherectomy (remove plaque), peripheral artery bypass w graft, endarterectomy (remove plaqe from artery), amputation  
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thrombosis and fibrosis occur inside the vessel causing exchemia   Buerger's Disease  
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Buerger's Disease   obstruction of small arteries, loss of phlanges, stop smoking  
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episodic vasospastic disorder fo small cutaneous arteries of fingers and toes   Raynaud's phenomenon, 15-20 yo women.  
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second leading cause of cancer in women   breast cancer, 1st is lung  
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increased exposure to estrogen causes   breast cancer  
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breast cancer tumors are   hard, circular, dimpling, immovable, non tender, large  
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risk for endometrial cancer   having 1 or more 1st relative w oravrian cancer, too much extrogen  
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beta blockers cause   ED  
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S/S prostate cancer   painless enlargement, heaviness, dragging sensatoin  
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most dangerous rooms for elderly   kitchen, bathroom, bedroom  
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medication risks   absorption, distribution, biotransformation, excretion  
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medication errors   polyphrmacy, living alone, inadequate education, inadequate finances, medication regime understanding. 60% take meds wrong  
🗑
drug toxicity   earliest sign.. mental confusion  
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4 types of extrapyramidal reactions. Early onset....   acute dystonia (severe spasm), Parkinsonism (mask face, drooling, tremors, shuffling, stoop, pill rolling), Akethisia (pacing, squirming)  
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late onset   tardive dyskinesia (tongue, face, swallowing dec) not reversiable  
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insulin reaction   speech disorder, slurring, confusion, disorientation. elderly w beta blockers have hpyoglycemic unawareness  
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fluid filled sac that cushions a jt. can become inflamed   bursa-bursitis  
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attaches muscle to bone   tendon-tendonitis is from overuse  
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inflammation of muscle   myositis  
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RICE   rest, ice, compression, elevation. Ice no more than 20min  
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tx for swelling and effusion   aspiration and rice  
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infection in bone   osteomyelitis by staff aureus  
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most common osteomyelitis   indirect via blood stream from elsewhere in body  
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direct osteomyelitis   contamination from surgical site, fx, DM foot ucler  
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bone deterioration   osteoporosis-deficit in bone replacement  
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ostoporosis   bone density test, post menopausal, kyphosis, fx, pain, Ca+vitD, no salt, caffeine, WB exercise, fosomax, evista  
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rehabilitation begins   the first day  
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loss of motor function   paralysis  
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lesser degree of paralysis   paresis  
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paralysis-legs lower body   paraplegic  
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paralysis all 4 limbs   tetraplegic  
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brain thru spinal cord   upper motor neuron  
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spinal cord to muscle   lower motor neuron  
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sensory impulse to corn, motor neuron, muscle or gland   motor reflex arc  
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sacral reflex arcs intact   sacral sparing  
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proprioception   ur understanding where ur limbs are  
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cervical injury   C1-2=freq fatal (vent dependant; C3-4 vent dependent, diaphragm weak; C5: if can shrug shoulders, no vent, only diaphragm to breath, no intercostals, has shoulder/elbow func, most common cervical injury  
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thoracic/lumbar:paraplegic   T1-6 confined to WC; T6down=parapareiss, WK, braces, they shake; above S2=reflex present for bowel/bladder emptying, S2-4, reflex arc destroyed  
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impaired resp-vent C1-4   frequent pulmonary inf, assisted cough, breathing exercises, possible phrenic nerve stim  
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impaired O2 transport:immobility   TEDS to prevent pooling, no IMs below injury, ROM q 8*  
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Bladder problems above S2 (UMN)Reflexic   bladder Hypertonic, <150CC capacity, reflex voiding, urgency, frequency, stroke trigger areas  
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Badder problems S2-4 Areflexic   reflex destroyed, hypotonic,>500cc residual(cant empty), overflow voiding, crede or St cath  
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Bowel problems above S2 (UMN) Reflexic   spastic bowel, dulcolax suppos, digital stim  
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Bowel problems S2-4 (LMN) Areflexic   Atonic bowel, enemas, digital removal,  
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sexuality   above C5 and up, women use birth control (not pill)  
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exaggerated ANS response to stimuli   Autonomic dysreflexia  
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autonomic dysreflexia   pt w lesion above T6 (often due to full bladder or bowel)can last 6yrs after injury.  
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s/s autonomic dysreflexia   generalized vasoconstriction first then sudden severe HTN. HA, brady, inc BP, dec P  
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autonomic dysreflexia can result in   CVA, SAH, seizure and death  
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Ns intervention for autonomic dysreflexia   fast action..raise HOB (90*), BP,check for full bladder, bowel, UTI, kinked cath, use anesthetic lubricant for digital stim and cath,  
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device that prevents neck flexion   halo traction  
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musculo-skeletal changes in the elderly primary   dec muscle mas/strength, jt mobility, shoulder width, disc space mineral loss in bones, fat to belly  
🗑
musculo-skeletal changes in the elderly secondary   unsteady gait, osteoporosis, fax, curvature of spine, osteo arthritis  
🗑
inflammation of freely moveable jts, bilateral, symmetric, poy-arthritis in all extremities   Rheumatoid arthritis-autoimmune disease, genetic, rheumatoid factor present  
🗑
systemic symptoms of RA   fever, wt loss, fatiue, feneralized aching, early morning stiffness, tachy, weakness, anemia  
🗑
local s/s of RA   jts stiff, pain, edema, prosimal finger jts enlarged, swan neck deformity, nodules on extensor surfaces  
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who are more susceptible for getting RA   Pima and Chipawa tribe women  
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meds for RA   DMARDS (Methotrexate #1), NSAIDS, asprin (enteric coated 4-6gm), Anit inflams (Prednisone),  
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Ns interventions for RA   pain, inflam, ROM warm H2O therapy, own ADLs, assistive devices, splints for extension not flexion  
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removal of synovia, reduces inflam and helps maintain jt func   synovectomy  
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jt replace ment   arthroplasty  
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cut bone to change alignment or correct deformity   osteotomy  
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fusion of jt   arthrodesis  
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chronic inflam autoimmune disease (vascular and connective tissue)   systemic lupus erythematosus-multi system disease, reversible form, poly arthritis, red butterfly rash  
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what caused lupus   procame  
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dx of lupus   +LE cell prep and ANA, Anti-DNA, Anti-Smith (most dx)  
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inflam disease of voluntary muscle   polymyositis-degeneration of muscle fibers. tx high dose steroids  
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s/s of polymyositis   muscle weakness, difficulty moving/swallowing, symmetrical, includes polyarthritis, arthralgias and Raynauds  
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polymyositis with rash,   dermatomyositis-more prone to malignancy with in 5 yrs. tx high dose steroids  
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disease that affects jts and surrounding tissue, loss of srticular cartilage in WB jts   osteoarthritis (DJD) from over use  
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s/s of DJD   pain, stiffness in jts after rest, crepitation, Bouchard(PIP) and Heberden(DIP)nodes  
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tx for DJD   tylenol (4gm),  
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metabolic disorder, too much uric acid   gout-no meat, alcohol  
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3 causes of gout   poor renal excretion of uric acid, metabolism of purine, inc in uric acid production,  
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s/s of gout   crystals in tissues cause pain (jts/kidneys), red swollen jts,  
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swollen red great toe   podagra  
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meds for gout   culchasine, fluids, uloric, cozar and zyloprim together  
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tx for gout   meds, ice, elevate, uric acid levels, foot craddle  
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benign bone tumor   osteoma  
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malignant bone tumor   osteosarcoma-young males 10-25yo and fatle, pain swelling in knee  
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dx of bone tumors   ALK phos and Ca will be inc, CAT scan, PET, MRI, biopsy  
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complications of amputations   hemorrhage, edema, contractures, inf  
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care for amputations   no elevations, chair rest x1hr, prone 3-4xdaily, monitor stump for bleeding, body image  
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