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Med Surge 3 Final

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Question
Answer
Central nervous system   Brain and Spinal cord  
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Peripheral nervous system   Peripheral nerves and Cranial nerves  
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Autonomic nervous system   Sympathetic (adrenergic) and Parasympathetic (cholinergic)  
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Frontal lobe   socialization, expressive speech (Broca's area)  
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Parietal lobe   Sensory interpretation, Proprioception, and Body image  
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Temporal lobe   Receptive language (Wernicke’s area)  
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Hippocampus   Memory - deep in the temporal lobe  
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Occipital lobe   Interpretation of vision  
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Brainstem   Vital life center  
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Cerebellum   "little brain" (Coordination, Fine motor movement, and Balance)  
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Vertebral column   33 vertebrae  
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First pair of spinal nerves   leaves the cord above C1  
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Sympathetic nervous system   Fight of flight (Dilated pupils) - general rule: speed up except GI tract  
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Parasympathetic nervous system   general rule: slow down except GI tract  
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CN 1   Olfactory (S)  
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CN 2   Optic (S)  
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CN 3   Oculomotor (M)  
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CN 4   Trochlear (M)  
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CN 5   Trigeminal (B)  
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CN 6   Abducens (M)  
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CN 7   Facial (B)  
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CN 8   Acoustic (S)  
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CN 9   Glossopharyngeal (B)  
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CN 10   Vagus (B)  
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CN 11   Spinal accessory (M)  
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CN 12   Hypoglossal (M)  
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CN 3, 4, and 6   Tested together for eye movement  
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Miotic   constricted pupils  
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Mydriatic   dilated pupils  
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Altered LOC   A decrease in response to surroundings  
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A&Ox4   person, place, time, situation  
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CSF in the brain   75 mL  
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3 things that work to keep balance   water, blood, and CSF  
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Epidural hematoma   between the skull and dura  
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Subdural hematoma   acute, subacute, or chronic (NOT acute, life-threatening arterial bleeds)  
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ICP normal range   10-20  
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The earliest sign of serious impairment of brain circulation related to ICP   throbbing headache  
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Mannitol   osmotic diuretic (pulls fluid from different places, VERY important to get electrolytes); it does not cross an intact blood brain barrier  
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CPP   Central Perfusion Pressure (needs to be >70 to have adequate O2)  
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CBF   Cerebral blood flow  
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CBV   Cerebral blood volume (75 mL at all times)  
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CPP   MAP-ICP (70 or > = good CBF; <50 = Irreversible brain damage)  
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IICP   Increased myocardial contractility  
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Herniation   the most common, compression of brain tissue  
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Cushing's reflex/response/triad   Increased BP, Decreased HR, Decreased RR  
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Widening pulse pressure   assessment finding of IICP  
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Most serious complication of IICP   brain herniation  
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IICP Diagnoses   History and physical, CT (if contrast is used force fluids after), and Lumbar Puncture  
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Relieve IICP   avoid trendelenberg position  
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Reduce metabolic demand of IICP   avoid shivering (it increases ICP)  
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Generalized seizures   whole brain Convulsive = Tonic (rigid muscle contraction - brief, sudden contraction), Tonic/Clonic (alternating contraction and relaxation) - most common Nonconvulsive = rigid and stiff (dead in the bed)  
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Epilepsy   reocurring seizures  
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EEG   electrical activity with or without stimulus that encompasses the whole head  
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CT for seizures   ordered with an EEG to confirm diagnosis of seizures  
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Dilantin (Phenytoin)   Treatment effect not immediate because it needs to get a therapeutic level - ALWAYS give Dilantin in NS - Stop tube feedings and hour before and after giving Dilantin and tube rinsed/flushed with water after SE = gingival hyperplasia  
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Celebrex (Fosphenytoin)   Short term management  
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Dilantin and Celebrex therapeutic level   10-20  
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New admit for seizures   most important plan of care is to place a sign above the bed  
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Post-ictal phase of a seizure   let them rest, without rest they will seize again  
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HA categories   Stress/tension, HTN, and vascular  
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Most effective way to diagnose a functional headache   get a thorough history  
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Migraine auras   Sensory - pain is NOT an aura  
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Migraine triggers   Stress, Caffeine, foods high in tyramine and nitrates, smoke, and bright lights. Avoid smoking, aged cheese, and red wine.  
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Preventative migraine medications   taken every day - Inderal (beta blockers)  
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Sumatriptan (Imitrex)   Abortive drug - take it to stop the entire migraine. Can only take twice a day and it has to have an hour in between doses  
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Antiemetics for headaches   Zofran when they are nauseous, once they have vomited they need phenergan (must be diluted in at least 10 mL NS and given slowly - at least over 5 minutes)  
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Patient with the "worst headache of their life"   tell them to go to the ED immediately  
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Malignancies   May be primary, secondary, or rapid growing  
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Gliomas   arise from neuroglia  
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Meningioma   arise from the meninges  
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Neuromas   arise from nerves  
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Pituitary Adenomas   Responsible for hormone release (Prolactin, HGH, ACTH)  
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Angiomas   Speculated that at least 40% of cerebral hemorrhage in people under 40 is related to angiomas  
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Grade 1 Tumor   The tumor grows slowly, has cells that look similar to normal cells, and rarely spreads. May be removed surgically. Most likely a menengioma  
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Grade 2 Tumor   The tumor grows slowly, but may spread into nearby tissue and may become a higher grade tumor. Most likely a glioma  
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Grade 3 Tumor   The tumor grows quickly, is likely to spread into nearby tissue and the tumor cells look very different from normal cells. Most likely an oligdendroglia  
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Grade 4 Tumor   Grows very aggressively Has cells that look very different from normal cells, and is difficult to treat successfully. Most likely an oligdendroglia  
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Embolic stroke   most serious because it is acute. Usually results in atrial fibrillation.  
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TIA   <24 hr in duration. One sided weakness (no bilateral paresthesias)  
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Left brain CVA   o Right hemiplegia Expressive, receptive, or global aphasia Cognitive impairment Slow cautious behavior Right visual field defects  
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Right brain CVA   Left hemiplegia Spatial-perceptual issues Hemi-neglect (Denial – neglect of the affected side (don’t realize that it is even still there)) Distractibility Poor judgment Left visual field defects  
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Patient who is right handed and experiences a left sided stroke   right hemiparesis and speech is affected  
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Anterior Cerebral Artery   Supplies the medial surface of the frontal lobe and the parietal lobes  
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Vertebral arteries   Supply cerebellum, brainstem, spinal cord, medial and inferior aspects of temporal lobes. And the occipital lobes  
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Middle Cerebral Artery   Artery that supplies the largest area of the cerebrum  
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Hemorrhagic CVA   primarily caused by uncontrolled HTN  
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CT with Contrast   Force fluids after and creatinine levels need to be checked  
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Dysphagia   Feed on UNAFFECTED side, HOB up, and thickened liquids  
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Verbal deficits after CVA   allow pt sufficient time - DON'T finish sentences for the pt  
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Walking aids after CVA   When transferring patient from a chair to a wheelchair, do not have them lean forward and keep their good leg straight  
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Contusion   Usually results in decreased LOC  
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Dextrose 5%   becomes hypotonic when in the body but may be identified as isotonic  
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Hypertonic Solutions   D5/0.45 NS and D5/0.9 NS  
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Isotonic Solutions   0.9% NS and LR  
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Spinal Cord Injuries   Concussion Contusion Laceration Transection Shearing of vessels Hemorrhage Edema  
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Patient admitted from the ED following a MVC on a backboard with a c-collar and is alert and oriented   talk to the trauma doctor, obtain a written order to remove the backboard after his c-spine has been cleared and leave the collar in place  
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Spinal Shock   Flaccid paralysis, loss of reflexes below injury, hypotension, lasts less than 48 hours, temporary loss of motor and sensory function, temporary loss of reflexes and autonomic function  
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Autonomic Dysreflexia   Drives BP dangerously high (300/160), severe HA and blurred vision  
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MS   Progressive demyelinating disease of the CNS (progressive increasing weakness)  
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MS Tremor   Intentional (Not resting)  
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Guillain Barre   Number 1 concern = airway (check pulse ox and pulmonary function - peak flow)  
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Guillain Barre Diagnosis   Serial peak flow rates - essential to evaluate ascending progression that involves the phrenic nerve  
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Guillain Barre Management   Ventilatory/intubation/trach and IV immunioglobulin (must be hung at the same time every time)  
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Trigeminal Neuralgia   Painful twitch  
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Trigeminal Neuralgia Medication   Antiseizure medicaitons - Tegretol (Carbamazepine)  
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Trigeminal Neuralgia Goal   prevent pain  
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Bells palsy   more common in younger patients  
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Parkinson's   Insufficient amounts of dopamine in the basil ganglia  
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Parkinson's cardinal findings   Tremor at rest, Muscle rigidity, Bradykenesia, Shuffling gait  
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Parkinson's Mask like expression   priority is the ability to chew and swallow  
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Parkinson's Medication toxicity   acute confusion  
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Parkinson's Medication   Sinemet – Ldopa often given in combination with Carbidopa,  
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Parkinson's walking education   Teach pt to stop periodically when walking to prevent falling from propulsive gait – reduces the risk of falling  
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Myasthenia Gravis   Easily fatigability, diplopia, ptosis (weak eye closure)  
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MG Diagnosis   Tensilon Test  
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Anticholinesterase drugs   give before meals to give the best chance of preventing aspiration – if breakfast is at 8:30, it needs to be given at least by 8. This medication must be given on time every time.  
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Myasthenia Crisis   Sudden exacerbation of symptoms (ventilatory distress, increased muscle weakness, difficulty swallowing and talking), Tensilon test = pt improves, Give anticholinesterase medications  
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Cholinergic Crisis   Sudden exacerbation of symptoms (abd cramping, diaphoresis, ventilatory distress, increased muscle weakness), Tensilon test = pt worsens, Give atropine antidote IV  
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MG Medication administration   administer medications 30 - 60 minutes BEFORE meals to help with chewing and swallowing  
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Orthopedic overuse   Keyboarding and Other repetitive joint actions  
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Bursitis   Inflammation of the fluid filled sacs that prevent friction between bones in joints  
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Tendonitis   Inflammation of the tendon sheaths and the synovial membrane  
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Bursitis and tendonitis treatment   prolonged rest, intermittent ice and heat, NSAIDs  
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Carpel Tunnel Syndrome   Irritation and Inflammation of the medial nerve  
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Carpel Tunnel Treatment   Rest in neutral position Cockup splints (especially at hs)- Wrists in neutral position NSAIDS Surgery if risk of permanent nerve damage or extreme pain  
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Ganglion Cyst   "Bible bumps" - forms at the dorsum of the wrist usually resulting from chronic overuse irritation. Tx = stop activity and possible surgery  
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Epicondylitis (tennis elbow)   Pain radiates to dorsal, extensor surface of the forearm, NSAIDS and rest = treatment of choice  
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Effects of Chronic Immobility   Pneumonia, DVT, pressure ulcers  
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Immobility   Can produce degenerative changes. Caused by nonuse, lack of exercise, pain, weakened muscles, joint contractures (takes 24-48 hrs to develop), prolonged bed rest, and incorrect positioning in bed  
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Preventing immobility   MOVE! (PT and OT can help), correct full turning, isometric exercises, ROM  
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Deep Tissue Injury   Blue to deep bruising that you cannot tell whether the skin has been eradicated or not (patient’s who have fallen and laid in the same position for a period of time). Unable to be staged (usually found to be greater than a 4th stage pressure ulcer)  
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Rehab of immobility   begins the instant nurses begin working with someone who has an immobility issue. Move what can be moved and do it often  
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Joint Strains   Injury to a muscle or muscle and tendon  
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Strain Treatment   Rest with moderate movement NSAIDS Analgesics Apply ice and elevate Ace bandage No heat in the first 24 hours  
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Joint Sprains   Twisting injury to ligaments. Usually involves stretching and tearing of ligaments  
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Dislocation or subluxation of joints   Pain with movement. Both require reduction and may require conscious sedation  
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Rotator Cuff Tear   Tear in 1 or more of the SITs muscles, + drop test, Tender AC joint, unable to perform over the head activities, MRI needed, NSAIDS and possible surgery  
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Torn Ligaments   Tear = higher risk in girls, causes knee instability issue, pain and loss of functions, rest, NSAIDs, and surgery. Prevention = 4 point knee muscle exercises  
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Meniscal Tears   2 crescent shaped cartilage discs attached to head of the tibia, allows knee to bed without pain, knee may "give away", loose cartilage may become trapped preventing extension, Rest NSAIDs and surgery  
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Osgood-Schlatter disease   Painful swelling of the bump on the upper part of the shinbone, just below the knee (tibial tubercle). Thought to be caused by small injuries due to repeated overuse before the area has finished growing. Affects boys more than girls  
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Ruptured Achilles   May occur suddenly, common in runners especially those that don't warm up. Sharp pain with inability to plantar flex, surgical repair and a cast (long term immobility of joint). Will never get back to 100%  
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Fractures   Open or closed, complete or imcomplete, simple or compound, stable or unstable  
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Most dangerous fracture   Open compound (r/t infection)  
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Oblique fx   45 degree angle  
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Spiral fx   curves up or down the bone  
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Greenstick fx   broken on one side - bone looks bent. Incomplete stable fracture  
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FX treatment (medication)   NSAIDs/ Prostaglandins, morphine, possible surgery  
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Prostaglandins   produced with the help of an enzyme called cyclooxygenase (AKA as Cox).  
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FX treatment (ortho)   Splints to avoid joint movement, crutches to avoid weight bearing, canes to limit weight bearing  
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FX ER management   RICE, if open - cover with sterile dressing, stop bleeding, check pulses, splint  
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Traction for fx   helps maintain alignment and reduce muscle spasms  
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Pt in traction reports severe pain from muscle spasms   Check body alignment  
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Bucks Traction   Decrease spasm, hip fractures  
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Russell's traction   Buck;s with a double pulley and knee sling  
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Bryant's traction   for children with a fractured femur  
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Pelvic traction   helps with muscle spasm (back and pelvic fx)  
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Skeletal traction   pins/external fixation  
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Electrical stimulation   used in fx that are not healing appropriately, can be internal or external  
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Pin care   sterile supplies but clean procedure. Site can be covered with a 4x4 but is not needed if the area is dry  
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Casting   rigid immobilization device applied after swelling as past peak point (usually 72 hrs)  
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Plaster Cast   48 hrs to dry and must be kept dry  
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Fiberglass cast   1 hr to dry and may blow dry if it gets wet (no more than 10 min on low heat)  
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Nursing priorities in cast care   Distal vascular checks (swelling), distal neuro checks (sensation and motion), checking for hot spots indicating infection or possible cast compression  
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Rib fractures   Biggest issue is pain and the ability to deep breathe. No use of restrictive devices, pillow used for splinting with coughing  
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Flail chest   nonsymmetrical chest with breathing. Highly unstable chest wall that interferes with ability to ventilate. may require ventilator and PEEP.  
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Complications of fx   Hemorrhage, infection, mal/non/dys alignment, compartment syndrome, DVT, gangrene, fat emboli,  
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Compartment syndrome   Muscle pressure builds up too much and the blood supply is impeded and the muscles do not get adequate oxygen  
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Forearm Compartments   2  
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Thigh Compartments   3  
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Lower leg compartments   4  
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Compartment Syndrome s/s   deep, throbbing, unrelenting PAIN  
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Volkman’s Contracture   Contractured wrist and fingers of affected hand – secondary to impaired arterial blood flow Unable to extend fingers Diminished color, temperature, and pulsations distally Permanent damage in hours if arterial circulation not restored  
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Amputations   Surgical removal of all or part of a limb.  
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Diabetic foot amputation   Partial sims - best type of amputation to get because the patient can still walk (with a stump sock on)  
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Stump Care   Elevate, ACE, Clean and DRY surgical wound, encourage movement of affected limb, monitor drainage pain and infection, stump sock after dressing comes off. Goal is to keep the limb health for fitting of prosthesis.  
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Disorders of the back   Pain or discomfort associated with the actual vertebral column, the nerves emanating from within the vertebral column, and, or the muscles articulating with the vertebral column.  
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Low back pain   L4-5 and L5-S1  
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Low cervical region pain   C2-5 is most common  
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Back strain   twisting, bending, and lifting  
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Acute sharp pain   muscular  
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burning and sharp pain   nerve involvement  
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dull and aching pain   chronic issue  
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Sciatica or radiculopathy   pain that radiates down one or both legs  
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Back pain management   Focused on history and physical. Pt is treated symptomatically for up to 4 weeks (rest and relaxation, analgesics, stress reduction, muscle relaxants), no heavy lifting, NSAIDs, Medrol dose pack for anti-inflammatory, heat and cold therapy  
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Back pain may be self limited   40% remit in one week 60 -80 % remit in 3 weeks 90 % remit in two months May be chronic (up to 3 mo)  
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Degenerative Disk Disease   May be injured by trauma and/ or wear and tear disorders that cause the nucleus pulposus to herniated through the annulus  
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Degeneration and/or herniation of intervertebral discs   Discs become more fibrous with age because of wear and tear L4 to S1 most common sites Cervical vertebrae the 2nd most common site  
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Back pain diagnosis   MRI = diagnostic tool of choice, myelogram, EMG possibility  
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Medical Management: Cervical Neck   Non-operative, rest, cervical collar, pain management, possible traction  
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Discectomy   Removal of the disc. May be donw with spinal fusion  
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Laminectomy   Removal of boney processes that allows the visualization of the nerves and the removal of pathology  
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Hemilaminectomy   Removal of the lamina and part of the vertebral arch  
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Foraminotomy   Increasing the space in the intervertebral foramen to allow more space for the nerve  
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Nursing Management: Neck   Cervical Discectomy and fusion, neck collar, bedrest, immobility of neck, pillow under shoulders and head, log rolling, routine dressing care (do not change)  
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Bone Cancer   Malignancy within bone structure. Metatstatic is most common. CT, MRI, labs - Hypercalcemia is commonly found because of bone breakdown.  
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Hypercalcemia   weakness, incoordination, anorexia, n/v, short QT and ST, bradycardia, LOC changes  
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Osteoarthritis/DJD   Chronic, progressive degeneration of the hands and weight bearing joints such as the knee, hip, and lumbar vertebrae. Most common disorder of joints Causes chronic join pain especially with early movement  
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Rheumatoid Arthritis   A connective tissue disorder Inflammatory based arthritis Autoimmune response occurs in the synovial fluid  
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Osteoarthritis Risk Factors   Over 40, obesity, wear and tear, trauma, congenital, inflammatory, poor posture, prolonged steroid use  
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Osteoarthritis Goals   Prevent disease or halt progression, maintain joint mobility, prevent deformity, reduce pain  
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Osteoarthritis joint pain   deep aching pain, especially upon wakening, limited ROM, improves with movement, related to temperature. Joint stiffness usually lasts less than 30 minutes  
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Rheumatoid Arthritis Joint pain   swelling, warmth, erythema, bilateral and symmetric. Joint stiffness > 30 minutes  
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Heberden’s Nodes   hard nodules or boney swellings around the distal interphangeal joints - late stages of OA  
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Bouchard’s Nodes   Similar to Heberden’s but develop around the proximal interphangeal joint  
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OA and RA diagnosis   X-rays, lab tests (inflammatory indicators: ANA, ESR, CRP)  
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OA and RA prevention   maintain normal weight, physical activity, minimal wear and tear  
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OA Management   No cure - goal is to stop progresion. Pain management: tylenol, NSAIDs  
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Cox 1   Ibuprofen, Advil  
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Cox 2 Inhibitors   Celbrex  
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Joint replacement   Need for joint replacement commonly associated with mobility, pain and/or stability issues secondary to Osteoprosis, OA, RA, Fx, trauma  
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Hip replacement   Removal of the head of the femur followed by placement of a prosthetic implant of the head of the femur and/or socket  
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Knee Replacement   Replacement of knee joint with femoral (distal) component and tibial component  
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Complications of joint replacement   infection, hemorrhage  
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Hip Dislocation Prevention: Posterior Surgical Approach   worry more about dislocation. NO internal rotation, do not adduct hip past midline, do not sit on low chairs, lean forward, or put on shoes without extension equipment  
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Hip Dislocation Prevention: Anterior Surgical Approach   Greater chance for them to have hemorrhage because of vessel location. No external rotation, do not flex hip greater than 90 degrees, don't sit on low chairs or lean forward while sitting. Do not raise knee higher than hip, don't pivot on involved leg  
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Hemorrhage r/t joint replacement   Joint replacements commonly lose a lot of blood - Surgeons will often have patients donate their own prior to surgery: Autologous - Can only donate 2 units within a 59 day period. Monitor VS, drainage, and H & H  
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Neurovascular Compromise   Assess color, temperature, and movement. Monitor for swelling and/or deep throbbing unrelenting pain, pain with movement  
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DVT   Assess for pain, swelling, temperature changes. TEDS - remove no more than 1 hour tid. SCDs  
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Time line for complications   Wrong Move (disslocation) - anytime Wind (pneumonia) - 48 hrs Water (UTI/Bladder)- 48 to 72 hrs Wound (Infection)- 72 to 96 hrs Walk (DVT)- 72 to 120 hrs  
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Osteoporosis   A metabolic, age related bone disorder in which bone demineralization causes decreased bone density, bone loss, decreased bone mass, and bone weakening  
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Primary Osteoporosis   More in women  
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Secondary Osteoporosis   More in men r/t steroids and smokers  
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Osteoporosis Goals   prevention of bone loss, deformity and complications, and fractures  
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Osteoporosis Assessment   loss of height, low back pain, kyphosis, pathologic fractures (wrist, hip, vertebral)  
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Osteoporosis Prevention   Adequate calcium, vitamin d, and protein, weight bearing exercise, avoid ETOH, caffeine, and smoking, HRT  
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Osteoporosis Diagnosis   Decrease in serial height, DEXA scan, spot scans, labs (Ca, Vit D3, Ph, alkaline phosphatase)  
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Osteoporosis Management   Encourage regular weight bearing exercises (walking, biking, low impact aerobics)  
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Bishosphonates   inhibit osteoclast function thus suppressing bone loss. Fosamax, Boniva, Calcitonin, Reclast, selective estrogen receptor modulators, parathyroid hormone agonist, biophosphate. Medications can be given daily  
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Fosamax   once a week, sitting or standing up 30 min  
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Boniva   Once a month, sitting or standing up 30 min  
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Calcitonin   Daily nasal sprain then IM/SQ  
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Reclast   IV once a year  
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Selective estrogen receptor modulators   Evista  
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Parathyroid Hormone Agonist   Forteo  
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The nurse is reviewing a prescription for a pt receiving drug therapy for the prevention of osteoporosis and the Pt has HTN   the nurse should question a prescription for hormone replacement therapy  
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Systemic Lupus Erythematosus   An auto immune disorder that results in an exaggerated production of autoantibodies. Characterized by exacerbations and remissions  
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SLE symptoms   vague symptoms: x-rays to rule out arthritis, labs and endocrine studies r/t weight loss, weakness….play the rule out game to properly diagnose  
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SLE skin   Butterfly rash across the face, papules, erythema, purpura, sensitivity to sunlight  
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SLE Diagnosis   history and physical. Blood tests revealing anemia, thrombocytopenia, leukocytosis or leukeopenia, positive ANA (antinuclear antibodies), urine to see if there is any hematuria  
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SLE treatment   no cure - supportive treatment. NSAIDs and Corticosteroids (single most important medication - need to check BS, they mask infection), immunosuppressive agents (reserved for the most seriously ill patients)  
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Vagnintis   vaginal discharge, may be watery or thick, cottage cheese appearance, itching redness, and irritation  
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Gardnerella vaginalis   Fish like odor, plated or glitter cells  
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Oral Candidiasis   oral lesions. redness, pain, common after inhaled steroid use and chemo. Give nystatin  
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The nurse is performing an oral assessment on a pt and notes white-plaque-like lesions on the tongue, palate, pharynx, and buccal mucosa. When patches are wiped away the underlying surfaces are red and sore. What disorder does the nurse suspect?   candida albicans  
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HIV   a retrovirus that invades certain lymphatic cells such as helper T cells or CD4 cells (cell wall receptor cells) and macrophages and leads to their destruction and ultimately loss of effective immune functioning.  
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AIDS   a wasting syndrome resulting from HIV infections that is characterized by immune system failure  
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B cells   offer humoral (Body fluid/blood) immunity and are antibody producing  
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T cells   offer cellular immunity and directly attack viruses and other invaders  
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Stem cells from bone marrow   migrate to the thymus where they are converted to T cells  
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Helper Ts   react to antigens and activate the immune system; T cells secrete cytokines that attract and activate B cells  
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seroconversion   Once the HIV infection occurs and despite the virus being inactive antibodies are formed against it. Go from HIV (+) to (-)  
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Classification/Stages of HIV/AIDS   Once the patient reaches a particular level or category he/she stays in that category  
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Primary Infection (Acute HIV infection)   The point from infection to the development of antibodies to HIV  
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viral set point   the balance between HIV and the body’s response to it. The higher the viral set point the poorer the prognosis.  
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HIV Asymptomatic (CDC Category A)   Virus present but in low enough levels that it does not cause sx except for persistent lymphadnopathy. Focus on maintaining health and good defense responses.  
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HIV Symptomatic (CDC Category B)   Symptoms begin to arise (treat the symptoms). Immunocompromised, fever, diarrhea, hairy leukoplakia, thrombocytopenic purpura  
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AIDS (CDC Category C)   Virus infection is now considered full blown AIDS. Symptoms expand and entitlements begin  
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HIV/AIDS diagnosis   Often not diagnosed until viral load is high and symptoms are advanced. Requires several screenings.  
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Ora Quick Rapid HIV-1 test   Uses less than a drop of blood is much more rapid, which allows for earlier intervention and education. Tests the progression of HIV into AIDS  
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HAART (combination therapy)   a regimen consisting of two antiretroviral agents inhibitors plus a protease inhibitor OR two protease inhibitors and one antiretroviral agent (2 and 1). Antivirals and Protease inhibitors  
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HIV/AIDS nursing management   Supportive approach by body systems symptoms and stage. Emotional support, education, and social stigma and confidentiality.  
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Snellen Alphabet/Chart   Numerator = the distance between the patient and the chart Denominator = the distance from which a person with normal vision could read the lettering  
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Low Vision – Blindness   a general term describing visual impairment that requires patients to use devices and strategies (magnifying glass) in addition of corrective lenses to perform visually based tasks  
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Best Corrected Visual Acuity (BCVA)   20/70 - 20/200  
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20/80 to 20/100   still be able to read at near normal levels with optic aids  
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20/200 t0 20/400   may be able to read slowly with optical aids  
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loss of visual light perception   BCVA that does not exceed 20/200 in the best eye and a widest visual field diameter of 20 degrees or less  
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Blindness causes   diabetic retinopathy, macular degeneration, glaucoma, cataracts, aging  
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Blindness management   Visual aides for magnification and vision enhancement, orientation to and frequent scanning of environment, using a clock for placement  
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Impaired vision   decreased ability to see objects clearly. refractory or nonrefractory  
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Refractory: Hyperopia   Inability to see NEAR objects clearly because of a failure to accommodate - Light focuses in front of retina. Most common (Far sighted)  
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Refractory: Myopia   Inability to see DISTANT objects clearly because of a failure to accommodate - Light focuses behind the retina. Most common in children (Near sighted)  
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Refractory: Presbyopia   Natural loss of accommodation caused by changes in lens accommodation (usually hyperopic in nature)- Poor near vision, Almost 100% of 60 year old require glasses  
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Refractory: Astigmatism   Curvature issues in the lens or other part of the optic apparatus that results in a refractive error- Easily corrected  
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Refractory: Anisometria   Different refractory errors in each eye. May be congenital or acquired.  
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Nonrefractory: Retinal disorders   degeneration, tears, detachments, hemorrhages  
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Nonrefractory: Glaucoma   A group of ocular disorders characterized by increased intraocular pressure that if unresolved will result in damage to the optic nerve and microinfarction causing loss of visual field  
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Glaucoma   May be open angle or closed angle - 3rd leading cause of blindness in the US  
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Primary Open Angle Glaucoma (POAG)   Is a primary issue of angled tissue in the anterior chamber that results in resistant flow of the aqueous humor - Most common form  
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Primary Angle Closure Glaucoma (closed angle)   Resistance/blocking of aqueous humor between the posterior surface of the iris and lens, which places pressure against the pupil close proximity to the pupil - Rare, constitutes a medical emergency  
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Cataracts   A decrease in transparency of the lens that interferes with visual acuity, protein coagulations cause the formation of opaque areas in the lens  
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Macular Degeneration   results in loss of central vision, usually in older adults - gradual onset  
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Strabismus   deviation from symmetrical movement. AKA Tropias  
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Esotrophic   crossed eyed  
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Exotrophic   deviates outward  
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Hypertrophic   deviates up  
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Hypotrophic   deviates down  
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Conjunctivitis   Acute injection or redness of the conjunctiva. May be viral (watery drainage), bacterial(muco-purulent drainage), allergic (watery drainage).  
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Eye trauma   occurs as the result of blunt or penetration injury because of foreign bodies, chemicals, lacerations, blunt force trauma, traumatic enuculation, chronic eye dryness  
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Presbyopia   Progressive decrease in near vision - may still have 20/20 vision, requires reading glasses, usually after age 40  
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Open angle glaucoma s/s   silent in early stages, bilateral, mild to dull ache, halos around lights, blurred vision, loss of visual acuity is not corrected by glasses, headache  
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Closed angle glaucoma s/s   ACUTE - rapid onset, unilateral pain, conjunctivitis, cloudy cornea, photophobia, blurred vision  
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Cataracts s/s   decreased visual acuity, glare, distorted vision, altered color perception  
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Retinal detachment s/s   flashing lights, veiling (most common), floaters  
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Fluorescence tape and a Wood Lamp   ascertain abrasive trauma - Paper tape with orange dye that stains the abrasion green so it is easier to see  
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Ear infections: external canal   otitis externa - more in swimmers  
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Ear infections: inner ear   otitis media - more in kids  
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External Otitis   Inflammation of the external canal. Pain and fullness, pain when lifting up the pinna, yellow/green foul smelling discharge, canal is red and edematous  
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Cholesteatoma   In growth of skin, debris that leads to chronic infection of the mastoid bone and possible the inner ear  
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Otitis Media   Acute infection of the inner ear. Complication of URI, sinusitis, tonsillitus, allergic rhinitis. Usually short term. Otalgia, fullenss, fever, hearing loss  
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Meniere’s Disease   Abnormal fluid in the inner ear. May be vestibular or cochlear. Usually unilateral.  
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Meniere’s Disease: Cochlear   sensoineural hearing loss, tinnitus  
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Meniere’s Disease: Vestibular   extreme vertigo, n/v  
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Meniere’s Disease Management   low Na diet, antihistamines, antiemetic, diuretics  
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Vertigo   Eyes get very big when changing positions to try to stop themselves if they are spinning. Patient has to get used to it because there is no cure  
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Hearing Loss   3rd most common disorder above 65. Can be conductive or sensoinural.  
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Conductive hearing loss   Loss of sound conduction from the external and middle ear to the inner ear  
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Sensoineural hearing loss   Hearing loss related to impaired function of the inner ear, primarily the cochlea and CN VIII  
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Weber test   Bone conduction to test for lateralization of sound. Conductive loss will lateralize to the affected ear Sensorineural loss will lateralize to the better ear  
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Rinne test   Bone and air conduction timing test Usually air conducts longer and louder than bone Conductive loss BC ≥ AC Sensorineural loss AC > BC  
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Skin layers   Epidermis, Dermis, Subcutaneous layer  
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Skin assessment equipment   strong direct lighting, small centimeter ruler, penlight, gloves, special procedures: wood's light and magnifying glass  
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Koilonychia   anemia  
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Paronychia   inflammation of surrounding tissue  
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Leukonychia   related to trauma  
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Clubbing   COPD  
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ABCDE rule   Asymmetry, border, color, diameter, elevation  
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Macule   flat  
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Papule   raised <.5cm  
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plaque   cirrhosis  
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nodule   little bumps that feel hard  
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urticaria   hives  
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vesicle   <.5cm, small pox or chicken pox. raised and fluid filled  
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bulla   > .5cm. raised fluid filled lesion  
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pustule   raised lesion with puss in it  
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crust   impetigo  
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scale   cirrhosis  
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fissure   cracks  
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erosion   pressure ulcers  
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ulcer   stages 1-3 and unstagable  
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excoriation   children and older people who do not have their diaper changed  
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Primary lesions   macule, papule, patch, plaque, nodule, wheal, tumor, uticaria, vesicle, cyst, bulla, pustule  
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Secondary lesions   crust, scale, fissure, erosion, ulcer, excoriation, scar  
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Open Comedones   blackheads  
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Meningococcemia   Petechiae, purpura, ecchymoses Child appears ill, hypotensive, and tachycardic  
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Rockey Mountain Spotted Fever   Maculopapular to petechial. Rash appears on the thenar eminence and flexor surfaces of the wrist and ankles. Palms and soles are usually affected HA, myalgia  
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Stevens-Johnson Syndrome (SJS)   Vesicles to bullae. More shallow that SJS and TEN Child appears less ill that with SJS and TEN Positive Nikolsky sign – skin reddens, fluid collects underneath, and the skin rubs off leaving a raw base  
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Malignant Skin Lesions   use ABCDE rules of examination  
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Beau’s line   Transverse furrow or groove across the nail that extends to the nail bed. Occurs with any trauma that temporarily impairs nail formation, such as acute illness, toxic reaction, or local trauma.  
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Habit-Tic Dystrophy   Depression down middle of nail or multiple horizontal ridges. Due to continuous picking of cuticle by another finger of same hand, which causes injury to nail base and nail matrix.  
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Burns   an alteration in skin integrity resulting in tissue loss or injury caused by heat, chemical, electrical or radiation  
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Dry heat   burn  
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moist heat   hot water  
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chemical burn   battery acid  
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electrical burn   a/c and d/c  
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radiation burn   gamma, beta  
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First-degree burn   superficial, involves only the epidermal layer, never blisters, heals spontaneously  
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Second-degree burn   dermal partial thickness, involves epidermal and dermal layers, pink moist and painful, gets blisters  
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Hand burns   bad due to loss of functionality  
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Third-degree burn   full thickness, nerve, muscle, and tendons, involves epidermal, dermal, subcutaneous layers and nerve endings. Formed eschar on skin  
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First-degree burn presentation   erythema, edema, painful, blanching  
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Second-degree burn presentation   very painful, ooxing, erythema, shiny, wet  
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Third-degree burn presentation   eschar, edema, white or charred, little or no pain  
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Immediate or Emergent Phase   Resuscitative stage lasts from the onset of injury through the successful fluid resuscitation – Stabilization. Determination whether to transport patient to a burn center - family travel is not considered  
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Estimation of Burn Size   Fluid amounts are calculated using the % of body burned. Rule of Nines - Divides teh TBSA into areas - best for adults and children over 10  
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Rule of Nines   face = 9% arms (shoulder through fingers) = 9% each front of body = 18% back of body = 18% legs (front and back) = 18% each Perineum = 1% each  
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Consensus Formula   2-4 mL x kg of body weight x % TBSA burned. Half is given in the first 8 hours then the next half given over 16 hours.  
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Acute or Intermediate Phase   begins 48-72 hours after a burn injury. continue to reassess respiratory and CV status, F&E, and nutritional status. Pharmacological therapy and wound management  
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Rehabilitative Phase   most costly and longest phase. Must be interdisiplinary.  
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Burns Blood Chemistry   Increased K, Decreased Na, total protein, and decreased albumin  
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Burn Hematoloty   Increased HGB and HCT, decreased fibinogen, platelets, and WBCs  
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Burns Assessment   Assess airway, breathing, and circulation, neck and face burns need to be intubated immediately, give O2, check for signs of shock, establish 2 large bore IVs (14 or 16), determine history of injury (AMPLE)  
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Transferring burn patient   do not apply creams, family travel is not considered, and a physician must call the burn center and speak to the physician to get the patient transferred  
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Nurse's role in transferring a burn patient   make sure paperwork is complete and goes with pt, and give the nurse receiving the pt a complete report.  
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