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Pharm Ch 48 anti dysrhythmics

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Question
Answer
dysrhythmias can be supraventricular or ventricular - which are more dangerous   ventricular far more dangerous  
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classes of drugs used to treat dysrthymias   sodium channel blockers---Beta blockers---K channel blockers---Ca channel blockers---digoxin ---and others  
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if SA node is damaged, what is outcome   dec impulse formation results-->tachydysrhythmia  
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if AV block, what happens   AV block --> disturbance of conduction-->tachydysrythmia  
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Sodium channel blockers MOA   this class slows impulse conduction---delays repolarizaiton---with ANTICHOLINERGIC properties (increased SA automaticity, AV node conduction--> inc. HR)  
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name 6 Na channel blockers   quinidine---procainamide---lidocaine---phenytoin---mexiletine---tocainide  
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What Na channel blocker with SE of diarrhea, must be given with food   quinidine  
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Is quinidine cardiotoxic   yes, it is as well as procainamide  
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so if quinidine slows HR, but it's anticholinergic SE increases HR, how do we dose with digoxin   often digoxin given first, then quinidine  
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procainamide ADRs   serious SEs more serious/frequent than others in class---lupus like syndrome---cardiotoxicity---Torsade ---blood dyscrasias (dec platelets, WBCs)  
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lidocaine special admin   only IV admin used exclusively for ventricular dysrhythmias, esp post-MI. huge first pass effect. no anticholinergic effect  
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phenytoin also special admin   IV only---hypoTN more likely due to solvent alkalinity-->phlebitis.  
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which 2 Na channel blockers most likely TdeP   quinidine, procainamide share this  
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Which Na channel blockers not particularly prodysrhythmic   lidocaine, phenytoin, mexiletine, tocainide  
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2 Na channel blockers that are derivatives of lidocaine---but ar ORALLY bioavailable   mexiletine and tocainide ---not commonlyused due to wicked SEs  
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mexiletine SEs   40% compliance limiting n/v/d/c-----neuro disturbances  
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tocainide SEs   pulmonary fibrosis --- serious blood dyscrasias/agranulocytosis  
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MOA of Beta Blockers in dysrhythmias   dec SA automaticity---dec AV conductivity---dec contractility--------------coupled with Ca++ channels, so CCB actions are similar  
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Indication of Na channel blockers   used for both supraventricular and ventricular dysrhythmias  
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indication of Beta blockers   used in tachydysrhythmias - esp caused by sympathetic stimulation  
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ADRs Beta-blocker - propanolol   AV block---bronchospasm---HF bwo negative inotropy dec contractility---sinus arrest bwo dec SA automaticity  
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acebutolol unusual ADR   is B-1 specific yet causes BRONCHOSPASM---oral admin  
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esmolol Beta Blocker admin   given IV due to 9 minute half life  
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what are the only 3 beta blockers used in dysrhythmia tx   propanolol, acebutolol and esmolol  
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Name 1 K channel blocker   amiodarone  
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amiodarone is last-line drug EXCEPT for tx of ventricular tachycardia and has this half-life   a half life of 1-4 MONTHS  
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Amiodarone has wicked, wicked SEs that are   brady/AV block/HF---hepatotoxic---thyroid dysfunction---CNS disturbances, along with many others---including pulmonary fibrosis with long term use  
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Calcium channel blockers used to treat only   supraventricular dysrhythmias bwo of decreasing SA/AV/contractility  
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ADRs of calcium channel blockers being used here - verapamil and diltiazem   ADRs of brady---AV block---HF ---hypoTN---periph edema ---CONSTIPATION  
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diltiazem SEs relative to verapamil   less likely to cause cardiac effects and constipation  
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What is digoxin MOA in dysrhythmias   mostly related to dec SA/AV conduction, which is mediated by vagal stimulation----unfortunately digoxin increases HPS automaticity which contributes to its cardiac toxicitys and dysrhythmias  
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balancing act of all anti-dysrhythmics   many also have significant toxicities including pro-dysrhythmias that lead to cardiotoxicities or TdeP----limit their use or consider non-drug txs  
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what drug produces lupus-like syndrome   procainamide  
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what drug LEAST likely to cause torsade de pointes   phenytoin  
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drug LEAST likely to cause pulmonary fibrosis witih short-term use   amiodarone - only causes pulmonary fibrosis long-term, dose related use  
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