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Lunch Expansion Therapy RCP 111

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Question
Answer
The respiratory group is located in which areas of the brain?   The Pons and Medulla Oblongata  
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The Dorsal Respiratory group   is located in the posterior medulla. Inspiratory Center Neurons Responsible for the rythem of breathing.  
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The Ventral Respiratory Group   is located in various areas of the medulla. Controlls inspiration and expiration. In active during normal breathing. Active during exercise/stress. Inspiratory and experiatory neurons.  
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What are included in the pontine respiratory centers?   Apneustic/pneumotaxic (homeostatic mechanism)  
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Apneustic   lower portion of the pons. sends impulses to activate inspiration. Takes over if pneumotaxic is damaged.  
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Pneumotaxic   located bilaterally upper 1/3 of pons. Restrains apneustic/cuts off the inspiration. Innervated by the vagus nerve  
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What are the respiratory monitoring system chemoreceptors?   Central chemoreceptors peripheral chemoreceptors  
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Central chemoreceptors   respond to an increas in hydrogen ions in CSF. Hydrogen ions porportional to co2.  
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Peripheral chemoreceptors   special 02 sensitive cells that react to a decrease in oxygen levels. Stimulate an increased respiratory rate. Located in cotoid and aortic arch.  
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Lung expansion therapy is designed to treat and prevent   atelectasis  
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What are the two types of atelectasis?   passive resorption  
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Passive atelectasis is   the result of shallow breathing. Caused by persistent use of small tidal volume.  
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Passive atelectasis can occur with the following   surgery medications (CNS depressents) Neurolgical disorder neuromuscular weakness bed rest immobility  
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Resorption atelectasis is the result of   an airway obstruction. Muscus plugs are present in the airway and block ventilation. Capillaries/blood flow absorb gas  
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What is lobar atelectasis   An entire lobe of atelectasis; a large plug can also be caused by tumors.  
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what are factors causing atelectasis?   obesity neuromuscular disease sedation surgery spinal injury bedridden immobility decreased cough  
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What are clinical signs of atelectasis?   breath sounds: decreased/crackles tachycardia, tachypnea,cyanosis; secondary to hypoxemia. CXR: increased opacity  
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Normal breathing physics   Transpulmonary Pressure (Ptp)  
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Transpulmonary Pressure (Ptp)   (Palv)-(Ppl) alveolar pressure-pleural pressure creates a gradient  
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Lung expansion therapy   increases lung volume by increasing the transpulmonary pressure gradient.  
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The greater the transpulmonary pressure gradient   the more the lung expands.  
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What are the types of lung expansion therapy?   Incentive Spirometry Intermittent Positive Pressure Breathing.  
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Incentive Spirometry (IS)   increases the transpulmonary pressure gradient by lowering pleural pressure. Most effective b/c mimics normal physiology of breathing.  
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IPPB   increases the transpulmonary pressure gradient by increasing alveolar pressure. (increased risk of damaging lung)  
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How do you know what to choose?   Needed equipment Personnel Risk Cost  
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Incentive spirometry can be done with   mothpiece or a trache.  
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Incentive spirometry   mimics natural sighing by encouraging a slow, deep breathing.  
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The therapist determines   the volume and repetitions during IS  
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Icentive Spirometry Procedure   Slow, deep breath in from resting exhalation, followed by a 3-5 second breath hold. Repeat every hour; 5 to 10 reps  
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Vital Capaicity   65-75 ml/kg (-10 ml/kg) not an effective therapy  
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THe indications of incentive spirometry.   Treat and prevent atelectasis presence of restrictive lung disease  
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What are the contraindications of IS?   patients unable to take a deep breath lack of consciousness/cooperation  
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What are the hazards/complications of IS?   hyperventilation barotrauma discomfort due to pain hypoxia due to interrupted 02 therapy bronchospasm fatigue  
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What are the three IS devices?   indirect volume measuring device volume oriented flow oriented  
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Indirect volume measuring device   flow through a fixed orifice over time displaces volume  
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Volume oriented   not used anymore. measures volume via bellows bulky/large  
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Flow oriented   indirectly measures volume  
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What should you Chart after Incentive Spirometry?   Vitals Volume Achieved Repetitions Good breath hold or not If they understood. Assessment of cough Effort/motivation set goal  
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IPPB   invented by forest bird in 1947. aka hyperinflation therapy used for a broad range of clinical conditions. 1st ventilator  
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IPPB is used short term or long term?   Short term  
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IPPB csn be administered   several times a day or as frequently as once every hour.  
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What does IPPB require   spontaneously breathing patient  
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How can IPPB be given   with a mouthpiece or a mask (Requires a tight seal)  
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IPPB is administered with a   pneumatic machine  
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Usually IPPB therapy is given accompanying   aerosol 32% less effective than hand held nebulizer. 3cc normal saline if ordered w/out treatment  
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IPPB Therapy lasts   15 minutes  
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IPPB Requires a what?   50 PSI sources  
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IPPB Indications   Prevent/Treat atelectasis Inability to clear secretions due to inneffective ventilation and coughing. short-term ventilatory support Deliver aerosol medication.  
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Condraindications for IPPB   untreated pneumothorax hemodynamic instability increased ICP Recent facial or esophageal surgery tracheosophageal fistula Acive hemoptisis  
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More contraindications for IPPB   Active/untreated TB Evidence of blebs (over distension) Singulations Air swallowing nausea  
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One important fact...   Increased thorax pressure clamps down on the great vessels and drops the blood pressure.  
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Hazards and complications of IPPB   barotrauma hemodynamic instability increased ICP (clamping of great vessels) Air trapping Nosocomial infection Hemoptysis Hypocarbia Hyperoxia or hypoxemia  
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more hazards and complications of IPPB   Gastric distension/aspiration Increased airway resistance increased V/Q mismatch Physchologic dependence bronchospasm  
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Facts about the Bird Mark 7   pneumatically powered requires a closed circuit with exhalation valve and nebulizer  
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The machine incorporates a venturi or air entrainment jet to   enhance flow capabilities and decrease Fi02  
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What are the IPPB controls?   Pressure, Flow, Sensitivity, Air mix control and apnea timer.  
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Pressure   directly controls tidal volume. Indirectly affects inspiratory time  
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Patients lung characteristics also affect tidal volume   lung compliance/tidal volume directly proportional. Airway resistance/tidal volume indirectly proportional  
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Flow   directly controls speed (i time) indirectly affects tidal volume  
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Sensitivity   controls patient effort needed to trigger machine  
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Air mix control   when used increases flow output and decreases Fi02  
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Apnea Timer   backup rate  
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Ventilator Class (Bird Mark 7) Pressure controller   Pressure does not change as a result of compliance and resistance changes  
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Volume Controller   Volume does not change as a result of compliance and resistance changes -measures volume directly  
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Flow controller   volume does not change as a result of compliance and resistance changes -measures volume indirectly by measuring flow  
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Phase 1   change from exhalation to inspiration  
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Phase 2   Inhalation  
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Phase 3   Change from inhalation to exhalation  
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Phase 4   Exhalation  
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Trigger: Phase 1   Variable that triggers (starts) breath delivery. Pressure (patient), manual or time. Other trigger variables flow  
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Limit:Phase 2   Variable not eexceeded above the preset value during inspiration. Inspiration does not end when the variable reaches the preset value. Flow, other limit variables (pressure)  
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Cycle: Phase 3   Variable that cycles (stops) breath delivery. Pressure. Other cycle variables: volume, flow or time.  
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Phase 4   Exhalation is passive  
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The circuit   Pressure drive line- powers nebulizer/ exhalation valve  
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Exhalation valve   close on inspiration/ opens on exhalation Mushroom type valve  
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The IPPB can have a   mouthpiece or a mask must have a tight seal  
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The left side of the IPPB machine is   the ambient side- atmospheric pressure  
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The right side is the   pressure side  
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The pressure control toggle   10-40 cm H20  
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If pressure increases   tidal volume increases  
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If pressure decreases   Tidal Volume decreases  
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This is a pressure cycle machine and pressure   indirectly affects inspiratory time  
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TLC   Trigger, Limit, Cycle  
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Pressure Cycles the machine   off  
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Patient lung characteristics affect   tidal volume  
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Overly compliant lungs take   longer to reach pressure (longer i time)  
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Stiff lungs   Take less time to reach pressure, shorter i time  
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The longer its on   the more volume  
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lung compliance and tidal volume   are directly proportional  
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Airway resistance   and tidal volume are indirectly porportional  
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Flow control   5-40 liters per minute  
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Flow control   directly controls speed The higher the speed the less time  
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Flow indirectly affects tidal volume   More flow less tidal volume less flow more tidal volume  
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Sensitivity (trigger)   Controls patient effort need to trigger machine one  
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The trigger variable   initiates the machine  
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Manual trigger   is red  
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Patient trigger   based on their effort  
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time trigger   black (apnea)  
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Patient effort for sensitivity   5-40 5 easier to trigger 40 more difficult to trigger  
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Apnea   makes machine trigger by itself  
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The closer the magnets   the more difficult to trigger machine  
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The farther away the magnets are   the easier it is to trigger machine  
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The initial setting on the Bird Mark 7   is 15/15/15  
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The Air max control   in 100% Fi02 out 40-60% FI02  
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Limit variable   Flow Can't get more or less flow than set  
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Manomometer   Green +, Pink (-)  
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How can you measure volume   Wrights respirometer Flows over time to give volume measurement Hooks on exhalation valve.  
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