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spinal cord trauma

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Question
Answer
spinal cord injury is   any person w/a head injury until proven otherwise  
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spinal cord injury can result from   fracturing/dislocating one or more vertebrae  
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approximately how many people sustain spinal cord injuries annually   7000-10000  
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what percentage are tetraplegic   57%  
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what percentage are below the age of 25yrs   50%  
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tetraplegia is also known as   quadriplegia  
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what is the largest nerve in the body   spinal cord  
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what do nerve fibers do   carry signals to & from brain  
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where is the spinal cord extended from   base of brain-level to waist  
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ascending tracts are   nerves in spinal cord that send messages for pain, temp and touch from the body to the brain  
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ascending tracts carry   subconscious information such as body position  
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descending tracts are responsible for   muscle movements and transmit impulses from cortex ot periheral nerves  
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upper neurons are a   voluntary motor system  
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upper motor neurons start in   cerbral cortex, cross over in brain stem and end in the spinal cord  
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upper motor neurons synapse with   lower motor neurons  
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damage to the upper motor neurons causes   spastic paralysis and hyperreflexia  
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upper motor neuron damage can be caused by   lesions at T11 and above  
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lower motor neurons are   final pathway for descending motor tracts to cause skeletal muscle innervation  
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lower motor neurons are found   in anterior horn of corresponding areas of spinal cord  
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lower motor neuron damage can cause   flaccid paralysis  
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lower motor neuron damage can be caused by   lesions at or below T12  
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Central Nervous System   brain and spinal cord  
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peripheral nerves   sensory nerves outside of the CNS  
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sympathetic/parasympathetic nervous system control   blood pressure, temperature regualtion  
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Spinal cord is surrounded by   rings of bone called vertebra  
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vertebra named according to   location  
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the higher the injury to vertebra and spinal cord the   more disability in a person  
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cervical vertebrae   7  
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thoracic vertebrae   12  
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lumbar vertebrae   5  
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sacrum vertebrae   5 fused into 1  
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coccyx vertebrae   4 fused into 1  
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cervical nerves   head & neck, diaphragm, deltoids, biceps, wrist extenders, triceps, hands  
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thoracic nerves   chest muscles and abd muscles  
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lumbar nerves   leg muscles  
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sacral nerves   bowel, bladder and sexual function  
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acceleration   hyperextension-whiplash, rear-end collision  
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deceleration   hyperflexion- front-end collision  
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accleration-deceleration   hyperextension and hyperflexion  
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excessive rotation   hanging  
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axial loading   vertical compression- dive, falls  
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penetrating wound   open injury- knife,missile  
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concussion of spinal cord   temp loss of function 254-48 hrs b/c of inflammation(edema)  
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contusion   bruising and bleeding  
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necrosis occurs from   compromised capillary circulation and venous return  
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complete cord transection   total loss of motor, sensory, and primary reflex activity below the level of the lesion  
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tetraplegia CCT   C 1-8 (respiratory compromise)  
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paraplegia CCT   T1-L2  
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cauda equina CCT   L2 and below  
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sacral CCT   loss of bowel, bladder, & sexual function  
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Incomplete Transection   anterior cord, central cord and brown-sequard syndrome  
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anterior cord syndrome   loss of motor, pain & temp below the level of injury. Intact touuch, position & vibration (50%>40;50-70yo-hyperflexion)  
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central cord syndrome   motor paralysis of upper ext & lower ext but uper ext affected more than lower- hyperextension injuries  
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brown-sequard syndrome   hemisection; ipsilateral(same side) motor loss, proprioception, vibration, deep touch; contralateral loss of pain, temp light touch, penetrating trauma  
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emergency care & tx   establish & maintain open airway  
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emergency care & tx   jaw thrust maneuver, cervical collar,sandbag, C1-4 require constant ventilatory support, C5 requires intermittent support, C5 and below-independent resp function, log roll pt  
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emergency care & tx   IV line, possible Dopamine(to prevent muscle tremors)foley, methylprednisolone-gold standard b/c to decrease inflammation-give w/in 8 hrs  
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diagnostic tests   spinal x-rays, ct scan, MRI(changes in cord and tissues surrounding), ABG levels of O2, cystogram, CXR(atelectasis), EMG, peripheral nerves stimulated  
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neurogenic bladder   lacks bladder control due to a brain or nerve condition--bladder can explode-need foley  
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kinetic therapy   movement  
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nursing assessments   assessment of sensation & motor function, cardiovascular & resp assessment, GI/GU assessment(ileus,distention,areflexic bladder, stasis, overflow), psychosocial assessment(dependence,support,role,self-esteem,body image,family,financial)  
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paralytic ileus   no movement causes air to be there which causes distention & impaction  
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nursing outcomes   no further deterioration in neuro status, no complications of immobility(kinetic therapy), stool & urine output maintained, maintain VS's r/t neurogenic shock, resolution of spinal shock, no incidence of autonomic dysreflexia  
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care of client in halo device   inspect for tightness, pin sites inspect for infection, bleeding, skin integrity-turn client, inspect skin, assess muscle function, ROM  
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effects of injury: Resp paralysis is common in   C1-4  
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injuries here may have edema above the injury causing temporary repsiratory compormise-ventilator.   C5-T1  
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when does edema usually subside in which client wont need a ventilator   several weeks  
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neurogenic shock occurs when injury form this level and above occur   T6  
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neurogenic shock   decreased heart rate, hypotension  
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what is given for neurogenic shock   atropine  
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what does atropine do in a pt with neurogenic shock?   increase heart rate  
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vasopressors (dopamine)   rasie BP  
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what tx is used in a pt with neurogenic shock   atropine, **fluids and dopamine  
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effects of injury: GI/GU   decreased motility, stress ulcers, foley until resolution of spinal shock(30-60mins after injury, paralytic ileus  
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bethanechol chloride (urecholine)   bladder tone  
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long term nursing interventions   oxygenation, suctioning, prevent pressure sores, contractures, DVT, PE, prevent Ortho hypotension, promote self care, establish bowel/bladder retraining program, assess for resolution of spinal shock  
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complications of injury   spinal shock, neurogenic shock, autonomic dysreflexia  
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long term complications   spasticity, bladder & bowel dysfunction  
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spinal shock   temp suppression of function below the level of injury, occurs 30-60 mins after injury, flaccid paralysis, loss of tendon reflexes, loss of sensation(called areflexia)  
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recovery from spinal shock takes   2 wks, average 1-6 wks  
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what signifies that spinal shock is resolved   appearance of involuntary reflexes  
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neurogenic shock occurs at level   T6 and above  
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neurogenic shock s/s   decreased BP(systolic <80), decreased Pulse(<60), decreased RR (apnea r/t cervical injury), decreased temp, immediate areflexia, flaccid paralysis, loss of skin sensations, priapism in males(erection that lasts for 4 hrs or more b/c of pulling of blood)  
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life-threatening complication is   autonomiv dysreflexia that occurs w/upper motor neuron involvement  
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autonomic dysreflexia   visceral stimuli in pt's w/injuries above T6, occurs after the spinal shock is over, trigger:visceral distention  
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autonomic dysreflexia s/s   HTN, h/a, flushed, bradycardia, pilomotor spasm(goosebumps), nasal stuffiness  
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complications of autonomic dysreflexia   stroke, seizures, hemorrhage, blindness  
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interventions for autonomic dysreflexia   HOB up, sitting, loosen clothing, call MD, find & remove cause immediatedly-check foley-last BM, impaction-nupercaine  
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interventions for autonomic dysreflexia   nifedipine(procardia), Nitropursside Sodium(nipride)-IV to lower BP quickly  
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long term complications spasticity   occurs after spinal shock-UMN, physical activity: stretching exercises, whirlpool, warm tub baths, baclofen, flexeril, diaxepam  
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s/e of spasticity   drowsiness, diplopia, GI upset  
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baclofen   muscle relaxant & effective on involuntary spastic movement  
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bowel retraining   consistent time for bowel elimination, high fluid intake(at least 2L daily)/high fiber diet, supository program(UMN injury-above T12-use lidocaine coated supp prior), stool softener  
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bowel retraining LMN   flaccid-manual disimpaction; optimize gastrocolic reflex, valsalva, massaging L to R, privacy, nonstressful, sitting position  
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bladder retraining   as soon as stable, d/c foley; intermittent cath q4 hrs  
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bowel retraining UMN   spastic-stroking the inner thigh, pullin on hair, warm water poured over perineum, tapping the bladder/detrusor muscle, bethanechol(urecholine)  
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bowel retraining LMN   flaccid-valsalva, tightening the abd muscles, cath for residual urine  
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sexuality UMN injuries for men   reflexogenic erections; orgasm & ejaculation absent; poor sperm quality  
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sexuality LMN injuries for men   complete lesion absent psychogenic or reflexogenic erections  
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incomplete lesion LMN   psychogenic erections w/ejaculation  
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sexuality women   meses may cease for 6 months, orgasms possible, fertility intact-loss of lubrication, pregnancy complications-autonomic dysreflexia in UMN injuries  
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