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Gastrointestinal System

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Question
Answer
this sphincter prevents reflux of gastric contents into the esophagus   cardiac (lower esophageal) sphincter  
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this sphincter regulates the rate of stomach emptying into the small intestin   pylorlic sphincter  
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the chief coenzyme of gastric juice which converts proteins into proteases and peptones   pepsin  
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necessary for the absorption of vitamin B12   intrinsic factor  
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enzyme that controls gastric acidinty   gastrin  
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this part of the small intestine contains the openings of the bile and pancreatic ducts   duodenum  
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digests starch to maltose   amylase  
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reduces maltose to monosaccharide glucose   maltase  
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splits lactose into galactose and glucose   lactase  
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reduces sucrose to fructose and glucose   sucrase  
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splits nuclec acids to nucleotides   nucleose  
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activates trypsinogen to trypsin   enterokinase  
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these play a vital role in the synthesis of some B vitamins and vitamin K   intestinal bacteria  
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prevents contents of the large intestine from entering the ileum   ileocecal valve  
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this organ synthesizes glucose, amino acids, and fats   liver  
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this organ stores 200 to 400 mL of blood and also filters the blood   liver  
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stores and concentrates bile and contracts to force bile into the duodenum during the digestion of fates   gallbladder  
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the cystic duct joins the hepatic duct to form   common bile duct  
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the presence of fatty materials in the duodenum stimulates the liberation of this, which causes contraction of the gallbladder and relaxation of the sphincter of Oddi   cholecystokinin  
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secretes sodium bicarbonate to neutralize the acidity of the stomach contents that enter the duodenum   pancreas  
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examination of the upper GI tract under fluoroscopy after the patient drinks barium sulfate   barium swallow (upper GI seies)  
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after a barium swallow test, the client should be instructed to   increase po fluid until stools resume their normal color  
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a fluoroscopic and radiographic examination of the large intestine after the rectal instillation of barium sulfate   barium enema (lower GI series  
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the diet restrictions before a barium enema study include   low residue diet for 1-2 days, clear liquid diet day before and laxative the evening before, NPO after midnight  
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requires the passage of a NG tube into the stomach to aspirate gastric contents for analysis of acidity, appearance, and volume   gastric analysis  
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how long should a patient be NPO before gastric analysis   8 to 12 hours  
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upper GI endoscopy is also known as   esophagogastroduodenoscopy(EGD)  
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following sedation, an endoscope is passed down the esophagus to view the gastric wall, sphincters, and duodenum   EGD  
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how long should a patient remain NPO following EGD   1 to 2 hours  
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use of a rigid scope to examine the anal canal   anoscopy  
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before anoscopy, proctoscopy, and sigmoidoxcopy enemas are given until   returns are clear  
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a fiberoptic endoscopy study in which the lining of the large intestine is visually examined   colonoscopy  
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following endoscopic examinations, guarding of the abdomen, increased fever and chills, pallor, abdominal distensiona and pain, restlessness, tachycardia, and tachypnea are signs of   bowel perforation; peritonitis  
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performed with a fiberoptic laparoscope that allows direct visualization of organs and structures within the abdomen   laparoscopy (peritoneoscopy)  
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performed to detect gallstones and to assess the ability of the gallbladder to fill, concentrate its contents, contract, and empty   cholecystography  
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before colecystography, patient should be assessed for allergies to   iodine or seafood  
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examination of the hepatiobiliary system performed via a flexible endoscope inserted into the esophagus to the descending duodenum   endoscopic retrograde cholangiopancreatography (ERCP)  
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examination involving injection of dye directly into the biliary tree   percutaneous transhepatic cholangiography  
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transabdominal removal of fluid from the peritoneal cavity for analysis   paracentesis  
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why should a patient void before paracentesis is performed?   to move the bladder out of the way of the paracentesis needle  
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how is a patient positioned for parcentesis?   upright on edge of bed, or fowlers if bedridden  
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needle inserted through the abdominal wall to the liver to obtain a tissue sample for biopsy and microscopic evaluation   liver biopsy  
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these laboratory values should be checked before a liver biopsy is performed   PT, PTT, INR  
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patient should like on this side for this long after a liver biopsy   right side, two hours  
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urea breath test detects the presence of ________, which is the bacteria that causes perptic ulcer disease   heicobacter pylori  
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__________ is released during liver damage or biliary obstruction   alkaline phosphatase, (bilirubin is also an acceptable answer)  
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prothrombin time is _________ with liver damage   prolonged  
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_________________ assesses the ability of the liver to deanimate protein byproducts   serum ammonia  
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liver enzymes are ______________ with liver damage   elevated  
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an increase in _________ indicates pancreatitis or biliary obstruction   cholesterol  
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increased values of amylase and lipase indicate __________   pancreatitis  
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normal bowel sounds occur every __ to ___ seconds   5 to 15  
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how long must you listen before assuming that bowel sounds are absent?   5 minutes  
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backflow of gastric and duodenal contents into the esophagus   gastroesophageal reflux  
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causes of GERD   incompetent lower esophageal sphincter, pyloric stenosis, or motility disorders  
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also known as esophageal or diaphragmatic hernia   hiatal  
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inflammation of the stomach or gastric mucosa   gastritis  
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in chronic gastritis, a deficiency of this vitamin may develop   B12  
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ulceration in the mucosal wall of the stomach, pylorus, duodenum, or esophagus   peptic ulcer  
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the proper order for performing an abdominal assessment   inspect, auscultate, percuss, palpate  
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chronic gastritis is distuinguished from acute gastritis by the following symptoms in addition to nausea, vomiting, and anorexia   belching, heartburn after eating, sour taste in mouth, vitamin B12 deficiency  
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ulcers are named according to their _____   location  
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most common sites of peptic ulcers   stomach and duodenum  
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mucosal barrier protectants should be administered...   1 hour before meals  
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pain with gastric ulcers is usually located in ___________ and occurs ________ after meals   mid or left epigastric, 30 to 60 minutes  
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pain with duodenal ulcers is usually located in ___________ and occurs _________ after meals   midepigastric, 1.5 to 3 hours  
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removal of the stomach with attachment of the esophagus to the jejunum or duodenum   total gastrectomy (esophagojejunostomy or esophagoduodenostomy)  
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surgical division of the vagus nerve to eliminate the vagal impulses that stimulate hydrochloric acid secretion in the stomach   vagotomy  
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removal of the lower halv of the stomach and usually includes a vagotomy   gastric resection or antrectomy  
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partial gastrectomy with the remaining segment anastomosed to the duodenum   billroth I, or gastroduodenostomy  
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partial gastrectomy with the remaining segment anastomosed to the jejunum   billroth II, or gastrojejunostomy  
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enlargement of the pylorus to prevent or decrease pyloric obstruction and enhance gastric emptying   pyloroplasty  
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should an NG tube be irrigated following gastric surgery?   no  
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rapid emptying of the gastric contents into the small intestine that occurs following gastric resection   dumping syndrome  
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loud gurgles indicating hyperperistalsis; stomach growling   borborygmi  
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results from a deficiency of intrinsic factor   pernicious anemia (vitamin B12 deficiency)  
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signs of pernicious anemia   smooth, beefy red tongue, parasthesias of hands and feet, gait a balance disturbances  
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to prevent dumping syndrome, this diet should be followed   high protein, high fat, low carbohydrate  
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foods that contain vitamin B12   yeast, citrus fruits, dried beans, green leafy vegetables, liver, nuts, organ meats  
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following bariatric surgery, the patient is restricted to liquid and pureed food for how many weeks   6  
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dilated and tortuous veins in the submucosa of the esophagus   esophageal varices  
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esophageal varices are caused by ______ _________   portal hypertension  
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how should a patient with esophageal varices be positioned?   head elevated  
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this medication is administered to induce vasoconstriction and reduce bleeding of esophageal varices   vasopressin  
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this medication is administered in conjunction with vasopressin to prevent vasoconstriction of the coronary arteries (in treatment of bleeding esophageal varices)   nitroglycerin  
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this should be avoided by a patient with esophageal varices   vasovagal responses  
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the injection of a sclerosing agent into and around bleeding varices   sclerotherapy or endoscopic injection  
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ligation of varices with an elastic rubber band   endoscopic variceal ligation  
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this type of ulcerative colitis results in vascular congestion, hemorrhage, edema, and ulceration of the bowel mucosa   acute  
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this type of ulcerative colitis causes muscular hypertrophy, fat deposits, and fibrous tissue with bowel shortening, thickening, and narrowing   chronic  
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key signs of ulcerative colitis   severe diarrhea with blood and mucus, dehydration and electrolyte imbalance, vitamin K deficiency  
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ulcerative colitis can be cured by   protocolectomy with permanent ileostomy  
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intraabdominal pouch that stores the feces constructed from the terminal ileum   kock pouch  
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before colostomy/ileostomy, intestinal antiseptics and antibiotics are administered to   decrease baceria in the colon  
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normal stoma color is   bright pink or red, and shiny  
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key signs of crohn's disease   crampy colicky pain after meals, semisolid diarrhea, dehydration, electrolyte imbalances  
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surgery for this inflammatory bowel disease is avoided because recurrence of the disease process is likely to occur   crohn's disease  
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outpouching or herniation of the intestinal mucosa   diverticulosis  
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inflammation of one or more diverticula   diverticulitis  
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a perforated diverticulum can progress to   peritonitis  
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key signs of diverticulosis/itis   LLQ pain that increases with coughing, straining or lifting, palpable, tender rectal mass, blood in stools  
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dilated varicose veins of the anal canal   hemorrhoids  
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_________ are caused by portal hypertension, straining, irritation, or increased venous or abdominal pressure   hemorrhoids  
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key sign of hemorrhoids   pain and bright red bleeding on defecation  
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how often should sitz baths be used   3 to 4 times per day  
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key sign for appendicitis   abdominal pain most intense at McBurney's point, rebound tenderness  
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how will a patient with acute appendicitis usually position themselves?   side lying with abdominal guarding and knees flexed  
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inflammation of the peritoneum   peritonitis  
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following appendectomy with appendix rupture, how should a patient be positioned   right side lying or low to semi fowlers with legs flexed to facilitat drainage  
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chronic, progressive disease of the liver characterized by diffuse degeneration and destruction of hepatocytes   cirrhosis  
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cirrhosis that is alcohol induced, nutritional, or portal   laennec's  
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cirrhosis as a complication of acute viral hepatitis or exposure to hepatotoxins   postnecrotic  
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cirrhosis as a complication of chronic biliary obstruction, bile stasis, or obstructive jaundice   biliary  
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cirrhosis associated with severe right sided congestive heart failure   cardiac  
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persistent increase in pressure within the portal vein that develops as a result of obstruction to flow   portal hypertension  
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the accumulation of fluid within the peritoneal cavity that results from venous congestion of the hepatic capillaries   ascites  
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progressive renal failure associated with hepatic failure   hepatorenal syndrome  
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a patient with ascites would have a dietary restriction of   sodium and fluid  
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coarse tremor chacterized by rapid, nonrhythmic extension and flexion in the wrist and fingers   asterixis  
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fruity, musty breath odor of severe chronic liver disease   fetor hepaticus  
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this medication decreases the pH of the bowel, decreases production of ammonia by bacteria in the bowel, and facilitates the excretion of ammonia   lactulose (chronulac)  
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this mecications inhibits protein synthesis in bacteria and decreases the production of ammonia   neomycin; metronidazole (flagyl)  
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this type of cholecystitis results when inefficient bile emptying and gallbladder muscle wall disease cause a fibrotic and contracted gallbladder   chronic  
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this type of cholecystitis occurs in the absence of gallstones and is due to bacterial invasion via the lymphatic or vascular systems   acalucous  
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key signs of cholecystitis   epigastric pain 2 to 4 hours after eating fatty foods, pain in RUQ, Murphy's sign (inability to take a deep breath when the examiner's fingers are passed below the hepatic margin)  
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removal of the gallbladder   cholecystectomy  
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incision into the common bile duct to remove a stone   choledocholithectomy  
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these two activities should be encouraged following gallbladder surgery to avoid respiratory complications   coughing, deep breathing  
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acute or chronic inflammation of the pancreas with associated escape of pancreatic enzymes into surrounding tissue   pancreatitis  
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key signs of pancreatitis   midepigastric or LUQ pain that is aggravated by a fatty meal, alcohol, or lying in a recumbent position  
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discoloration of the abdomen and periumbilical area (pancreatitis)   cullen's sign  
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discoloration of the flanks (pancreatitis)   turner's sign  
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elevated serum lipase and amylase are indicators of   pancreatitis  
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signs of chronic pancreatitis   LUQ mass, seatorrhea and foul smelling stools, diabetes mellitus  
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the first stage of hepatitis preceding the appearance of jaundice; flu like symptoms   preicteric stage  
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the second stage of hepatitis which includes the appearance of jaundice and associated symptoms such as elevated bilirubin levels, dark urine, and clay colored stools   icteric stage  
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the convalexcent stage of hepatitis in which jaundice decreases and urine and stool color return to noromal   posticteric stage  
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how is hepatitis A most commonly transmitted   fecal-oral route  
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how is hepatitis B most commonly transmitted   parenterally  
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how is hepatitis C most commonly transmitted   parenterally  
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how is hepatitis D most commonly transmitted   parenterally, and only in conjunction with hepatitis B  
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How is hepatitis E most commonly transmitted   contaminated water  
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how is hepatitis G most commonly transmitted   parenterally  
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