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Integumentary Nursing from Lewis

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Question
Answer
Epidermis   thin superficial layer of skin (0.05 – 0.1 mm in thickness) protects  
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Dermis   connective tissue below the epidermis (1-4 mm) Blood supply, blood pressure because the skin can vasodilate and decrease pressure  
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Subcutaneous tissue   below the dermis, not part of the skin. Attaches skin to underlying tissue (such as bone and muscle) loose connective tissue and fat cells that provide insulation Meds given here for slow use because of the lack of blood supply  
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Skin appendages   develop from the epidermal layer and receive nutrients, electrolytes and fluids from the dermis  
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Hair   forms from keratin  
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Nails   forms from keratin  
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Sebaceous glands   secrete sebum which is emptied into the hair follicles. (prevents dryness of the skin)  
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apocrine sweat glands   axillae, breast areola, umbilical and anogenital aread, ear canals and eyelids. Thick milky substance tat becomes odoriferous when altered by skin bacteria  
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eccerine sweat glands   transparent watery solution, cools body  
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Functions of the Skin   protect the underlying tissues of the body by serving as a surface barrier to environment, barrier against invasion by bacteria and viruses, Barrier against excessive water loss, Insulation, and protection from trauma, Sensory perception, Heat regulation  
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Functions of the skin continued   Synthesis of Vitamin D, Administer meds, Express emotion  
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Primary Skin Lesions   are physical changes in the skin considered to be caused directly by the disease process  
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Macule   circumscribed flat area with a change in skin color less than 1 cm. eg Freckles, petechiae, measels  
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Papule   elevated solid lesion. Eg mole, wart  
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Vesicle   circumscribed superficial collection of serous fluid, less than 1 cm. eg. Chicken pox, shingles, second degree burn  
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Plaque   circumscribed, elevated superficial SOLID lesion, greater than 1 cm. eg psoriasis  
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Wheal   firm, edematous, irregularly shaped area. Eg. Insect bite  
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Pustule   elevated, superficial lesion filled with purulent fluid. Eg. acne  
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Secondary Skin Lesions   may evolve from primary lesions, or may be caused by external forces such as scratching, trauma, infection, or the healing process. The distinction between a primary and secondary lesion is not always clear  
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Fissure   linear crack or break from the epidermis to dermis, dry or moist. Eg. Athlete’s foot, crack in mouth corners  
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Scale   excess, dead epidermal cells produced by abnormal keratinization and shedding. Eg. Scarlet fever, flaking of skin after drug reaction  
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Scar   abnormal formation of connective tissue that replaces normal skin. In dark skin, higher incidence of Keloid devoloping.  
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Ulcer   loss of the epidermis and dermis, crater like, irregular shape  
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Atrophy   depression in the skin resulting from thinning of the epidermis and dermis  
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Excoriation   area in which the epidermis is missing exposing the dermis. Eg. Scabies, abrasion  
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Lesion Configuration Terms    
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Annular   ring shaped  
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Gyrate   spiral shaped  
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Iris lesions   concentric rings (bulls eye) lyme disease is and example  
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Linear   in a line  
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Nummular, discoid   coinlike  
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Polymorphous   occurring in several forms  
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Punctate   marked by points or dots  
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Serpiginous   snakelike  
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Lesion Distribution Terms    
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Asymmetric   unilateral distribution, one side of body  
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Confluent   merging together, overlapping  
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Diffuse   wide distribution, all over  
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Discrete   separate from other lesions  
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Generalized   diffuse distribution  
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Grouped   cluster of lesions  
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Localized   limited area of involvement that are clearly defined  
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Satellite   single lesion in close proximity to a large grouping  
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Solitary   single lesion  
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Symmetric   bilateral distribution  
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Zosteriform   bandlike distribution along a dermatome area, like shingles  
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Common Assessment Abnormalities    
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Alopecia   loss of hair  
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Angioma   tumor consisting of blood or lymph vessels  
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Carotenemia   yellow discoloration of skin, no yellowing of sclera  
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Comedo   enlarged hair follicle plugged with sebum, bacteria and skin cells (blackhead, whitehead)  
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Cyanosis   slightly bluish grey discoloration of the skin, in dark skin ashen or gray color (check in eye and nail beds)  
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Cyst   sac containing fluid or semisolid material  
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Ecchymosis   large bruiselike lesion caused by collection of extravascular blood in dermis and subq. In dark skin deeper purple to brownish black, difficult to see.  
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Erythema   redness occurring in patches of variable size and shape. In dark skin Deeper brown or purple skin tone with increase in skin temp.  
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Hematoma   extravasation of blood of sufficient size to cause visible swelling  
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Hirsutism   male distribution of hair in women  
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Hypopigmentation   congential or acquired loss of pigment resulting in white patchy areas  
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Intertrigo   dermatitis of overlying surfaces of the skin  
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Jaundice   yellow discoloration of skin. In dark skin yellowish green color most apparent in sclera of eyes, palms of hands, soles of feet.  
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Keloid   hypertrophied scar beyond wound margins  
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Lichenification   thickening of the skin with accentuated skin markings  
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Mole   benign overgrowth of melanocytes  
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Petechiae   pinpoint discrete deposit of blood less then 1-2 mm in the extravascular tissues. In dark skin difficult to see but may be evident in mouth or eyes  
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Telangiectasia   visibly dilated superficial cutaneous small blood vessels (spider veins)  
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Tenting   failure of skin to return immediately to normal position after gentle pinching  
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Varicosity   increased prominence of superficial veins  
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Diagnostic Studies    
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Biopsy   punch, excisional, incisional, shave  
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Microscopic tests   culture of pustules  
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Wood’s lamp   long wave uv light (black light) - tinea shows as green  
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Patch test/allergy test   allergy testing  
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Malignant Skin Neoplasms    
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Diagnosis:ABCD   Asymmetry, Border irregularity, Color change/ variation – tan, brown, black, Diameter of 6 mm or more  
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Risk Factors   Fair skin, hx of chronic skin exposure, family hx of skin cancer, exposure to tar and systemic arsenicals. Living near the equator, outdoor living, tanning smoking  
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Nonmelanoma skin cancers    
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Actinic Keratosis (solar keratosis)   premaligant form of squamous cell carcinoma. Affects nearly all older whites, most common. Irregularly shaped, flat, slightly reddened papule with indistinct border.  
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Basal Cell Carcinoma   locally invasive malignancy arising from epidermal basal cells. Most common type of skin cancer (least deadly). B is best, rarely metastisizes  
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Squamous Cell Carcinoma   malignant neoplasm of keratinizing epidermal cells. Less common than BCC. Aggressive, metastatic  
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Malignant Melanoma   tumor arising in melanocytes which are the cells that produce melanin., metastisize to ANY organ. Most deadly skin cancer. Incidence increasing faster than others as well  
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Treatment of skin cancer   Surgical removal  
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Moh’s Surgery Incision   for suspected or known cancer. Pt under anesthesia, check right then to see if they got all the cancer. Removed in small slices, checked under microscope to see if there was still cancer, if so another slice. Repeat until the slide is clear.  
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Tx of Skin Cancer   Metastisized also needs chemotherapy  
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Skin Infections and Infestations    
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Bacterial Skin Infections   bacteria on skin cause infection. Staph A. and group B strep are most responsible for skin infections.  
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Impetigo   kids, mouth, nose and pierced ears. Bacterial postules, itchy. Antibiotics and warm compresses  
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Folliculitis   infected hair follicles, postules, painful, itchy cysts. antibiotics  
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Furuncles   hair follicles infection, necrotic sacs requiring incision and drainage. Antibiotics  
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Cellulitis   subq secondary infection. Hot, tender, red, fever, chills, malaise. Tx elevation, moist heat antibiotics  
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Viral Skin infections   virus can cause lesions. Antivirals for treatment  
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Herpes Simplex Virus   painful vesicles on the skin. (cold sore). Red, crusty, life long virus  
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Herpes Zoster   Shingles (varicella) common in immunocompromised. Happens along dermatomes  
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Veruca Vulgaris   wart (HPV) TX freezing  
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Plantar warts   foot warts TX freezing  
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Fungal skin infections   treat with antifungals  
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Candidiasis   yeast infection, any warm moist area  
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Tinea Corporis   ringworm, anywhere on body  
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Tinea Cruris   jock itch  
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Tinea pedis   athlete’s foot  
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Infestations and Insect Bites   bees, bedbugs, lice, scabies, ticks  
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Rash   in light skin may be visualized as well as felt. In dark skin not easy to see but can be felt  
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Common Allergic Conditions of the Skin    
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Allergic contact dermatitis   hypersensitivity reaction, red papules and plaqued, usually puss producing, frequently takes shape of causative agent. TX- topical corticosteroids, antihistamines  
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Urticaria (hives)   (histamine response) spontaneously occurring elevation, varying sizes, multiple, very itchy, can go with dermatitis. Tx- removal of cause, antihistamines, cool compresses  
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Drug Reaction   rash as last as 14 days after cessastion of drug common on face and chest. Tx- dc drug, antihistamine, corticosteroids  
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Atopic Dermatitis   pruritic, oozing, scaly discolored. Usually associated with autoimmune issues (associated with Asthma and allergic rhinitis in children) Tx lubrication of skin, corticosteroids  
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Common Benign Conditions of the Skin    
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Acne   noninflammatory lesions of skin. Tx- topicaly benzoyl peroxide, accutane  
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Nevi (moles)   hyperpigmented areas of skin. Tx-non necessary  
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Psoriasis   chronic dermatitis, silvery scales. Tx- reduce inflammation, corticosteroids, tars, UV light, immunosuppressants  
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Seborrhic Keratoses   irregularly round or oval papules or plaques. Well defined, itchy. Removal  
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Acrodhordons (skin tags)   small skin colored soft papules. Tx – none necessary  
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Lipoma   benign tumor of adipose tissue. Tx- none  
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Vitiligo   focal amelanosis (loss of pigment). Tx- topical steroids  
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Lentigo   (liver spots) areas of hyperpigmentation Tx. None, but monitor  
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Pallor   in light skin white or ashen. In dark skin underlying red tones are missing and they look ashen or grey.  
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