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Thrombocytopenia and DIC

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Question
Answer
thrombocytopenia   platelet count < 150,000  
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immune thrombocytopenia   platelets function normally but are destroyed by macrophages due to their coating with antibodies  
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treatment for ITP   corticosteroids, IV immunoglobulin, splenectomy  
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signs and symptoms of ITP   bruising, headache, SOB, confusion, fever, weakness  
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idiopathic causes of ITP   H.Pylori, flu or unknown  
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Thrombotic Thrombocytopenia Purpura   always associated with hemolytic-uremic syndrome and platelets aggregate which form micro-thrombi that are deposited into arterioles and veins  
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causes of TTP   idiopathic, drug reactions, pregnancy or autoimmune  
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TTP treatment   treat underlying cause, plasmapheresis (reverses platelet consumption), immunosuppressants  
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signs and symptoms of TTP   fever, neruo, renal, anemia, platelets  
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heparin induced thrombocytopenia and thrombosis syndrome   usually occurs 5-10 days after onset of heparin therapy  
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when to suspect HITTS   if platelet count falls past 50% or < 150,000  
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signs and symptoms of HITTS   bleeding, petechiae, pain/ tenderness, weakness of arm/ leg  
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clinical manifestations of internal blood loss   fainting, dizziness, tachycardia, weakness, abdominal pain, hypotension  
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treatment of HITTS   direct thrombin inhibitor to maintain anticoagulation (Lepirudin, Argatroban) plasmapheresis if severe to clear blood of platelet aggregating substances can give warfarin for thrombosis after platelet count is > 150,000  
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Disseminated Intravascular Coagulation (DIC)   results from abnormally initiated and accelerated clotting subsequent decreases in clotting factors and platelets ensue which can lead to uncontrollable hemorrhage  
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acute DIC risk factors   shock, septicemia, tissue damage, snake bite, head injury  
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subacute DIC risk factors   malignancy, obstetric  
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chronic DIC risk factors   liver disease, lupus, malignancy  
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bleeding clinical manifestations   skin - pallor, petechiae, hematomas respiratory- tachypnea, hemoptysis, orthopnea cardio- hypotension and tachypnea neurologic - vision changes, dizziness, changes in mental status, irritability  
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thrombotic clinical manifestations   skin- cyanosis, ischemic tissue necrosis, hemorrhagic necrosis, bloody stools respiratory- tachypnea, dyspnea, pulmonary emboli, ARDs cardio- ECG changes, venous distention renal failure, AKI, oliguria  
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diagnostic studies   prolonged PT, prolonged PTT, low fibrinogen, low platelets, elevated fibrin, elevated D-Dimer  
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medical management of DIC   stabilize (oxygen and volume replacement) heparin (Thrombosis) chronic DIC with no bleeding - treat cause chronic DIC with bleeding- blood products chronic DIC who does not respond to oral anticoagulants- long term heparin  
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nursing management of DIC   active assessment, minimize damage to tissue and protect patient from additional sources of bleeding early detection of bleeding  
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