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NPTE Neuromuscular

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Question
Answer
Myotatic reflex stimulus   muscle stretch  
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Myotatic reflex arc   Afferent Ia from muscle spindle to alpha MN and back to muscle  
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Myotatic reflex function   maintenance of muscle tone, support agonsist muscle contraction, provide feedback about muscle length  
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Myotatic reflex testing   DTR  
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Via an inhibitory IN the myotatic reflex inhibits the antagonist   Reciprocal inhibition  
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Myotatic reflex effect on synergistic muscles   facilitation  
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Inverse myotatic reflex stimulus   muscle contraction  
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Inverse myotatic reflex arc   Afferent Ib from GTO via inhibitory IN to muscle  
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Function of inverse myotatic reflex arc   provides agonist inhibition, decreases force of agonist, stretch protection reflex  
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Allows muscle tension to come under control of descending pathways   Gamma reflex loop  
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Gamma reflex loop path   excite gamma MN causing muscle spindle contraction then increased stretch sensitivity and increased firing from spindle afferents then conveyed to alpha MNs  
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Flexor withdrawal reflex stimulus   cutaneous sensory stimuli  
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Flexor withdrawal reflex arc   cutaneous receptors via Ins to flexor muscles  
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Flexor withdrawal reflex arc function   protective withdrawal mechanism  
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Crossed extension reflex stimulus and response   noxious stimuli – Flexors excited with extensor inhibition, opposite on CL side  
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Crossed extension reflex function   coordinates reciprocal limb activities such as gait  
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3 elements of Glasgow Coma Scale   eye opening, motor response, verbal response  
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Scoring of GCS   3-8 severe, 9-12 moderate, 13-15 minor  
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State defined by no eye opening even to pain, failure to obey commands, inability to speak   Coma  
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Return of sleep/wake cycles, normalization of basic functions, lack of cognitive responsiveness   Vegetative state  
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Central language DO with speech is awkward, restricted, interrupted, produced with effort   Expressive aphasia (Broca’s, nonfluent, motor)  
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Expressive aphasia result of   L hemisphere – Broca’s area  
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Impairment of volitional articulatory control 2/2 cortical dominant hemisphere lesion   Verbal apraxia  
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Impairment of speech production   Dysarthria  
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Central language DO where spontaneous speech preserved/smooth while auditory comprehension impaired   Receptive or Wernicke’s aphasa  
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Receptive aphasia is result of damage to   Posterior frist temporal gyrus of L hemisphere (Wernicke’s area)  
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Severe aphasia with impairments in comprehension & production of language   Global aphasia  
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Periods of apnea followed by gradually increasing depth/frequency of respirations   Cheyne Stokes respiration  
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Cheyne Stokes caused by   depression of frontal lobe and diencephalic dysfunction  
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Increased rate and depth of respirations   Hyperventilation  
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Hyperventilation can be caused by dysfunction of   lowere midbrain and pons  
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Abnormal respiration with prolonged inspiration   Apneustic breathing  
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Apneustic breathing is result of damage to   upper pons  
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Elevation of temperature may be damage to   hypothalamus or brainstem  
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Signs of meningeal irritation   Kernig’s sign, Brudinski’s sign, guarding in neck flexion, photophobia, disorientation, restlessness, persistent HA that increases with head down, altered vitals, weakness  
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Kernig’s sign   meningeal irritation. Supine with flexed hip/knee to chest then extend knee.  
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Brudzinski’s sign   meningeal irritation. Supine, flex neck. Causes flexion of hips/knees  
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Increased ICP leads to   restless, confused, decr LOC, incr BP, widening pulse P & slowed pulse, Cheyne-Stokes, elevated temp, HA, vomiting, unequal pupils, slowed PLRs, dilated pupils*, papilledema, weakness, hemiplegia, Babinski, decorticate or decerebrate rigidity, seizures  
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Joint position sense   test for ability to perceive joint position at rest in response to passive positioning  
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Kinesthesia   movement sense  
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Pallesthesia   vibration sense with tuning fork  
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Combined cortical sensation testing   discriminative sensory tests including Steriognosis, Tactile Localization, Two Point Discrimination, Bilateral Simultaneous Discrimination, Barognosis, Graphesthesia, Texture recognition  
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Stereognosis   ID familiar objects by touch  
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Two point discrimination   ability to recognize one or two blunt points applied to skin simultaneously  
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Barognosis   ability to differentiate weights  
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Graphesthesia   ability to ID numbers, letters or symbols traced on skin  
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Homonymous hemianopsia   loss of half of visual field in each eye contralateral to side of cerebral hemisphere lesion  
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Somatognosia   body scheme disorder – unable to ID body parts or relations to each other  
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Unilateral neglect   patient ignores one side of body and stimuli from that side  
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Anosognosia   severe neglect or denial of severity of condition  
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Figure ground discrimination   spatial relations syndrome with lack of ability to pick out object  
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Form constancy   pick out object from array of similar shapes but different sizes  
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Spatial relations   pt duplicates a pattern of 2-3 blocks  
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Topographical disorientation   navigation of a familiar route  
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Vertical disorientation   inability to accurately determine what is upright  
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Agnosia   inability to recognize familiar objects with one sensory modality  
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Apraxia   inability to perform voluntary learned movements in the absence of loss of sensation , strength, coordination, attention, or comprehension. Breakdown in conceptual or motor production system or both  
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Ideomotor apraxia   cannot perform task on command, but can do independently  
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Ideational apraxia   cannot perform the task at all, either on command or independently  
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Clasp-knife response   marked resistance to PROM suddenly gives way  
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Clonus   maintained stretch stimulus produces cyclical spasmodic contraction, usu plantar flexors or wrist flexors  
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Babinski   DF of great toe with fanning of other toes with stroke to lateral bottom of foot  
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Babinski indicates   corticospinal (pyramidal) tract disruption  
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Hyperreflexia   increased DTRs  
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Lead pipe Rigidity   uniform throughout range  
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Cogwheel rigidity   interrupted by series of jerks  
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Decerebrate posturing   increased tone in extension, seen in brainstem lesions between superior colliculus and vestibular nucleus  
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Decorticate posturing   UEs in flexion, LEs in extension, seen in brainstem lesions above superior colliculus  
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Opisthotonos   arching back of head back and heels with UEs rigidly flexed. Seen in severe meningitis, tetanus, epilepsy, strychnine poisoning  
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Plantar reflex   Normal is PF of toes in response to stroking lateral sole of foot from calcaneus to 5th met, S1-2, tibial nerve  
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Abdominal reflex   T6-L1, lateral to medial scratching of skin to umbilicus in each of 4 quadrants should cause deviation of umbilicus to stimulus  
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Cremasteric reflex   L1-L2, stroking of skin on inner thigh elevates testicle, lost in SCI and Corticospinal lesions  
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Sources of fatigue   CNS/central fatigue, neural/myoneural fatigue, muscle contractile failure  
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CNS/Central fatigue   in MS, ALS, CFS  
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Neural/myoneural junction fatigue   MS, Post Polio syndrome, GBS, myasthenia gravis  
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Muscle contractile failure fatigue   metabolic changes at muscle, muscular dystrophies  
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Extrapyramidal disorders (basal ganglia dysfunction)   Tics, Chorea, athetosis, tremors, myoclonus  
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Tics   spasmodic contractions of specific muscles  
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Chorea   relatively quick twitches or dancing movments  
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Athetosis   slow, irregular, twisting movements, esp in UEs  
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Tremor   continuous quivering mvmt, rhythmic, oscillatory observed at rest  
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Myoclonus   single, quick jerk  
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Cerebellar disorders cause   intention tremors  
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Cortical disorders cause   seizures, tonic/clonic convulsive mvmts  
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Dyssynergia   impaired ability to associate muscles together for complex mvmt  
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Dysmetria   impaired ability to judge distance or range of movement  
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Dysdiadochokinesia   impaired ability to perform rapid alternating movements  
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