Ch. 1-5
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| HOW MANY AMERICANS DIE FROM CARDIOVASCULAR DISEASE EACH YEAR? | 950,000
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| WHAT IS THE LEADING CAUSE OF PREMATURE, PERMANENT DISABILITY AMONG WORKING ADULTS? | CORONARY HEART DISEASE
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| NONMODIFIABLE FACTORS OF CARDIOVASCULAR DISEASE | HEREDITY, RACE, GENDER, AGE
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| MODIFIABLE FACTORS OF CARDIOVASCULAR DISEASE | HIGH BP, ELEVATED SERUM CHOLESTEROL LEVELS, TOBACCO USE, DIABETES, PHYSICAL INACTIVITY, OBESITY, METABOLIC SYNDROME
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| NORMAL BP | 120/80
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| PREHYPERTENSION | 120-139/80-89
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| STAGE 1 HIGH BP | 140-159/90-99
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| STAGE 2 HIGH BP | GREATER THAN OR = TO 160/100
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| QUITTING SMOKING REDUCES THE RISK OF HEART DISEASE BY WHAT % AFTER 1 YEAR | 50%
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| STUDIES HAVE SHOWN THAT EVEN WHAT % REDUCTION IN BODY WEIGHT REDUCES THE RISKS ASSOCIATED WITH OBESITY | 10%
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| WHAT IS THE BODY MAX INDEX FORMULA | WEIGHT IN LBS / HEIGHT IN INCHES * 704.4
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| CARDIAC CAUSES OF CARDIAC ARREST | CORONARY ARTERY DISEASE (MOST COMMON), DYSRHYTHMIAS, ACUTE MI, VALVULAR HEART DISEASE, CHD, INTRACARDIAC TUMOR
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| NON-CARDIAC CAUSES OF CARDIAC ARREST | PULMONARY EMBOLISM, CHOKING, ASPHYXIA, DRUGS, STROKE, HYPOXIA, ALCOHOLISM
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| CHAIN OF SURVIVAL FOR CARDIAC | EARLY ACCESS, EARLY CPR, EARLY DEFIB, EARLY ACLS
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| COMPONENTS OF BASIC LIFE SUPPORT | RECOGNITION OF SIGNS OF HEART ATTACK, CARDIAC ARREST, STROKE, FBAO, RELIEF OF FBAO, CPR, DEFIB
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| SHOCKABLE RHYTHMS | V-TACH, V-FIB
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| NON-SHOCKABLE RHYTHMS | ASYSTOLE, NO PULSE
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| COMPONENTS OF ADVANCED CARDIAC CARE | BASIC LIFE SUPPORT, ADVANCED AIRWAY MNG, VENTILATION SUPPORT, ECG RECOGNITION, ECG INTERPRETATION, VASCULAR ACCESS AND FLUID RESUSCITATION, DEFIB, SYNCHRONIZED CARDIOVERSION, PACING, MEDS, CORONARY ARTERY BYPASS, STENT INSERTION, ANGIOPLASTY
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| PHASES OF CPR | ELECTRICAL PHASE (FIRST 5 MIN), CIRCULATORY PHASE (5 MIN TO 10-15 MINS), METABOLIC PHASE (AFTER 10-15MIN)
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| WHEN DO YOU REPEAT THE PRIMARY SURVEY | CHANGE IN PT'S CONDITION, INTERVENTIONS NOT WORKING, VITALS UNSTABLE, BEFORE ANY PROCEDURES, CHANGE IN RHYTHM
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| WHAT IS SECONDARY SURVEY | ADANCED AIRWAY, BREATHING, CIRCULATION, DIAGNOSIS, EVALUATE, FACILITATE
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| TYPES OF ADVANCE DIRECTIVES | LIVING WILL, PATIENT SELF DETERMINATION ACT, DURABLE POWER OF ATTORNEY FOR HEALTHCARE
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| DEFINE CARDIAC ARREST | ABSENCE OF CARDIAC MECHANICAL ACTIVITY, NO PULSE, UNRESPONSIVE, ABNEA OR AGONAL BREATHING
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| WHAT DOES THE UPPER AIRWAY CONSIST OF | NASOPHARYNX, OROPHARYNX, LARYNGOPHARYNX
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| NASAL CANULA LITERS AND FIO2 | 1L=24%2L=28%3L=32%4L=36%5L=40%6L=44%
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| SIMPLE MASK LITERS AND FIO2 | 8-10L & 40%-60%
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| THE RIGHT CORONARY ARTERY ORIGINATES FROM | RIGHT SIDE OF THE AORTA
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| LEFT CORONARY ARTERY ORIGINATES FROM | LEFT SIDE OF THE AORTA
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| WHAT IS DEPOLARIZATION | BEFORE A CONTRACTION, GETTING READY TO CONTRACT, PULSELESS ELECTRICAL ACTIVITY
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| POLARIZATION IS | READY STATE
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| DEPOLARIZATION | STIMULATION
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| REPOLARIZATION | RECOVERY
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| THE POSITION OF THE __ ELECTRODE ON THE BODY DETERMINES THE PORTION OF THE HEART "SEEN" BY EACH LEAD | POSITIVE
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| 3 LEADS THAT LOOK AT THE INFERIOR WALL OF THE LEFT VENTRICLE | LEADS 2, 3 AND AVF
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| 2 LEADS THAT LOOK AT THE ANTERIOR WALL OF THE LEFT VENTRICLE | V3 & V4
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| 4 LEADS THAT LOOK AT THE LATERAL WALL OF THE LEFT VENTRICLE | LEAD 1, AVL, V5, V6
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| ___ PLANE LEADS VIEW THE HEART AS IF THE BODY WERE SLICED IN HALF | HORIZONTAL/TRANSVERSE
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| ECG PAPER... WHAT SIZE ARE SMALL BOXES | 1MM WIDE AND 1MM HIGH
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| ECG PAPER RECORDS AT A SPEED OF | 25MM/SEC
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| EACH HORIZONTAL UNIT REPRESENTS HOW MANY SEC | .04 SEC OR 1MM
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| A LARGE BOX REPRESENTS | .20 SEC
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| DEFINE WAVEFORM | A MOVEMENT AWAY FROM BASELINE EITHER POS OR NEG
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| DEFINE SEGMENT | A LINE BETWEEN WAVEFORMS
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| DEFINE INTERVAL | A WAVEFORM AND A SEGMENT
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| DEFINE COMPLEX | SEVERAL WAVEFORMS
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| WHAT IS A P WAVE | FIRST WAVE IN CARDIAC CYCLE, ATRIAL DEPOLARIZATION, SMOOTH, ROUNDED AND ABOUT 0.11 SEC
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| WHAT IS QRS COMPLEX | Q IS FIRST AND ALWAYS NEG, R IS POS, S IS NEG, VENTRICULAR DEPOLARIZATION
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| WHAT IS T WAVE | VENTRICULAR REPOLARIZATION, UPRIGHT EXCEPT IN LEAD AVR
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| NEGATIVE (INVERTED) T WAVE = | MYOCARDIAL ISCHEMIA
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| PEAKED T WAVE = | HYPERKALEMIA
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| LOW AMPLITUDE T WAVES = | HYPOKALEMIA
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| PR INTERVAL MEASURES | 0.12-0.20 SEC
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| ST SEGMENT REPRESENTS | EARLY PART OF REPOLARIZATION OF THE RIGHT AND LEFT VENTRICLES
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| QT INTERVALS REPRESENTS | TOTAL VENTRICULAR ACTIVITY
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| QT MEASURES | 0.36-0.44 SEC
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| PROLONGED QT = | LENGTHENED RELATIVE REFRACTORY PERIOD
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| 3 STEPS TO ASSESS THE RATE ON STRIPS | 6 SEC METHOD, LARGE BOXES, SMALL BOXES
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| STEPS TO ANALYZE A RHYTHM STRIP | ASSESS THE RATE, ASSESS RHYTHM, EXAMINE P WAVES, ASSESS INTERVALS, OVERALL APPEARANCE, INTERPRET
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| CHARACTERISTICS OF SINUS ARRHYTHMIA | RATE= 60-100RHYTHM= IRREGULARP WAVES= UNIFORMPR INTERVAL= CONSTANTQRS DURATION= 0.10 SEC OR LESS
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| CHARACTERISTICS OF SINUS TACHY | RATE= 101-180RHYTHM= REGULARP WAVES= UNIFORMPR INTERVAL= CONSTANTQRS DURATION= 0.10 SEC OR LESS
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| CAUSES OF SINUS TACH | EXERCISE, FEVER, PAIN, FEAR, HYPOXIA, INFECTION, SHOCK, CAFFEINE, NICOTINE
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| HOW IS ATRIAL TACH DIFFERENT FROM SINUS TACH | ATRIAL P WAVES DIFFER IN SHAPE
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| CHARACTERISTICS OF ATRIAL TACHY | RATE=150-250; RHYTHM=REG; P WAVES= DIFFER IN SHAPE; PR INTERVAL=SHORTER OR LONGER, P WAVE MAY BE HIDDEN IN T WAVE; QRS DURATION=0.10 SEC
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| WHAT IS AVNRT | ATRIOVENTRICULAR NODAL REENTRANT TACHYCARDIA
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| CHARACTERISTICS OF AVNRT | RATE=150-250; RHYTHM=NORM; P WAVES=HIDDEN IN QRS; PR INTERV=NOT MEASURED; QRS DUR=0.10 SEC
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| CHARACTERISTICS OF WOLFF-PARKINSON-WHITE SYNDROME | RATE=60-100;
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| HOW DO YOU RECOGNIZE WPW | SHORT PR INTERVAL, DELTA WAVE, WIDENING OF THE QRS
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| WHAT ARE VAGAL MANEUVERS | METHODS USED TO STIMULATE BARORECEPTORS LOCATED IN THE INTERNAL CAROTID ARTERIES AND THE AORTIC ARCH
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| MAT IS MOST OFTEN SEEN IN | SEVERE COPD, HYPOXIA, ACUTE CORONARY SYNDROME, DIGOXIN TOXICITY, RHEUMATIC HEART DISEASE, THEOPHYLLINE TOXICITY, ELECTROLYTE IMBALANCES
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| ATRIAL FLUTTER IS | ECTOPIC, SAW TOOTH
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| CONDITIONS ASSOCIATED WITH A-FLUTTER | HYPOXIA, PULMONARY EMBOLISM, CHRONIC LUNG DISEASE, PNEUMONIA, CARDIAC SURGERY
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| FIRST DEGREE AV BLOCK | P WAVES CONDUCTED BUT DELAYED
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| SECOND DEGREE AV BLOCK | SOME P WAVES CONDUCTED
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| THIRD DEGREE AV BLOCK | NO P WAVES CONDUCTED
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| DEFIB INDICATIONS | PULSELESS VT, VF, SUSTAINED POLYMORPHIC VT
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| WHAT ARE THE MOST IMPORTANT TREATMENTS FOR THE PTS IN CARDIAC ARREST DUE TO PULSELESS VT OR VE | DEFIB AND CPR
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| ENERGY (JOULES)= | AMPS * VOLTS * TIME
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| TRANSTHORACIC RESISTANCE IS ALSO KNOWN AS | IMPEDANCE
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| WHAT FACTORS AFFECT IMPEDANCE | PADDLE SIZE, POSITION, USE OF CONDUCTIVE MATERIAL, PHASE OF PT'S RESPIRATION, PRESSURE, ENERGY
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| INCREASED RESISTANCE = | DECREASED CURRENT DELIVERY
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| CRITICAL RESUSCITATION TASKS | AIRWAY MNG, CHEST COMPRESSIONS, MONITORING AND DEFIB, VASCULAR ACCESS AND MEDS
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| WHAT DO U DO WHEN A "FLAT LINE" IS OBSERVED ON A CARDIAC MONITOR | MAKE SURE POWER IS ON, CHECK CONNECTIONS, MAKE SURE CORRECT LEAD IS SELECTED, TURN UP ECG SIZE ON MONITOR
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| AED OPERATION | TURN ON, ATTACH, ANALYZE, DELIVER
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| SYNCHRONIZED CARDIOVERSION INDICATIONS | UNSTABLE SUPRAVENTRICULAR TACHY, UNSTABLE ATRIAL FIB WITH RAPID VENT RESPONSE, UNSTABLE A FLUTTER WITH A RAPID VENT RESPONSE, UNSTABLE WIDE-COMPLEX TACHY, UNSTABLE VT WITH A PULSE
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| DEFIB AND CARDIOVERSION COMPLICATIONS | SKIN BURNS, RISK OF FIRE, MYOCARDIAL DAMAGE, EMBOLIC EPISODES, DYSRHYTHMIAS, INJURY TO OPERATOR
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| DEFIB AND CARDIOVERSWION POSSIBLE ERRORS | TREATING THE MONITOR, NOT THE PATIENT, OPERATOR UNFAMILIAR WITH EQUIPMENT, FAILURE TO PROPERLY MAINTAIN EQUIPMENT
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| TRANSCUTANEOUS PACING INDICATIONS | SYMPTOMATIC BRADY, NARROW QRS THAT DOES NOT RESPOND TO ATROPINE, WIDE QRS BRADY
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| PACEMAKER COMPLICATIONS | COUGHING, SKIN BURNS, PAIN, TISSUE DAMAGE,
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| FAILURE TO PACE | FAILS TO DELIVER AN ELECTRICAL STIMULUS OR WHEN IT FAILS TO DELIVER THE CORRECT NUMB OF ELECTRICAL STIMULATIONS PER MIN.
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| FRAILURE TO CAPTURE (PACE) | INABILITY OF A PACEMAKER STIMULUS TO DEPOLARIZE THE MYOCARDIUM.
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| WHAT FACTORS DO YOU CONSIDER WHEN SELECTING IV SITES | PURPOSE, AMOUNT AND TYPE OF FLUIDS, DURATION, ACCESSIBILITY, SIVE OF VEIN, EXPERIENCE
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