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These drugs affect the Autonomic Nervous System

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Drug Name
Drug Classification
Trade Name
Used to Treat
Carbamylcholine   Direct-Acting Cholinergic   N/A   Atony of the GI tract and to stimulate uterine contractions in swine  
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Bethanechol   Direct-Acting Cholinergic   Urecholine   GI and urinary tract atony  
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Pilocarpine   Direct-Acting Cholinergic   Isopto Carpine, Akarpine, Pilocar   Reduces intraocular pressure associated with glaucoma  
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Metoclopramide   Direct-Acting Cholinergic   Reglan   Control vomiting and to promote gastric tract emptying  
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Edrophonium   Indirect-Acting Cholinergic   Tensilon   Diagnose myasthenia gravis  
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Neostigmine   Indirect-Acting Cholinergic   Prostigmine, Stiglyn   Urine retention, GI atony, and used as antidote to neuromuscular blocking agents  
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Physostigmine   Indirect-Acting Cholinergic   Antilirium, Eserine   Urine retention, GI atony, and used as antidote to neuromuscular blocking agents  
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Organophosphate compounds   Indirect-Acting Cholinergic   N/A   Used as insecticide dips  
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Demecarium   Indirect-Acting Cholinergic   Humorsol   Preventive management of glaucoma  
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Pyridostigmine   Indirect-Acting Cholinergic   Mestinon   Treatment of myasthenia gravis  
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Atropine   Cholinergic Blocking Agent   Numerous generic/trade names available for parenteral or opthalmic administration   Preanesthetic, counteract organophosphate poisoning, dilate pupils for opthalmic exam, control ciliary spasms of eye, treat bradycardia, slow hypermotile gut  
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Methscopolamine   Cholinergic Blocking Agent   Biosol-M   Control diarrhea  
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Glycopyrrolate   Cholinergic Blocking Agent   Robinul-V   Similar to atropine (longer duration of action), primarily used as preanesthetic  
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Aminopentamide   Cholinergic Blocking Agent   Centrine   Control vomiting and diarrhea in dogs and cats  
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Propantheline   Cholinergic Blocking Agent   Pro-Banthine   Diarrhea, urinary incontinece, bradycardia; also used to reduce colonic peristalsis in horses to allow rectal exam  
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Pralidoxime   Cholinergic Blocking Agent   Protopam, 2-PAM   Organophosphate intoxication  
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Epinephrine   Adrenergic Agent   Adrenalin   Stimulates all receptors to cause increase in HR and CO, constriction of blood vessels in skin, dilation of blood vessels in muscle dilation of bronchioles, increase of metabolic rate  
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Norepinephrine   Adrenergic Agent   Levophed   Vasopressor (raise blood pressure)  
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Isoproterenol   Adrenergic Agent   Isuprel   Bronchodilation  
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Phenylephrine   Adrenergic Agent   Neo-Synephrine   Nasal vasoconstrictor  
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Dopamine   Adrenergic Agent   Intropin   Shock, congestive heart failure, increase renal perfusion  
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Phenylpropanolamine   Adrenergic Agent   Proin   Urinary incontinence in dogs  
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Dobutamine   Adrenergic Agent   Dobutrex   Short-term treatment of heart failure  
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Albuterol   Adrenergic Agent   Proventil   Bronchodilation  
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Phenoxybenzamine   Adrenergic Blocking Agent (Alpha Blocker)   Dibenyline   Hypotensive (vasodilator) agent  
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Aacepromazine   Adrenergic Blocking Agent (Alpha Blocker)   Ace   Tranquilizer; vasodilation  
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Prazosin   Adrenergic Blocking Agent (Alpha Blocker)   Minipress   Hypotensive agent  
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Yohimbine   Adrenergic Blocking Agent (Alpha Blocker)   Yobine   Antidote for xylazine toxicity  
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Atipamezole   Adrenergic Blocking Agent (Alpha Blocker)   Antisedan   Reversal agent for dexmedetomidine  
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Propranolol   Adrenergic Blocking Agent (Beta Blocker)   Inderal   Cardiac arrythmias and hypertrophic cardiomyopathy  
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Timolol   Adrenergic Blocking Agent (Beta Blocker)   Timoptic   Opthalmic preparation to treat glaucoma  
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Atenolol   Adrenergic Blocking Agent (Beta Blocker)   Tenormin   Cardiac arrythmias and hypertrophic cardiomyopathy  
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Carteolol   Adrenergic Blocking Agent (Beta Blocker)   Ocupress   Human labeled antiglaucoma medication  
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Levobunolol   Adrenergic Blocking Agent (Beta Blocker)   Betagan   Human labeled antiglaucoma medication  
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Metipranolol   Adrenergic Blocking Agent (Beta Blocker)   OptiPranolol   Human labeled antiglaucoma medication  
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Acetylcholine   Direct-Acting Cholinergic   N/A   seldom used clinically because it is rapidly broken down by acetylcholinesterase  
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