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Problems of Oxygenation & Perfusion: Nrs Management of Dysrhythmias

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Question
Answer
perfusion   movement of oxygen - bringing O2 to the cells (delivering)  
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telemetry monitoring   remote monitoring of a patient. 3 electrodes & a little box (usually kept in the pocket).  
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EKG   tracing. road map of the heart. where does the message originate, which is it communicating with, where does it go to make the contraction happen.  
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dysrhythmias   electrical message in the heart "goes a different way." there is some problem that makes the impulse take a different route.  
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properties of cardiac tissue   automaticity; excitability; conductivity; contractility.  
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automaticity   the ability to initiate an impulse. SA node (where the impulse starts)  
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excitability   ability to be electrically stimulated.  
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conductivity   ability to transmit an impulse along the system. SA node communicates to the AV node.  
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Contractility   heart muscle responds to impulse and contracts  
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conduction system of the heart   originates in SA node -> (intraatrial pathways to LA) internodal pathways to AV node -> bundle of His -> R&L bundle branch -> purkinje fibers --> ventricular contraction  
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heart block   something is blocking the conduction pathway.  
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Autonomic NS   responsible for normal, regular control (rate of impulse formation, speed of conduction, strength of contraction).  
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parasympathetic NS   Vagus nerve is main nerve.  
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Vagal Nerve Stimulation   dec HR, slowed conduction, decreased force of contraction,  
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causes of Vagal Nerve Stimulation   valsalva maneuver (bearing down) -- this is a nursing action for high HR, panic attack  
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sympathetic NS   opposite of parasympathetic. increased HR, increased force of contraction  
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P wave   atrial depolarization  
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PR interval   beginning of P wave to beginning of QRS complex. time it takes for the atria to depolarize and repolarize  
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QRS complex   ventricular depolarization.  
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ST segment   flat line in normal heart. interval from the end of ventricular depolarization to the beginning of ventricular repolarization.  
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T wave   ventricular repolarization  
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QT interval   total time for ventricular depolarization and repolarization  
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ECG - vertical axis   electrical potential  
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ECG - horizontal axis   time - how long it takes  
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ECG - 5mm square   0.2 seconds x 0.5 mV  
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ECG 1mm square   0.04 seconds x 0.1 mV  
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Normal Sinus Rhythm   60-100 bpm. follows normal conductive pathway. look for P wave - P wave present = NSR.  
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Sinus Bradycardia   sinus fires < 60 bpm. normal rhythm in aerobically trained athletes. can be problem w vagal nerve stim. some drugs do this (parasymphathomimetics). hypothermia can cause.  
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Sinus Bradycardia: Sx and Clinical Manifestations   hypotensive. hypoxia. weak. dizzy. nauseous (if due to vagal nerve stim). angina. confusion.  
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Parasymphathomimetic Drugs   mimic the parasympathetic NS. side effect = slows HR. ex: Pilocarpine (for dry mouth).  
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Sinus Bradycardia: Treatment   Atropine. Pacemaker  
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Atropine   anticholinergic drug. increases HR. may be given to reverse effects of muscarinic drugs (parasympathomimetics).  
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Sinus Tachycardia   discharge rate from the sinus node is increased as a result of vagal inhibition. > 100 bpm.  
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Sinus Tachycardia: Causes   anxiety. exercise. pain. fever. hypovolemia.  
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Sinus Tachycardia: Clinical Manifestations   hypotension. dizzy, lightheadedness, SOB, angina (not enough O2 to the heart)  
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Sinus Tachycardia: Treatment   beta-blocker (not for BP, to control/decrease HR). antipyretic (if caused by fever). analgesia (if caused by pain). IV fluids, increase oral intake (if caused by hypovolemia)  
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Treatment of Sinus Rhythms   treat the CAUSE to get the patient out of that rhythm  
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Normal Sinus Rhythms   NRS, sinus bradycardia, sinus tachycardia.  
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Premature Atrial Contraction   NOT NSR. irregular. ectopic beat occurs occasionally, originating not from SA node. no lethal.  
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Premature Atrial Contraction: causes   emotional stress, too much caffeine, too much alcohol, too much tobacco, electrolyte imbalances (some), COPD.  
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Premature Atrial Contraction: significance with heart disease   can be a warning. check labs (K & Na). have meds been taken?take apical pulse 1 min. inform provider.  
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Premature Atrial Contraction: Treatment   depends on the symptoms. find and eliminate the source. drugs secondary. beta-blocker (to control the rate). decrease caffeine/nicotine intake, halter monitor.  
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Paroxysmal Supraventrical Tachycardia (PSVT)   person is in NSR, run of tachy, then back to NSR. > 130 bpm in 6s strip (up to 200 bpm). sustained or non-sustained.  
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PSVT - sustained   emergency. BP can bottom out.  
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PSVT: Cauess   over exertion. stimulants. emotional stress. digitoxicity. known CAD.  
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Digitoxicity signs   nausea. lack of appetite. "yellow halos" in vision. blurry vision.  
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PSVT: causes in known CAD   could mean things are not going well. take vitals, symptoms, info from tele-clerk. Report to Dr.  
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PSVT: Clinical Manifestations   c/o palpitations. Hypotensive. dyspnea. angina  
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PSVT: Treatment   vagal maneuvers. if not effective, then drugs. Adenosine  
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Adenosine   potent anti-arhythmic. interrupts pathway to the AV node, slows AV node conduction, which slows HR. rapid response team will push this (not you - unless told to). want monitor by bedside.  
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Atrial Flutter   originates from a single ectopic focus w/in atria. very typical pattern (sawtooth mtns). tx depends on what pattern looks like.  
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Atrial Flutter - rapid ventricular response   TX directed at controlling vent rate (~160 bpm).BP drops, dizzy lightheaded, decreased CO.  
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Atrial Flutter: associated with...   CAD, HTN, valve disorders, lung disease  
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Atrial Flutter: risks   risk for stroke. risk for clots (blood not ejected from As, stasis --> clots --> stroke).  
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Atrial Flutter: clinical significance   has to do with how fast the ventricles are contracting.  
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Atrial Flutter: Treatment   goal: slow HR. beta-blockers, Ca Channel Blockers, anti-dysrhythmic. elective cardioversion.  
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Amdiodarone   anti-dysrhythmic. slows sinus rate, increases PR interval. many side effects. IV push under controlled circumstances. more potent than beta- or Ca- blockers.  
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elective cardioversion   controlled shock to get the heart out of a bad rhythm.  
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Atrial Fibrillation   irregular impulse coming from multiple ectopic focuses in atria. less organized than flutter, atria become very disorganized. most common dysrhythmia. prevalence increases w age.  
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Atrial Fibrillation: common with...   CAD. HF (sometimes first sign of HF). alcohol intoxication. too much caffeine. electrolyte imbalances (K & Na). post-cardiac surgery.  
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Atrial Fibrillation: Clinical Manifestations   exactly same as A flutter. decreased CO. some may have rapid ventricular response. high risk for embolus.  
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Atrial Fibrillation: Goals of Treatment   rapid ventricular response: slow HR, prevent thrombus formation. drugs: beta-blockers, Ca channel blockers, amiodarone. elective cardioversion.  
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Atrial Fibrillation: Elective Cardioversion   can only be done if we know when the AFib started. Don't want to cardiovert someone with thrombi. unknown: anticoagulant for 3-4 weeks.  
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Premature Ventricular Contractions (PVC): causes   stimulants (caffeine, alcohol, epinephrine, recreational drugs). electrolyte imbalances (K, usually too low). Known CAD.  
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PVC: known CAD or lasix usage   watch for these patients. lasix - pulls fluid off, K follows fluid, K imbalance.  
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PVC: Clinical Manifestations   CO can fall. hypoxia. angina.  
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PVC: Treatment   O2 (hypoxia, angina). electrolyte replacement (K and Na). drugs (beta-blockers, amiodarone, lidocaine).  
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Multifocal PVCs   QRSs are not the same. tells us that an impulse is coming from different places (weaker/stronger). not much of Q at first glance (distorted and wide = ineffective CO). patterns (couplets or triplets = both are bad).  
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PVC: Ventricular Bigeminy   one normal complex, then wide distorted one, repeate  
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Ventricular Tachycardia   EMERGENCY. can move from pulse VTach to pulseless VTach. activate rapid response team.  
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V Tach: Treatment   shock them. ACLS protocol.  
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Ventricular Fibrillation   can see this when they have an MI and progress to VFib.  
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