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a MCPHS- Provider I- Ch 27 Management of Pts w/Dysrhythmias

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Question
Answer
Dysrhythmias are disorders of   Formation/Conduction of heart’s electrical impulses  
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Diagnose dysrhythmias   ECG  
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Dysrhythmia naming   Site of origin, Mechanism of formation, Conduction involved  
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Sympathetic stimulation vs. Parasympathetic stimulation r/t Heart rate, AV node conduction & Force of myocardial contraction   S:all increase, P:all decrease  
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Chronotropy vs. Dromotropy vs. Inotropy   C:heart rate, D:AV node conduction, I:force of myocardial contraction  
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# of electrodes r/t 12-lead ECG   10 w/6 on chest and 4 on limbs  
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Limb electrode placement   Non-bony areas w/out major movement  
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12-lead ECG reflects electrical activity in   Left ventricle  
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Horizontal axis r/t ECG strip   Time & Rate  
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Vertical axis r/t ECG strip   Voltage/Amplitude  
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Positive deflection vs. Negative deflection   P:ECG waveform moves to top of strip, N:ECG waveform moves to bottom of strip  
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P Wave r/t Atria   Atrial depolarization  
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QRS Complex r/t Ventricle   Ventricular depolarization  
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T Wave r/t Ventricle   Ventricular repolarization  
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Atrial Repolarization r/t ECG strip   Occurs during QRS Complex  
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U Wave   Repolarization of Purkinje fibers  
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PR Interval   Time from SA node stimulation to conduction through AV node, Does not include ventricular depolarization  
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ST Segment r/t Isoelectric line   Analyzed to determine if below/above isoelectric line  
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QT Interval   Total time for ventricular depolarization and repolarization  
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Prolonged QT intervals put Pt at risk for   Torsade de pointes  
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Type of dysrhythmia r/t Torsade de pointes   Ventricular  
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TP Interval   No electrical activity, Isoelectric line  
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PP Interval vs. RR Interval   PP:determines atrial rhythm & rate, RR:determines ventricular rate & rhythm  
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Each small box on ECG strip represents   .04 seconds  
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If RR & PP intervals are same or < .8 seconds   Regular rhythm  
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Contributing factors r/t Bradycardia   H’s and T’s  
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3 H’s r/t Bradycardia   Hypovolemia, Hydrogen ions(acidosis), Hypoglycemia  
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3 T’s r/t Bradycardia   Toxins, Thrombosis, Trauma  
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Sinus bradycardia vs. Sinus tachycardia vs. Normal sinus rhythm   Only difference is rate  
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Sinus arrhythmia r/t Respiration   Increases w/inspiration, Decreases w/expiration  
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Atrial flutter r/t AV node conduction   Not all atrial impulses are conducted into ventricle, Therapeutic block at AV node  
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P-wave shape r/t Atrial flutter   Saw-tooth shape  
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Occurs when AV node becomes pacemaker   Junctional/Idionodal rhythm  
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When P waves cannot be identified   Supraventricular tachycardia(SVT)  
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SVT indicates only that rhythm is not   Ventricular tachycardia  
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Emergencies r/t Dysrhythmia   Ventricular tachycardia, Ventricular fibrillation, Idioventricular rhythm, Ventricular asystole  
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Ventricular fibrillation characteristics   Absence of: heartbeat, palpable pulse & respirations  
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AHA guidelines r/t Unconscious adults who experience cardiac arrest d/t Ventricular fibrillation   Induce mild hypothermia for 12-24 hours  
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Idioventricular rhythm   Purkinje fibers become pacemaker  
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Ventricular asystole synonym   Flatline  
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AV blocks occur when   AV node conduction is decreased/stopped  
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