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mott ob peds 104

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Answer
functions of amniotic fluid   Allows free movement _ Prevent adherence _ Cushions _ Maintains temperature _ Provides homeostasis _ Prevents cord compression  
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ductus arteriosus   diverts blood from pulmonary artery to aorta. closes functionally 15h completely 3weeks  
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Foramen ovale   window left atrium to right, closes temp 2h completely 3months  
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ductus venosus   diverts blood away from liver as it returns from placenta, closes functionally when cord is cut and completely in one week  
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3 categories of pregnancy signs   *presumptive:signs and symptoms that may resemble pregnancy *probable:indicate pregnancy the majority of the time *positive:medically proven  
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presumptive   amenorrhea, N&V, breast tenderness, urinary frequency, fatigue, quickening(tummy movement)  
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Probable   Uterine enlargement _ Pigmentation changes _ Positive pregnancy test _ Chadwick’s sign _ Ballottement _ Braxton Hick’s _ Goodell’s sign _ Hegar’s sign  
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positive   fetal heart sounds palpating active fetal movments visualizing fetus by U/S  
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changes in endocrine system during pregnancy   first produced in luteum and then by placenta, maintain pregnancy ESTROGEN, AND PROGESTERONE  
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HPL   affects the metabolic system of the maternal organism. HPL decreases maternal insulin sensitivity, and, therefore, raises maternal blood glucose levels, whilst decreasing maternal glucose utilization  
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human chorionic gonadotropin or simply (hCG).   This hormone is produced by fetal trophoblast cells. It binds to the luteinizing hormone receptor on cells of the corpus luteum, which prevents luteal regression. Thus, hCG serves as the signal for maternal recognition of pregnancy.  
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changes of the reproductive system during pregnancy. (continued)   breast preped to lactate by hpl, estrogen, progesterone,prolactin. rapidly increase in size first 8weeks from vascular engorgment  
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changes of the cardiovascular system during pregnancy.   orthostatic hypertension, venacava compression by uterus+baby(supine hypotensive syndrome)cardiac output doubles, 40% increase in volume. clotting factors increase  
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when is the placenta fully functional   at 3 months the placenta is fully operational, begins to function 4th week  
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scheduled prenatal visits   every month for first 7months. every 2 weeks during the 8th month. weekly during the 9th month  
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discomforts of the first trimester   N&V, frequency, breast tenderness vaginal discharge  
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interventions for first trimester discomforts   N&V (eat dry crackers in am)supportive bra for tenderness. vaginal discharge=hygiene cotton panties contact dr for change in color odor character  
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interventions for second trimester   heart burn(pyrosis)maalox or gelusil no sodium bicarb or alka seltzer.gas and constipation=water 8glasses,exercise roughage. hemorrhoids= cool witch hazel, sitz bath, up dietary fiber leg cramps= eval calcium intake. round lig pain= heat pack, pillow abd  
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vaccinations for pregnancy   tetnus during second trimester. Tdap given after delivery. in short viruses can cross placenta.!!!!!MMR 28 days later  
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danger signs of pregnancy   headaches visual disturbances. increase in systolic 30+ diastolic pressure of 15+,or over 140/80. pain,s/s of UTI, decreased fetal movement, fever 100.4+, bleeding fluid loss(wt loss) seizures  
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normal wt gain   25-35 lbs  
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biophysical profile (BPP)   assess fetal oxygenation done after 26 weeks.1 breathing movements,2 gross fetal movements, 3 FHR variability and reactivity (NST),4 fetal heart tones 5 amniotic volume  
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percutaneous umbilical blood sampling   obtain fetal blood sample from umbilicus detects anemia,Rh isoimmunizaton, chromosomal disorders and acid base balance done after 16 weeks  
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chorionic villus sampling   Small amount of placenta is obtained _ Vaginally or abdominally _ To analyze fetal cells for genetic abnormalities(sickle cell, downs duchanes)risk of abortion higher than amniocentesis potential limb reduction defect in fetus _ Done at 10-12 weeks  
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amniocentesis   Sample of amniotic fluid _ To detect chromosomal abnormalities, fetal lung maturity, and AFP level _ Done at 16-20 weeks  
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fetal lung maturity   _ Lecithin/ sphingomyelin ratio (2:1) _ Phosphatidylglycerol PG=good, no PG=bad test for surfactant(foam stability index)or shake test 15 min bubble stability= surfactant is present  
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alpha fetoprotein produced by baby   _ Glycoprotein excreted by fetus _ Via amniocentesis or maternal blood sample _ Assess for genetic disorders/ neural tube defects _ Done at 15-20 weeks higher with multibirths  
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daily fetal movement count KICK COUNT   Kick count” _ Mother counts number of fetal movements for 30-60 minutes TID _ To assess fetal well-being _ Done after 28 weeks (7months)  
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fetal activity study (NST)   Non stress test _ Fetal monitor to mother’s abdomen _ Assess acceleration of FHR in association with movementTo assess fetal well being _ Normal = reactive _ Done after 28 weeks  
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oxytocin challenge test (Stress test)   Stress Test _ Induce contractions _ Oxytocin or nipple stimulation _ Evaluate FHR in response to contractions _ Assess fetal well-being during labor _ Normal = negative _ Done at 30 weeks FHR deceleration in response is bad news for baby!  
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internal monitoring   Rupture membranes • Cervical dilation +2 • Spiral electrode (head or butt) • Uterine catheter (intensity measured from within)  
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Fetal Heart patterns   Rate: 110-160, variability:6-25/min Decells are bad accells good= reassuring  
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accelerations   15BPM with fetal activity (exercise) also with vaginal exams, contractions, fundal pressure, and breach presentations.  
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early Decelerations   _ Decrease in FHR _ Starts and ends with contraction (V-shape) _ Head compression (vagal response) intervention: monitor and reasure mother.  
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late deceleration   _ Decrease in FHR _ Starts at peak of contraction and ends after contraction ends _ Uteroplacental insufficiency  
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late deceleration cont. intervention!!!!!LIONPIT   (drop of 30/min or change in base line variability less than 3-5 beats/min****significant indicator of fetal distress) _ Intervene!!!: repositioning admin 8-10L PIT stop, increase IV fluids evaluate vitals(ID hypotension)  
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LION PIT   Left side, IV, O2, Notify, stop the PITocin  
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VEAL CHOP V-C, E-H, A-O, L-P   Varibility---cordcompression Early--------headcompression aceleration--Ok Late---------placenta  
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variable deceleration   _ Sudden decrease in FHR _ “U” or “V” or “W” shape _ Before, during, or after contractions _ Cord compression (may be nucal cord) BPM down by 15, in 15 sec-2min _ Intervene change moms position give O2, call doc, prob C-section  
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normal oxygenation of fetus DURING LABOR   40-70%  
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NITROZINE PAPER   BLUE OR BLUE-GREEN indicates that fluid is alkaline and most likely amniotic fluid  
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signs of onset labor   lightening(fetal drop), bloody show,plug, energy spurt, false labor (braxton hicks),SROM,cervical changes  
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Engagement vs crowning   engagement is 0 station. crowning is +2 or +3  
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duration   no longer than 90 sec  
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intervals   no shorter than 60 seconds  
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mechanisms of labor   Engagement & descent _ Flexion _ Internal rotation _ Extension _ External rotation _ Expulsion  
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engagement& decent   presenting part at 0 station  
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flexion   the fetal head flexes so that the chin rest of the chest  
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internal rotation   fetal head rotates from transverse to anterior position assisted by the pelvic inlet pressure.  
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extension   begins when fetal head is at the pelvic floor, and it pivots under the pubic symphysis and advances upward. extension is complete when head is born.  
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external rotation (restitution)   the head immediatly rotates back to the transverse position in alignment with the shoulders, and the shoulders align themselves with the AP diameter of the pelvic outlet  
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expulsion   anterior shoulder rotates forward under pub symph and delivered,the rest of the body delivered quickly. this ends the second stage of labor  
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stages of labor   _ Stage of dilatation & effacement _ Stage of expulsion _ Placental separation stage _ Recovery stage  
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STAGE ONE:dilatation and effacement   LATENT:0-3cm,15-20min (longest in primapara) ACTIVE:4-7cm,30-60sec TRANSITION:8-10cm,60-90sec. Begins with contractions _ Ends with cervix fully dilated & effaced _ Lasts 5-20 hours  
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STAGE TWO: expulsion   _ Begins when cervix fully dilated & effaced _ Ends with birth of baby _ Lasts minutes to hours  
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STAGE THREE:Placenta   _ Begins with !!BIRTH!!! of baby _ Ends with expulsion of placenta _ Lasts 5-30 minutes  
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STAGE FOUR: recovery   _ Starts with delivery of placenta _ Ends 1-4 hours after placenta delivered _ Lasts up to four hours  
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intervention for stage one phases LATENT   answer questions and teach  
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intervention for stage one phases ACTIVE   assist in breathing techniques, she may walk until she is uncomfertable or ROM, at this point anxiety may kick in  
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intervention for stage one phases TRANSITION   stay with mom as support,remind how to relax and focus on contractions. she may not want to be touched  
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vaginal exam labor process   _ Assess progress of labor _ Dilatation & effacement _ Membranes _ Position of fetus _ Station _ After ROM, sterile gloves _ Not if bleeding  
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vital signs labor process   every hour latent phase every 30 min active phase  
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ROM amniotic fluid   _ Rupture of membranes: sterility _ Color, amount, & odor=infection, green is maconium, yellow is acidic from urin,wine is blood stained _ Nitrazine paper will be blue. _ Ferning conferms amniotic fluid and not other  
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interventions during labor   rapport,comfort,coach during stage one, monitor check for hemorage, encourage bonding, privacy  
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bladder care during labor   every 2h  
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vitals during labor   latent Q1hr, active Q30min, transition Q15min  
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non pharm pain relief   effleurage, sacral pressure(cutainous),(thermal)shower, hot or cold pack nipple stimulation cause contractions by releasing Oxytocin(pituitary origination ). mental diversion focal point , immagery, music. breathing  
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neuromuscular dissociation   contraction of all muscle groups and slowly releasing tension, helps focus of the relese of tension where needed. best if practiced before labor.  
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breathing techniques   woman to decide what type is most comfortable. S/S of hyperventilation:dizzy,tingles in hands or feet.numbness of nose and mouth. Measure to correct:hold breath before exhale, cupped hand breathing or into a moist washcloth  
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pharmacological pain relief   at increased risk for hypoxia, due to uterus against diaphragm,decreased GI motility leads to risk of aspirating emesis,consider effect on fetus  
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advantages of pharmacological pain relief   reduce "stress response" which decreases platlet perfusion,place mother and fetus at risk for metabolic acidosis. good for fat and hypertensive moms  
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limits of pharmacological pain relief   cardiodysfunction may limit the amount of IV fluids needed by some drugs,dosage and time titration prevent baby from respratory distress, fetal drug metabolism slower than moms,  
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limits of pharmacological pain relief   labor should be well established, dialatation of 4cm, and active phase. vitals and FHR considered.  
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Demerol   most common IV opoid.rapid onset and 50 min peek. birth should be estimated to occur more than 2hours after admin to prevent CNS depression of newborn(resusatation prevention)*hypotension*sedation *nausea *pruritis(itching)*decrease FHR and variablility  
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sublimaze (fentanyl)   rapid onset,short acting OPOID AGONIST,last 1-2 hr *respiratory depression, may be used in combo with regional anesthesia.  
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Butorphanol(stadol) nalbuphine (Nubian)   combo opioid agonist antagonist,relief pain and Nausea with out respiratory depression. not to be given to **drug addicted moms cause withdraw in mother and baby**  
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labor: analgesia given as early as   one hour before delivery, must be 4cm dilated (ACTIVE LABOR)  
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NARCAN (naloxone)   used to relieve pruritis(itching)common side effect of epidural anesthesia. also used in conjunct with newborn O2,venting and gentle stimulation, in new born experiencing respiratory depression.  
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fun opioid fact five   opioids remain in newborn system for 7days, narcan caused withdraw in mother and baby addicted to drugs  
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anesthesia during labor   REGIONAL:pudendal, epidural, sub arachnoid. GENERAL: bad situations, crosses placenta, increases risk of mom hemorrhage  
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anesthesia during labor   monitor (Q5min-VS, FHR). monitor ability to push, urin output freq  
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blood patch for subarachnoid(spinal block)   10-15 ml inj into epidural space to prevent leaking of CSF  
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pudendal block   local anesthetic inj trans vag into pudental nerve. late second stage of labor 10-20 before delivery, assess for broad ligamint hematoma  
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epidural block   local anesthetic inj into epidural space at 5th lumbar vertebrae. umbilical down affected. given at active labor 4cm. assess for maternal hypotension, fetal bradycardia.loss of Bering down reflex passably causing URINARY RETENTION.  
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spinal block (subarachnoid)   local anesthetic injected into subarachnoid space. blood patch to keep CSF from leeking,affects all sensations from the level of the nipple down,given for vaginal and Csection.  
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spinal block (subarachnoid)   Disadvantages:limited duration of action,maternal hypotension, fetal bradycardia, potential spinal headache, URINARY RETENTION risk. monitor vitals 5-10min,contractions FHR, safety when mom moves.recognise signs of impending birth  
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Newborns respiratory function   surfactant made by mature set of lungs, is a phospholipid.holds open aveoli by reducing surface tension.  
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functional residual capacity   small amount of air that ramains in aveoli. causing each breath to get easier.  
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thermoregulation prevent cold stress   keep enviroment 89-93 degrees, normal temp (97.7)  
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NB GI system   glycogen stors are low, will begin to use brown fat(thermogenic)for energy needs, can feed immediately after birth. burping reduces air pocketing(air obstruction)erly feeding reduces biliruben lvls. glucose levels should be around 60-70 by day 3,  
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TORCH   TOXOPLASMOSIS,RUBELLA,CYTOMEGALOVIRUS, AND HERPESINFECTIONS(NO PRETECTION FROM THESE INFECTIONS)  
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COLD STRESS COMPLICATIONS   increase of O2 consumption hypoxia then leads to acidosis,glucose increase relese of lactic acid hypoglycemia, reduced surfactant production collapse of alveoli reopening of shunts(foramen ovale, ductus arteriosus)  
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brown fat(thermogenic) BAT(brown adapose tissue)   nonshivering thermogenesis or called chemical thermogenesis, demerol may interfere with utilization causing hypothermia  
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molding resolves when   2-3 days, not seen in c section or breech babies  
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caput succedaneum   soft flactuaant mass that may cross the suture lines. ABSORBED IN A FEW DAYS AND REQUIRES NO INTERVENTION  
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cephalhematoma   swelling with in the bone of the skull, subperiosteal, (WILL NOT CROSS THE SUTURE LINE)but may be bilateral.emerges in one to two days, gone in 3 weeks, common with prolonged or difficult labor(dystocia)head circomference altered.  
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NB GI   iron stores last baby for 5months.  
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kirnicterus newborn   Kernicterus is a rare neurological condition that occurs in some newborns with very high levels of bilirubin jaundice.results from cold stress atributing to metabolic acidosis  
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microcephaly newborn   (small head)detected by measureding head circumference. caused by toxoplasmosis, rubella during fetal development  
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desquamation newborn   peeling of the skin at birth (postmaturity)instruct parents to avoid harsh soaps(also seen in vigorously removing the vernix)  
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mongolian spots newborn   dark blue or slate grey lumbosacral region, no medical significance.fades in time(up to 2 years)  
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nevi(stork bites)   gradual fade no medical significance,  
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port-wine stain newborn   darkens with age,will not blanch on pressure. can be associated with genetic disorders. pg169 (laser surgery for self image elective)  
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craniosynostosis newborn   small or early closure of ANTERIOR FONTENELLE. associated with abnormal brain development.can be caused by FAS,fetal hypoxia, chromosomal abnormality  
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eyes of the newborn   color is established 3-6months may be longer. can see 7-10 inc away. tracking 10sec.tears not normal until 2months of age(conjunctiveitis or glaucoma)  
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newborn sleep   deep sleep aprox 3hr. (quiet alert 30 min-1hr. active alert 23hr  
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chest of newborn   nipples that are abnormaly wide may indicate defect.-----{nipples may secreat substance caused from mothers hormones (witches milk)gone in about a week.  
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hearing test of newborn   ALGO screener, sends a tone to babies ear, reads a brain wave that indicates acknowledgement of sound and compares to data base, of other babies responses  
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newborn bowel sounds   can be heard 15 min -2hr after birth  
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genitalia of new born   hymen tags(few weeks) and psudomenstruation (maternal hormones) males: phimosis is foreskin that covers urethra. check for descent of testes  
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anus of newborn   poo in first 24, assess patent anus. 3 stools a day for breast feed and one every other day for bottle fed  
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simian crease   linear crease running across the palm indicates downs syndrome  
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clubfoot   inward downward  
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unilateral moros reflex   broken clavical,  
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APGAR   (activity):flacid to flexed, (pulse): absent to 100+, (grimace): none to vigorus cry, (appearance): blue to compleatly pink, (respiration):absent to good cry  
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SGA, LGA, AGA   small for gestational age is less the 10th percentile, large for gestational age is over 90th percentile. BALLARD SCORE to differentiate from premature  
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maple syrup urine disease   disorder passed down through families in which the body cannot break down certain parts of proteins. Urine in persons with this condition can smell like maple syrup.  
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maple syrup urine disease treatment   life long diet free of amino acids  
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vitamin K   0.5-1mg aquamephyton, phytonadione. normal flora not present to make K,  
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circumcision   petrolium jelly, sterile gauze. assess bleeding q15min first hour,q30 min 2-3hours. change gauze and ointment with each diaper change. heals 3-5 days.  
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circumcision home care   change gauze and ointment with each diaper change.wash warm water only. no debridment of any type. prevent pressure on site. report less than 6 wets a day.report redness bleeding drainage to HCP  
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feeding time at the zoo!   galactogogues:breast mild stimulators include beer and brewers yeast or sesame tea. breast feeding speeds uterin return. 500+kcals needed.8-10glasses of non cafinated.estrogen,progesterone and human placental lactogen. feeds should be 15-20mins  
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let down reflex of breast feeding   suckling stimulates posterior pituitary to secreat oxytocin. causes cramping (involution)and breast tingles  
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breast milk types   colostum, 3-4 days immunity,transitional milk 5 days to 2 weeks has a little more calories and fat and lactose. mature milk is 22.5 kcals per oz and is plenty sufficient to meet infants needs.  
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dont feed if   HIV, HBV with out vaccine.HepC if liver failure. herps and vericella zoster are contraindicated until leasions are on the breast. small pox vaccination is contra. you been drinking,  
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wash your hands and knockers   hands are primary source of infection. careful what u wash the boobies with,some soaps may cause drying cracking and can lead to mastitis.  
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late sign of hunger   crying, rooting and hand to mouth sucking and alertness are early signs of hunger (trust vs mistrust)  
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storage of breast milk   room temp: 4hours max. fridge at39.2 for 24hrs. freezer 20 deg for 3months. polypropylene not suggested for freezing. microwaves not suggested for warming  
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breast engorgment   cold between feedings and warm before feeds help. massage helps express milk. wear a suportive bra  
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puerperium   postpartum up to 6 weeks (return to normalcy)  
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first 1-2 hours of postpartum   risks are hemmorage and hyypovolemic shock. subsaquent danger is infection  
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the primary cause of involution is   the sudden withdraw of estrogen and progesterone. releases proteolytic enzymes to endometrium  
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factors that neg impact involution   prolonged labor, incomplete expultion of placenta, distended bladder,anesthesia, prevoius labors  
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factors that pos impact involution   breastfeeding, uncomplicated labor, ambulation  
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after pains   2-3 days postpartum, oxytocin and uterine contractions  
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what is lochia   blood, mucus and particles of necrotic decidua, heaviest 1-2hrs postpartum. count pads assess amt, color smell, clots ect  
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lochia rubra   1-3days may contain SMALL CLOTS.  
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lochia serosa   4-10days), pinkish brown,serosang  
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lochia alba   10days to 6 weeks,yellow white  
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episiotomy assessment   REEDA redness, edema, ecchymosis, approximation  
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hemorrhiods   assess for size, number and discomfert, and relieving factors. witch hazzel, cortico suppository, local anesthestics  
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temp and wbc post postpartum   wbc 15000 first 10days. temp due to dehydration in first 24 hrs, hbg and hct elevated as well as fibrinogens  
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postpartum urinary   increased volume decreased muscle tone, local swelling and anesthesia make it difficult to pee. RISK for infection. diuresis U/O 3000. protein but not glucose in urine  
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postpartum GI   woman has worked up a hunger. constipation has multipal factors. fear of pain progesterone,decreased tone in abdominal muscles  
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postpartum Neuro   carpaltunel resolves as edema decreases, hypertension still a concern  
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postpartum endocrine   lactation and menstration starts as a result of declining hormones. menstration may return later if lactating up to 6mo, nonlactating 1-3 months  
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phases of maternal adaptation   taking in, taking hold, letting go  
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taking in phase   concerned for her self and newborn, passive dependent behavior  
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taking hold phase   ready to assert her independence, ready to initiate newborn care  
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letting go phase   assumes her position in the home (aka the kitchen or ass up face down)which makes her sad  
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postpartum BLUES not depression is   up to 10 days. tearfulness, insomnia, lack of appetite, and a feeling of being disappointed. multifaceted: hormones, anesthesia, fear, pain, new adjustments and grief over freedoms lost. rest empathy and support to manage  
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abortion   before 20 weeks,complications: hemorrhage infection.treatment bed rest, D&C, fluids,monitor for inf and bleeding  
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threatened abortion   vaginal bleeding and cramping  
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inevitable abortion   ROM and Cervical changes  
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incomplete abortion   part of product of conception retained in uterus  
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complete abortion   all product of conception is expelled  
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missed abortion   products of conception retained in uterus less than 2 mo. mommy dident know  
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methotrexate and tubal pregnancy   chemotherapy agent,avoid alcohol and folic acid to prevent toxic response to the drug. prevent sun exposure(photosensitivity)  
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gestational trophoblastic disease   hydatidiform mole, invasive mole, and choriocarcinoma.trophoblast poliferate, chorion becomes fluid filld grape sacs.  
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gestational trophoblastic disease   avascular, no ICM. vaginal bleeding(prune juice),hypertension and hyperemesis gravidarum(elevated hCG)treatment is D&C and follow up hCG lvls  
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placenta previa   painless bleeding, observe, bed rest tocolytics MAG SULFATE(stop labor) no vag exams, c section  
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abrupto placenta   placental detatchment, PAINFUL BLEEDING. FHR changes. shock and DIC!!!! monitor transfusion csection  
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Rh sensitization   affects second child, erythroblastosis fetalis Rhogam 28 weeks and 72hr of PP  
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ABO incompatibility   mother -O, fetus is -A,B,or AB. affects first child, causes patho jaundice  
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antidote for mag sulfate   calcium gluconate, to prevent pre eclampsia  
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gestational hypertension   eclampsia pre-eclampsia, treatment MgSo4 mag sulfate, assess deep tendon reflex to titrate MgSo4.needs more if she kicks you through the fucking wall  
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pre-eclampsia   proteinemia, edema/wt gain. renal involvement leads to proteinuria  
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eclampsia   results in seizures,HELLP, CNS involvement  
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HELLP   H:hemolysis, EL:elevated liver enzymes, LP:low platlets  
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preterm labor   poor preterm care, infection,over distention of uterus, nutritional status. SES, tocolytics slow labor process, other measures (stop infection,restrict activity ensuring hydration)  
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tocolytics before 3 cm(point of no return) not effective in 5cm or more   Yutopar,brethine procardia indocin vasodilation, (assess for hemorrhage) headache is common  
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methods of induction and augmentation   prostaglandin, AROM, Oxytocin  
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preterm delivery (promoting fetal lung maturity)   steroids given to mother 24-48 hours before delivery, betamethazone or dexamethasone  
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posterm delivery   after 42 weeks,fetal hypoxia due to an aging placenta 42weeks exp date, birth trauma due to size of baby, CPD  
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precipitate labor   less than 3hr from first true contraction,:fetal hypoxia from little time between contractions (fetal respiration)  
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hydraminos   associated with fetal swallowing and voiding, removal of excess hydraminos can result in placenta abrupto, prolapsed cord is also concern. amniocentesis is done to prevent overdistention of uterus and preterm labor  
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factors that increase the incidence of prolapsed cord   hydraminos, long cord, small fetus, ROM before engagment of fetus,multi fetal birth, breech or transverse lie  
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PKU   build up of keytones, unable to break down phenylalanine. life long diet, detection using guthrie test 48-72 hr after birth  
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low birth wt.   under 2500g(5.5lbs)(2.5kg)  
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ballard scoring posture,and physcial atrabutes   -10=20gestational weeks, 50=44gestational weeks  
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ECMO   heart and lung machine, used for meconium aspiration as well as other reasons  
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immature livers of newborns   hypoglycemia(30 or less), and hypocalcemia,symptoms are twitching convulsions and high pitched cry  
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atony (sloppy soft bleeding uterus)   MgSo4 can impair calcium that is needed to tighten that shit up bro.as well as calcium channel blockers such as the mother fuckin nifedipine used in da preterm bitchs. and you know that bitchs be trippin bro!you need to tighten that shit up hoe  
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atony managment   shock (tach and low BP)boggy high uteruses. get base line labs and anticipate dextran/albumin, and blood products, high doses of oxytocin/pitocin. admin of vit K. factor 7 can reverse symptoms DIC  
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blood vs lochia   lochia darker red, blood brighter red and continuous.  
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oxytotics   expell retained placenta frags, if not effecient enough a D&C is in order  
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DIC   is suspected when usual means of contraction the uterus fails to stop bleeding. signs of DIC are horrific, she will bleed out of every little interuption in skin integraty, IV, SHOCK IS LATE SIGN, blood volume delays signs of blood loss.  
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DIC blocker(treatment)   factor 7(VIIa)  
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how does DIC happen   infection, abrupto,GTD  
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