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PHM1810 Chp 10 Drug

Pharmacology I: Chapter 10: Musculoskeletal (Entire)

QuestionAnswer
Anectine succinylcholine chl; neuromuscular blocking agent
Tracrium atracurium besylate; neuromuscular blocking agent
Nimbex cisatracurium besylate; neuromuscular blocking agent
Zemuron rocuronium bromide; neuromuscular blocking agent
Nuromax doxacurium chloride; neuromuscular blocking agent
Mivacron mivacurium chloride; neuromuscular blocking agent
Pavulon pancuronium bromide; neuromuscular blocking agents
Lioresal baclofen; centrally acting skeletal muscle relaxant
Soma carisoprodol; centrally acting skeletal muscle relaxant
Maolate chlorphenesin carbamate; centrally acting skeletal muscle relaxant
Parafon Forte/Paraflex chlorzoxazone; centrally acting skeletal muscle relaxant
Flexeril cyclobenzaprine; centrally acting skeletal muscle relaxant
Valium diazepam; centrally acting skeletal muscle relaxant
Robaxin methocarbamol; centrally acting skeletal muscle relaxant
Norflex orphanadrine citrate; centrally acting skeletal muscle relaxant
Botox botulinium toxin type A; Direct-acting anti-spasmodic agents
Myoblock botulinium toxin type B; Direct-acting antispasmodic agents
Dantrium dantrolene sodium; Direct-acting antispasmodic agents
Quinamm quinine sulfate; diect-acting antispasmodic agent
Ridaura auranofin; 2nd line agents for Rheumatoid Arthritis
Aurolate gold sodium thiomalate; 2nd line agents for Rheumatoid Arthritis
Mexate methotrexate; 2nd line agents for Rheumatoid Arthritis
Plaquenil hydrochloroquine; 2nd line agents for Rheumatoid Arthritis
Depen penicillamine; 2nd line agents for Rheumatoid Arthritis
Azulfadine sulfasalazine; 2nd line agents for Rheumatoid Arthritis
Mechanism of Action of Neuromuscular Agents prevent somatic motor nerve impulses, which affect skeletal muscles
Adverse Effects of Neuromuscular Agents drowsiness, dry mouth, loss of strength, hypotension, muscle weakness, cardiac arrythmias, increase in seizures in epileptics
Mechanism of Action of Centrally-Acting Skeletal Muscle Relaxants Unknown; May act in the CNS to depress polysynaptic reflexes.
Adverse Effects of Centrally-Acting Agents weakness, fatigue, drowsiness, dizziness
Mechanism of Action of Direct Acting Agents Inhibit release of calcium ions, making the musces less responsive to nerve impulses.
Adverse Effects of Direct Acting Agents muscle weakness, drowsiness, dizziness, nausea, diarrhea, seizures, tachycardia, erratic BP and pericarditis
Mechanism of Action for RA Drugs Unknown; Gold compounds, however, suppress or prevent inflammation but do not sure it
Adverse Effects of RA Drugs gold compounds and penicillamine: GI disturbances, dermatitis, lesions of mucous membranes; Corticosteroids: GI bleeding, poor wound healing, hyperglycemia, hypertension and osteoporosis (long term use)
Colcrys colchicine; used for gout; mechanism of action unknown; adverse effects: very toxic!! stop if diarrhea, vomiting, abdominal pain
Zyloprim allopurinol; used for gout; blocks the formation of /enhances the excretion of uric acid; adverse effects: drowsiness, headache, vertigo, nausea, GI discomfort, hepatoxicity, renal insufficiency
Created by: MrsAFlaherty
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