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blood values
lymphatic disorder
| Question | Answer |
|---|---|
| 1. what are the normal values for RBC | males:4.7-6.1 million/mm3 females: 4.2-5.4 million/mm3 actual cell count |
| 2. what are the normal values for Hgb | males: 14-18 g/dl females: 12-16 g/dl |
| 3. what are the normal values for Hct | Males: 42%-52% females:37%-47% |
| 4. what are the normal values for WBC | 5000-10,000/mm3 cell count |
| 5. what are the normal values for Neutrophils | 60%-70% 3000-7000 |
| 6. what are the normal values for Eosinophils | 1%-4% 50-400 |
| 7. what are the normal values for Basophils | 0.5%-1% 25-100 |
| 8. what are the normal vallues for Lymphocytes | 20%-40% 1000-4000 |
| 9. what are the normal values for monocytes | 2%-6% 100-600 |
| 10.what are the normal values for (ESR) Erythrocyte Sedimentation Rate | male: 0-15 mm/hr female:0-20 mm/hr |
| 11.what are the normal values for Platelet count | 150,000/mm3-40,000/mm3 actual cell count |
| 12. what are the normal values for Prothrombin time(PT) | 11-12.5 seconds rapidity of blood clotting |
| 13. what are the normal values for INR | 0.7-1.8 |
| 14.what are the normal values for bleeding time | 1-9 minutes (lvy method) |
| 15.what are the normal values fr clotting time | 3-9 seconds |
| 16.what blood type is called the universal donor | type O |
| 17.Type A blood the RBCs contains what anti antigens | type A antigen and the plasma conains anti-B antigens |
| 18.In type B blood, the RBCS contain | type B antigen and the plasma contains anti-A antigen |
| 19.In type AB blood, the RBCs contain both | A and B antigens, nd the plasma contains neither anti-A nor anti-B antibodies |
| 20. Blood type O RBCs contain | neither type A nor type B antigens and the plasma contains both anti-A and anti-B antibodies |
| 21. Define agglutination | donor cells clump together because the antibodies; this occludes arteries and can result in death. |
| 22. Define hemolyzation | The antibodiescause the RBCs of the recipient to rupture and release thier cell contents; this can lead to death |
| 23. RH factor | People who have RH factor which is located on he surface of the RBCS, are said to be RH positive. |
| 24. what is the name of the desensitization drug for RH incompatibility | RhoGam |
| 25. What is RH incompatibility | If a RH-negative person reseives RH -positive blood, within 2 weeks Rh antibodies are produced and reman in the blood. If RH negative person recieves more RH-positive blood , a severe reaction occurs. |
| 26. What is Anemia | A disorder characterized by RBC and hemoglobin and hematocrit levels below normal range |
| 27. What are some of the clinical manifistations of anemia | The older adult Hbg levels less than 10 g/dl. signs and symptoms: Anorexia, disorientaion, Fatigue, |
| 28. Define 3 nursing diagnosis for anemia | Ineffective tissue perfusion, impaired gas exchange and activity intolerance. |
| 29. what is pernicious anemia | This type of anemia is a result of a metabolic defect: the absence of a glycoprotien intrinsic factor secreted by the gastric mucosa |
| 30. The intrinsic factor is essential for the absorbtion of what vitamin? | vitamin b12(CYANOCOBALAMIN) |
| 31. Where is vitamin b12 absorbed | vitamin b12 is obsorbed in the distal iliem |
| 32. what are the clinical manifestations of pernicious anemia | Extreme weakness, dyspnea, fever,and hypoxia, jaundice(iterus),edema of the legs intermittent constipation and diarrea. |
| 33. what is te medical management for pernicious anemia? | viamin B12 replacement therapy |
| 34.Define Aplastic anemia | Aplastic anemia or aplasia is a hematological term for a failure of the normal process of cell generation and development. |
| 35. what are the two etiologic classifications for aplastic anemia? | Congenital (caused by chromosomal alterations) and acquired(directly related to exposure to viral invasion. |
| 36. what are the clinical manifestations of aplastic anemia | Repeated infections with high fevers, along with fatique,weakness,general malaisedyspnea and palatations. Mortality is high from complications of infections and hemorrhage. bleedng gum, petechiae, ecchymoses,GI bleed. epitaxis, bleeding |
| 37. Iron deficiency anemia | A condition in which RBC contain decreased levels of hemoglobin. |
| 38. what is the most common cause of iron deficiency anemia? | excession iron loss. |
| 39. In adults, what are the common sources of iron loss? | Chronic intestinal or uterine bleeding. |
| 40. what are the chief symptoms of iron deficiency anemia? | Ploor, and glossitis(imflammation f the tongue). |
| 41. when a patient cannot tolerate oral preparations of iron,parental iron therapy is used used. Define parentliron therapy | The Z-tract method of giving iron dextran (Dexerrum) intramusclarl is prefererable , to prevent staining. |
| 42. what is sickle celll anemia | A abnormal cresent shaped RBG containing hemaglobin S (Hg-S)- a defective hemaglobin molecule. |
| 43.the sickle cell conditio ocurrs in people who are? | Homozygous: having two identical genes inherited from each parent for a given hereditary characteristic for Hg-S. |
| 44. A sickle cel crisis is a ? | episode of od acute "sickling" of erthrocytes, which causes occusion and eschemia( in the distal blood vessals |
| 45. what is the cycle cell trait? | The cycle cell trait is heterozymous Having two differnt genes) form a cycle cell where by the individuaL Has the Hg-S and thehemoglobin A (Hg-A) in then RBGS. |
| 46. what are the clinical manifestations of cycle cell anemia? | Physical factors include:events that cause dehydration, or change the oygen tension in the body, such as infection, overexcertion, weather changes(cold) ingestion of alcohol, and smoking. Loss of appetite. |
| 47. Medical management for sickle cell anemia | There is no specific treatment. Therapy is directed towards elleviating the symptoms. |
| 48. Nursing interventions and patient teaching are? | Suppoted treatment follow sign and symptom presentastion: hydration and analgesia during crisis and dilution of blood with increased fluid intake to reverse sickling for evidence of transfusion reaction. Monitor infusion therapy. |
| 49. Define Disseminated intravascular coagulation (DIC) | A coagulopathy resulting from the overstimulation of clotting and anticlotting process in responce to disease and or injury. |
| 50. What is the medical managment for (DIC) | Transfusion replacement and cryoprecipitate are ordered. Heparin therapy given, Fresh frozen plasma (FFP) is administered to repace other coagulation factors. |