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pharm 4 moa

QuestionAnswer
procainamide, quinidine stabilize ventricular fibrillation
lidocaine, propafenone stabilize atrial and ventricular afibrillation
flecainide stabilize atrial fibrillation
adenosins activates adenosine receptors (give fast)
digoxin increase intracellular pressure
esmolol, propranolol decrease hr, excitability, contractability
amiodarone, dofetilide, sotalol blocks potassium channels which prolong action potential
diltiazem, veramapil cardiac depressant
hydrochlorothiazide, metolazone blocks chloride pump, keeping sodium and chloride in the distal convoluted tubule
bumetanide, furosemide blocks chloride pump, keeping sodium and chloride in the loop of henle (may need k supplrements)
acetazolamide slow movement of hydrogen ions, increasing sodium and bicarbonate loss in urine
spironolactone lodd of sodium and water but keeps potassium
mannitol increased osmotic pull in renal tube to increase fluid excretion (must bi given iv through filter)
fosfomycin, nitrofurantoin, trimethoprim-sulfamethoxazole 1-time dose with unpleasant gi effects and incomplicated cystitis
oxybutynin, tolterodine anticholinergic (dry mouth)
mirabegron b3 agonist
phenazopyridine pain relief with uti or trauma to bladder (red-orange tint)
pentosan polysulfate sodium coats bladder (monitor coagulation)
doxazosin, prazosin, tamsulosin, terazosin dilutes and relaxes ( - htn, tam - kidney stone)
dutasteride, finasteride blocks enzyme that converts testosterone
cholestyramine, colesevelam, colestipol binds with bile acids to be excreted in stool (may need fat-soluble vitamin supplements)
atorvastatin, rosuvastatin, simvastatin decreased cholesterol production and LDL's in blood
ezetumibe decreases absorption in small intestine
evolocumab monoclonal antibody that binds to receptor and recycles it
fenofibrate, gemfibrozil stimulates breakdown and removal of lipoproteins
vitamine b3 decrease free fats circulating
omega-3 fatty acids decrease liver enzyme which increases bleeding time
digoxin stronger, slower, more efficient pump
milrinone stronger contraction and prolongs effect of sympathetic stimulation
ivabradine slows SA node to decrease hr which allows for ventricular filling
valsartan binds to all receptor and blocks smooth muscle contraction and aldosterone
sacubitril blocks breakdown of natriuretic peptide which increases sodium and water loss
isosorbide dinitrate, isosorbide mononitrate, nitroglycerin increases coronary flow and decreases cardiac workload
nadolol, nebivolol, propranolol, sotalol decreases hr, contractibility, excitability
diltiazem, verapamil cardiac depressant
ranolazine decreases myocardial workload whihc results in better perfusion
benazepril, captopril, enalapril, lisinopril stops angiotensin 1 from becoming angiotensin 2
losartan, valsartan blocks smooth muscle and aldosterone
aliskiren inhibits renin
amlodipine, nicardipine, nifedipine blocks muscle cell contraction
diltiazem, verapamil cardiac depressant
hydralazine, nitroglycerin, nitroprusside muscle relaxation
hydrochlorothiazide, chlorthalidone, metolazone excretes sodium and chloride
spironolactone keeps potassium
beta blockers decreases hr, contractibility, excitability
alpha- and beta-blockers rest and digest, decrease bp
alpha-blockers decreases
alpha2 - agonists decrease bp
dobutamine, dopamine, epinephrine, norepinephrine, phenylephrine increases bp, strengthens cardiac contraction
midrodrine activates alpha to increase vascular tone
droxidopa vasocontracts
Created by: KimBriann
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