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GI

Drug/ClassMOAIndicationConsiderations
H2 Receptor Antagonists (H2RA) Blocks H2 receptors; reduces volume and acidity of gastric secretions
Cimetidine H2RA -Healing of ulcers -GERD works faster than PPI but less effective
PPI Suppress acid secretion by blocking the enzyme that makes gastric acid
Omeprazole PPI -Ulcers -GERD no longer than necessary
Pantoprazole PPI Prevents stress ulcers in clinical setting convert to PO when possible
Sucralfate Forms protective barrier over ulcer from acid/pepsin Acute maintenance and therapy of duodenal ulcers lasts about 6 hours, does not reduce or neutralize acid
Misoprostol Suppresses acid secretion Promotes bicarb secretion Cytoprotective mucous Prevent ulcers in long-term NSAID therapy Pregnancy test (Can terminate pregnancy)
Promethazine Suppresses emesis by blocking dopamine receptors in vomit center emesis MONITOR RESP. Depression
Scopalamine Muscarinic antagonist (anticholinergic) -suppresses nerve traffic from ear to vomit center motion sickness DOC
Antihistamines in GI usage Motion sickness
Loperamide Opioid Antidiarrheal preggy safe low abuse
Diphenoxylate Opioid Most effective antidiarrheal abuse added with atropine
Metoclopramide H2RA suppresses emesis n/v in preggy GERD Chemo diabetic gastroparesis
Alosetron increases colonic transit time, reduces intestinal secretions women with severe IBS-D[diarrhea]
Lubiprostone same as alosetron IBS-C (constipation)
Amino salicylate Sulfasalazine reduces inflammation in GUt Ulcerative colitis Chrohns RA CBC for agranulocytosis and anemia
Inflixamab Immunomodulator: tumor necrosis inhib, Severe disease or perianal chrohns disease
Palifermin synthetic form of Keratinocyte growth factor (KGF) Oral mucositis in chemo patients 24 hours before or after chemo
Created by: tapolk
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