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bios ch. 19

QuestionAnswer
necrosis plasma membrnae damaged --> cell swells and bursts --> contents released --> inflammation --> debris removed
programmed cell death varied pathways all controlled by signals
pro-apoptotic proteins stimulate apoptosis
Bcl-2 family cytosolic proteins which interact with one another
Bad, Bax, Bak, Bid (Bcl-2 proteins) promote cell death via apoptosis
some Bcl-2 proteins inhibit cell death via apoptosis
pro-death pro-apoptotic (decision to undergo apoptosis)
pro-life anti-apoptopic
effector pro-apoptotic (Bak/Bax) form complexes that insert into outer mito. mem. and form hole big enough for cytocrome C to enter cytosol
apoptosome cuts procaspases to form active effector caspases
caspases cysteine aspartic proteases
effector caspases cut DNase inhibitor
scramblase specific plasma mem. flippase
TNF tumor nercrosis factor
platelets release Platelet-Derived Growth Factor (PDGF), found on fibroblast
PDGF is a receptor tyrosine kinase
fibroblasts motile cells (fiber making), secrete, organize
epidermis interconnected layers of cells forming a epithelium
dermis ECM + cells
hypodermis loose ECM + cells
stem cells in epidermis are basal
MHC Major Histocompatibility Complex
hematopoietic stem cells make blood cells (in bone marrow)
stem cells in intestine epithelial cells,
stem cells skin epedermis, dermis, hypodermis
stem cells in skeletal muslces called what satellite cells
Embryonic stem cells (ES) grow indefinitely. (can differentiate into varied cell types)
induced pluripotent cells treat adult stem cells/self-replacing cells so they become pluripotent cells. use signals to cause them to differentiate
Created by: lutmanlucy21
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