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NRSG Pharm CH 33

Inflammation and Fever

QuestionAnswer
Inflammation purpose Limits spread of injury or antigen, destroys microorganisms
5 basic steps of inflammation 1.Vasodilation, 2.Vascular permeability (edema) 3.Cellular infiltration (pus) 4. Thrombosis (clots) 5. Stim of nerve endings (pain)
s/s of inflammation Swelling, pain, warmth, redness, loss of function
Chem mediators of inflammation Bradykinin, Complement, C-Reactive Protein, Cytokines, Histamine, Leukotrienes, Prostaglandins
Histamine Receptor H1 In vascular smooth muscle, bronchi, sensory nerves stim= itching, pain, edema, vasodilation, bronchoconstriction, characteristic of inflammation and allergy
H2 receptors Located in stomach, stim = secretion of hydrochloric acid
COX1 and COX2 inhibitors Ibuprofen (Motrin, Advil)
COX2 inhibitors Celecoxib (Celebrex)
NSAIDS Analgesics, antipyretics
Prednisone (Meticorten) classes, mech, Corticosteroid, glucocorticoid. Immunosuppression and anti-inflammatory. Relieve sx of ex: arthritis, asthma exacerbation, autoimmune disease
Prednisone (Meticorten) considerations Lowest effective dose for shortest period of time to achieve effect TAPER OFF - abrupt d/c can result in adrenal insufficiency
Prednisone (Meticorten) side/adverse: Immunosuppression - inc risk of infection, delayed wound healing. Hyperglycemia, hypernatremia, hyopkalemia, wt gain, fat redist, edema, gastric ulcer formation, osteoporosis. Most related to dose/duration
Created by: kmulla
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