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cardiovascular med
| Term | Definition |
|---|---|
| NITRATES | relaxes the smooth muscle surrounding blood vessels. Dilates arteries and veins. Decreased O2 use by cardiac muscle |
| ANTICOAGULANTS | Interrupt the Vitamin K mediated production of proteins called cofactors that help blood to clot. |
| ANTIPLATELET AGENTS | Interfere with the plasma membrane of platelets and prevent them from sticking together and forming clots |
| THROMBOLYTICS / FIBRINOLYTICS | Converts plasminogen to plasmin. Plasmin attaches to the surface of the clot and breaks down the fibrin that holds the clot together. |
| ANTIFIBRINOLYTICS | Inhibits the conversion of plasminogen to plasmin, preventing the breakdown of fibrin and maintains the clot, stopping bleeding |
| HEMATOPOIETIC STIMULATES | Depends on the cause of anemia. Iron combines with porphyrin and globin chains to form hemoglobin, which is critical for oxygen delivery from the lungs to other tissues. |
| ACE INHABITOR | Works on the kidneys to prevent Angiotensin I from converting to Angiotensin II. decreased blood pressure. |
| ANGIOTENSIN RECEPTOR BLOCKERS | Blocks angiotensin II from attaching to receptor sites and causing vasoconstriction which increases BP |
| BETA BLOCKERS | Beta blockers cause your heart to beat more slowly and with less force, which lowers blood pressure. |
| DIURETICS | Increases urinary excretion of Na+, K+, and H20; Increases urinary excretion of Na and H20 increased retention of K+3 |
| CALCIUM CHANNEL BLOCKERS | Dilates arteries, decreases blood pressure, and decreases work load of the heart |
| CARDIAC GLYCOSIDES | Deactivates the Na+/K+ ATPase pump resulting in high levels of Na+ within cardiac muscle cells. |
| VASODILATORS | Relax the smooth muscles in blood vessels which causes blood vessels to dilate. |
| POTASSIUM CHANNEL BLOCKERS | Block potassium channels on cardiac muscle cells and prevents K+ from leaving the cells. |
| HMG-COA REDUCTASE INHIBITORS | Lower cholesterol levels by blocking the HMG- CoA reductase enzyme in the liver. This slows down the liver's production of cholesterol. |
| BILE ACID SEQUESTRANTS | Lowers (LDL) cholesterol levels by blocking bile acid in your stomach from being absorbed into the bloodstream. |
| FIBRIC ACID DERIVATIVES | Inhibit the secretion of triglycerides from the liver, which reduces levels of blood triglycerides and stimulates the clearance of triglycerides by activating lipoprotein lipase. |
| NICOTINIC ACID | Niacin works on LDL and HDL cholesterol in the liver which results in decreased TG production by the liver and lowers overall TG levels in the body. |
| ESTERIFIED FISH OILS | Decreased lipogenesis in the liver, and increased plasma lipoprotein lipase activity |
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