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HEMODYNAMIC

HEMO

QuestionAnswer
Swan-Ganz is also called flow-directed balloon tip catheter
pulmonary artery catheter enables medical practitioners to acquire important hemodynamic info about the cardiovascular system as well as function of the right and left ventricles.
Indications for Swan-Ganz catheter -Measure cardiac output via thermodilution technique -measure hemodynamic parameters associated with central venous pressure and ventricular function -acquire blood samples for mixed-venous analysis
Swan-Ganz catheter is approximately how long? 110cm or 28in
Swan-Ganz consists of -electronic thermistor cable to attach cardiac output monitor -balloon(or cuff) on distal end to inflate and acquire pulmonary capillary wedge pressure (PCWP) -Ports or openings in the catheter
Thermistor temperature sensor imbedded near the tip of the catheter
Balloon or cuff distal end to inflate and acquire the pulmonary capillary wedge pressure (PCWP)
Distal port tip of catheter to measure pulmonary capillary wedge pressure, pulmonary artery pressure and draw blood for analysis
Proximal port placed where? about 30cm from tip of catheter
Proximal port measures what? right atrial pressure or CVP and for injection of iced saline to assess the cardiac output via thermodilution technique.
Insertion site flow-directed balloon-tipped catheter (Swan-Ganz) internal jugular or subclavian veins
What technique is used to place catheter? sterile technique
Where should the Pulmonary artery catheter tip enter during insertion? large vein, into vessels until catheter tip enters the right side of the heart.
What happens after entry to the right side of the heart with the pulmonary artery catheter? Balloon is inflated
How much air is used to inflate balloon on pulmonary artery catheter? .8 to 1.5 depending on size of catheter
After balloon inflation what does the catheter do? floats through the right atrium and right ventricle and into pulmonary artery
What does pressure waveforms note during insertion? indicate position of the catheter
Right atrial pressure (RAP) is the same as Central venous pressure (CVP)
Right atrial pressure is measured with Swan-Ganz catheter in place at the proximal port which lies in the right atrium
Normal Diastolic RAP/CVP 2-6 mmHg
Increase or decrease in RAP is most indicative of patients volume status
Normal Systolic RVP pressure 15-30 mmHg same as systolic PAP
Where does diastolic pressure read and what does it indicate? Reads in right ventricle and indicates right ventricular preload or right ventricular end diastolic pressure (RVEDP)
Distention of RVP compared to RAP RVP will have taller upstrokes reflecting higher pressure in ventricle.
Increased RVP may indicate Decreased RV function ( increased preload; decreased RV ejection fraction) or hypervolemia
Decreased RVP indicates hypovolemia
Ventricular preload or end-diastolic pressure pressure in ventricle prior to contraction, during resting and filling stage.
Higher preload is more blood in ventricle
Higher preload indicates one or two things: -Decreased ventricular function (
Pulmonary artery pressure reflects RV peak systolic pressure
Where is the dicrotic notch located? downslope of the PAP waveform
What does dicrotic notch indicate? closure of pulmonic valve
Normal pulmonary artery pressure (PAP): (MPAP) 9-18 mmHg
Normal pulmonary artery pressure (PAP) systolic 15-30 mmHg
Normal PAP diastolic 8-15 mmHg
Decreased PAP with hypovolemia or dehydration
Increased PAP with hypervolemia, increased pulmonary vascular resistance, pulmonary embolus, and left ventricular failure (increased pulmonary venous pressure)
Where is the pulmonary capillary wedge obtained from (PCWP)? distal port of catheter with balloon inflated or wedged in place
What does the PCWP measure? "Down-stream" preload or filling pressure of left side of the heart. Left ventricular end-diastolic pressure (LVEDP)
PCWP balloon should not be inflated more than 15 sec during measurement to prevent pulmonary infarction
Normal PCWP or pulmonary artery occlusion pressure is 6-12 mm Hg
PCWP sometimes referred to pulmonary artery wedge pressure (PAWP) but not common.
PCWP measurement used to assess preload in left side of heart
Increased left ventricular end-diastolic pressure (LVEDP) or preload indicates decreased contractility of the left ventricle or fluid overload
PCWP in non-cardiogenic pulmonary edema fairly normal
PCWP in cardiogenic pulmonary edema will be elevated
PCWP Increased -decreased left ventricular function -hypervolemia/ fluid overload -mitral valve stenosis
PCWP decreased - systemic vasodilation -sepsis -hypovolemia
Cardiogenic shock inability of the heart to pump blood adequately due to loss of contractility
Cardiogenic shock results of major Myocardial infarction that destroys heart tissue or left ventricular hyper
Diminished left ventricular function characterized by low cardiac output/ cardiac index, elevated PAP and PCWP, increased SVR (systemic vascular resistance)
SVR formula map-cvp/ QT x80
PVR formula mpap-pcwp/ QT x 80
MPAP normal 10-20 mm Hg
MAP formula systolic PAP + 2(diastolic) /3
Normal PVR 110-250 dynes/sec
systole contraction phase of cardiac cycle.
Diastole resting/ filling phase of cardiac cycle.
Mean arterial pressure pressure propelling blood from LV to RA.
Mean arterial pressure related to overall adequacy of tissue perfusion.
Frank Stallings law greater preload, greater force of contraction, to the point where ventricular failure occurs.
preload prior to contraction
Preload assessed by measuring ventricular end-diastolic pressure
Mean arterial pressure normal 70-105 mmHg
Stroke volume influences blood pressure by: hypervolemia and hypovolemia
Arterial compliance influences blood pressure by: arteriosclerosis
Arterial resistance influences blood pressure by: Vasoconstriction, atherosclerosis, increased blood viscosity (increased hematocrit)
Pulse pressure difference between systolic and diastolic pressure
Influences on pulse pressure Widening (increasing) pulse pressure which indicates stroke volume or cardiac output. Narrowing (decreasing) pulse pressure which indicates decreasing stroke volume or cardiac output.
Contractility inotropic state of myocardium or velocity of myo fiber shortening during systole. STRENGTH
Decreased myocardial contractility results to: reduction in stroke volume and cardiac output leads to decrease perfusion and diminished oxygen delivery to tissues.
stroke volume blood ejected during single ventricular contraction
Stroke volume formula QT/HR x 1000
normal stroke volume 60-130 mL/ beats
cardiac out put (QT) amount of blood ejected from heart in one min
Ejection fraction percentage of blood pumped from ventricle during single contraction
left ventricular ejection fraction 60-70% healthy, reduced with decreased contractility or valvular disease
End diastolic volume after systole, blood remaining in ventricle
EF formula SV/EDV
low EF reaches about 50%
EF treatment inotropic agents
EF decreased when contractility of heart is impaired
oxygen delivery (DO2) amount of O2 delivered to tissues each mi, primary function of cardiovascular system.
Delivery of O2 dependent among blood flow(QT) and O2 content in arterial blood (CaO2).
amount of O2 delivered to tissues in a min 1000 mL
O2 consumption (VO2) in a min 250 mL/min
reserve of O2 750 mL
O2 content content or total amount of O2 carried in the blood
Normal arterial O2 content (CaO2) 18-20 mL/dl
normal venous O2 content (CVO2) 14-16 ml/dl
O2 content takes into play with hemoglobin and O2 stat.
CaO2 formula: (Hb X 1.34 X SaO2) + (PaO2 X .003)
CVO2 formula : (Hb X 1.34X SvO2) + ( PvO2 X .003)
one gram of hemoglobin combine with O2 is 1.34
O2 dissolved in every dl of plasma is .003 mL
difference in venous and arterial O2 content arteriovenous O2 content difference C(a-v) O2
Normal arteriovenous O2 content difference 3.5-5 ml/dl
Increase in arteriovenous O2 content difference indicates decreased Qt
greater blood flow less O2 removed in blood and smaller C(a-v) O2
smaller the difference equals greater Qt
VO2= [C(a-v) O2 X 10] x Qt
Fick equation: Qt= VO2/ CaO2 - CvO2 x 10
Created by: Summerj
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