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adrenergic agonistsx

QuestionAnswer
alpha 1 effects contraction of vascular and genital SM, increase glycogenolysis in liver, increased cardiac inotropy
alpha 2 effects decreased NE release from post synaptic nerve terminals
beta 1 effects increased cardiac inotopy and chronotropy
beta 2 effects decrease bronchial and vascular SM contraction, decrease uterine SM contraction
beta 3 efects lipolysis in adipose tissue
D1- dopamine receptor produce vascular (renal, mesenteric, coronary) SM relaxation, increase NE release from sympathetic nerves
alpha 1 signaling system phosphoinositide hydrolysis
alpha 2 signaling system open K+ channels, inhibit adenylyl cyclase, open voltage-dependent Ca2+ channels
beta 1,2,3 signaling system activate adenylyl cylase
D1-dopamine signaling system activate adenylyl cylase
activate all beta receptors non-selectively isoproterenol
beta 1 selective agonist dobutamine
beta 2 selective agonist terbutaline, albuterol
D1-dopamine agonist fenoldopam
regulates smooth muscle by activating PKA Beta adrenergic agonists
regulates smooth muscle by activating PLC alpha 1 adrenergic agonists
PLC hydrolyzes phosphoinositol 4, 5 bisphosphate to DAG and IP3
Diffuses into the cytoplasm causing release stored calcium from SR IP3
remains membrane associated and activates PKC DAG
use coupling to ROCs to produce smooth muscle contraction alpha 2 adrenergic agonists
activated when cytoplasmic calcium binds to calmodulin myosin light chain kinase
increases the affinity for actin causing muscle contraction MLCK activation
Created by: 53500854
Popular Pharmacology sets

 

 



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