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CHF pharm

pharmacology of CHF

QuestionAnswer
glycosides inhibits Na,K-ATPase resulting in increased intracellular sodium and decreased intracellular potassium
glycosides positive inotrpic effects, increases systemic vascular resistance and the constriction of smooth muscle in veins
glycosides improves renal blood flow, increases vagal activity, decreased sympathetic tone
digoxin oral or IV enteric bacteria may inactivate some
digoxin rapid clearance, eliminated renally
digitoxin excreted by bile, metabolized by liver, may set up enterhepatic circulation
glycosides can be fatal, arrythmias, GI( anorexia, nausea, vomiting, diarrhea) CNS( disorientation and visual disturbances--> can be treated with potassium, phenytoin or lidocaine
bipyridines inhibit phosphodiesterase, increase myocardial contractility by increasing inward calcium flux in heart during the action potential, vasodilating effects, increases in cAMP
bipyridines inamrinone and milrinone
inamrinone nausea and vomiting, arrythmias, thrombocytopenia, and liver enzyme changes
milrinone less bone marrow and liver toxicities, still cause arrythmias
beta one agonist dobuatmine
beta one agonist produces increase in cardiac output with a decrease in ventricular filling pressure
beta one agonist some tachycardia and and increased in myocardial oxygen consumption have been reported
Created by: swohlers
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