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Phase 2 - Block 2 RX

Phase 2 - block 2 cardiac drugs

Agonist drugs possessing receptor affinity and intrinsic activity
Antagonist drugs that have receptor affinity for particular receptor but unable to produce intrinsic activity
Intrinsic activity biochemical response by the cell once a receptor have been activated
Drugs facilitate or inhibit the normal functions of a particular tissue
Receptor affinity drugs that bind to a specific receptor are claimed to have receptor affinity and may or may not generate intrinsic activity within the cell
Receptor chemical component on the surface of cell membranes (are the site of action)
Somatic voluntary, motor neurons, stimulates contraction of skeletal muscle
Autonomic involuntary
Sympathetic fight or flight - release norepinephrine
Parasympathetic conservation - release acetylcholine
Adrenergic agonists also known as sympathetic or adrenergic drugs
Sympathomimetics drugs that imitate sympathetic stimulation / alpha and beta receptors
Alpha receptor smooth muscle contraction
Beta 2 receptor smooth muscle relaxation
Beta 1 receptor cardiac stimulation
Adrenergic antagonists block actions of neurotransmitters; adrenergic blockers, antagonists, beta blockers, alpha blockers, inotropic drugs (vasoconstrictor / vasodilator, anti-hypertensives, anti-anginyls)
Beta blockers inhibits beta receptors - decrease HR, contractility, myocardial O2 demands and cardiac output. SA node discharge rate is reduced and conduction time of AV node increased.
Beta blockers used to treat angina, arrhythmias, and systemic hypertension
Beta blocker drugs -olol ; B CANT TLL MC TIC
Calcium channel blockers affect electrophysiology and mechanical properties of the heart, vasodilator
Calcium channel blockers treat a-fib, svt, a-flutter; systemic hypertension and hypertrophic cardiomyopathy
Angiotension-converting enzymes (ACE Inhibitors) - (-il) prevent angiotensin I from converting to a substance that retains salt and water (causing vessel relaxation)
Angiotension Receptor Blockers - ARBs blood vessels constrict
Antiadrenergic agents stimulates alpha-adrenergic receptors of CNS - dropping BP
Antianginal/Nitrates treats angina
Diuretics prevent fluid retention, reducing BP
Direct Acting on vascular dilation of vascular system - arterial or venous
Vasopressor/Inotropes vasoconstriction and release norepinephrine
Cardiac Glycosides treats CHF, SVT, tachy, a-fib; decrease AV node conduction rates
Created by: hspointon